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Cell, ISSN 0092-8674, 07/2014, Volume 158, Issue 2, pp. 288 - 299
The etiology of colorectal cancer (CRC) has been linked to deficiencies in mismatch repair and adenomatous polyposis coli (APC) proteins, diet, inflammatory... 
MULTIPLE INTESTINAL NEOPLASIA | ACTIVATION | DIETARY GLYCEMIC LOAD | COLORECTAL-CANCER | INFLAMMATION | BIOCHEMISTRY & MOLECULAR BIOLOGY | DNA-DAMAGE | MISMATCH-REPAIR | BUTYRATE | TUMORIGENESIS | DEFICIENCY | CELL BIOLOGY | MutL Protein Homolog 1 | Butyrates - metabolism | Cell Proliferation | Epithelial Cells - metabolism | Colonic Polyps - pathology | Colorectal Neoplasms - genetics | Colonic Polyps - metabolism | Dietary Carbohydrates - metabolism | MutS Homolog 2 Protein - metabolism | Inflammation - metabolism | Colonic Polyps - microbiology | DNA Mismatch Repair | Colorectal Neoplasms - metabolism | Adenomatous Polyposis Coli Protein - genetics | Inflammation - microbiology | Specific Pathogen-Free Organisms | Mice, Inbred C57BL | Colorectal Neoplasms - microbiology | Nuclear Proteins - metabolism | MutS Homolog 2 Protein - genetics | beta Catenin - metabolism | Animals | Cell Transformation, Neoplastic | Epithelial Cells - microbiology | Adenomatous Polyposis Coli Protein - metabolism | Inflammation - genetics | Mice | Adaptor Proteins, Signal Transducing - metabolism | Colorectal Neoplasms - pathology | Proteins | Colon cancer | Metabolites | Microbiota (Symbiotic organisms) | Analysis | Physiological aspects | Development and progression | Esters | Cell differentiation | Cells | Colorectal cancer | Index Medicus
Journal Article
Journal Article
Immunity, ISSN 1074-7613, 01/2014, Volume 40, Issue 1, pp. 140 - 152
Journal Article
Journal Article
Gut, ISSN 0017-5749, 07/2003, Volume 52, Issue 7, pp. 988 - 997
Background: The colonic epithelium maintains a life long reciprocally beneficial interaction with the colonic microbiota. Disruption is associated with mucosal... 
TIGHT JUNCTIONS | IN-VITRO | SACCHAROMYCES-BOULARDII | MAINTENANCE TREATMENT | ULCERATIVE-COLITIS | BARRIER FUNCTION | DOUBLE-BLIND | EPIDERMAL GROWTH-FACTOR | GASTROENTEROLOGY & HEPATOLOGY | FACTOR RECEPTOR | CHLORIDE SECRETION | Phosphorylation | Epithelial Cells - metabolism | Escherichia coli Infections - prevention & control | Humans | Diarrhea - prevention & control | Colony Count, Microbial | Lactobacillus acidophilus | Inflammatory Bowel Diseases - metabolism | Immunoblotting - methods | Diarrhea - microbiology | Chlorides - metabolism | Colonic Diseases - prevention & control | Membrane Proteins - metabolism | Colonic Diseases - microbiology | Cell Line | Inflammatory Bowel Diseases - prevention & control | ErbB Receptors - metabolism | Colonic Diseases - metabolism | Escherichia coli Infections - microbiology | Escherichia coli Infections - metabolism | Streptococcus | Cell Membrane Permeability - physiology | Cell Adhesion - physiology | Epithelial Cells - microbiology | Cytoskeleton - metabolism | Diarrhea - metabolism | Inflammatory Bowel Diseases - microbiology | Probiotics - therapeutic use | Studies | Inflammatory bowel disease | Oxidative stress | Signal transduction | Probiotics | Epidermal growth factor | E coli | Gastroenterology | Bacteria | Infections | Small intestine | Index Medicus | Abridged Index Medicus | Inflammatory Bowel Disease | enteroinvasive Escherichia coli | epidermal growth factor | probiotics
Journal Article
PLoS ONE, ISSN 1932-6203, 09/2012, Volume 7, Issue 9, pp. e38801 - e38801
Background: Intestinal epithelium is essential for maintaining normal intestinal homeostasis; its breakdown leads to chronic inflammatory pathologies, such as... 
INTESTINAL INFLAMMATION | CALCIUM-BINDING PROTEINS | INFLAMMATORY-BOWEL-DISEASE | MULTIPLE-SCLEROSIS | CROHNS-DISEASE | COLORECTAL-CANCER | MULTIDISCIPLINARY SCIENCES | SERUM-LEVELS | NEUTROPHILS | MRP14 | IL-6 | Dextran Sulfate | RNA, Small Interfering - genetics | Calgranulin B - metabolism | Intestinal Mucosa - metabolism | Cytokine Receptor gp130 - genetics | Humans | Colitis, Ulcerative - genetics | Colon - drug effects | Male | Intestinal Mucosa - drug effects | Chromatin Immunoprecipitation | Calgranulin B - genetics | Interleukin-6 - metabolism | STAT3 Transcription Factor - genetics | STAT3 Transcription Factor - metabolism | Disease Models, Animal | Caco-2 Cells | Promoter Regions, Genetic | Cytokine Receptor gp130 - metabolism | Interleukin-6 - genetics | Colon - pathology | Colitis, Ulcerative - metabolism | Mice, Inbred C57BL | Colitis, Ulcerative - pathology | Colon - metabolism | Gene Expression Regulation - drug effects | Animals | Interleukin-6 - pharmacology | Signal Transduction - drug effects | Colitis, Ulcerative - chemically induced | Cytokine Receptor gp130 - pharmacology | Mice | Intestinal Mucosa - pathology | Dextran | Chromatin | RNA | Interleukins | Analysis | Research | Sulfates | Ulcerative colitis | Phosphorylation | Inflammatory bowel diseases | Immunoprecipitation | Transcription | Physicians | Epithelial cells | Interleukin | Homeostasis | Activation | Kinases | Small intestine | Interleukin 6 | Intestine | Rodents | Animal tissues | Dextran sulfate | Colon | Glycoprotein gp130 | Medical treatment | Stat3 protein | siRNA | Epithelium | Ribonucleic acid--RNA | Inflammatory bowel disease | Immune systems | Fc receptors | Signaling | S100A9 protein | Sodium | Infiltration | Sulfate | Leukocytes (granulocytic) | Index Medicus | Animal models | Inflammation | Promoters | Molecular modelling | Ribonucleic acid
Journal Article
American Journal of Physiology - Gastrointestinal and Liver Physiology, ISSN 0193-1857, 06/2013, Volume 304, Issue 11, pp. 1025 - 1037
Short-chain fatty acids (SCFAs), such as butyrate and propionate, are metabolic products of carbohydrate fermentation by the microbiota and constitute the main... 
Short-chain fatty acids | Fasting-induced adipose factor | Clostridium tyrobutyricum | Peroxisome proliferator-activated receptor-γ | Angiopoietin-like protein 4 | PHYSIOLOGY | COLONIC FUNCTION | GERM-FREE-MICE | BEHAVIOR | fasting-induced adipose factor | angiopoietin-like protein 4 | ANGIOPOIETIN-LIKE PROTEIN-4 | GUT MICROBIOTA | peroxisome proliferator-activated receptor-gamma | PPAR | METABOLISM | CHAIN FATTY-ACIDS | SODIUM-BUTYRATE | ACTIVATED-RECEPTOR-GAMMA | short-chain fatty acids | GASTROENTEROLOGY & HEPATOLOGY | Caco-2 Cells | Transcription, Genetic - drug effects | Angiopoietins - metabolism | Intestinal Mucosa - metabolism | Response Elements | Enterocytes - metabolism | Butyrates - pharmacology | HCT116 Cells | Humans | Mice, Inbred C57BL | Intestinal Mucosa - microbiology | PPAR gamma - metabolism | Hypoglycemic Agents - pharmacology | HT29 Cells | Angiopoietin-like 4 Protein | Animals | Transcription Initiation Site | Germ-Free Life | PPAR gamma - agonists | Mice | Angiopoietins - genetics | Thiazolidinediones - pharmacology | Metagenome | Epithelial cells | Physiological aspects | Genetic aspects | Research | Gene expression | Binding proteins | Health aspects | Fatty acids | Carbohydrates | Metabolites | Digestive system | Bacteria | Fermentation | Metabolism | Index Medicus | Life Sciences | Agricultural sciences | Medicinsk bioteknologi (inriktn. mot cellbiologi (inkl. stamcellsbiologi), molekylärbiologi, mikrobiologi, biokemi eller biofarmaci) | Klinisk medicin | Medicin och hälsovetenskap | Gastroenterologi | Medicinsk bioteknologi
Journal Article
International Journal of Cancer, ISSN 0020-7136, 04/2018, Volume 142, Issue 7, pp. 1453 - 1466
Journal Article
PLoS ONE, ISSN 1932-6203, 10/2013, Volume 8, Issue 10, pp. e75664 - e75664
Journal Article