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Journal Article
Journal Article
The Journal of Physiology, ISSN 0022-3751, 06/2017, Volume 595, Issue 12, pp. 3987 - 3999
Key points Hypertrophic cardiomyopathy (HCM) is the most common inherited cardiac illness and can lead to diastolic dysfunction, sudden cardiac death and heart... 
cardiac myosin‐binding protein C | diltiazem | hypertrophic cardiomyopathy | hypertrophy | cardiac myosin-binding protein C | HEART | BINDING-PROTEIN-C | SARCOMERIC CARDIOMYOPATHIES | PHYSIOLOGY | FAMILIAL HYPERTROPHIC CARDIOMYOPATHY | MESSENGER-RNA | MYOFILAMENT CA2+ SENSITIVITY | MOUSE MODEL | DISEASE | KNOCK-IN MICE | DYSFUNCTION | NEUROSCIENCES | Calcium - metabolism | Sarcomeres - drug effects | Sarcomeres - metabolism | Cardiomyopathy, Hypertrophic - metabolism | Myocardial Contraction - drug effects | Mutation - genetics | Diltiazem - pharmacology | Phenotype | Animals | Carrier Proteins - metabolism | Myocytes, Cardiac - drug effects | Isoproterenol - pharmacology | Myocardium - metabolism | Myocytes, Cardiac - metabolism | Diastole - drug effects | Heart Ventricles - metabolism | Mice | Heart Ventricles - drug effects | Heart | Cardiomyopathy | Heart cells | Protein C | Muscle proteins | Diltiazem | Prevention | Analysis | Myosin | Stress management | Genetic aspects | Heart diseases | Protein binding | Calcium antagonists | Arrhythmia | Activation | Antagonists | Contraction | Guidelines | Eutrophication | Genotype & phenotype | Stress relaxation | Rodents | Attenuation | Pretreatment | Dihydropyridine | Heart failure | Effectiveness | Fetuses | Contractility | Cardiomyocytes | Pharmacology | Patients | Muscle contraction | Fibrosis | Death | Adrenergic receptors | Mutation | Ventricle | Hypertrophy | Regulation of Contractile Function in Health and Disease | Symposium Related Research Paper
Journal Article
Circulation Research, ISSN 0009-7330, 10/2016, Volume 119, Issue 9, pp. 992 - 995
Hypertrophic cardiomyopathy (HCM) is an inherited cardiac disease with an incidence of about 1 in 500 individuals.(1) It is characterized by asymmetrical... 
cardiomyocytes | heart failure | hypertrophic | contractile protein | cardiomyopathy | myosin heavy chains | allelic imbalance | FIBROSIS | CARDIAC & CARDIOVASCULAR SYSTEMS | MYOSIN | DILATED CARDIOMYOPATHY | BETA | PERIPHERAL VASCULAR DISEASE | MUTATIONS | HEMATOLOGY | TROPONIN
Journal Article
American Journal of Gastroenterology, ISSN 0002-9270, 01/2012, Volume 107, Issue 1, pp. 37 - 45
OBJECTIVES: This study aimed to refine the criteria for esophageal hypercontractility in high-resolution esophageal pressure topography (EPT) and to examine... 
DYSPHAGIA | WAVES | ASYNCHRONY | MOTILITY ABNORMALITIES | LONGITUDINAL MUSCLE-CONTRACTION | CHEST PAIN | CLASSIFICATION | MANOMETRY | NUTCRACKER ESOPHAGUS | GASTROENTEROLOGY & HEPATOLOGY | PERISTALSIS | Manometry | Humans | Middle Aged | Male | Peristalsis | Young Adult | Esophagus - physiopathology | Phenotype | Muscle Contraction | Adult | Female | Surveys and Questionnaires | Retrospective Studies
Journal Article
The Journal of Physiology, ISSN 0022-3751, 03/2011, Volume 589, Issue 6, pp. 1443 - 1454
Non‐technical summary  Muscle fibres change when they are used differently, such as by exercise. Genetic studies have shown that a hyperactive form of the gene... 
OBSTRUCTIVE PULMONARY-DISEASE | INTERMITTENT HYPOXIA | TRANSCRIPTIONAL ACTIVITY | PHYSIOLOGY | CONTRACTILE PROPERTIES | VASTUS LATERALIS MUSCLE | FIBER-TYPE | HIF-1-ALPHA EXPRESSION | NEUROSCIENCES | MYOSIN HEAVY-CHAIN | HUMAN SKELETAL-MUSCLE | ENDOTHELIAL GROWTH-FACTOR | Skeletal Muscle and Exercise
Journal Article
Journal Article
Journal of the American College of Cardiology, ISSN 0735-1097, 05/2019, Volume 73, Issue 18, pp. 2267 - 2282
Journal Article
Journal Article
Current Opinion in Hematology, ISSN 1065-6251, 05/2012, Volume 19, Issue 3, pp. 224 - 231
Journal Article
Atherosclerosis, ISSN 0021-9150, 01/2019, Volume 280, pp. 183 - 191
Atherosclerosis is a widespread and complicated disease involving phenotypic modulation and transdifferentiation of vascular smooth muscle cells (SMCs), the... 
Contractile phenotype | Chromatin | Enhancers | Atherosclerosis | DNA methylation | Super-enhancer | Smooth muscle cells | CARDIAC & CARDIOVASCULAR SYSTEMS | EPIGENETIC CONTROL | TET2 | IDENTITY | SUPER-ENHANCERS | INTEGRATIVE ANALYSIS | PERIPHERAL VASCULAR DISEASE | SMOOTH-MUSCLE-CELLS | CONTRIBUTES | EXPRESSION
Journal Article