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Plant, Cell & Environment, ISSN 0140-7791, 10/2012, Volume 35, Issue 10, pp. 1707 - 1728
Journal Article
Genes & Development, ISSN 0890-9369, 03/2014, Volume 28, Issue 6, pp. 548 - 560
textabstractThe disruption of the NRF2 (nuclear factor erythroid-derived 2-like 2)/glutathione-mediated antioxidant defense pathway is a critical step in the... 
Circadian clock | Bleomycin | Pulmonary fibrosis | NRF2 | Glutathione
Journal Article
Pharmacology and Therapeutics, ISSN 0163-7258, 2014, Volume 144, Issue 1, pp. 60 - 70
Journal Article
The Plant Cell, ISSN 1040-4651, 5/2011, Volume 23, Issue 5, pp. 1741 - 1755
Journal Article
Nature, ISSN 0028-0836, 2013, Volume 502, Issue 7473, pp. 689 - 692
Journal Article
Nature, ISSN 0028-0836, 01/2018, Volume 553, Issue 7688, pp. 351 - 355
The circadian clock imposes daily rhythms in cell proliferation, metabolism, inflammation and DNA damage response(1,2). Perturbations of these processes are... 
CIRCADIAN CLOCK | TARGET | STEM-CELLS | METABOLISM | DNA-DAMAGE RESPONSE | BEHAVIOR | MULTIDISCIPLINARY SCIENCES | ALPHA | AUTOPHAGY | MODULATION | Apoptosis - drug effects | Humans | Male | Circadian Clocks - genetics | Autophagy - drug effects | Pyrrolidines - pharmacology | Neoplasms - genetics | Female | Membrane Proteins - metabolism | Nevus - drug therapy | Nuclear Receptor Subfamily 1, Group D, Member 1 - metabolism | Cell Survival - drug effects | Oncogenes - genetics | Nuclear Receptor Subfamily 1, Group D, Member 1 - agonists | Membrane Proteins - genetics | Mice, Inbred C57BL | Thiophenes - pharmacology | Neoplasms - drug therapy | Animals | GTP Phosphohydrolases - metabolism | Signal Transduction - drug effects | GTP Phosphohydrolases - genetics | Nevus - pathology | Glioblastoma - pathology | Lipogenesis - drug effects | Cell Line, Tumor | Glioblastoma - drug therapy | Neoplasms - pathology | Circadian Clocks - physiology | Physiological aspects | Development and progression | Genetic aspects | Cancer | Cell proliferation | Oxidative stress | Regulators | Senescence | Toxicity | p53 Protein | DNA damage | Glioblastoma | Cytotoxicity | Genomes | Autophagy | Cancer therapies | Neoplasms | Anticancer properties | Receptors | Antitumor agents | Metabolites | Animal tissues | Cell cycle | Biocompatibility | Lipogenesis | Deoxyribonucleic acid--DNA | Circadian rhythms | Enzymes | Pharmacology | Circadian rhythm | Gene expression | Nuclear receptors | Metabolism | Fatty acids | Stem cells | Hypoxia | Antitumor activity | Phagocytosis | Tumors | Apoptosis | Index Medicus
Journal Article