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Journal Article
Immunological Reviews, ISSN 0105-2896, 11/2016, Volume 274, Issue 1, pp. 33 - 58
Journal Article
Journal Article
PLoS ONE, ISSN 1932-6203, 07/2012, Volume 7, Issue 7, p. e40495
Journal Article
JOURNAL OF BIOLOGICAL CHEMISTRY, ISSN 0021-9258, 01/2013, Volume 288, Issue 3, pp. 1726 - 1738
Complement C3 is a pivotal component of three cascades of complement activation. The liver is the main source of C3 in circulation and expression and secretion... 
PPAR-ALPHA | 3RD COMPONENT | FACTOR-B | PATHWAY | NUCLEAR RECEPTORS | INFLAMMATION | BIOCHEMISTRY & MOLECULAR BIOLOGY | ACYLATION-STIMULATING PROTEIN | X-RECEPTOR | FATTY-ACIDS | KAPPA-B | Inflammation - chemically induced | Humans | Male | NF-kappa B - metabolism | Hepatocytes - metabolism | Gene Knockdown Techniques | Lipopolysaccharides | MAP Kinase Kinase 1 - genetics | Complement C3 - antagonists & inhibitors | Inflammation - metabolism | Transfection | p38 Mitogen-Activated Protein Kinases - metabolism | Complement C3 - genetics | MAP Kinase Kinase 2 - genetics | Hepatocytes - drug effects | PPAR alpha - immunology | Response Elements | Signal Transduction | Complement C3 - agonists | Mice, Inbred C57BL | p38 Mitogen-Activated Protein Kinases - genetics | MAP Kinase Kinase 1 - metabolism | PPAR alpha - genetics | Peroxisome Proliferators - pharmacology | Inflammation - immunology | MAP Kinase Kinase 2 - metabolism | Pyrimidines - pharmacology | Hepatocytes - immunology | Complement C3 - immunology | Hep G2 Cells | Gene Expression Regulation - drug effects | Tumor Necrosis Factor-alpha - pharmacology | Animals | NF-kappa B - genetics | Inflammation - genetics | Protein Binding | Mice | PPARalpha | Immunology | Complement C3 | Tumor Necrosis Factor (TNF) | Complement | Peroxisome Proliferator-activated Receptor (PPAR) | NF-kB Transcription Factor | Hepatocellular Carcinoma
Journal Article
Lupus, ISSN 0961-2033, 11/2012, Volume 21, Issue 13, pp. 1423 - 1432
Objective: Patients with systemic lupus erythematosus (SLE) have an increased risk of developing vascular diseases (VD) such as myocardial infarction, stroke... 
cardiovascular disease | platelet | Systemic lupus erythematosus | complement | venous thrombosis | COMPLEMENT ACTIVATION | COMPLEXES | ANTIPHOSPHOLIPID ANTIBODIES | RECEPTOR | RHEUMATOLOGY | DAMAGE | DISEASE | INDEX | BINDING | ASSOCIATION | Lupus Coagulation Inhibitor - blood | Up-Regulation | Lupus Erythematosus, Systemic - complications | Blood Platelets - immunology | Humans | Middle Aged | Venous Thrombosis - blood | Male | Lupus Erythematosus, Systemic - blood | Case-Control Studies | Complement C4b - analysis | Complement C1q - analysis | Young Adult | Flow Cytometry | Venous Thrombosis - immunology | Lupus Erythematosus, Systemic - immunology | Aged, 80 and over | Adult | Female | Complement Pathway, Classical | Complement C3d - analysis | Risk Factors | Biomarkers - blood | Venous Thrombosis - etiology | Peptide Fragments - analysis | Platelet Activation | Aged | Myocardial infarction | complement component C4 | Anticoagulants | Flow cytometry | Stroke | Centrifugation | Data processing | Thrombosis | Blood | Risk factors | antiphospholipid antibodies | Complement component C1q | Complement activation | Vascular diseases | Complement component C3 | Platelets | Sexually-transmitted diseases | Rheumatology and Autoimmunity | Clinical Medicine | Medical and Health Sciences | Klinisk medicin | Medicin och hälsovetenskap | Reumatologi och inflammation
Journal Article
Immunobiology, ISSN 0171-2985, 2012, Volume 217, Issue 11, pp. 1106 - 1110
Journal Article
Kidney International, ISSN 0085-2538, 04/2018, Volume 93, Issue 4, pp. 977 - 985
C3 glomerulonephritis (C3GN) and dense deposit disease comprise the two classes of C3 glomerulopathy. Studies from Europe and Asia have aided our understanding... 
dense deposit disease | glomerulonephritis | C3 glomerulopathy | alternative complement pathway | COMPLEMENT ABNORMALITIES | VARIANTS | PATHOLOGY | MPGN | CONSENSUS REPORT | FEATURES | OXFORD CLASSIFICATION | UROLOGY & NEPHROLOGY | IGA NEPHROPATHY | MONOCLONAL GAMMOPATHY | Complement C3 - analysis | United States - epidemiology | Kidney - pathology | Humans | Middle Aged | Kidney - immunology | Male | Atrophy | Renal Insufficiency, Chronic - epidemiology | Young Adult | Time Factors | Kidney Failure, Chronic - pathology | Adult | Female | Retrospective Studies | Complement Pathway, Alternative - genetics | Kidney - physiopathology | Glomerulonephritis, Membranoproliferative - pathology | Kidney Failure, Chronic - immunology | Renal Insufficiency, Chronic - immunology | Risk Factors | Biomarkers - analysis | Treatment Outcome | Glomerulonephritis, Membranoproliferative - therapy | Remission Induction | Disease Progression | Renal Insufficiency, Chronic - pathology | Autoantibodies - immunology | Glomerulonephritis, Membranoproliferative - immunology | Kidney Failure, Chronic - epidemiology | Biopsy | Complement Pathway, Alternative - immunology | Adolescent | Fibrosis | Glomerulonephritis, Membranoproliferative - epidemiology | Glomerulonephritis | End-stage renal disease | Pediatrics | Autoantibodies | Disease | Patients | Glomerular filtration rate | Histopathology | Microscopy | Complement component C3 | Remission | Kidney diseases | Age
Journal Article