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1. Full Text Immunity, Inflammation, and Cancer
by Grivennikov, Sergei I and Greten, Florian R and Karin, Michael
Cell, ISSN 0092-8674, 2010, Volume 140, Issue 6, pp. 883 - 899
Inflammatory responses play decisive roles at different stages of tumor development, including initiation, promotion, malignant conversion, invasion, and... 
PROMOTE TUMOR-GROWTH | EPITHELIAL-MESENCHYMAL TRANSITION | T-CELL RESPONSES | TGF-BETA | NONSTEROIDAL ANTIINFLAMMATORY DRUGS | BIOCHEMISTRY & MOLECULAR BIOLOGY | CHRONIC LYMPHOCYTIC-LEUKEMIA | COLITIS-ASSOCIATED CANCER | NECROSIS-FACTOR-ALPHA | MYELOID CELLS | NF-KAPPA-B | CELL BIOLOGY | Cell Transformation, Neoplastic - immunology | Animals | Neoplasms - immunology | Humans | Inflammation - immunology | Immune System - cytology | Inflammation - physiopathology | NF-κB | cytokines | immunity | inflammation | STAT3 | Cancer
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2. Colon Cancer: Inflammation-Associated Cancer
by Shawki, Sherief and Ashburn, Jean and Signs, Steven A and Huang, Emina
Surgical Oncology Clinics of North America, ISSN 1055-3207, 04/2018, Volume 27, Issue 2, pp. 269 - 287
Colitis-associated cancer is a relatively rare form of cancer with an unclear pathogenesis. Colitis-associated cancer serves as a prototype of... 
Colitis-associated cancer | Colitis-associated cancer management | 3D human models | Colitis cancer surveillance | SURGERY | ULCERATIVE-COLITIS | COLONOSCOPIC SURVEILLANCE | ILEAL POUCH | INTRAEPITHELIAL NEOPLASIA | RECTAL-CANCER | ONCOLOGY | ILEORECTAL ANASTOMOSIS | BOWEL-DISEASE PATIENTS | COLORECTAL-CANCER | RESTORATIVE PROCTOCOLECTOMY | POUCH-ANAL ANASTOMOSIS | Inflammation - complications | Prognosis | Colonic Neoplasms - pathology | Humans | Colonic Neoplasms - etiology | Colitis - complications
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3. Full Text Cancer-related inflammation
by Mantovani, Alberto and Allavena, Paola and Sica, Antonio and Balkwill, Frances
Nature, ISSN 0028-0836, 07/2008, Volume 454, Issue 7203, pp. 436 - 444
The mediators and cellular effectors of inflammation are important constituents of the local environment of tumours. In some types of cancer, inflammatory... 
CELL LUNG-CANCER | SIGNALING PATHWAY | CHEMOKINE RECEPTOR CXCR4 | COLORECTAL-CANCER | MULTIDISCIPLINARY SCIENCES | BONE-MARROW | TUMOR-ASSOCIATED MACROPHAGES | NECROSIS-FACTOR-ALPHA | COLITIS-ASSOCIATED CANCER | OVARIAN-CANCER | NF-KAPPA-B | Inflammation - pathology | Oncogenes - genetics | Gonadal Steroid Hormones - metabolism | Neoplasm Invasiveness | Humans | Inflammation - immunology | Oncogenes - physiology | Leukocytes - immunology | Neoplasms - therapy | Neoplasms - genetics | Neoplasms - immunology | Inflammation - genetics | Neoplasms - pathology | Leukocytes - metabolism | Development and progression | Inflammation | Prognosis | Research | Analysis | Cancer | Oncology | Cellular biology | Molecular biology | Inflammatory diseases
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4. Full Text Intestinal Inflammation and Cancer
by Ullman, Thomas A and Itzkowitz, Steven H
Gastroenterology, ISSN 0016-5085, 2011, Volume 140, Issue 6, pp. 1807 - 1816.e1
Patients with ulcerative colitis and Crohn's disease are at increased risk for developing colorectal cancer (CRC). Chronic inflammation is believed to promote... 
Gastroenterology and Hepatology | Adaptive Immunity | IBD | Animal Model | Neoplasia | Intestine | Innate Immunity | NITRIC-OXIDE SYNTHASE | MICROSATELLITE INSTABILITY | ULCERATIVE-COLITIS | COLITIS-ASSOCIATED CARCINOGENESIS | BOWEL-DISEASE | COLON-CANCER | K-RAS GENE | EPITHELIAL-CELLS | COLORECTAL-CANCER | MOUSE MODEL | GASTROENTEROLOGY & HEPATOLOGY | Colitis, Ulcerative - complications | Oxidative Stress | Colitis, Ulcerative - metabolism | Humans | Risk Factors | Crohn Disease - metabolism | Gastrointestinal Tract - microbiology | Immunity, Innate | Crohn Disease - complications | Colonic Neoplasms - physiopathology | Bacterial Infections - complications | Animals | Colonic Neoplasms - pathology | Crohn Disease - drug therapy | Anti-Inflammatory Agents - therapeutic use | Colitis, Ulcerative - drug therapy | Colonic Neoplasms - etiology | Development and progression | Ursodiol | Crohn's disease | Inflammation | DNA binding proteins | Sulfates | Dextran | Colon cancer | Analysis | Nitric oxide | DNA | Genetically modified organisms | Tumor proteins | Ulcerative colitis | Cancer
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5. Full Text Inflammation and Colon Cancer
by Terzić, Janoš and Grivennikov, Sergei and Karin, Eliad and Karin, Michael
Gastroenterology, ISSN 0016-5085, 2010, Volume 138, Issue 6, pp. 2101 - 2114.e5
The connection between inflammation and tumorigenesis is well-established and in the last decade has received a great deal of supporting evidence from genetic,... 
Gastroenterology and Hepatology | Inflammation | Cytokines | Immunity | Colon Cancer | CHRONIC INTESTINAL INFLAMMATION | COLITIS-ASSOCIATED CANCER | SEVERE CROHNS-DISEASE | EPITHELIAL-MESENCHYMAL TRANSITION | TUMOR-NECROSIS-FACTOR | NONSTEROIDAL ANTIINFLAMMATORY DRUGS | KINASE-I-EPSILON | TOLL-LIKE RECEPTORS | GASTROENTEROLOGY & HEPATOLOGY | NF-KAPPA-B | GENOME-WIDE ASSOCIATION | Colonic Neoplasms - genetics | Colonic Neoplasms - prevention & control | Colonic Neoplasms - drug therapy | Colorectal Neoplasms - genetics | Humans | Inflammatory Bowel Diseases - immunology | Gene Expression Regulation, Neoplastic | Colon - immunology | Cell Transformation, Neoplastic - genetics | Colonic Neoplasms - immunology | Colorectal Neoplasms - drug therapy | Inflammation Mediators - metabolism | Inflammatory Bowel Diseases - genetics | Anti-Inflammatory Agents - therapeutic use | Inflammatory Bowel Diseases - complications | Anticarcinogenic Agents - therapeutic use | Inflammatory Bowel Diseases - drug therapy | Genetic Predisposition to Disease | Cytokines - metabolism | Risk Assessment | Signal Transduction | Neoplasm Invasiveness | Risk Factors | Colorectal Neoplasms - prevention & control | Colonic Neoplasms - secondary | Cell Transformation, Neoplastic - immunology | Animals | Colorectal Neoplasms - immunology | Colorectal Neoplasms - secondary | Colon - microbiology | Dendritic cells | Measles-mumps-rubella vaccine | Biological response modifiers | Sulfates | Risk factors | Dextran | Prostaglandins | Colon cancer | Gastrointestinal diseases | Vascular endothelial growth factor | Growth factors | Tumors
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6. Full Text NF-κB and STAT3 - key players in liver inflammation and cancer
by He, Guobin and Karin, Michael
Cell Research, ISSN 1001-0602, 01/2011, Volume 21, Issue 1, pp. 159 - 168
Hepatocellular carcinoma (HCC), the major form of primary liver cancer, is one of the most deadly human cancers. The pathogenesis of HCC is frequently linked... 
signaling transduction | NF-κB | carcinogenesis | inflammation | STAT3 | HCC | liver cancer | REVERSIBLE ACETYLATION | IKK-BETA | COLITIS-ASSOCIATED CANCER | DEFICIENT MICE | CELL BIOLOGY | HEPATOCELLULAR-CARCINOMA | NF-kappa B | CHEMICAL HEPATOCARCINOGENESIS | COMPENSATORY PROLIFERATION | EMBRYONIC LETHALITY | TNF-ALPHA | HEPATITIS-C | Liver - pathology | Signal Transduction | Liver Neoplasms - immunology | NF-kappa B - metabolism | Hepatitis - complications | Inflammation - metabolism | Animals | STAT3 Transcription Factor - physiology | Liver Neoplasms - metabolism | NF-kappa B - physiology | Carcinoma, Hepatocellular - immunology | Mice | Interleukin-6 - metabolism | STAT3 Transcription Factor - metabolism | Carcinoma, Hepatocellular - metabolism | Review
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7. Full Text Inflammation and Cancer IV. Colorectal cancer in inflammatory bowel disease: the role of inflammation
by Steven H. Itzkowitz and Xianyang Yio
American Journal of Physiology - Gastrointestinal and Liver Physiology, ISSN 0193-1857, 07/2004, Volume 287, Issue 1, pp. 7 - 17
Patients with ulcerative colitis and Crohn's disease are at increased risk for developing colorectal cancer. To date, no known genetic basis has been... 
Inflammatory bowel disease | Oxidative stress | Animal models | Colitis | Colorectal cancer | animal models | PHYSIOLOGY | colorectal cancer | colitis | ACTIVATED RECEPTOR-GAMMA | NITRIC-OXIDE SYNTHASE | MICROSATELLITE INSTABILITY | COLITIS-ASSOCIATED NEOPLASIA | inflammatory bowel disease | INTERLEUKIN-10 KNOCKOUT MICE | CHROMOSOMAL INSTABILITY | COLON-CANCER | CHRONIC ULCERATIVE-COLITIS | K-RAS GENE | GASTROENTEROLOGY & HEPATOLOGY | oxidative stress | Colorectal Neoplasms - etiology | Animals | Colorectal Neoplasms - genetics | Humans | Colitis - complications | Inflammatory Bowel Diseases - complications | Loss of Heterozygosity | Chronic Disease | Disease Models, Animal
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