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Journal Article
The Journal of cell biology, ISSN 1540-8140, 2003, Volume 161, Issue 3, pp. 535 - 545
.... They also participate in cytokinesis and cell cycle progression in ways that are poorly understood... 
COS cells | Delta cells | Centrosomes | Cell cycle | Cell lines | HeLa cells | Centrioles | Cytokinesis | Cells | Daughter cells | MEN/SIN | Cell cycle progression | Maternal centriole | Centrosome | cytokinesis | SPINDLE POLE BODY | maternal centriole | centrosome | CELL-CYCLE PROGRESSION | MAMMALIAN-CELLS | MICROTUBULE ORGANIZATION | CELL BIOLOGY | ANIMAL-CELLS | EPITHELIAL-CELLS | VERTEBRATE CELLS | MITOTIC EXIT | cell cycle progression | GAMMA-TUBULIN | CENTROSOME STRUCTURE | Immunohistochemistry | Protein Binding - genetics | Cytoskeletal Proteins - genetics | Humans | Embryo, Nonmammalian - metabolism | Molecular Sequence Data | Eukaryotic Cells - metabolism | Amino Acid Sequence - genetics | Embryo, Nonmammalian - drug effects | Deoxyribonucleases - metabolism | Microtubules - metabolism | Eukaryotic Cells - ultrastructure | Cell Cycle Proteins - genetics | Cytoskeletal Proteins - metabolism | Centrioles - ultrastructure | Cell Division - genetics | DNA, Complementary - analysis | Base Sequence - genetics | Cell Cycle Proteins - isolation & purification | Cell Cycle Proteins - metabolism | Protein Structure, Tertiary - genetics | Saccharomyces cerevisiae Proteins - genetics | Microscopy, Electron | S Phase - genetics | Antibodies - pharmacology | Deoxyribonucleases - genetics | Animals | Microtubules - genetics | tRNA Methyltransferases | Saccharomyces cerevisiae Proteins - metabolism | RNA, Small Interfering | Centrioles - metabolism | HeLa Cells | COS Cells | Proteins | Research | Cytology | SIN | centrosome; maternal centriole; cytokinesis; cell cycle progression; MEN
Journal Article
Journal Article
Nature Neuroscience, ISSN 1097-6256, 10/2002, Volume 5, Issue 10, pp. 939 - 945
During development, Reelin acts on migrating neuronal precursors and controls correct cell positioning in the cortex and other brain structures by a hitherto unidentified mechanism... 
NEURONAL MIGRATION | NERVOUS-SYSTEM | FOREBRAIN | RADIAL GLIAL-CELLS | OLFACTORY-BULB | MESSENGER-RNA EXPRESSION | SUBVENTRICULAR ZONE | CAJAL-RETZIUS CELLS | NEUROSCIENCES | VLDL RECEPTOR | ADULT-MOUSE | Lateral Ventricles - transplantation | Animals, Newborn - genetics | Extracellular Matrix Proteins - biosynthesis | Coculture Techniques | Neurons - cytology | Cell Adhesion Molecules, Neuronal - physiology | Cell Movement - physiology | Mice, Neurologic Mutants | Cell Differentiation - genetics | Animals, Newborn - physiology | Neurons - metabolism | Prosencephalon - metabolism | Serine Endopeptidases | Cell Differentiation - physiology | Gene Expression Regulation, Developmental - physiology | Extracellular Matrix Proteins - genetics | Extracellular Matrix Proteins - physiology | Mice, Transgenic | Nerve Tissue Proteins | Cell Adhesion Molecules, Neuronal - genetics | Animals | Prosencephalon - cytology | Cell Adhesion Molecules, Neuronal - biosynthesis | Signal Transduction - physiology | Mice | Mice, Inbred BALB C | Brain Tissue Transplantation - methods | COS Cells | Neurons | Physiological aspects | Glycoproteins | Research | Properties | Cell migration | Protein binding | Animals, Newborn | Signal Transduction | Brain Tissue Transplantation | Neurons and Cognition | Lateral Ventricles | Prosencephalon | Life Sciences | Cell Adhesion Molecules, Neuronal | Extracellular Matrix Proteins | Gene Expression Regulation, Developmental | Cell Differentiation | Cell Movement
Journal Article
The Journal of cell biology, ISSN 0021-9525, 2/2002, Volume 156, Issue 3, pp. 531 - 542
.... To explore the mechanisms underlying the induction of apoptosis after cytokine withdrawal or FKHR-L1 activation, we used a cell line in which FKHR-L1 activity could be specifically induced... 
T lymphocytes | COS cells | Transcription factors | Cytokines | Cell lines | Cell cycle | Stem cells | Cytochromes | Cells | Apoptosis | Forkhead | Mitochondria | PI3K | Cytokine | cytokine | FORKHEAD TRANSCRIPTION FACTOR | ACTIVATION | INDUCED APOPTOSIS | mitochondria | PHOSPHATIDYLINOSITOL 3-KINASE | apoptosis | forkhead | AKT | PROTEASOME-DEPENDENT DEGRADATION | CELL BIOLOGY | CYTOCHROME-C | INHIBITOR P27(KIP1) | SIGNALING PATHWAY | BCL-2 FAMILY | Poly(ADP-ribose) Polymerases | Protein-Serine-Threonine Kinases | Male | Phosphatidylinositol 3-Kinases - metabolism | Phosphatidylinositol 3-Kinases - antagonists & inhibitors | DNA-Binding Proteins - metabolism | Caspases - metabolism | Bcl-2-Like Protein 11 | Female | Cell Death - drug effects | Cell Survival - physiology | Hematopoietic Stem Cells - drug effects | Proto-Oncogene Proteins - metabolism | Cell Survival - drug effects | Tumor Suppressor Proteins - metabolism | Cell Cycle Proteins - metabolism | Gene Expression Regulation - physiology | Hematopoietic Stem Cells - metabolism | Mitochondria - metabolism | Mitochondria - drug effects | Membrane Potentials - physiology | Cytochrome c Group - metabolism | Cyclin-Dependent Kinase Inhibitor p27 | Mice, Knockout | Gene Expression Regulation - drug effects | Membrane Proteins | Proto-Oncogene Proteins c-akt | Transcription Factors - metabolism | Annexin A5 - metabolism | Animals | Apoptosis Regulatory Proteins | Carrier Proteins - metabolism | Proteins - metabolism | Cell Death - physiology | Cell Cycle - physiology | Cytokines - deficiency | Forkhead Box Protein O1 | Mice | Poly (ADP-Ribose) Polymerase-1 | Cell Cycle - drug effects | Forkhead Transcription Factors | Cytokines - pharmacology | Research | Cell death | Health aspects | apoptosis; cytokine; mitochondria; PI3K; forkhead
Journal Article
PloS one, ISSN 1932-6203, 2019, Volume 14, Issue 5, p. e0216467
Journal Article
PloS one, ISSN 1932-6203, 2012, Volume 7, Issue 1, p. e29976
.... Results from a microarray screen and quantitative RT-PCR demonstrate that Wnt4 expression is regulated during muscle cell differentiation in vitro and muscle development in vivo, being highly... 
MOTOR-NEURONS | NEUROTRANSMITTER | POSTSYNAPTIC DIFFERENTIATION | AGRIN | ACETYLCHOLINESTERASE | BIOLOGY | GENE-EXPRESSION | MUSCLE | SYNAPSE FORMATION | RECEPTOR | KINASE MUSK | Phosphorylation | Wnt4 Protein - metabolism | Vertebrates - genetics | Neuromuscular Junction - metabolism | Humans | Vertebrates - embryology | Cercopithecus aethiops | Wnt4 Protein - deficiency | Muscles - pathology | Embryo, Mammalian - metabolism | Neuromuscular Junction - embryology | Wnt4 Protein - genetics | Gene Expression Regulation, Developmental | HEK293 Cells | Neuromuscular Junction - pathology | Biomarkers - metabolism | Muscle Cells - pathology | Muscles - ultrastructure | Receptors, Cholinergic - metabolism | Muscles - embryology | Muscle Cells - metabolism | Receptor Protein-Tyrosine Kinases - metabolism | Neuromuscular Junction - ultrastructure | Animals | Muscles - innervation | Protein Binding | Mice | Body Patterning - genetics | COS Cells | Cluster Analysis | Proteins | Cell differentiation | Analysis | Target recognition | Wnt protein | Synaptogenesis | Innervation | Differentiation (biology) | Kinases | Acetylcholinesterase | Signal transduction | Immunology | Rodents | Clusters | Localization | Rapsyn | Cues | Nerve endings | Motor neurons | Phenotypes | Myotubes | Muscles | Polymerase chain reaction | Vertebrates | Axons | Signaling | Microscopy | Neural networks | Acetylcholine receptors | Low density lipoprotein receptors | Neuromuscular junctions | Receptor density | Biological Markers | Embryo, Mammalian | Biochemistry, Molecular Biology | Receptor Protein-Tyrosine Kinases | Life Sciences | Neuromuscular Junction | Body Patterning | Muscle Cells | Molecular biology | Receptors, Cholinergic | Wnt4 Protein
Journal Article
Journal Article
PloS one, ISSN 1932-6203, 06/2014, Volume 9, Issue 6, p. e100013
Therapeutic use of immunoregulatory cells represents a promising approach for the treatment of uncontrolled immunity... 
SURVIVAL | RESPONSES | DENDRITIC CELLS | TOLERANCE | MULTIDISCIPLINARY SCIENCES | ACCUMULATE | DISEASE | REGULATORY T-CELLS | MONOCYTES | DIFFERENTIATION | PGE | Autoimmunity | Graft Rejection - therapy | Mice, Inbred C57BL | Cells, Cultured | Bone Marrow Cells - physiology | Cercopithecus aethiops | Male | Mice, Transgenic | Allografts | Animals | Adoptive Transfer - methods | Bone Marrow Transplantation | Autoimmune Diseases - therapy | Female | Myeloid Cells - transplantation | Mice | Graft Rejection - immunology | Autoimmune Diseases - pathology | COS Cells | Graft Rejection - pathology | Disease Models, Animal | Type 1 diabetes | Analysis | Transplantation of organs, tissues, etc | Inflammation | B cells | T cells | Health aspects | Cell proliferation | Cell culture | Flow cytometry | Animal models | Toxicity | CD8 antigen | Cytotoxicity | Transplantation | Lymphocytes T | Suppressor cells | Immunity | Adoptive transfer | Lipopolysaccharides | Interleukin 6 | Cell activation | Cell growth | Immunology | Lymphocytes | Bone marrow | Biocompatibility | Skin grafts | Pancreas | Spleen | Antigens | CD11b antigen | Cell survival | Cytokines | Immunoregulation | Granulocyte-macrophage colony-stimulating factor | Immunomodulation | Diabetes mellitus | T cell receptors | Injection | Survival | Graft rejection | Inflammatory bowel disease | Immunosuppression | Antigen-presenting cells | Diabetes | Prolongation
Journal Article