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1. Hematopoietic growth factors in oncology
by Dale, David C and Lyman, Gary H. 1946- (Gary Herbert)
2011, 1, Cancer treatment and research, ISBN 9781441970725, Volume 157., xv, 433
This volume reviews and integrates the major advances in our understanding of the underlying molecular biology and pharmacology of hematopoietic growth factors... 
drug therapy | pharmacology | Oncology | Mechanism of action | therapeutic use | Neoplasms | Hematopoietic growth factors | Colony-Stimulating Factors | Clinical & internal medicine | Colony-Stimulating Factors - therapeutic use | Medicine & Public Health | Cancer
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2. Full Text The effect of macrophage-colony stimulating factor and other humoral factors (interleukin-1, -3, -6, and -11, tumor necrosis factor-α, and granulocyte macrophage-colony stimulating factor) on human osteoclast formation from circulating cells
by Fujikawa, Y and Sabokbar, A and Neale, S.D and Itonaga, I and Torisu, T and Athanasou, N.A
Bone, ISSN 8756-3282, 2001, Volume 28, Issue 3, pp. 261 - 267
Macrophage-colony stimulating factor (M-CSF) is an essential requirement for human osteoclast formation, but its effect on the proliferation and... 
Bone resorption | Monocytes | Human osteoclasts | IL-3 | M-CSF | GM-CSF | SURVIVAL | monocytes | human osteoclasts | bone resorption | MOUSE | DIFFERENTIATION FACTOR | RECEPTOR | BONE-MARROW CULTURES | OSTEOBLASTS | MUTATION | GROWTH | ENDOCRINOLOGY & METABOLISM | STROMAL CELLS | OSTEOPETROSIS | Tumor Necrosis Factor-alpha - pharmacology | Humans | Granulocyte-Macrophage Colony-Stimulating Factor - pharmacology | Macrophage Colony-Stimulating Factor - pharmacology | Adult | Interleukins - pharmacology | Osteoclasts - cytology | Cell Division - drug effects | Osteoclasts - drug effects | TRANCE protein | Index Medicus
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3. The hemopoietic colony-stimulating factors
: from biology to clinical applications
by Metcalf, Donald and Nicola, Nicos, 1950
1995, ISBN 0521461588, xi, 329
The colony-stimulating factors (CSFs) are the major regulators in the body of the production and activity of two types of white blood cells - granulocytes and... 
Colony-stimulating factors (Physiology) | Receptors, Colony-Stimulating Factor | Hematopoiesis | Colony-Stimulating Factors
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4. Colony-stimulating factors
: molecular and cellular biology
by Garland, John M., 1942 and Hilton, Douglas J. 1964- (Douglas James) and Quesenberry, Peter J
1997, 2nd ed., rev. and expanded., ISBN 0824794923, xii, 566
Colony-stimulating factors (Physiology)
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5. Cytokines in cancer therapy
by Balkwill, Frances R and Balkwill, F. R
1989, Oxford medical publications., ISBN 019261715X, xvi, 297
Chemotherapy | Cytokines | Therapeutic use | Cancer | Testing
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6. Full Text Autologous Bone Marrow Stem Cell Mobilization Induced by Granulocyte Colony-Stimulating Factor After Subacute ST-Segment Elevation Myocardial Infarction Undergoing Late Revascularization. Final Results From the G-CSF-STEMI (Granulocyte Colony-Stimulating Factor ST-Segment Elevation Myocardial Infarction) Trial
by Engelmann, Markus G and Theiss, Hans D and Hennig-Theiss, Christine and Huber, Armin and Wintersperger, Bernd J and Werle-Ruedinger, Anja-Eva and Schoenberg, Stefan O and Steinbeck, Gerhard and Franz, Wolfgang-M
Journal of the American College of Cardiology, ISSN 0735-1097, 10/2006, Volume 48, Issue 8, pp. 1712 - 1721
Objectives: The purpose of this investigator-driven, prospective, randomized, double-blinded, placebo-controlled phase II study was to compare the effects of... 
ARTERIOGENESIS | HEART | REPAIR | CARDIAC & CARDIOVASCULAR SYSTEMS | ISCHEMIC CARDIOMYOPATHY | REGENERATION | GROWTH | DOUBLE-BLIND | RESTENOSIS | NEOVASCULARIZATION | Granulocyte Colony-Stimulating Factor - adverse effects | Heart - physiopathology | Myocardial Infarction - diagnosis | Cardiovascular Diseases - etiology | Double-Blind Method | Humans | Granulocyte Colony-Stimulating Factor - therapeutic use | Angioplasty, Balloon, Coronary | Magnetic Resonance Imaging | Myocardial Infarction - therapy | Time Factors | Electrocardiography | Myocardial Infarction - physiopathology | Coronary Circulation - drug effects | Hematopoietic Stem Cell Mobilization - adverse effects | Enzymes | Automation | Heart attacks | Nuclear magnetic resonance--NMR | Cardiology | Acute coronary syndromes | Veins & arteries | Index Medicus | Abridged Index Medicus
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7. Full Text Macrophage tumor necrosis factor-α induces epithelial expression of granulocyte-macrophage colony-stimulating factor: Impact on alveolar epithelial repair
by Cakarova, Lidija and Marsh, Leigh M and Wilhelm, Jochen and Mayer, Konstantin and Grimminger, Friedrich and Seeger, Werner and Lohmeyer, Juergen and Herold, Susanne
American Journal of Respiratory and Critical Care Medicine, ISSN 1073-449X, 09/2009, Volume 180, Issue 6, pp. 521 - 532
Rationale: Resident alveolar macrophages have been attributed a crucial role in host defense toward pulmonary infection. Their contribution to alveolar repair... 
Acute lung injury | Alveolar repair | Epithelial proliferation | HEPATOCYTE GROWTH-FACTOR | INNATE IMMUNE-RESPONSE | LUNG INJURY | KAPPA-B | acute lung injury | RESPIRATORY-DISTRESS-SYNDROME | IN-VITRO | GM-CSF | RESPIRATORY SYSTEM | INFLAMMATION | epithelial proliferation | alveolar repair | II CELLS | CSF-DEFICIENT MICE | CRITICAL CARE MEDICINE | Inflammation Mediators - immunology | Lung - pathology | Lung Injury - pathology | Enzyme-Linked Immunosorbent Assay | Macrophage Activation - immunology | Mice, Inbred C57BL | Epithelial Cells - pathology | Mice, Transgenic | Reverse Transcriptase Polymerase Chain Reaction | Blotting, Western | Animals | Flow Cytometry | Granulocyte-Macrophage Colony-Stimulating Factor - immunology | Granulocyte-Macrophage Colony-Stimulating Factor - biosynthesis | Epithelial Cells - immunology | Lipopolysaccharides - pharmacology | Tumor Necrosis Factor-alpha - immunology | Mice | In Vitro Techniques | Lung Injury - immunology | Lung - immunology | Macrophages, Alveolar - immunology | Index Medicus | Abridged Index Medicus
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8. Full Text Thyroid Transcription Factor 1 Reprograms Angiogenic Activities of Secretome
by Wood, Lauren W and Cox, Nicole I and Phelps, Cody A and Lai, Shao-Chiang and Poddar, Arjun and Talbot, Conover and Mu, David
Scientific Reports, ISSN 2045-2322, 02/2016, Volume 6, Issue 1, pp. 19857 - 19857
Through both gain-and loss-of-TTF-1 expression strategies, we show that TTF-1 positively regulates vascular endothelial growth factor (VEGF) and that the VEGF... 
SURVIVAL | MORPHOGENESIS | EPITHELIAL-CELLS | GM-CSF | VEGF | MULTIDISCIPLINARY SCIENCES | LUNG ADENOCARCINOMA | GENE-EXPRESSION | NKX2-1 | CANCER | ENDOTHELIAL GROWTH-FACTOR | Exosomes - metabolism | Angiogenic Proteins - genetics | Neovascularization, Physiologic - drug effects | Thyroid Nuclear Factor 1 | TOR Serine-Threonine Kinases - metabolism | Humans | Lung Neoplasms - metabolism | Culture Media, Conditioned - pharmacology | Lung Neoplasms - pathology | Vascular Endothelial Growth Factor A - metabolism | Vascular Endothelial Growth Factor Receptor-2 - immunology | Vascular Endothelial Growth Factor A - genetics | Cell Hypoxia | Transcription Factors - secretion | Nuclear Proteins - secretion | Vascular Endothelial Growth Factor Receptor-2 - antagonists & inhibitors | RNA Interference | Hypoxia-Inducible Factor 1, alpha Subunit - metabolism | Cytokines - genetics | Real-Time Polymerase Chain Reaction | Lung Neoplasms - blood supply | A549 Cells | Promoter Regions, Genetic | Cytokines - metabolism | Enzyme-Linked Immunosorbent Assay | Cell Survival | Granulocyte-Macrophage Colony-Stimulating Factor - pharmacology | Vascular Endothelial Growth Factor Receptor-2 - metabolism | Indoles - toxicity | Nuclear Proteins - metabolism | Transcription Factors - antagonists & inhibitors | Vascular Endothelial Growth Factor A - analysis | Vascular Endothelial Growth Factor Receptor-1 - metabolism | Blotting, Western | Antibodies - pharmacology | Transcription Factors - metabolism | Up-Regulation - drug effects | Granulocyte-Macrophage Colony-Stimulating Factor - immunology | Pyrroles - toxicity | Nuclear Proteins - antagonists & inhibitors | Cell Line, Tumor | Angiogenic Proteins - metabolism | RNA, Small Interfering - metabolism | TOR protein | Statistical analysis | Granulocyte-macrophage colony-stimulating factor | Lung | Lung cancer | Media | Rapamycin | Secretome | Exosomes | Macrophages | Angiogenesis | Hypoxia | Colony-stimulating factor | Vascular endothelial growth factor | Thyroid transcription factor 1 | Thyroid | Index Medicus
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9. Full Text Tumor necrosis factor α stimulates osteoclast differentiation by a mechanism independent of the ODF/RANKL-RANK interaction
by Kobayashi, Kanichiro and Takahashi, Naoyuki and Jimi, Eijiro and Udagawa, Nobuyuki and Takami, Masamichi and Kotake, Shigeru and Nakagawa, Nobuaki and Kinosaki, Masahiko and Yamaguchi, Kyoji and Shima, Nobuyuki and Yasuda, Hisataka and Morinaga, Tomonori and Higashio, Kanji and Martin, T. John and Suda, Tatsuo
Journal of Experimental Medicine, ISSN 0022-1007, 01/2000, Volume 191, Issue 2, pp. 275 - 285
Osteoclast differentiation factor (ODF, also called RANKL/TRANCE/OPGL) stimulates the differentiation of osteoclast progenitors-of the monocyte/macrophage... 
Bone resorption | Nuclear factor- κB | Interleukin-1 | Macrophage colony-stimulating factor | Tumor necrosis factor receptor | MEDICINE, RESEARCH & EXPERIMENTAL | INTERLEUKIN-1 | ACTIVATION | bone resorption | tumor necrosis factor receptor | nuclear factor-kappa B | INHIBITS OSTEOCLASTOGENESIS | OSTEOPROTEGERIN | IMMUNOLOGY | TNF RECEPTOR SUPERFAMILY | BONE-RESORPTION | CELL-DEATH | FAMILY | macrophage colony-stimulating factor interleukin-1 | LIGAND | NF-KAPPA-B | Tumor Necrosis Factor-alpha - metabolism | Receptor Activator of Nuclear Factor-kappa B | Membrane Glycoproteins - metabolism | Humans | Tartrate-Resistant Acid Phosphatase | Male | RANK Ligand | Osteoclasts - cytology | Antigens, CD - genetics | Antigens, CD - metabolism | Acid Phosphatase - metabolism | Isoenzymes - metabolism | Carrier Proteins - pharmacology | Receptors, Tumor Necrosis Factor, Type II | Macrophage Colony-Stimulating Factor - pharmacology | Bone Marrow Cells - drug effects | Cell Differentiation | Osteoclasts - physiology | Receptors, Tumor Necrosis Factor - genetics | Receptors, Tumor Necrosis Factor - metabolism | Interleukin-1 - metabolism | Gene Expression | Bone Marrow Cells - cytology | Membrane Glycoproteins - pharmacology | Mice, Inbred C57BL | Receptors, Tumor Necrosis Factor, Type I | Cells, Cultured | Macrophage Colony-Stimulating Factor - metabolism | Osteoclasts - metabolism | Mice, Knockout | Tumor Necrosis Factor-alpha - pharmacology | Animals | Carrier Proteins - metabolism | Mice | Bone Marrow Cells - metabolism | Osteoclasts - drug effects | RANK protein | RANKL protein | tumor necrosis factor receptors | osteoclast differentiation factor | macrophage colony-stimulating factor | tumor necrosis factor-^a | Index Medicus | Space life sciences | nuclear factor-κB | interleukin-1 | Original
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10. Cytokines and colony stimulating factors
: methods and protocols
by Körholz, Dieter and Kiess, W. (Wieland) and SpringerLink (Online service)
2003, Methods in molecular biology, ISBN 9781588290359, Volume 215, xv, 478
Enormous effort has been spent developing many new laboratory techniques for studying immune cells and the immune system, not only to increase fundamental... 
Cytokines | Colony-stimulating factors (Physiology) | Laboratory manuals | Immunology | Biomedicine
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11. Full Text Association between placental lesions, cytokines and angiogenic factors in pregnant women with preeclampsia
by Weel, Ingrid C and Baergen, Rebecca N and Romão-Veiga, Mariana and Borges, Vera T and Ribeiro, Vanessa R and Witkin, Steven S and Bannwart-Castro, Camila and Peraçoli, Jose C and De Oliveira, Leandro and Peraçoli, Maria T
PLoS ONE, ISSN 1932-6203, 06/2016, Volume 11, Issue 6, pp. e0157584 - e0157584
Preeclampsia (PE) is considered the leading cause of maternal and perinatal morbidity and mortality. The placenta seems to play an essential role in this... 
PATHOGENESIS | IN-VITRO | MULTIDISCIPLINARY SCIENCES | SYNCYTIAL KNOTS | EARLY-ONSET | SOLUBLE ENDOGLIN | NECROSIS-FACTOR-ALPHA | RESTRICTION | HYPOXIA | ENDOTHELIAL GROWTH-FACTOR | PATHOPHYSIOLOGY | Tumor Necrosis Factor-alpha - metabolism | Granulocyte-Macrophage Colony-Stimulating Factor - metabolism | Humans | Placenta Growth Factor - metabolism | Vascular Endothelial Growth Factor Receptor-1 - metabolism | Vascular Endothelial Growth Factor A - metabolism | Pre-Eclampsia - pathology | Placenta - metabolism | Pregnancy | Age of Onset | Placenta - pathology | Interleukin-10 - metabolism | Adult | Female | Pre-Eclampsia - metabolism | Immunohistochemistry | Laboratories | Interleukin | Kinases | Macrophages | Medical schools | Cell adhesion & migration | Angiogenesis | Endoglin | Immunology | Histopathology | Tumor necrosis factor-TNF | Colony-stimulating factor | Lesions | Vascular endothelial growth factor | Age | Knots | Tyrosine | Hypertension | Transforming growth factor-b1 | Cytokines | Granulocyte-macrophage colony-stimulating factor | Gynecology | Pre-eclampsia | Tumor necrosis factor-α | Obstetrics | Morbidity | Fibrin | Placenta | Womens health | Preeclampsia | Interleukin 10 | Hypoxia | Index Medicus
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12. Colony-stimulating factors
: molecular and cellular biology
by Dexter, T. Michael and Garland, John M and Testa, Nydia G
1990, ISBN 9780824780944, Volume 49., xvi, 475
Colony-stimulating factors (Physiology)
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13. Full Text Mast cells decrease efficacy of anti-angiogenic therapy by secreting matrix-degrading granzyme B
by Wroblewski, M and Bauer, R and Cubas Córdova, M and Udonta, F and Ben-Batalla, I and Legler, K and Hauser, C and Egberts, J and Janning, M and Velthaus, J and Schulze, C and Pantel, K and Bokemeyer, C and Loges, S
Nature Communications, ISSN 2041-1723, 12/2017, Volume 8, Issue 1, pp. 269 - 11
Resistance towards VEGF-centered anti-angiogenic therapy still represents a substantial clinical challenge. We report here that mast cells alter the... 
BREAST-CANCER | NEOADJUVANT CHEMOTHERAPY | TYROSINE KINASE | MULTIDISCIPLINARY SCIENCES | IN-VIVO | C-KIT | EXTRACELLULAR-MATRIX | HEPARAN-SULFATE PROTEOGLYCANS | TUMOR ANGIOGENESIS | VASCULAR-PERMEABILITY | ENDOTHELIAL GROWTH-FACTOR | Fibroblast Growth Factor 2 - drug effects | Human Umbilical Vein Endothelial Cells | Granulocyte-Macrophage Colony-Stimulating Factor - metabolism | Granzymes - metabolism | Extracellular Matrix - metabolism | Vascular Endothelial Growth Factor A - metabolism | Fibroblast Growth Factor 1 - drug effects | Cromolyn Sodium - pharmacology | Fibroblast Growth Factor 2 - metabolism | Vascular Endothelial Growth Factor A - drug effects | Anti-Asthmatic Agents - pharmacology | Vascular Endothelial Growth Factor Receptor-2 - drug effects | Endothelial Cells - metabolism | Extracellular Matrix - drug effects | Fibroblast Growth Factor 1 - metabolism | Angiogenesis Inhibitors - pharmacology | Vascular Endothelial Growth Factor Receptor-2 - metabolism | Neoplasms - blood supply | Vitronectin - metabolism | Mast Cells - drug effects | Granulocyte-Macrophage Colony-Stimulating Factor - drug effects | Neoplasms - drug therapy | Animals | Cell Line, Tumor | Mast Cells - secretion | Cell Proliferation - drug effects | Mice | Neovascularization, Pathologic - metabolism | Laminin - metabolism | Endothelial Cells - drug effects | Fibroblast growth factor 2 | Therapy | Fibroblast growth factor | Fibroblast growth factor 1 | Granulocyte-macrophage colony-stimulating factor | Secretion | Endothelial cells | Histamine | Angiogenesis | Granzyme B | Antiangiogenics | Mast cells | Vascular endothelial growth factor | Index Medicus
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14. Full Text Interleukin-1 beta and tumor necrosis factor-alpha induce gene expression and production of leukocyte chemotactic factors, colony- stimulating factors, and interleukin-6 in human mesangial cells
by Zoja, C and Wang, JM and Bettoni, S and Sironi, M and Renzi, D and Chiaffarino, F and Abboud, HE and Van Damme, J and Mantovani, A and Remuzzi, G
American Journal of Pathology, ISSN 0002-9440, 04/1991, Volume 138, Issue 4, pp. 991 - 1003
The capacity of human cultured mesangial cells to produce soluble factors potentially relevant for mechanisms of inflammation and immunity at the glomerular... 
HUMAN GLOMERULONEPHRITIS | COMPLEMENTARY-DNA | MESSENGER-RNAS | CYTOKINES | CELLULAR-IMMUNITY | ENDOTHELIAL-CELLS | PURIFICATION | MOLECULAR-CLONING | MONONUCLEAR LEUKOCYTES | GROWTH-FACTOR | PATHOLOGY | Chemotaxis, Leukocyte | Interleukin-8 - genetics | Interleukin-6 - genetics | Humans | Cells, Cultured | Colony-Stimulating Factors - biosynthesis | Colony-Stimulating Factors - genetics | Glomerular Mesangium - cytology | Glomerular Mesangium - physiology | Interleukin-1 - pharmacology | Gene Expression Regulation - drug effects | Tumor Necrosis Factor-alpha - pharmacology | Chemotactic Factors - biosynthesis | Glomerular Mesangium - metabolism | Chemotactic Factors - genetics | Interleukin-6 - biosynthesis | Chemokine CCL2 | Cytokines - pharmacology | Index Medicus | Abridged Index Medicus | Regular
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15. The hemopoietic colony stimulating factors
by Metcalf, D and Metcalf, Donald
1984, ISBN 0444805648, xv, 493
Hematopoiesis | Colony-stimulating factors (Physiology) | Hematopoietic stem cells
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16. Hematopoietic growth factors in oncology
by Lyman, Gary H., M.D and Dale, David C
2011, Cancer treatment and research, ISBN 9781441970725, Volume 157
drug therapy | pharmacology | Oncology | Mechanism of action | therapeutic use | Neoplasms | Hematopoietic growth factors | Colony-Stimulating Factors
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17. Full Text Osteoclast Differentiation Factor is a Ligand for Osteoprotegerin/Osteoclastogenesis-Inhibitory Factor and is Identical to TRANCE/RANKL
by Hisataka Yasuda and Nobuyuki Shima and Nobuaki Nakagawa and Kyoji Yamaguchi and Masahiko Kinosaki and Shin-Ichi Mochizuki and Akihiro Tomoyasu and Kazuki Yano and Masaaki Goto and Akihiko Murakami and Eisuke Tsuda and Tomonori Morinaga and Kanji Higashio and Nobuyuki Udagawa and Naoyuki Takahashi and Tatsuo Suda
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 3/1998, Volume 95, Issue 7, pp. 3597 - 3602
Osteoclasts, the multinucleated cells that resorb bone, develop from hematopoietic cells of monocyte/macrophage lineage. Osteoclast-like cells (OCLs) are... 
COS cells | Cell culture techniques | Spleen cells | Complementary DNA | Stromal cells | Cell lines | Stem cells | Osteoclasts | Cultured cells | Cells | IN-VITRO | COLONY-STIMULATING FACTOR | MULTIDISCIPLINARY SCIENCES | MOUSE | MONOCYTES | SUPERFAMILY | GENERATION | STROMAL CELLS | PROTEINS | MODULATION | OSTEOBLASTIC CELLS | Glycoproteins - agonists | Cell Line | Receptor Activator of Nuclear Factor-kappa B | Bone Marrow Cells - cytology | Membrane Glycoproteins - pharmacology | DNA, Complementary - genetics | Molecular Sequence Data | RANK Ligand | Glycoproteins - pharmacology | Osteoclasts - cytology | DNA, Complementary - isolation & purification | Receptors, Cytoplasmic and Nuclear | Membrane Glycoproteins - genetics | Osteoprotegerin | Animals | Receptors, Tumor Necrosis Factor | Cell Differentiation - drug effects | Cell Communication - drug effects | Cloning, Molecular | Ligands | Mice | Stromal Cells - cytology | Carrier Proteins | Physiological aspects | Bone cells | Research | Cell differentiation | Cellular biology | Cloning | Index Medicus | Biological Sciences
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