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The Journal of immunology (1950), ISSN 1550-6606, 2014, Volume 192, Issue 6, pp. 2837 - 2845
Inflammation is associated with development of atherosclerosis, and cholesterol crystals (CC) have long been recognized as a hallmark of atherosclerotic... 
ATHEROSCLEROTIC LESIONS | TUMOR-NECROSIS-FACTOR | C5A RECEPTOR | MEMBRANE ATTACK | STERILE INFLAMMATION | EPITHELIAL-CELLS | CARDIOVASCULAR EVENTS | INTERLEUKIN-1-BETA | OXIDATIVE BURST | ANAPHYLATOXINS | IMMUNOLOGY | Inflammasomes - metabolism | Reactive Oxygen Species - metabolism | Complement C1q - immunology | Humans | Complement Pathway, Classical - drug effects | Complement C5a - metabolism | Complement System Proteins - immunology | Caspase 1 - metabolism | Monocytes - metabolism | Caspase 1 - immunology | Macrophage-1 Antigen - immunology | Monocytes - immunology | Interleukin-1beta - genetics | Complement Activation - immunology | Complement C1q - metabolism | Complement System Proteins - metabolism | Inflammasomes - drug effects | Reactive Oxygen Species - immunology | Complement C5 - immunology | Tumor Necrosis Factor-alpha - immunology | Macrophage-1 Antigen - metabolism | Complement Activation - drug effects | Cytokines - immunology | Interleukin-1beta - secretion | Complement C5 - metabolism | Tumor Necrosis Factor-alpha - secretion | Cells, Cultured | Cytokines - secretion | Phagocytosis - immunology | Interleukin-1beta - immunology | Cholesterol - metabolism | Reverse Transcriptase Polymerase Chain Reaction | Blotting, Western | Monocytes - drug effects | Complement Pathway, Classical - immunology | Microscopy, Confocal | Complement Pathway, Alternative - drug effects | Complement Pathway, Alternative - immunology | Inflammasomes - immunology | Cholesterol - pharmacology | Complement C5a - immunology
Journal Article
Kidney international, ISSN 0085-2538, 2018, Volume 94, Issue 4, pp. 689 - 700
Journal Article
Proceedings of the National Academy of Sciences - PNAS, ISSN 0027-8424, 5/2013, Volume 110, Issue 21, pp. 8650 - 8655
Complement C1q is a hexameric molecule assembled from 18 polypeptide chains of three different types encoded by three genes. This versatile recognition protein... 
Proteins | Molecules | Cell lines | Materials | Cell aggregates | Ligands | Mass spectroscopy | Biochemistry | Electron microscopy | Molecular chains | C1 complex | Protein engineering | Innate immunity | B-CHAIN | ACTIVATION | 1ST COMPONENT | PROTEIN | MULTIDISCIPLINARY SCIENCES | LONG PENTRAXIN PTX3 | LIGAND-RECOGNITION | MANNAN-BINDING LECTIN | protein engineering | innate immunity | STRUCTURAL BASIS | PATHWAY | SUBCOMPONENT C1Q | Surface Plasmon Resonance | Complement C1r - genetics | Calcium - metabolism | Humans | Complement C1s - metabolism | Mutation, Missense | Complement C1q - metabolism | Complement C1r - metabolism | Complement C1s - genetics | Complement C1r - chemistry | Complement C1s - chemistry | C-Reactive Protein - metabolism | HEK293 Cells | Protein Structure, Quaternary | Binding Sites | Complement C1q - chemistry | Complement Activation - physiology | Protein Structure, Tertiary | Recombinant Proteins - metabolism | Gene Expression | Immunoglobulin G - chemistry | Serum Amyloid P-Component - chemistry | Recombinant Proteins - chemistry | Recombinant Proteins - genetics | Complement C1q - genetics | C-Reactive Protein - chemistry | Serum Amyloid P-Component - metabolism | Amino Acid Substitution | Immunoglobulin G - metabolism | Protein Binding - physiology | Bone morphogenetic proteins | Polypeptides | Research | Properties | Proteases | Protein binding | C-Reactive Protein | Calcium | Complement Activation | Immunoglobulin G | Biochemistry, Molecular Biology | Serum Amyloid P-Component | Recombinant Proteins | Complement C1q | Complement C1s | Complement C1r | Life Sciences | Protein Binding | Biomolecules | Biological Sciences
Journal Article
Obesity Research, ISSN 1930-7381, 06/2003, Volume 11, Issue 6, pp. 699 - 708
Journal Article
Blood, ISSN 1528-0020, 2010, Volume 115, Issue 2, pp. 379 - 387
Journal Article
American journal of transplantation, ISSN 1600-6135, 03/2013, Volume 13, Issue 3, pp. 663 - 675
Atypical hemolytic and uremic syndrome (aHUS) is a severe disease strongly associated with genetic abnormalities in the complement alternative pathway. In... 
Acute rejection | endothelial cell damage | mutations and polymorphisms in complement alternative pathway | thrombotic microangiopathy | atypical hemolytic and uremic syndrome | renal transplantation | SURGERY | FACTOR-B | HUS | FACTOR-I | FACTOR-H-AUTOANTIBODIES | TRANSPLANTATION | KIDNEY-TRANSPLANTATION | MUTATIONS | CD46 | PREDISPOSE | MEMBRANE COFACTOR PROTEIN | Recurrence | Genetic Testing | Prognosis | Humans | Middle Aged | Male | Young Adult | Hemolytic-Uremic Syndrome - genetics | Complement Factor B - genetics | Adult | Complement System Proteins - genetics | Female | Hemolytic-Uremic Syndrome - therapy | Retrospective Studies | Complement C3 - genetics | Biomarkers - metabolism | Risk Factors | Biomarkers - analysis | Kidney Transplantation | Mutation - genetics | Fibrinogen - genetics | Graft Survival - genetics | Adolescent | Membrane Cofactor Protein - genetics | Aged | Complement Factor H - genetics | Graft Rejection - genetics | Atypical Hemolytic Uremic Syndrome | Medical research | Kidneys | Genes | Organ transplant recipients | Medicine, Experimental | Genetic research | Transplantation | Clinical trials | Mutation | Transplants & implants | Drug therapy | Biological Markers | Complement C3 | Life Sciences | Immunology | Fibrinogen | Hemolytic-Uremic Syndrome | Antigens, CD46 | Graft Survival | Complement Factor H | Complement System Proteins | Complement Factor B | Graft Rejection
Journal Article
The Journal of biological chemistry, ISSN 0021-9258, 03/2012, Volume 287, Issue 11, pp. 8092 - 8100
PRELP is a 58-kDa proteoglycan found in a variety of extracellular matrices, including cartilage and at several basement membranes. In rheumatoid arthritis... 
C4B-BINDING PROTEIN | DOMAIN | BIOCHEMISTRY & MOLECULAR BIOLOGY | GLYCOPROTEINS | EXTRACELLULAR-MATRIX | CD59 | ARTHRITIS | HEPARIN | BINDING | Complement C9 - chemistry | Complement Membrane Attack Complex - metabolism | Humans | Glycoproteins - metabolism | Male | Complement Membrane Attack Complex - chemistry | Complement System Proteins - chemistry | Basement Membrane - pathology | Complement Membrane Attack Complex - genetics | Complement System Proteins - metabolism | Basement Membrane - chemistry | Arthritis, Rheumatoid - metabolism | Complement C4b-Binding Protein - genetics | HEK293 Cells | Complement System Proteins - genetics | Female | Glycoproteins - chemistry | Extracellular Matrix Proteins - metabolism | Glycoproteins - genetics | Complement C3-C5 Convertases - metabolism | Extracellular Matrix Proteins - chemistry | Complement C9 - metabolism | Extracellular Matrix Proteins - genetics | Complement C4b-Binding Protein - metabolism | Complement Membrane Attack Complex - antagonists & inhibitors | Basement Membrane - metabolism | Complement C3-C5 Convertases - chemistry | Down-Regulation - genetics | Arthritis, Rheumatoid - pathology | Complement Activation - genetics | Arthritis, Rheumatoid - genetics | Complement C3-C5 Convertases - genetics | Complement C4b-Binding Protein - chemistry | Complement C9 - genetics | Complement System | Ligand-binding Protein | Immunology | Extracellular Matrix Proteins | Arthritis | PRELP | Heparin-binding Protein | Rheumatology and Autoimmunity | Basic Medicine | Other Basic Medicine | Clinical Medicine | Andra medicinska och farmaceutiska grundvetenskaper | Medical and Health Sciences | Medicin och hälsovetenskap | Reumatologi och inflammation | Medicinska och farmaceutiska grundvetenskaper | Klinisk medicin
Journal Article
The Journal of immunology (1950), ISSN 1550-6606, 2006, Volume 177, Issue 3, pp. 1872 - 1878
The objective of this study was to explore the role of classical, lectin, and alternative pathways of complement activation in laser-induced choroidal... 
RNA INTERFERENCE | DRUSEN FORMATION | PATHOGENESIS | MEMBRANE ATTACK COMPLEX | ANGIOGENESIS | SUBRETINAL NEOVASCULARIZATION | MOUSE MODEL | DISEASE | GENES | MACULAR DEGENERATION | IMMUNOLOGY | Complement C5 - genetics | Complement Factor H - antagonists & inhibitors | Complement C1q - biosynthesis | Vascular Endothelial Growth Factor A - biosynthesis | Injections, Intravenous | Complement Membrane Attack Complex - metabolism | Male | Vascular Endothelial Growth Factor A - antagonists & inhibitors | Transforming Growth Factor beta - biosynthesis | Complement Factor B - genetics | Lasers | Transforming Growth Factor beta - antagonists & inhibitors | Complement Pathway, Alternative - genetics | Transforming Growth Factor beta2 | Complement Factor B - physiology | Complement Factor H - physiology | Choroidal Neovascularization - immunology | Mice, Inbred C57BL | Up-Regulation - genetics | Choroidal Neovascularization - prevention & control | Complement Factor H - biosynthesis | Complement C1q - genetics | Complement C5 - deficiency | Complement C4 - genetics | Complement Factor B - biosynthesis | Complement C4 - deficiency | Down-Regulation - genetics | Mice, Knockout | Animals | Up-Regulation - immunology | Complement C1q - antagonists & inhibitors | Down-Regulation - immunology | Complement Pathway, Alternative - immunology | Mice | RNA, Small Interfering - administration & dosage | Choroidal Neovascularization - genetics | Complement Factor B - antagonists & inhibitors
Journal Article
Clinical and experimental immunology, ISSN 0009-9104, 04/2016, Volume 184, Issue 1, pp. 118 - 125
Journal Article