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Journal of the American Society of Nephrology, ISSN 1046-6673, 02/2012, Volume 23, Issue 2, pp. 252 - 265
Journal Article
Journal Article
PLoS ONE, ISSN 1932-6203, 03/2013, Volume 8, Issue 3, p. e59226
Previously we showed that CCN family member 2/connective tissue growth factor (CCN2) promotes the proliferation, differentiation, and maturation of growth... 
CHONDROCYTE-SPECIFIC GENE | SKELETAL GROWTH | CTGF/HCS24 | ANGIOGENESIS IN-VIVO | CONSTITUTIVE ACTIVATION | MULTIDISCIPLINARY SCIENCES | PROLIFERATION | VASCULAR ENDOTHELIAL-CELLS | INTEGRIN ALPHA(V)BETA | DIFFERENTIATION | TRANSGENIC MICE | Chondrocytes - cytology | Cell Proliferation | Limb Buds - cytology | Insulin-Like Growth Factor I - genetics | Mesenchymal Stromal Cells - cytology | Insulin-Like Growth Factor II - genetics | Bone and Bones - metabolism | Cartilage - cytology | Chondrocytes - metabolism | Bone and Bones - cytology | Animals, Newborn | Biomarkers - metabolism | Cartilage - growth & development | Promoter Regions, Genetic | Gene Expression | Mesenchymal Stromal Cells - metabolism | Mice, Transgenic | Cartilage - metabolism | Insulin-Like Growth Factor II - metabolism | Animals | Limb Buds - metabolism | Receptors, Somatomedin - metabolism | Receptors, Somatomedin - genetics | Connective Tissue Growth Factor - genetics | Mice | Limb Buds - growth & development | Connective Tissue Growth Factor - metabolism | Insulin-Like Growth Factor I - metabolism | RNA | Collagen | Genes | Stem cells | Bones | Genetic engineering | Density | Cell culture | Biochemistry | Ossification | Proteins | Connective tissues | Cartilage | Angiogenesis | Bone growth | Computed tomography | Fibroblasts | Bone density | Insulin-like growth factor II | Chondrogenesis | Fusion protein | Bone (endochondral) | Proteoglycans | Cultures | Gene expression | Insulin | Orthodontics | Overexpression | Chondrocytes | Endocrinology | Cell proliferation | Dental schools | Phosphorylation | Mesenchyme | Insulin-like growth factor I | Collagen (type II) | mRNA | Insulin-like growth factors | Limb buds | Transgenic animals | Rodents | Extracellular matrix | Pharmaceutical sciences | University graduates | Recombinant | Heparan sulfate | Transgenic mice | Connective tissue growth factor | Dentistry | Bone mineral density | In vivo methods and tests | Bone | Aggrecan
Journal Article
PLoS ONE, ISSN 1932-6203, 01/2015, Volume 10, Issue 1, p. e0113334
Inflammatory response in the dental pulp can alter the collagen matrix formation by dental pulp stem cells and lead to a delay or poor healing of the pulp.... 
ACTIVATION | CHONDROCYTES | PROTEASOME INHIBITION | MULTIDISCIPLINARY SCIENCES | IN-VIVO | GENE-EXPRESSION | SYNOVIOCYTES | DEGRADATION | NECROSIS-FACTOR-ALPHA | INDUCTION | TNF | RNA, Small Interfering - genetics | Humans | Extracellular Matrix - metabolism | NF-kappa B - metabolism | Stem Cells - cytology | Stem Cells - metabolism | Gene Knockdown Techniques | Cell Differentiation - genetics | RNA Interference | Inflammation Mediators - pharmacology | Mesenchymal Stem Cells - cytology | Mesenchymal Stem Cells - drug effects | Collagen - genetics | Odontoblasts - cytology | Mesenchymal Stem Cells - metabolism | Gene Expression | Odontoblasts - metabolism | Cells, Cultured | Dental Pulp - cytology | Collagen - metabolism | Leupeptins - pharmacology | NF-kappa B - genetics | Cell Differentiation - drug effects | Odontoblasts - drug effects | Stem Cells - drug effects | Cytokines - pharmacology | Dentin | Alkaline phosphatase | Nestin | Interleukin | Differentiation (biology) | Cytology | Inflammatory response | Kinases | Cell morphology | Dental materials | Odontoblasts | Fibroblasts | Extracellular matrix | Tumor necrosis factor-TNF | Synaptotagmin | NF-κB protein | Cytokines | Tissue engineering | Inflammation | Dspp protein | siRNA | Dentistry | Tumor necrosis factor-α | Cell differentiation | Gene expression | Signaling | Rheumatoid arthritis | Collagen | Stem cells | Dental pulp | Pulp
Journal Article
Journal of Cellular and Molecular Medicine, ISSN 1582-1838, 11/2018, Volume 22, Issue 11, pp. 5486 - 5493
Journal Article
International journal of molecular medicine, ISSN 1107-3756, 08/2018, Volume 42, Issue 2, pp. 1168 - 1180
Journal Article
International Immunology, ISSN 0953-8178, 11/2011, Volume 23, Issue 11, pp. 701 - 712
Rheumatoid arthritis (RA) is a chronic inflammatory disease that causes irreversible joint damage and significant disability. However, the fundamental... 
Joint destruction | Chronic inflammation | Collagen-induced arthritis | Rheumatoid arthritis | IL-6 | RHEUMATOID-ARTHRITIS | INTERLEUKIN-1 RECEPTOR ANTAGONIST | STAT3 | MONOCLONAL-ANTIBODY | IMMUNOLOGY | rheumatoid arthritis | OSTEOCLASTOGENESIS | RODENT MODELS | TUMOR-NECROSIS-FACTOR | collagen-induced arthritis | REGULATORY T-CELLS | DOUBLE-BLIND | joint destruction | NF-KAPPA-B | chronic inflammation | Tumor Necrosis Factor-alpha - metabolism | Inflammation - pathology | Arthritis, Experimental - drug therapy | RANK Ligand - metabolism | Humans | Arthritis, Rheumatoid - complications | Interleukin-1beta - adverse effects | Arthritis, Experimental - pathology | RANK Ligand - immunology | Inflammation - complications | Signal Transduction - immunology | Osteoclasts - immunology | Arthritis, Rheumatoid - drug therapy | Inflammation - drug therapy | Interleukin-1beta - metabolism | Piperidines | Gene Expression - immunology | STAT3 Transcription Factor - deficiency | Tumor Necrosis Factor-alpha - immunology | Mice, Inbred DBA | Pyrroles - therapeutic use | Interleukin-6 - metabolism | Joints - metabolism | STAT3 Transcription Factor - genetics | Joints - drug effects | Mice, Inbred C57BL | Enzyme Inhibitors - pharmacology | Interleukin-1beta - immunology | Joints - pathology | Arthritis, Experimental - immunology | Inflammation - immunology | Pyrimidines - pharmacology | Arthritis, Rheumatoid - pathology | Pyrroles - pharmacology | Animals | Tumor Necrosis Factor-alpha - adverse effects | Interleukin-6 - pharmacology | RANK Ligand - antagonists & inhibitors | Pyrimidines - therapeutic use | Fibroblasts - drug effects | Interleukin-6 - immunology | Fibroblasts - immunology | Mice | STAT3 Transcription Factor - immunology | Arthritis, Rheumatoid - immunology | STAT3 Transcription Factor - antagonists & inhibitors | Chronic Disease | Osteoclasts - drug effects | Animal models | Transcription | Leukemia inhibitory factor | Stat3 protein | Data processing | Inflammation | Joint diseases | Osteoblasts | Inflammatory diseases | Interleukin 6 | Osteoclastogenesis | Collagen | Interleukin 1 | TRANCE protein | Tumor necrosis factor- alpha
Journal Article
Journal Article
PLoS ONE, ISSN 1932-6203, 02/2012, Volume 7, Issue 2, p. e32177
Nucleotide pyrophosphatase phosphodiesterase 1 (NPP1) is required for the conversion of extracellular ATP into inorganic pyrophosphate (PPi), a recognised... 
INFANTILE ARTERIAL CALCIFICATION | PATHOLOGICAL CALCIFICATION | POSTERIOR LONGITUDINAL LIGAMENT | INORGANIC PYROPHOSPHATE | MULTIDISCIPLINARY SCIENCES | NONSPECIFIC ALKALINE-PHOSPHATASE | VASCULAR CALCIFICATION | CELL MEMBRANE GLYCOPROTEIN-1 | SMOOTH-MUSCLE-CELLS | HYPEROSTOTIC MOUSE TWY/TWY | PATHOGENIC ROLE | Pyrophosphatases - physiology | Bone Development - drug effects | Calcium - metabolism | Pyrophosphatases - deficiency | Fibroblast Growth Factors - biosynthesis | Osteoblasts | Phosphoric Diester Hydrolases - physiology | Mice, Knockout | RNA, Messenger - biosynthesis | Phosphates - metabolism | Animals | Calcification, Physiologic | Mice | Phosphoric Diester Hydrolases - deficiency | Phosphates | Fibroblast growth factor | Surgical implants | Femur | Calcium | Fibroblast growth factor 23 | Syngeneic grafts | Spine | Homeostasis | Hydroxyapatite | Adipocytes | Kinases | Bone (trabecular) | Proteins | Cartilage | Biomedical materials | Computed tomography | Mineralization | Tibia | Fibroblasts | Aorta | Biocompatibility | Bones | Stiffness | Growth factors | Pyrophosphatase | Age | Adenosine triphosphate | Calcium homeostasis | Phosphodiesterase | Osteocalcin | Kidneys | Architecture | Mechanical properties | Gene expression | Insulin | Calcium phosphates | Studies | Ribonucleic acids | Collagen | Calcification | Plasma levels | Bone | Mutation | Disruption | Metabolic disorders
Journal Article