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Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 06/2011, Volume 108, Issue 26, pp. 10656 - 10661
Journal Article
Immunity, ISSN 1074-7613, 2012, Volume 36, Issue 1, pp. 142 - 152
Cytotoxic CD8 + T lymphocytes directly kill infected or aberrant cells and secrete proinflammatory cytokines. By using metal-labeled probes and mass... 
IMMUNE | RESPONSES | EFFECTOR FUNCTIONS | MEMORY | CLASS-I LIGANDS | SUBSETS | PEPTIDE COMPLEXES | IMMUNOLOGY | MHC TETRAMERS | ANTIGEN | LYMPHOCYTES | Antigens | Flow cytometry | Peptides | Cytokines | Cytotoxicity | Principal components analysis | Software | Viral infections
Journal Article
PLoS ONE, ISSN 1932-6203, 2014, Volume 9, Issue 9, p. e108925
Tumor-derived exosomes, which are nanometer-sized extracellular vesicles of endosomal origin, have emerged as promoters of tumor immune evasion but their role... 
IMMUNORECEPTOR | APOPTOSIS | CANCER-PATIENTS | LIGANDS | VESICLES | MULTIDISCIPLINARY SCIENCES | MICROVESICLES | RECEPTOR | IMMUNOSURVEILLANCE | RELEASE | CYTOTOXICITY | Exosomes - metabolism | Cell Line | Prostatic Neoplasms - metabolism | Prostatic Neoplasms - pathology | NK Cell Lectin-Like Receptor Subfamily K - chemistry | Antibodies, Monoclonal - pharmacology | Humans | Histocompatibility Antigens Class I - immunology | Intracellular Signaling Peptides and Proteins - immunology | Male | Intracellular Signaling Peptides and Proteins - metabolism | Down-Regulation - drug effects | Histocompatibility Antigens Class I - metabolism | Castration | NK Cell Lectin-Like Receptor Subfamily K - metabolism | Leukocytes, Mononuclear - immunology | Immune Evasion - drug effects | K562 Cells | CD8-Positive T-Lymphocytes - metabolism | Killer Cells, Natural - immunology | Leukocytes, Mononuclear - cytology | Killer Cells, Natural - metabolism | CD8-Positive T-Lymphocytes - immunology | Antibodies, Monoclonal - immunology | Plasma | Flow cytometry | Regulators | Toxicity | CD8 antigen | Cytotoxicity | Lymphocytes T | Exosomes | Cancer therapies | Proteins | Killer cells | Immunology | Lymphocytes | Rodents | Natural killer cells | Incubation | T cell receptors | Patients | Pathology | Cytometry | Surveillance | Pheochromocytoma cells | Ligands | Aberration | Prostate cancer | Prostate | Cancer | Apoptosis | Tumors | Clinical Medicine | Biological Sciences | Naturvetenskap | Immunologi | Medical and Health Sciences | Medicin och hälsovetenskap | Biologiska vetenskaper | Natural Sciences | Cancer and Oncology | Klinisk medicin | Cancer och onkologi
Journal Article
EMBO reports, ISSN 1469-221X, 09/2018, Volume 19, Issue 9, p. n/a
The cytokine interferon‐γ (IFNγ) can induce expression of MHC class II (MHCII) on many different cell types, leading to antigen presentation to CD4+ T cells... 
Keap1 | dimethyl fumarate | interferon‐γ | MHC class II | oxidative stress | interferon-γ | PLACEBO-CONTROLLED PHASE-3 | BIOCHEMISTRY & MOLECULAR BIOLOGY | CLASS-II TRANSACTIVATOR | CELL BIOLOGY | FUMARIC-ACID ESTERS | TRANSCRIPTION FACTOR NRF2 | MULTIPLE-SCLEROSIS | HISTONE DEACETYLASE INHIBITORS | interferon-gamma | NF-KAPPA-B | T-CELLS | Adaptive Immunity | Arsenites - pharmacology | Oxidative Stress | Antigens, Nuclear - metabolism | Humans | Kelch-Like ECH-Associated Protein 1 - metabolism | Antioxidants - pharmacology | Interferon-gamma - metabolism | Gene Expression Regulation - drug effects | Nerve Tissue Proteins - metabolism | Transcription Factors - metabolism | Up-Regulation - drug effects | Histocompatibility Antigens Class II - immunology | Antigen Presentation | Dimethyl Fumarate - pharmacology | Signal Transduction - drug effects | Interferon-gamma - immunology | Cullin Proteins - metabolism | Histocompatibility Antigens Class II - metabolism | HeLa Cells | Histocompatibility Antigens Class II - genetics | Sequestosome-1 Protein - metabolism | Interferon-gamma - pharmacology | Ubiquitin | Drugs | Oxidative stress | Regulators | Chromatin | Transcription | Lymphocytes T | Immunity | Autophagy | Immunosuppressive agents | Cullin | Antioxidants | Signal transduction | Cell activation | Pathways | Lymphocytes | Ubiquitin-protein ligase | Antigen presentation | Phenotypes | Genetic control | Medical treatment | CD4 antigen | Diseases | Organic chemistry | Major histocompatibility complex | γ-Interferon | Epigenetics | Interferon | Autoimmune diseases | Arsenite | Phagocytosis | Tumors | Immunology | Signal Transduction | Chromatin, Epigenetics, Genomics & Functional Genomics
Journal Article
Nature Genetics, ISSN 1061-4036, 04/2010, Volume 42, Issue 4, pp. 295 - U42
Journal Article
PLoS Biology, ISSN 1544-9173, 05/2008, Volume 6, Issue 5, pp. 1020 - 1032
Journal Article