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2004, Hematology/oncology clinics of North America, Volume 18, no. 3., xvi p., p. [525]-782
Book
1999, Basic and clinical oncology, ISBN 9780824799014, Volume 16., xi, 442
Book
Deutsche Medizinische Wochenschrift, ISSN 0012-0472, 2018, Volume 143, Issue 18, pp. 1304 - 1310
Journal Article
Tumor Diagnostik und Therapie, ISSN 0722-219X, 02/2019, Volume 40, Issue 1, pp. 43 - 49
Journal Article
Journal Article
Journal Article
Journal Article
JNCCN Journal of the National Comprehensive Cancer Network, ISSN 1540-1405, 01/2012, Volume 10, Issue 1, pp. 64 - 110
Journal Article
Journal of Clinical Investigation, ISSN 0021-9738, 10/2016, Volume 126, Issue 10, pp. 3961 - 3980
lmatinib-insensitive leukemia stem cells (LSCs) are believed to be responsible for resistance to BCR-ABL tyrosine kinase inhibitors and relapse of chronic... 
PROGENITOR CELLS | MEDICINE, RESEARCH & EXPERIMENTAL | TYROSINE KINASE | SYMMETRIC DIMETHYLATION | PHILADELPHIA-CHROMOSOME | ARGININE METHYLTRANSFERASE | BONE-MARROW | BETA-CATENIN | IMATINIB MESYLATE | CHRONIC MYELOID-LEUKEMIA | ONCOGENE ADDICTION | RNA, Small Interfering - genetics | Cell Proliferation | Neoplastic Stem Cells - drug effects | Humans | Gene Expression Regulation, Neoplastic | Leukemia, Myelogenous, Chronic, BCR-ABL Positive - drug therapy | Protein-Arginine N-Methyltransferases - antagonists & inhibitors | Molecular Targeted Therapy | Gene Knockdown Techniques | STAT5 Transcription Factor - metabolism | Leukemia, Myelogenous, Chronic, BCR-ABL Positive - pathology | 1-Naphthylamine - pharmacology | HEK293 Cells | Female | Antineoplastic Agents - pharmacology | Protein-Arginine N-Methyltransferases - genetics | Aminoquinolines - pharmacology | Leukemia, Myelogenous, Chronic, BCR-ABL Positive - enzymology | Gene Expression | Cell Survival | Imatinib Mesylate - pharmacology | Mice, Inbred C57BL | Enzyme Induction | Pyrimidines - pharmacology | Mice, SCID | Protein-Arginine N-Methyltransferases - metabolism | 1-Naphthylamine - analogs & derivatives | Xenograft Model Antitumor Assays | Animals | Cell Line, Tumor | Mice, Inbred NOD | Carbazoles - pharmacology | Neoplastic Stem Cells - enzymology | Fusion Proteins, bcr-abl - metabolism | Care and treatment | Methyltransferases | Development and progression | Genetic aspects | Chronic myeloid leukemia | Properties | Gene expression | Health aspects | Cloning | Leukemia | Mortality | Science | Grants | Kinases | Experiments | Proteins | Stem cells | DNA methylation | Epigenetics | Mutation | Deoxyribonucleic acid--DNA | Cancer | Index Medicus | Abridged Index Medicus
Journal Article
Journal Article
Blood, ISSN 0006-4971, 04/2011, Volume 117, Issue 17, pp. 4501 - 4510
Journal Article
Journal Article
Cancer Medicine, ISSN 2045-7634, 07/2018, Volume 7, Issue 7, pp. 3406 - 3410
Chronic myelogenous leukemia (CML) is commonly treated with tyrosine kinase inhibitors (TKIs) that inhibit the pro‐leukemic activity of the BCR‐ABL1... 
BCR | | 1 | antisense therapy | chronic myelogenous leukemia | ABL | selective interaction | target recognition | BCR-ABL1 | THERAPEUTICS | STEM-CELLS | CML | ANTISENSE | ABL GENE-EXPRESSION | CHRONIC MYELOID-LEUKEMIA | CURRENT PROGRESS | TYROSINE KINASE INHIBITORS | ONCOLOGY | IMATINIB | QUIESCENT | Drug resistance | Stem cells | Chronic myeloid leukemia | Leukemia
Journal Article