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Journal Article
Journal Article
Journal Article
Nature, ISSN 0028-0836, 09/2017, Volume 549, Issue 7673, pp. 523 - 527
APOE4 is the strongest genetic risk factor for late-onset Alzheimer disease. ApoE4 increases brain amyloid-beta pathology relative to other ApoE isoforms(1).... 
BRAIN ATROPHY | MICROGLIA | ALZHEIMERS-DISEASE | MULTIDISCIPLINARY SCIENCES | FRONTOTEMPORAL DEMENTIA | IN-VIVO | FRONTAL-LOBE DEMENTIA | EPSILON-4 | ASSOCIATION | APOLIPOPROTEIN-E | TRANSGENIC MICE | Tumor Necrosis Factor-alpha - metabolism | Inflammation - pathology | Microglia - metabolism | Neurons - pathology | Phosphorylation | Apolipoprotein E4 - genetics | Tauopathies - genetics | Coculture Techniques | Humans | Tauopathies - pathology | tau Proteins - metabolism | Phosphoproteins - metabolism | Inflammation - metabolism | Microglia - immunology | tau Proteins - genetics | Apolipoprotein E4 - deficiency | Phosphoproteins - analysis | Apolipoprotein E4 - toxicity | Neurons - metabolism | Neurons - drug effects | Disease Models, Animal | Cell Survival - drug effects | Apolipoprotein E4 - metabolism | Genotype | Mice, Transgenic | Phosphoproteins - genetics | Immunity, Innate | Disease Progression | Gene Knock-In Techniques | Mice, Knockout | Tauopathies - metabolism | Animals | Alleles | Inflammation - genetics | Mice | Nervous system | Development and progression | Degeneration | Models | Apolipoproteins | Health aspects | Tau proteins | Brain | Pathogenesis | Genomes | Autophagy | Neuronal-glial interactions | Risk factors | Atrophy | Neurodegeneration | Apolipoprotein E | Rodents | Aging | Alzheimer's disease | Genotypes | Recombinant | Apolipoprotein E4 | Neurodegenerative diseases | Neurons | Secretion | Health risks | Transgenic mice | Inflammation | Gene expression | Microglia | Pathology | Tau protein | Tumor necrosis factor | β-Amyloid | Alzheimers disease | Viability | Tumors | Dementia
Journal Article