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American Journal of Pathology, ISSN 0002-9440, 06/2006, Volume 168, Issue 6, pp. 1861 - 1868
.... The reduction in collagen synthesis due to fibroblast aging was demonstrated by a lower in vitro production of type I procollagen by dermal fibroblasts isolated from skin... 
TOPICAL TRETINOIN | PHOTOAGED SKIN | IN-VITRO | INHIBITION | MATRIX METALLOPROTEINASE-1 | DERMAL CONNECTIVE-TISSUE | PROCOLLAGEN SYNTHESIS | TRANS-RETINOIC ACID | PATHOLOGY | STRESS | EXPRESSION | Skin - metabolism | Humans | Stress, Mechanical | Skin Aging | Collagen - metabolism | Biopsy | Adolescent | Aged, 80 and over | Aging | Adult | Collagen - biosynthesis | Skin - pathology | Fibroblasts - metabolism | Regular
Journal Article
PloS one, ISSN 1932-6203, 2016, Volume 11, Issue 4, p. e0153118
.... It is known that HSCs are themselves able to produce cytokines and chemokines, and that this production may be a key event in the initiation of fibrogenesis... 
FIBROSIS | TGF-BETA | MULTIDISCIPLINARY SCIENCES | DOUBLE-BLIND | NECROSIS-FACTOR-ALPHA | NONALCOHOLIC STEATOHEPATITIS | MICE | ALCOHOLIC HEPATITIS | CARBON-TETRACHLORIDE | FACTOR RECEPTOR | INDUCED LIVER-INJURY | Tumor Necrosis Factor-alpha - metabolism | Cell Line | Matrix Metalloproteinases - genetics | Cytokines - metabolism | Interleukin-1beta - metabolism | Humans | Hepatic Stellate Cells - metabolism | Collagen - biosynthesis | Gene Expression Profiling | Matrix Metalloproteinases - metabolism | Tissue Inhibitor of Metalloproteinases - metabolism | Tissue Inhibitor of Metalloproteinases - genetics | Drugs | Tissue inhibitor of metalloproteinases | Cell culture | Collagen (type I) | Endothelin | Platelet-derived growth factor | Liver | Clinical trials | Gallbladder diseases | Activation | Matrix metalloproteinase | Drug development | Experiments | Interleukin 6 | Proteins | Hepatitis | Infliximab | Rodents | Interleukin 1 | Tumor necrosis factor-TNF | Collagen (type IV) | Metalloproteinase | Growth factors | Interleukin 8 | Interstitial collagenase | Stellate cells | Liver diseases | Cytokines | Interleukin 1 receptor antagonist | Inflammation | Endothelin 1 | Platelet-derived growth factor BB | Gelatinase B | Signaling | Collagen | Fibrosis | Monoclonal antibodies | Sepsis | Chemokines | Monocyte chemoattractant protein 1 | Bile | Life Sciences
Journal Article
Science (American Association for the Advancement of Science), ISSN 1095-9203, 2010, Volume 327, Issue 5965, pp. 580 - 583
Journal Article
PloS one, ISSN 1932-6203, 2012, Volume 7, Issue 5, p. e35144
Interleukin-6 (IL-6) is an important cytokine participating in multiple biologic activities in immune regulation and inflammation. IL-6 has been associated... 
HYPERTROPHY | INFLAMMATION | MULTIDISCIPLINARY SCIENCES | RECEPTOR | MECHANISMS | DYSFUNCTION | INDUCTION | INTERLEUKIN-6 | TRANSFORMING GROWTH FACTOR-BETA | PRECURSORS | EXPRESSION | Angiotensin II - pharmacology | Interleukin-6 - genetics | Mice, Inbred C57BL | Transforming Growth Factor beta1 - metabolism | Male | Myocardium - pathology | Smad3 Protein - metabolism | Interleukin-6 - deficiency | Gene Expression Regulation - drug effects | Macrophages - metabolism | Animals | Fibroblasts - drug effects | Myocardium - metabolism | Fibrosis | Interleukin-6 - biosynthesis | Collagen - biosynthesis | Mice | Phosphorylation - drug effects | Transforming Growth Factor beta1 - biosynthesis | Fibroblasts - metabolism | Immunohistochemistry | Heart | Oxidative stress | Collagen (type I) | Phosphorylation | Smad protein | Cardiomyopathy | Transforming growth factor | Interleukin | Staining | Smooth muscle | Cardiovascular disease | Smad3 protein | Kinases | Macrophages | Interleukin 6 | Infusion | Immunology | Actin | Rodents | Fibroblasts | Angiotensin II | Growth factors | Heart diseases | Heart failure | Cardiovascular system | Hypertension | Cytokines | Immunoregulation | Muscles | Cardiomyocytes | Inflammation | Gene expression | Hospitals | Angiotensin | Sepsis | Laboratory animals | Cardiac muscle | Antibodies | Transforming growth factor- beta 1 | Transforming growth factor- beta | cardiomyocytes | Cell activation
Journal Article
Arthritis research & therapy, ISSN 1478-6354, 2015, Volume 17, Issue 1, p. 235
Journal Article
Journal Article
Immunological reviews, ISSN 1600-065X, 2008, Volume 226, Issue 1, pp. 112 - 131
Journal Article
Clinical Orthopaedics and Related Research, ISSN 0009-921X, 10/2011, Volume 469, Issue 10, pp. 2773 - 2784
...) the spontaneous production of cytokines and (2) the response to interleukin (IL)-1β.Twelve pellets/donor prepared with monolayer-expanded HACs (n = 6 donors... 
Surgical Orthopedics | Medicine & Public Health | Sports Medicine | Surgery | Orthopedics | Conservative Orthopedics | Medicine/Public Health, general | OSTEOARTHRITIC CHONDROCYTES | SURGERY | II COLLAGEN | MATRIX-METALLOPROTEINASE | GROWTH-FACTORS | GENE-EXPRESSION | NECROSIS-FACTOR-ALPHA | HUMAN ARTICULAR CHONDROCYTES | CHEMOKINE RECEPTORS | ORTHOPEDICS | MESENCHYMAL STEM-CELLS | CATHEPSIN-B | Chondrocytes - immunology | Humans | Middle Aged | Transforming Growth Factor beta1 - metabolism | Male | RNA, Messenger - metabolism | Cartilage, Articular - enzymology | Time Factors | Interleukin-1beta - metabolism | Inflammation Mediators - metabolism | Adult | Female | Chemokine CCL2 - metabolism | Interleukin-8 - metabolism | Chondrogenesis | Cytokines - genetics | Tissue Engineering | Cytokines - metabolism | Cells, Cultured | Gene Expression Regulation | Matrix Metalloproteinase 13 - metabolism | Chondrocytes - enzymology | Aged | Matrix Metalloproteinase 1 - metabolism | Cartilage, Articular - immunology | Transforming growth factor | Inflammation | Transforming growth factor- beta 1 | Matrix metalloproteinase | Gene expression | Joint diseases | Collagenase 3 | Survival | Cartilage | Donors | Monocyte chemoattractant protein | Chondrocytes | Interleukin 1 | Metalloproteinase | Chemokines | Interleukin 8 | Interstitial collagenase | Monocyte chemoattractant protein 1 | Clinically Relevant Strategies for Treating Cartilage and Meniscal Pathology | Symposium
Journal Article
European Journal of Immunology, ISSN 0014-2980, 05/2009, Volume 39, Issue 5, pp. 1301 - 1312
.... In inflamed tissues, production of prostaglandins by COX‐2 has been proposed to favor Th17 responses indirectly by increasing IL‐23 and blocking IL‐12 release from APC... 
Memory T cells | Prostaglandin | IL‐17 | IFN‐γ | Th17 | IFN-γ | IL-17 | RHEUMATOID-ARTHRITIS | COLLAGEN-INDUCED ARTHRITIS | INDUCTION | IMMUNOLOGY | JOINT INFLAMMATION | CYTOKINE PROFILE | GROWTH-FACTOR-BETA | PROSTANOID RECEPTORS | IFN-gamma | MULTIPLE-SCLEROSIS | T(H)17 | DIFFERENTIATION | T-Lymphocyte Subsets - immunology | Humans | Interleukin-17 - immunology | T-Box Domain Proteins - immunology | RNA, Messenger - metabolism | Th1 Cells - immunology | Dinoprostone - immunology | Receptors, Retinoic Acid - genetics | CD4-Positive T-Lymphocytes - immunology | Receptors, Prostaglandin E, EP4 Subtype | Receptors, Thyroid Hormone - genetics | Autoimmunity - immunology | T-Lymphocyte Subsets - drug effects | Receptors, Retinoic Acid - immunology | Receptors, Prostaglandin E - immunology | Interferon-gamma - genetics | Receptors, Thyroid Hormone - immunology | Immunologic Memory - immunology | Nuclear Receptor Subfamily 1, Group F, Member 3 | Dinoprostone - pharmacology | Th1 Cells - drug effects | RNA, Messenger - genetics | Interleukin-17 - genetics | Interleukin-17 - biosynthesis | Reverse Transcriptase Polymerase Chain Reaction | T-Box Domain Proteins - genetics | Interleukin-17 - antagonists & inhibitors | Interferon-gamma - immunology | Receptors, Prostaglandin E, EP2 Subtype | Autoimmunity - drug effects | CD4-Positive T-Lymphocytes - drug effects | Interferon-gamma - biosynthesis
Journal Article