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Oncotarget, ISSN 1949-2553, 2018, Volume 9, Issue 16, pp. 13023 - 13035
GDC-0853 is a selective, reversible, and non-covalent inhibitor of Bruton's tyrosine kinase (BTK) that does not require interaction with the Cys481 residue for... 
BTK | CLL | GCD0853
Journal Article
Haematologica (Roma), ISSN 1592-8721, 2019, Volume 104, Issue 10, pp. 2053 - 2060
Olaptesed pegol (NOX-A12) is a pegylated structured L-oligoribonucleotide that binds and neutralizes CXCL12, a chemokine tightly regulating the life cycle of... 
CLL | SPIEGELMER | COMBINATION | INHIBITOR | HEMATOLOGY | Index Medicus
Journal Article
Oncotarget, ISSN 1949-2553, 09/2018, Volume 9, Issue 72, pp. 33710 - 33718
Signal transducer and activator of transcription (STAT)-3 might be phosphorylated or acetylated. Unlike the phosphorylation of STAT3, little is known about the... 
CLL | STAT3 | Acetylation
Journal Article
Journal Article
Blood, ISSN 1528-0020, 2018, Volume 131, Issue 15, pp. 1704 - 1711
ORR was 67% with venetoclax for 36 patients with CLL refractory to or who relapsed after idelalisib; median PFS has not yet been reached. Common adverse events... 
CLL | OPEN-LABEL | INHIBITOR | HEMATOLOGY | Clinical Trials And Observations
Journal Article
Asian Pacific Journal of Cancer Prevention, ISSN 1513-7368, 12/2018, Volume 19, Issue 12, pp. 3383 - 3392
Journal Article
Cancer Research, ISSN 0008-5472, 03/2018, Volume 78, Issue 5, pp. 1225 - 1240
The angiotensin II receptor AGTR1, which mediates vasoconstrictive and inflammatory signaling in vascular disease, is overexpressed aberrantly in some breast... 
SYSTEM | OVEREXPRESSION | ABC-DLBCL | ANGIOGENESIS | ONCOLOGY | INFLAMMATION | SENSITIVITY | PROTEIN-COUPLED RECEPTORS | MALT1 PROTEASE | TYPE-1 RECEPTOR | CELL-LINE | Receptors, Angiotensin - metabolism | RNA, Small Interfering - genetics | Cell Proliferation | Prognosis | Follow-Up Studies | Humans | Gene Expression Regulation, Neoplastic | Neovascularization, Pathologic | NF-kappa B - metabolism | Breast Neoplasms - metabolism | CARD Signaling Adaptor Proteins - genetics | CARD Signaling Adaptor Proteins - metabolism | Receptor, Angiotensin, Type 1 - genetics | Biomarkers, Tumor - metabolism | Receptors, Angiotensin - chemistry | Female | Tumor Cells, Cultured | B-Cell CLL-Lymphoma 10 Protein - genetics | B-Cell CLL-Lymphoma 10 Protein - metabolism | Survival Rate | Mucosa-Associated Lymphoid Tissue Lymphoma Translocation 1 Protein - genetics | Receptors, Angiotensin - genetics | Chick Embryo | Xenograft Model Antitumor Assays | CARD Signaling Adaptor Proteins - antagonists & inhibitors | Animals | Breast Neoplasms - genetics | NF-kappa B - genetics | Breast Neoplasms - pathology | Mice, Nude | Receptor, Angiotensin, Type 1 - metabolism | Mucosa-Associated Lymphoid Tissue Lymphoma Translocation 1 Protein - metabolism | Biomarkers, Tumor - genetics | Mice | Mucosa-Associated Lymphoid Tissue Lymphoma Translocation 1 Protein - antagonists & inhibitors | B-Cell CLL-Lymphoma 10 Protein - antagonists & inhibitors | Apoptosis | Cell Movement | Drugs | Aggressive behavior | Activation | Breast cancer | Cancer therapies | ErbB-2 protein | Endothelial cells | Lymph nodes | Metastases | Angiogenesis | Signal transduction | Signaling | Vascular diseases | Angiotensin | Bcl-10 protein | Ligands | Aberration | Angiotensin II | Cell migration | Cancer | Bcl10 | invasion | breast carcinoma | angiogenesis | MALT1
Journal Article
Oncotarget, ISSN 1949-2553, 2018, Volume 9, Issue 98, pp. 37173 - 37184
Earlier we have shown the expression of a constitutively active receptor tyrosine kinase Axl in CLL B-cells from previously untreated CLL patients, and that... 
CLL | TP-0903 | AXL | Ibrutinib | Apoptosis
Journal Article
Journal Article
Journal Article
Journal Article
Nature communications, ISSN 2041-1723, 2018, Volume 9, Issue 1, pp. 4822 - 12
Pathogen immune responses are profoundly attenuated in fetuses and premature infants, yet the mechanisms underlying this developmental immaturity remain... 
SYSTEMATIC ANALYSIS | PHAGOCYTOSIS | CANDIDA-ALBICANS | INTERFERON-GAMMA | RECOGNITION | NEWBORN | MULTIDISCIPLINARY SCIENCES | PRETERM | HUMAN MONOCYTES | UNDER-5 MORTALITY | DECTIN-1 | Mucosa-Associated Lymphoid Tissue Lymphoma Translocation 1 Protein - deficiency | Monocytes - cytology | Humans | Transcription Factors - deficiency | Tumor Necrosis Factor-alpha - genetics | Lectins, C-Type - immunology | Protein Biosynthesis - immunology | CARD Signaling Adaptor Proteins - deficiency | PPAR gamma - immunology | Monocytes - immunology | Transcriptome - immunology | Tumor Necrosis Factor-alpha - deficiency | Lectins, C-Type - genetics | TOR Serine-Threonine Kinases - deficiency | CARD Signaling Adaptor Proteins - genetics | Interleukins - genetics | TOR Serine-Threonine Kinases - genetics | Mucosa-Associated Lymphoid Tissue Lymphoma Translocation 1 Protein - immunology | Gene Expression Regulation, Developmental | Microarray Analysis | Interleukins - immunology | PPAR gamma - deficiency | Tumor Necrosis Factor-alpha - immunology | Candida parapsilosis - immunology | Adult | Lectins, C-Type - deficiency | Transcription Factors - immunology | Infant, Newborn | PPAR gamma - genetics | B-Cell CLL-Lymphoma 10 Protein - genetics | CARD Signaling Adaptor Proteins - immunology | Mucosa-Associated Lymphoid Tissue Lymphoma Translocation 1 Protein - genetics | B-Cell CLL-Lymphoma 10 Protein - immunology | Transcription Factors - genetics | Candida albicans - immunology | Immunity, Innate | B-Cell CLL-Lymphoma 10 Protein - deficiency | Monocytes - drug effects | TOR Serine-Threonine Kinases - immunology | Infant, Premature | Lipopolysaccharides - pharmacology | Interleukins - deficiency | Primary Cell Culture | TOR protein | Neonates | Pathogens | Immune response | Transcription | Cytokines | Fetuses | DNA damage | Ontogeny | Infants | Gestation | Metabolism | Gene expression | Monocytes | Glycolysis | Metabolic pathways | Peroxisome proliferator-activated receptors | Human behavior | Deoxyribonucleic acid--DNA | Phagocytosis | Immune system
Journal Article
by Kepp, Oliver and Senovilla, Laura and Vitale, Ilio and Vacchelli, Erika and Adjemian, Sandy and Agostinis, Patrizia and Apetoh, Lionel and Aranda, Fernando and Barnaba, Vincenzo and Bloy, Norma and Bracci, Laura and Breckpot, Karine and Brough, David and Buqué, Aitziber and Castro, Maria G and Cirone, Mara and Colombo, Maria I and Cremer, Isabelle and Demaria, Sandra and Dini, Luciana and Eliopoulos, Aristides G and Faggioni, Alberto and Formenti, Silvia C and Fučíková, Jitka and Gabriele, Lucia and Gaipl, Udo S and Galon, Jérôme and Garg, Abhishek and Ghiringhelli, François and Giese, Nathalia A and Guo, Zong Sheng and Hemminki, Akseli and Herrmann, Martin and Hodge, James W and Holdenrieder, Stefan and Honeychurch, Jamie and Hu, Hong-Min and Huang, Xing and Illidge, Tim M and Kono, Koji and Korbelik, Mladen and Krysko, Dmitri V and Loi, Sherene and Lowenstein, Pedro R and Lugli, Enrico and Ma, Yuting and Madeo, Frank and Manfredi, Angelo A and Martins, Isabelle and Mavilio, Domenico and Menger, Laurie and Merendino, Nicolò and Michaud, Michael and Mignot, Gregoire and Mossman, Karen L and Multhoff, Gabriele and Oehler, Rudolf and Palombo, Fabio and Panaretakis, Theocharis and Pol, Jonathan and Proietti, Enrico and Ricci, Jean-Ehrland and Riganti, Chiara and Rovere-Querini, Patrizia and Rubartelli, Anna and Sistigu, Antonella and Smyth, Mark J and Sonnemann, Juergen and Spisek, Radek and Stagg, John and Sukkurwala, Abdul Qader and Tartour, Eric and Thorburn, Andrew and Thorne, Stephen H and Vandenabeele, Peter and Velotti, Francesca and Workenhe, Samuel T and Yang, Haining and Zong, Wei-Xing and Zitvogel, Laurence and Kroemer, Guido and Galluzzi, Lorenzo
Oncoimmunology, ISSN 2162-402X, 2014, Volume 3, Issue 9, p. e955691
Apoptotic cells have long been considered as intrinsically tolerogenic or unable to elicit immune responses specific for dead cell-associated antigens.... 
TLR, Toll-like receptor | ATF6, activating transcription factor 6 | HMGB1 | EIF2A, eukaryotic translation initiation factor 2A | H2B, histone 2B | endoplasmic reticulum stress | HMGB1, high mobility group box 1 | APC, antigen-presenting cell | ATP release | immunotherapy | BCL2, B-cell CLL/lymphoma 2 protein | mitochondrial transmembrane potential | DiOC | MOMP, mitochondrial outer membrane permeabilization | RFP, red fluorescent protein | HSV-1, herpes simplex virus type I | ICD, immunogenic cell death | IFN, interferon | XBP1, X-box binding protein 1 | GFP, green fluorescent protein | autophagy | DAMP, damage-associated molecular pattern | PI, propidium iodide | G3BP1, GTPase activating protein (SH3 domain) binding protein 1 | PDIA3, protein disulfide isomerase family A | member 3 | DAPI, 4′,6-diamidino-2-phenylindole | FLT3LG, fms-related tyrosine kinase 3 ligand | BAX, BCL2-associated X protein | CALR, calreticulin | 3,3′-dihexyloxacarbocyanine iodide | CTL, cytotoxic T lymphocyte | IL, interleukin | calreticulin | HSP, heat shock protein | ER, endoplasmic reticulum | BAK1, BCL2-antagonist/killer 1 | Calreticulin | Autophagy | Immunotherapy | Endoplasmic reticulum stress | FLOW-CYTOMETRIC DETECTION | TUMOR-CELLS | IMMUNOLOGY | MOLECULAR-PATTERN | CALRETICULIN EXPOSURE | MITOCHONDRIAL-MEMBRANE PERMEABILIZATION | HEAT-SHOCK PROTEINS | ONCOLOGY | IMMUNE-RESPONSES | APOPTOTIC CELLS | FIND-ME SIGNAL
Journal Article