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Journal Article
Journal of Neuroimmunology, ISSN 0165-5728, 2010, Volume 229, Issue 1, pp. 157 - 168
Abstract Massive programmed cell death (PCD) of developing chick embryo motoneurons (MNs) occurs in a well defined temporal and spatial sequence between... 
Neurology | Allergy and Immunology | Immunoglobulins | Chick embryo | Amyotrophic lateral sclerosis | Programmed cell death | Motoneuron | Cerebrospinal fluid | Semaphorin | ANTIBODIES | SPINAL-CORD CULTURES | CA2+ CHANNELS | ALS-IGG | SERUM | AMYOTROPHIC-LATERAL-SCLEROSIS | MOTOR-NEURON DISEASE | IMMUNOLOGY | NEUROSCIENCES | CALCIUM-CHANNELS | CEREBROSPINAL-FLUID PROTEIN | MOUSE MODEL | Enzyme-Linked Immunosorbent Assay - methods | Immunoglobulins - immunology | Apoptosis - drug effects | Neuromuscular Junction - drug effects | Humans | Motor Neuron Disease - blood | Motor Neuron Disease - immunology | Cercopithecus aethiops | Male | Muscle, Skeletal - innervation | Ganglia, Spinal - cytology | Statistics as Topic | Dose-Response Relationship, Drug | Serum - immunology | Tubulin - metabolism | Transfection - methods | Motor Neurons - cytology | Statistics, Nonparametric | Female | Muscle, Skeletal - embryology | Proteomics - methods | Motor Neurons - drug effects | Cell Survival - drug effects | Immunoglobulins - pharmacology | Cells, Cultured | Electrophoresis, Gel, Two-Dimensional - methods | Spectrometry, Mass, Matrix-Assisted Laser Desorption-Ionization - methods | Semaphorins - metabolism | Chick Embryo | Animals | Analysis of Variance | Growth Cones - drug effects | In Vitro Techniques | Neuromuscular Junction - physiology | Serum - chemistry | Index Medicus
Journal Article
Neuroscience Letters, ISSN 0304-3940, 2006, Volume 401, Issue 1, pp. 119 - 124
In our previous double-labeling studies, the fluorescent anatomical tracers Fluorogold (FG) and Texas red dextran amine (TRDA) were used to demonstrate that... 
Cell death | Retrograde labeling | Fluorogold | LONG-TERM | cell death | AXONAL REGENERATION | fluorogold | TOXICITY | retrograde labeling | FLUORESCENT TRACERS | NEUROSCIENCES | RETROGRADE | FAST-BLUE | SPINAL-CORD-INJURY | PROJECTIONS | FACIAL NUCLEUS | MOTONEURONS | Staining and Labeling - methods | Reticular Formation - drug effects | Cell Count | Nerve Regeneration - physiology | Brain Stem - metabolism | Nerve Degeneration - physiopathology | Fluorescent Dyes - metabolism | Brain Stem - cytology | Brain Stem - drug effects | Nerve Degeneration - chemically induced | Efferent Pathways - drug effects | Lampreys - metabolism | Reticular Formation - metabolism | Time Factors | Cell Death - drug effects | Lampreys - anatomy & histology | Larva - anatomy & histology | Stilbamidines - metabolism | Stilbamidines - toxicity | Horseradish Peroxidase - toxicity | Larva - metabolism | Neurotoxins - toxicity | Lampreys - growth & development | Efferent Pathways - cytology | Efferent Pathways - metabolism | Animals | Growth Cones - metabolism | Reticular Formation - cytology | Cell Death - physiology | Fluorescent Dyes - toxicity | Horseradish Peroxidase - metabolism | Neurotoxins - metabolism | Spinal Cord Injuries - physiopathology | Growth Cones - drug effects | Dextran | Cell research | Neurosciences | Tracers (Biology) | Neurons | Biochemistry | Index Medicus
Journal Article
INVESTIGATIVE OPHTHALMOLOGY & VISUAL SCIENCE, ISSN 0146-0404, 07/2019, Volume 60, Issue 8, pp. 3221 - 3235
PURPOSE. Retinitis pigmentosa (RP) refers to a group of inherited blinding retinal diseases, whereby the death of mutated rod photoreceptors is followed... 
OXIDATIVE STRESS | ACTIVATED PROTEIN-KINASE | rd10 | progesterone | RECEPTOR | PROLIFERATION | CELL-DEATH | DEGENERATION | BREAST-CANCER | cone photoreceptors | PATHWAY | neuroprotection | retinitis pigmentosa | OPHTHALMOLOGY | GROWTH-FACTOR | NEURAL REGENERATION | Index Medicus
Journal Article
JAMA Ophthalmology, ISSN 2168-6165, 2019, Volume 137, Issue 6, pp. 603 - 609
ImportanceTargeting the early pathogenic steps in Stargardt disease type 1 (STGD1) is critical to advance our understanding of this condition and to develop... 
LIPOFUSCIN FLUOROPHORE | CELLS | A2E | PHENOTYPE | OPHTHALMOLOGY | RETINA | CONE | DEGENERATION | Phenotypes | Scanning | Fluorescence | Optics | Retina | Rods | Acuity | Atrophy | Spatial distribution | Cones | Cell death | Photoreceptors | Ophthalmology | Index Medicus | Abridged Index Medicus
Journal Article
Investigative Ophthalmology and Visual Science, ISSN 0146-0404, 09/2016, Volume 57, Issue 11, pp. 4847 - 4858
PURPOSE. Previously, patients with RHO mutations and a class A phenotype were found to have severe early-onset loss of rod function, whereas patients with a... 
Cone | Perimetry | Optical coherence tomography | Rod | LIGHT-INDUCED DAMAGE | optical coherence tomography | rod | MODEL | cone | CELL-DEATH | RETINAL HISTOPATHOLOGY | HETEROGENEITY | DISEASE | TRANSVERSION MUTATION | OPHTHALMOLOGY | GENE-THERAPY | perimetry | PHOTORECEPTOR DEGENERATION | Retina
Journal Article
Molecular Vision, ISSN 1090-0535, 01/2017, Volume 23, p. 844
Purpose: Previous studies have shown that melatonin (MEL) signaling is involved in the modulation of photoreceptor viability during aging. Recent work by our... 
Melatonin | FasL protein | Hydrogen peroxide | Cones | Immunocytochemistry | Cell death | Aging | Caspase | Photoreceptors | mRNA | Caspase-3 | Apoptosis
Journal Article
Journal of Clinical Investigation, ISSN 0021-9738, 04/2015, Volume 125, Issue 4, pp. 1433 - 1445
Oxidative stress contributes to the loss of neurons in many disease conditions as well as during normal aging; however, small-molecule agents that reduce... 
RETINAL DEGENERATION | MEDICINE, RESEARCH & EXPERIMENTAL | DIFFUSIBLE FACTOR | OXIDATIVE STRESS | OPTIC-NERVE | PLACEBO-CONTROLLED PHASE-3 | MOUSE MODEL | AXON REGENERATION | PARTIAL RECOVERY | RETINITIS-PIGMENTOSA | CONE CELL-DEATH | NF-E2-Related Factor 2 - physiology | Dependovirus - genetics | Genetic Therapy | Catalase - therapeutic use | NF-E2-Related Factor 2 - therapeutic use | Oxidative Stress - physiology | Genetic Vectors - therapeutic use | Mice, Mutant Strains | Superoxide Dismutase - physiology | Peroxisome Proliferator-Activated Receptor Gamma Coactivator 1-alpha | Neurons - physiology | NF-E2-Related Factor 2 - genetics | Optic Nerve Injuries - therapy | Transcription Factors - therapeutic use | Retinal Ganglion Cells - physiology | Transcription Factors - physiology | Catalase - physiology | Mice, Inbred C57BL | Optic Nerve Injuries - physiopathology | Nerve Crush | Retinal Cone Photoreceptor Cells - physiology | Genetic Vectors - genetics | Superoxide Dismutase - therapeutic use | Animals | Retinitis Pigmentosa - therapy | Mice | Nerve Degeneration | Apoptosis | Oxidative stress | Transcription factors | Nervous system | Development and progression | Degeneration | Genetic aspects | Health aspects | Antioxidants | Proteins | Enzymes | Statistical analysis | Optic nerve | Disease | Genes | Lipids | Photoreceptors | Physiology | Index Medicus | Abridged Index Medicus | Genetics | Neuroscience | Ophthalmology | Therapeutics
Journal Article
Investigative Ophthalmology and Visual Science, ISSN 0146-0404, 09/2018, Volume 59, Issue 11, pp. 4362 - 4374
PURPOSE. Glutathione-S-transferase omega 1-1 (GSTO1-1) is a cytosolic glutathione transferase enzyme, involved in glutathionylation, toll-like receptor... 
Retinal degeneration | Knockout animals | Inflammation | Oxidative damage | GSTO1-1 | retinal degeneration | OXIDATIVE STRESS | ACTIVATION | PROTEIN | MACROPHAGES | oxidative damage | MACULAR DEGENERATION | ENDOPLASMIC-RETICULUM STRESS | S-TRANSFERASE OMEGA | CONE CELL-DEATH | MITOCHONDRIAL-DNA DAMAGE | knockout animals | inflammation | GLUTATHIONE | OPHTHALMOLOGY | Index Medicus
Journal Article
Journal Article