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Molecular and Cellular Proteomics, ISSN 1535-9476, 06/2012, Volume 11, Issue 6, p. M111.013946
Journal Article
Biochimica et biophysica acta. Molecular basis of disease, ISSN 0925-4439, 2014, Volume 1842, Issue 8, pp. 1240 - 1247
Journal Article
Journal Article
American Journal of Physiology - Cell Physiology, ISSN 0363-6143, 06/2015, Volume 308, Issue 11, pp. C919 - C931
Journal Article
Journal Article
Molecular and Cellular Biochemistry, ISSN 0300-8177, 12/2007, Volume 306, Issue 1, pp. 153 - 162
The creatine kinase (CK) system is essential for cellular energetics in tissues or cells with high and fluctuating energy requirements. Creatine itself is... 
Life Sciences | Biochemistry, general | Mitochondria | Creatine kinase | Oncology | Medical Biochemistry | siRNA | Cardiology | Creatine | TRANSPORTER | mitochondria | PERFORMANCE | ADAPTATIONS | NEUROPROTECTION | DEFICIENCY | PERMEABILITY TRANSITION | CELL BIOLOGY | SUPPLEMENTATION | METABOLISM | creatine | CONSEQUENCES | creatine kinase | COMPARTMENTATION | HeLa Cells - enzymology | Mitochondria - enzymology | Isoenzymes | Humans | RNA, Messenger - genetics | RNA, Small Interfering - pharmacology | Creatine Kinase, BB Form - antagonists & inhibitors | Creatine Kinase, Mitochondrial Form - antagonists & inhibitors | Creatine Kinase, Mitochondrial Form - genetics | Mitochondria - drug effects | RNA, Messenger - metabolism | HeLa Cells - drug effects | Creatine Kinase, BB Form - biosynthesis | Creatine Kinase, BB Form - genetics | Gene Expression Regulation - drug effects | Cytosol - enzymology | Keratinocytes - metabolism | Phosphocreatine - metabolism | Creatine Kinase, Mitochondrial Form - biosynthesis | Cell Survival - physiology | Proteins | Cellular biology | Kinases | Apoptosis | Cell Survival | Creatine Kinase, Mitochondrial Form | Biochemistry, Molecular Biology | Gene Expression Regulation | Keratinocytes | Phosphocreatine | Cytosol | Creatine Kinase, BB Form | RNA, Small Interfering | RNA, Messenger | Hela Cells
Journal Article
PloS one, ISSN 1932-6203, 2017, Volume 12, Issue 3, p. e0173798
Mild traumatic brain injury (TBI) accounts for the vast majority of the nearly two million brain injuries suffered in the United States each year. Mild TBI is... 
C-REACTIVE PROTEIN | DIAGNOSTIC-TEST | NEW-ORLEANS CRITERIA | ACUTE-PHASE | MULTIDISCIPLINARY SCIENCES | CREATINE-KINASE-BB | NECROSIS-FACTOR-ALPHA | EXTERNAL VALIDATION | COMPUTED-TOMOGRAPHY SCANS | MINOR HEAD TRAUMA | CEREBROSPINAL-FLUID | Neuroimaging | Predictive Value of Tests | Prospective Studies | Brain Concussion - pathology | Humans | Malondialdehyde - analogs & derivatives | Middle Aged | Male | Tomography, X-Ray Computed | Biomarkers - blood | Creatine Kinase, BB Form - blood | Matrix Metalloproteinase 2 - blood | C-Reactive Protein - metabolism | Glasgow Coma Scale | Malondialdehyde - blood | Fatty Acid-Binding Proteins - blood | Adult | Brain Concussion - blood | Brain Concussion - diagnostic imaging | Aged | Granulocyte-Macrophage Colony-Stimulating Factor - blood | Lipoproteins, LDL - blood | Brain | Usage | Biological markers | Injuries | Risk factors | Coma | C-reactive protein | Traumatic brain injury | Physicians | Cognitive ability | Cerebrospinal fluid | Macrophages | Neuronal-glial interactions | Head injuries | Computed tomography | Vomiting | Amnesia | Children | Adolescents | Age | Stroke | Immunoglobulins | Medical imaging | Consciousness | Decision making | Health risks | Inflammation | Trauma | Radiography | Algorithms | Hospitals | Biomarkers | Death | Ventricle | Monocyte chemoattractant protein 1
Journal Article
Brain Research, ISSN 0006-8993, 2009, Volume 1279, pp. 21 - 28
Abstract Drugs that bind to imidazoline binding proteins have major physiological actions. To date, three subtypes of such proteins, I1 , I2 and I3 , have been... 
Neurology | Harmane and psychiatric disorders | Imidazoline binding protein | 2-BFI | Creatine kinase | MONOAMINE-OXIDASE | ALZHEIMERS-DISEASE | RECEPTOR PROTEINS | SELECTIVE LIGAND | NEUROSCIENCES | ELECTRIC ORGAN | I-2-IMIDAZOLINE RECEPTORS | IN-VIVO | RAT-BRAIN | HUNTINGTONS-DISEASE | I-2 RECEPTORS | Rabbits | Rats, Wistar | Creatine Kinase, BB Form - antagonists & inhibitors | Brain - enzymology | Models, Molecular | Rats | Benzofurans - pharmacology | Male | Imidazoles - pharmacology | Enzyme Activation - drug effects | Imidazoline Receptors - antagonists & inhibitors | Creatine Kinase, BB Form - metabolism | Brain - metabolism | Dose-Response Relationship, Drug | Creatine Kinase, BB Form - chemistry | Animals | Imidazoline Receptors - metabolism | Chickens | Female | Imidazoline Receptors - chemistry | Cell Membrane - metabolism | Binding Sites | Boats and boating | Monoamine oxidase | Neurons | Analysis | Physiological aspects | Bookbinding | Quinoline | Chemical properties | Binding proteins | Mental illness | Protein binding | KATP channel, ATP sensitive potassium channel | MOE, molecular operating environment | BU224, 2-(4,5-dihydroimidaz-2-yl)quinoline | CK, creatine kinase | MAO, monoamine oxidase | 2-BFI, 2-(2-benzofuranyl)2-imidazoline | BU99006, 5-isothiocyanoato-2-benzofuranyl-2-imidazoline | GR, glucose-responsive | Research Report | GOLD, genetic optimisation for ligand docking | B-CK, brain creatine kinase | I2, imidazoline-2 subtype
Journal Article