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PLoS ONE, ISSN 1932-6203, 2010, Volume 5, Issue 8, pp. e12304 - e12304
Gliomas are morbid brain tumors that are extremely resistant to available chemotherapy and radiology treatments. Some studies have suggested that... 
BREAST-CANCER CELLS | HUMAN PROSTATE-CANCER | APOPTOSIS | IN-VITRO | PROTEIN-KINASE | GROWTH | BIOLOGY | CA2+-ACTIVATED K+ CHANNEL | INTERMEDIATE-CONDUCTANCE | BRAIN-TUMORS | CYCLE | Potassium - metabolism | RNA, Small Interfering - genetics | Large-Conductance Calcium-Activated Potassium Channels - metabolism | Small-Conductance Calcium-Activated Potassium Channels - genetics | Intermediate-Conductance Calcium-Activated Potassium Channels - genetics | Humans | Large-Conductance Calcium-Activated Potassium Channels - deficiency | Molecular Sequence Data | Electric Conductivity | Large-Conductance Calcium-Activated Potassium Channels - antagonists & inhibitors | Intermediate-Conductance Calcium-Activated Potassium Channels - metabolism | RNA, Messenger - metabolism | Gene Knockdown Techniques | Glioblastoma - genetics | Intermediate-Conductance Calcium-Activated Potassium Channels - antagonists & inhibitors | Large-Conductance Calcium-Activated Potassium Channels - genetics | Gene Expression Regulation, Neoplastic - drug effects | Potassium Channel Blockers - pharmacology | Amino Acid Sequence | RNA, Messenger - genetics | Down-Regulation - genetics | Glioblastoma - surgery | Glioblastoma - pathology | Intermediate-Conductance Calcium-Activated Potassium Channels - deficiency | Cell Line, Tumor | Cell Proliferation - drug effects | Chemotherapy | Gliomas | Genes | Brain tumors | Genetic engineering | Potassium channels | Cancer | Cell proliferation | Brain | Biotechnology | Neurosciences | Calcium | Brain cancer | Glioblastoma | Smooth muscle | Potassium conductance | Kinases | Cancer therapies | Channels | Potassium channels (calcium-gated) | Proteins | Cell growth | Surgery | Glioma cells | Cell cycle | Physiology | Calcium channels | Channel gating | Tumor cells | Cultures | siRNA | Pharmacology | Glioblastoma multiforme | Resistance | Studies | Polymerase chain reaction | Gene silencing | Inhibitors | Cell lines | Radiology | Conductance | Glioblastoma cells | Potassium | Prostate cancer | Clotrimazole | Tumors | Index Medicus
Journal Article
Physica C: Superconductivity, ISSN 0921-4534, 11/2013, Volume 494, pp. 89 - 94
Superconducting layered compound beta -ZrNCl(x) (x-0.7) with the critical temperature T(c)= 13-14 K was investigated by means of scanning tunnelling... 
Conductance
Journal Article
Journal Article
Journal of Cellular Physiology, ISSN 0021-9541, 12/2017, Volume 232, Issue 12, pp. 3409 - 3421
Compound #326‐stimulated activity of BK Ca channels is explained by a shortening of mean closed time, despite its inability to alter single‐channel... 
channel | current | large‐conductance Ca | inflammation | thiourea derivative | activated K | large-conductance Ca | POTENT ACTIVATION | PHYSIOLOGY | Na+ current | CA2+-ACTIVATED K+ CHANNELS | KB-R7943 | CURRENTS | GH CELLS | large-conductance Ca2+-activated K plus channel | CELL BIOLOGY | INHIBITION | PITUITARY-TUMOR CELLS | Ca2+-activated K+ current | Microglia - metabolism | Calcium - metabolism | Humans | Interleukin-1beta - genetics | Thiourea - pharmacology | Dose-Response Relationship, Drug | Transfection | Interleukin-1beta - metabolism | HEK293 Cells | Large-Conductance Calcium-Activated Potassium Channel alpha Subunits - agonists | Microglia - drug effects | Anti-Inflammatory Agents - pharmacology | Rats | Large-Conductance Calcium-Activated Potassium Channel alpha Subunits - metabolism | Pituitary Neoplasms - metabolism | Animals | Large-Conductance Calcium-Activated Potassium Channel alpha Subunits - genetics | Membrane Potentials | Nitric Oxide Synthase Type II - genetics | Calcium Signaling - drug effects | Lipopolysaccharides - pharmacology | Cell Line, Tumor | Calcium Channel Agonists - pharmacology | Mice, Inbred BALB C | Kinetics | Thiourea - analogs & derivatives | Ion Channel Gating - drug effects | Nitric Oxide Synthase Type II - metabolism | Nitric oxide | Potassium channels | Electric potential | Calcium | Interleukin | Potassium conductance | Immunity | Potassium channels (calcium-gated) | Interleukin 1 | Sodium channels (voltage-gated) | Thiourea | Ion channels | Potassium channels (voltage-gated) | Gating | Calcium channels | Channel gating | Tolbutamide | Inflammation | Free energy | Nitric-oxide synthase | Microglia | Resistance | Calcium conductance | Pituitary | Conductance | Potassium currents | Potassium | Index Medicus
Journal Article
Science, ISSN 0036-8075, 05/2018, Volume 360, Issue 6388, pp. 508 - 513
Journal Article
New Phytologist, ISSN 0028-646X, 1/2013, Volume 197, Issue 1, pp. 65 - 72
The classical view that the drought-related hormone ABA simply acts locally at the guard cell level to induce stomatal closure is questioned by differences... 
Transpiration | Plant physiology | Hydraulics | Aquaporins | Stomatal conductance | Guard cells | Stomata | Plants | Bundle sheath cells | Plant cells | Rapid reports | ABA | Arabidopsis thaliana | isohydric/anisohydric | leaf hydraulic conductance | stomatal conductance | transpiration | PIP | aquaporin | leaf | Isohydric/anisohydric | Stomatal conductance (g | Leaf hydraulic conductance (K | Aquaporin (PIP) | aquaporin (PIP) | isohydric | anisohydric | ARABIDOPSIS-THALIANA | PLANT WATER STATUS | DROUGHT STRESS | CELL-PRESSURE | PLANT SCIENCES | SIGNAL-TRANSDUCTION | RESPONSES | leaf hydraulic conductance (Kleaf) | HYDRAULIC CONDUCTIVITY | stomatal conductance (gs) | PRESSURE PROBE | LEAF EXPANSION | MAIZE ROOTS | Plant Transpiration - drug effects | Arabidopsis Proteins - genetics | Arabidopsis - drug effects | Plant Cells - metabolism | Phosphoprotein Phosphatases - metabolism | Water - metabolism | Plant Epidermis - drug effects | Plant Stomata - metabolism | Plant Epidermis - metabolism | Arabidopsis - metabolism | Arabidopsis - genetics | Arabidopsis Proteins - metabolism | Abscisic Acid - pharmacology | Argon - metabolism | Phosphoprotein Phosphatases - genetics | Biological Transport | Plant Leaves - metabolism | Droughts | Plant Stomata - drug effects | Plant Leaves - drug effects | Mutation | Abscisic acid | Permeability | Index Medicus
Journal Article
Journal of Cellular Physiology, ISSN 0021-9541, 09/2017, Volume 232, Issue 9, pp. 2478 - 2488
We investigated the expression of BK channels in GBM stem‐like cells using electrophysiological and immunochemical techniques, and assessed their involvement... 
INVASION | PHYSIOLOGY | BK CHANNELS | MITOCHONDRIAL | GLIOMA | MOUSE | CA2+-ACTIVATED K+ CHANNEL | BETA-SUBUNIT | EXPRESSION | MODULATION | CD133 | CELL BIOLOGY | Up-Regulation | Large-Conductance Calcium-Activated Potassium Channels - metabolism | Neoplastic Stem Cells - drug effects | Humans | Brain Neoplasms - pathology | Gene Expression Regulation, Neoplastic | Large-Conductance Calcium-Activated Potassium Channels - antagonists & inhibitors | Brain Neoplasms - metabolism | Glioblastoma - genetics | Neoplastic Stem Cells - metabolism | Time Factors | Biomarkers, Tumor - metabolism | Large-Conductance Calcium-Activated Potassium Channels - genetics | Neoplastic Stem Cells - pathology | Glioblastoma - metabolism | Tumor Cells, Cultured | Spheroids, Cellular | Potassium Channel Blockers - pharmacology | Signal Transduction | Neoplasm Invasiveness | Brain Neoplasms - genetics | Large-Conductance Calcium-Activated Potassium Channel alpha Subunits - metabolism | Cell Movement - drug effects | Phenotype | Membrane Potentials | AC133 Antigen - metabolism | Glioblastoma - pathology | Cell Line, Tumor | Primary Cell Culture | Potassium channels | Analysis | Brain tumors | Stem cells | Growth conditions | Brain | Cell culture | Calcium channels | Calcium | Leukocyte migration | Invasiveness | Brain cancer | Glioblastoma | Parenchyma | Potassium conductance | Channels | Cell adhesion & migration | Potassium channels (calcium-gated) | Resistance | Calcium conductance | Conductance | Potassium | Cell migration | Cancer | Tumors | Index Medicus
Journal Article
American Journal of Physiology - Heart and Circulatory Physiology, ISSN 0363-6135, 05/2016, Volume 310, Issue 9, pp. H1151 - H1163
Activation of vascular endothelial small-(K(Ca)2.3, SK3) or intermediate-(K(Ca)3.1, IK1) conductance Ca2+-activated potassium channels induces vasorelaxation... 
Small conductance CaCa | Calcium microdomain | Intermediateconductance CaCa | Transient receptor potential vanilloid 4 | Endothelium | activated potassium channel | endothelium | K(CA)3.1 CHANNELS | CARDIAC & CARDIOVASCULAR SYSTEMS | PHYSIOLOGY | SKCA CHANNELS | CA2+-ACTIVATED K+ CHANNELS | SIGNALS | transient receptor potential vanilloid 4 | calcium microdomain | HYPERPOLARIZING FACTOR | small conductance Ca2+-activated potassium channel | FUNCTIONAL EXPRESSION | TRPV4 CHANNELS | PERIPHERAL VASCULAR DISEASE | ARTERY | intermediate-conductance Ca2+-activated potassium channel | EDHF | SMALL-CONDUCTANCE | Blood Pressure | Genetic Predisposition to Disease | Small-Conductance Calcium-Activated Potassium Channels - genetics | Calcium - metabolism | Endothelial Cells - metabolism | Membrane Microdomains - metabolism | Mice, Inbred C57BL | Small-Conductance Calcium-Activated Potassium Channels - deficiency | Intermediate-Conductance Calcium-Activated Potassium Channels - metabolism | Mesenteric Arteries - metabolism | Hypertension - physiopathology | Small-Conductance Calcium-Activated Potassium Channels - metabolism | Vasodilation | Hypertension - metabolism | Mice, Knockout | TRPV Cation Channels - metabolism | Phenotype | Animals | Membrane Potentials | Time Factors | Activity Cycles | Hypertension - genetics | Calcium Signaling | Index Medicus | Vascular Biology and Microcirculation
Journal Article
Journal Article
American Journal of Physiology - Heart and Circulatory Physiology, ISSN 0363-6135, 10/2014, Volume 307, Issue 8, pp. H1093 - H1102
Impaired endothelial function, which is dysregulated in diabetes, also precedes hypertension. We hypothesized that in Type 2 diabetes, the impaired... 
Mesenteric artery | Calcium-activated potassium channel | Nitric oxide | Diabetes | Endothelium-derived hyperpolarization | Vasodilation | Endothelium | endothelium | nitric oxide | CARDIAC & CARDIOVASCULAR SYSTEMS | PHYSIOLOGY | ENDOTHELIAL DYSFUNCTION | vasodilation | CA2+-ACTIVATED K+ CHANNELS | ACTIVATED POTASSIUM CHANNELS | endothelium-derived hyperpolarization | mesenteric artery | HEME OXYGENASE | EDHF-TYPE RELAXATION | HYPERPOLARIZING FACTOR | WORKING GROUP-REPORT | NITRIC-OXIDE | PERIPHERAL VASCULAR DISEASE | MYOENDOTHELIAL GAP-JUNCTIONS | calcium-activated potassium channel | diabetes | EPOXYEICOSATRIENOIC ACIDS | Large-Conductance Calcium-Activated Potassium Channels - metabolism | Mesenteric Arteries - physiology | Small-Conductance Calcium-Activated Potassium Channels - genetics | Cyclooxygenase Inhibitors | Intermediate-Conductance Calcium-Activated Potassium Channels - genetics | Diabetes Mellitus, Type 2 - genetics | Endothelium, Vascular - drug effects | Male | Diabetes Mellitus, Type 2 - metabolism | Apamin - pharmacology | Intermediate-Conductance Calcium-Activated Potassium Channels - metabolism | RNA, Messenger - metabolism | Endothelium, Vascular - physiology | Charybdotoxin - pharmacology | Intermediate-Conductance Calcium-Activated Potassium Channels - antagonists & inhibitors | Large-Conductance Calcium-Activated Potassium Channels - genetics | Potassium Channel Blockers - pharmacology | Pyrazoles - pharmacology | Acetylcholine - pharmacology | RNA, Messenger - genetics | Large-Conductance Calcium-Activated Potassium Channels - agonists | Rats | Mesenteric Arteries - metabolism | Small-Conductance Calcium-Activated Potassium Channels - metabolism | Nitric Oxide Synthase Type III - antagonists & inhibitors | Small-Conductance Calcium-Activated Potassium Channels - antagonists & inhibitors | Rats, Zucker | Homozygote | Animals | Diabetes Mellitus, Type 2 - physiopathology | Membrane Potentials | Endothelium, Vascular - metabolism | Benzimidazoles - pharmacology | Calcium Channel Agonists - pharmacology | Heterozygote | Index Medicus
Journal Article
The New England Journal of Medicine, ISSN 0028-4793, 11/2017, Volume 377, Issue 21, pp. 2024 - 2035
Approximately 5% of patients with cystic fibrosis express one allele with some retained CFTR function. In a prospective trial, tezacaftor–ivacaftor had a... 
POPULATION | REGULATOR | MEDICINE, GENERAL & INTERNAL | LUMACAFTOR | EFFICACY | SAFETY | PHENOTYPE | AGED 6-11 YEARS | RANDOMIZED CONTROLLED-TRIAL | F508DEL-CFTR | G551D-CFTR MUTATION | Forced Expiratory Volume - drug effects | Cystic Fibrosis - physiopathology | Humans | Male | Aminophenols - therapeutic use | Quinolones - pharmacology | Aminophenols - adverse effects | Aminophenols - pharmacology | Young Adult | Quinolones - adverse effects | Quinolones - therapeutic use | Cystic Fibrosis Transmembrane Conductance Regulator - pharmacology | Adult | Benzodioxoles - adverse effects | Female | Indoles - pharmacology | Benzodioxoles - therapeutic use | Child | Double-Blind Method | Cross-Over Studies | Cystic Fibrosis Transmembrane Conductance Regulator - therapeutic use | Indoles - adverse effects | Cystic Fibrosis - genetics | Cystic Fibrosis Transmembrane Conductance Regulator - adverse effects | Adolescent | Quality of Life | Cystic Fibrosis Transmembrane Conductance Regulator - genetics | Heterozygote | Indoles - therapeutic use | Mutation | Benzodioxoles - pharmacology | Cystic Fibrosis - drug therapy | Drug Combinations | Proteins | Clonal deletion | Clinical trials | Heterozygotes | Cystic fibrosis | Cystic fibrosis transmembrane conductance regulator | Gene deletion | Drug therapy | Patients | Pharmaceuticals | Index Medicus | Abridged Index Medicus
Journal Article