X
Search Filters
Format Format
Subjects Subjects
Subjects Subjects
X
Sort by Item Count (A-Z)
Filter by Count
commd1 (50) 50
index medicus (32) 32
humans (27) 27
animals (26) 26
biochemistry & molecular biology (26) 26
copper (18) 18
gene (17) 17
adaptor proteins, signal transducing - metabolism (15) 15
cell biology (15) 15
murr1 (15) 15
proteins (15) 15
adaptor proteins, signal transducing - genetics (14) 14
female (14) 14
copper - metabolism (13) 13
mice (13) 13
nf-kappa-b (13) 13
article (12) 12
dogs (11) 11
activation (10) 10
atp7b (10) 10
male (10) 10
protein (10) 10
adaptor proteins, signal transducing (9) 9
copper-transporting atpases (9) 9
genes (9) 9
analysis (8) 8
copper toxicosis (8) 8
liver (8) 8
mutation (8) 8
nf-κb (8) 8
physiological aspects (8) 8
cancer (7) 7
cation transport proteins - genetics (7) 7
hepatolenticular degeneration - genetics (7) 7
homeostasis (7) 7
life sciences (7) 7
mutations (7) 7
nf-kappa b (7) 7
ubiquitination (7) 7
adenosine triphosphatases - genetics (6) 6
apoptosis (6) 6
bedlington terriers (6) 6
disease (6) 6
endocrinology & metabolism (6) 6
epithelial sodium-channel (6) 6
metabolism (6) 6
nf-kappa b - metabolism (6) 6
oncology (6) 6
proteins - genetics (6) 6
amino acid sequence (5) 5
bedlington terrier (5) 5
carrier proteins - metabolism (5) 5
cation transport proteins - metabolism (5) 5
copper - toxicity (5) 5
degradation (5) 5
disease models, animal (5) 5
dog diseases - genetics (5) 5
expression (5) 5
hek293 cells (5) 5
hepatolenticular degeneration - metabolism (5) 5
identification (5) 5
inflammation (5) 5
menkes disease (5) 5
toxicosis (5) 5
trafficking (5) 5
transcription (5) 5
wilson disease (5) 5
xiap (5) 5
adenosine triphosphatases - metabolism (4) 4
atp7a (4) 4
binding (4) 4
canine copper toxicosis (4) 4
carrier proteins - genetics (4) 4
cullin (4) 4
dog (4) 4
gene expression (4) 4
genetics & heredity (4) 4
hypoxia (4) 4
liver-disease (4) 4
localization (4) 4
metal metabolism, inborn errors - genetics (4) 4
molecular sequence data (4) 4
oxidative stress (4) 4
phosphorylation (4) 4
population (4) 4
proteolysis (4) 4
transporting atpase (4) 4
ubiquitin (4) 4
ubiquitin ligase (4) 4
veterinary sciences (4) 4
wilson-disease (4) 4
wilson-disease gene (4) 4
acetylation (3) 3
adaptor proteins, signal transducing - physiology (3) 3
adult (3) 3
alpha (3) 3
antineoplastic agents - pharmacology (3) 3
atpase (3) 3
biochemistry, general (3) 3
biophysics (3) 3
more...
Language Language
Publication Date Publication Date
Click on a bar to filter by decade
Slide to change publication date range


JOURNAL OF CELL SCIENCE, ISSN 0021-9533, 10/2019, Volume 132, Issue 19
Copper-responsive intracellular ATP7B trafficking is crucial for maintaining the copper balance in mammalian hepatocytes and thus copper levels in organs. The... 
PROTEIN | Trafficking | Lysosome | COMPLEXES | COMMD1 | PHOSPHOINOSITIDES | ATPASES | CELL BIOLOGY | ATP7B | GENE | ROLES | TOXICOSIS | Endosome | ARF6
Journal Article
Veterinary Sciences, ISSN 2306-7381, 09/2019, Volume 6, Issue 4, p. 78
Wilson’s disease (WD), an autosomal recessive disorder, results in copper accumulation in the liver as a consequence of mutations in the gene ATPase copper... 
copper toxicosis | Bedlington terrier | nuclear receptor | COMMD1
Journal Article
The EMBO Journal, ISSN 0261-4189, 01/2007, Volume 26, Issue 2, pp. 436 - 447
NF‐κB is a pleiotropic transcription factor involved in multiple processes, including inflammation and oncogenesis. We have previously reported that COMMD1... 
NF‐κB | COMMD1 | cullin | transcription | ubiquitination | NF-κB | Ubiquitination | Transcription | Cullin | ACTIVATION | DOMAIN | PHOSPHORYLATION | PROTEASOME PATHWAY | BIOCHEMISTRY & MOLECULAR BIOLOGY | ALPHA | SOCS-1 | CELL BIOLOGY | NF-kappa B | GENE | DEGRADATION | PROTEINS | KINASES
Journal Article
Trends in Genetics, ISSN 0168-9525, 10/2019, Volume 35, Issue 10, pp. 768 - 780
The nuclear organelle the nucleolus and the transcription factor nuclear factor of κ-light-chain-enhancer of activated B cells (NF-κB) are both central to the... 
RRN3 | TIF-IA | NF-kappaB | COMMD1 | nucleoli | nucleophosmin | RelA | FACTOR TIF-IA | ACTIVATION | NUCLEAR | PHOSPHORYLATION | GENETICS & HEREDITY | GENE-EXPRESSION | RIBOSOMAL-RNA SYNTHESIS | CELL-CYCLE | TRANSCRIPTION FACTOR | CANCER | POLYMERASE-I
Journal Article
Cell Reports, ISSN 2211-1247, 08/2018, Volume 24, Issue 9, pp. 2342 - 2355
Physiologic microbe-host interactions in the intestine require the maintenance of the microbiota in a luminal compartment through a complex interplay between... 
intestinal microbiota | myeloid cells | goblet cells | COX-2 | colon cancer | Tuft cells | CCC complex | PGE-2 | Commd1 | IMMUNITY | GENE | INFLAMMATION | DISEASE | MICE | TUFT CELLS | COMMD1 | CANCER | TUMORIGENESIS | CYCLOOXYGENASE 2 | CELL BIOLOGY
Journal Article
Molecular Cancer Research, ISSN 1541-7786, 01/2010, Volume 8, Issue 1, pp. 119 - 130
Secretory clusterin (sCLU) is a stress-activated, cytoprotective chaperone that confers broad-spectrum cancer treatment resistance, and its targeted inhibitor... 
APOPTOSIS | ONCOLOGY | UBIQUITIN LIGASE | ANTISENSE OLIGODEOXYNUCLEOTIDE | GENE-EXPRESSION | ALPHA | BETA-CATENIN | ANDROGEN-INDEPENDENCE | UP-REGULATION | PHASE-I | PROGRESSION | CELL BIOLOGY | clusterin | NF-κB | prostate cancer | COMMD1 | I-κB
Journal Article
Genes and Development, ISSN 0890-9369, 04/2009, Volume 23, Issue 7, pp. 849 - 861
The transcription factor NF-kappa B is a critical regulator of inflammatory and cell survival signals. Proteasomal degradation of NF-kappa B subunits plays an... 
Ubiquitin | Cul2 | NF-κB | GCN5 | COMMD1 | RelA | ACTIVATION | PHOSPHORYLATION | ACETYLATION | B-DEPENDENT TRANSCRIPTION | ALPHA | DEVELOPMENTAL BIOLOGY | PROTEOLYSIS | MURR1 | ubiquitin | CELL BIOLOGY | P65 SUBUNIT | NF-kappa B | GENE | GENETICS & HEREDITY | DEGRADATION | Research Paper
Journal Article
Journal of Veterinary Clinics, ISSN 1598-298X, 2015, Volume 32, Issue 1, pp. 1 - 4
This study was performed to survey prevalence of Copper metabolism domain containing 1 (COMMD1) mutation using molecular diagnostic method in a population of... 
Copper toxicosis | COMMD1 mutation | Bedlington terriers | 수의학
Journal Article
Journal of Peptide Science, ISSN 1075-2617, 11/2014, Volume 20, Issue 11, pp. 850 - 859
Journal Article
Metallomics, ISSN 1756-5901, 2018, Volume 10, Issue 3, pp. 378 - 387
Wilson disease (WD), a Mendelian disorder of copper metabolism caused by mutations in the ATP7B gene, manifests a large spectrum of phenotypic variability.... 
Journal Article
Bioscience, Biotechnology, and Biochemistry, ISSN 0916-8451, 05/2019, Volume 83, Issue 5, pp. 845 - 850
Journal Article
Immunity, ISSN 1074-7613, 07/2017, Volume 47, Issue 1, pp. 66 - 79.e5
Hypoxia augments inflammatory responses and osteoclastogenesis by incompletely understood mechanisms. We identified COMMD1 as a cell-intrinsic negative... 
hypoxia | arthritis | inflammation | glycolysis | E2F | macrophage | metabolism | COMMD1 | CKB | osteoclast | RHEUMATOID-ARTHRITIS | ACTIVATION | MITOCHONDRIAL BIOGENESIS | RNA-SEQ | NEGATIVE REGULATION | JOINT DESTRUCTION | MECHANISMS | DIFFERENTIATION | IMMUNOLOGY | CREATINE-KINASE | BONE-RESORPTION | RNA, Small Interfering - genetics | Signal Transduction | Humans | Mice, Inbred C57BL | Middle Aged | Cells, Cultured | Male | NF-kappa B - metabolism | E2F1 Transcription Factor - metabolism | Hypoxia - metabolism | Mice, Knockout | Animals | Adaptor Proteins, Signal Transducing - genetics | Female | Mice | Adaptor Proteins, Signal Transducing - metabolism | Arthritis, Rheumatoid - immunology | Macrophages - immunology | Osteogenesis - genetics | Disease Models, Animal | Glucose metabolism | Yuan (China) | Medical colleges | Rheumatoid factor | Analysis | Physiological aspects | Arthritis | Inflammation | Macrophages | Population genetics | Regulators | Phosphorylation | Transcription factors | Destruction | Genes | Biosynthesis | Kinases | Osteolysis | Proteins | E2F protein | Signal transduction | Clonal deletion | Metabolites | Rodents | Deletion | Tumor necrosis factor-TNF | Biocompatibility | TRANCE protein | Copper | Erosion | Cytokines | Metabolism | Gene expression | E2F1 protein | Quantitative trait loci | Osteoclastogenesis | Rheumatoid arthritis | Glycolysis | Hypoxia | Bone | Cancer
Journal Article