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Journal of Nutritional Science and Vitaminology, ISSN 0301-4800, 2012, Volume 58, Issue 6, pp. 377 - 383
Journal Article
Neuroscience, ISSN 0306-4522, 02/2019, Volume 399, pp. 53 - 64
•Adamts18 mRNA is highly abundant in developing brains.•Adamts18 deficiency enhances dendritic branching complexity and spine density.•Adamts18 deficiency... 
laminin | dendritic branching | depression | ADAMTS18 | DISINTEGRIN | METALLOPROTEINASE | CRMP-2 | IDENTIFICATION | EXPRESSION | NEUROSCIENCES | MULTIPLE
Journal Article
Neurochemistry International, ISSN 0197-0186, 10/2018, Volume 119, pp. 207 - 217
CRMP2, alternatively designated as DPYSL2, was the first CRMP family member to be identified as an intracellular molecule mediating the signaling of the axon... 
Phosphorylation | Behavior | Knock-in | CRMP2 | Proteomics | BIOCHEMISTRY & MOLECULAR BIOLOGY | SCHIZOPHRENIA | CRMP-2 | MATURATION | NEUROSCIENCES | LACKING | CDK5 | NEURONS
Journal Article
Journal of Neuroscience, ISSN 0270-6474, 10/2004, Volume 24, Issue 41, pp. 8994 - 9004
Neurite outgrowth is influenced by positive and negative signals that include the semaphorins, an important family of axonal outgrowth inhibitors. Here we... 
RacGTPase | Cdk5/p35 | α2-chimaerin | Sema 3A | CRMP-2 | Growth cone collapse | Cellular | p35 | Cdk5 | growth cone collapse | Molecular
Journal Article
by Zhao, J and Qu, Y and Wu, J and Cao, M and Ferriero, DM and Zhang, L and Mu, D
NEUROSCIENCE, ISSN 0306-4522, 05/2013, Volume 238, pp. 242 - 251
Alterations in axon-dendrite polarity impair functional recovery in the developing CNS after hypoxia-ischemia (HI) injury. PTEN (phosphatase and tensin homolog... 
hypoxia-ischemia | POLARITY | PHOSPHORYLATION | PHOSPHATASE | INVOLVEMENT | GSK-3-BETA | neuron | signaling pathway | PTEN | MECHANISMS | CRMP-2 | ESTABLISHMENT | NEUROSCIENCES | PATHWAY | GSK-3 beta | BRAIN
Journal Article
by Fang, W and Gao, GD and Zhao, HK and Xia, Y and Guo, XD and Li, N and Li, YQ and Yang, Y and Chen, L and Wang, Q and Li, LH
BRAIN RESEARCH, ISSN 0006-8993, 03/2015, Volume 1602, pp. 9 - 19
Parkinson's disease (PD) is the most common neurodegenerative disease of the basal ganglia. Earlier reports suggest that the main pathological change in PD is... 
Parkinson's disease | POLARITY | Pathogenesis | MICROTUBULE | PHOSPHORYLATION | ALZHEIMERS-DISEASE | WALLERIAN DEGENERATION | Axon degeneration | MECHANISMS | DEATH | Akt | CRMP-2 | SUBSTANTIA-NIGRA | NEUROSCIENCES | TRANSPORT | GSK-3 beta | PARKINSONS-DISEASE
Journal Article
Cold Spring Harbor perspectives in biology, ISSN 1943-0264, 2009, Volume 1, Issue 3, pp. a001644 - a001644
Journal Article
by Wu, ZT and Wang, GH and Wei, YY and Xiao, L and Wang, HL
NEUROREPORT, ISSN 0959-4965, 10/2018, Volume 29, Issue 15, pp. 1256 - 1263
Journal Article
Journal of Neurosurgery, ISSN 0022-3085, 10/2017, Volume 127, Issue 4, pp. 716 - 724
OBJECTIVE Inflammation and apoptosis are two key factors contributing to secondary brain injury after intracerebral hemorrhage (ICH). The objective of this... 
Neurological deficit | Glycogen synthase kinase-3β | Lithium | Inflammation | Vascular disorders | Intracerebral hemorrhage | Apoptosis | SURGERY | ACTIVATION | glycogen synthase kinase-3 beta | PHOSPHORYLATION | apoptosis | DISORDERS | CRMP-2 | BETA-CATENIN | vascular disorders | CLINICAL NEUROLOGY | CELL-DEATH | intracerebral hemorrhage | lithium | RECOVERY | inflammation | neurological deficit | EXPRESSION
Journal Article
Epilepsy Research, ISSN 0920-1211, 09/2018, Volume 145, pp. 93 - 101
•Effect of three doses of lacosamide was studied on pilocarpine-induced SE in C57BL/6 mice.•Lacosamide exhibited inverted-U-response against... 
Oxidative stress | Pilocarpine | CRMP-2 | Status epilepticus | Lacosamide | Spatial memory | TEMPORAL-LOBE EPILEPSY | BEHAVIORAL ALTERATIONS | RATS | ANTICONVULSANT | MODEL | CLINICAL NEUROLOGY | SPONTANEOUS SEIZURES | INTRAVENOUS LACOSAMIDE | NEUROGENESIS | ANTIEPILEPTIC DRUGS | BRAIN | Mice | Seizures (Medicine) | Analysis
Journal Article
Journal of Biological Chemistry, ISSN 0021-9258, 10/2013, Volume 288, Issue 43, pp. 31229 - 31240
The Rho-associated protein kinases (ROCK I and II) are central regulators of important cellular processes such as migration and invasion downstream of the... 
SURVIVAL | TARGET | SIGNALING PATHWAYS | ACTIVATION | MYOSIN | BIOCHEMISTRY & MOLECULAR BIOLOGY | GLYCOGEN-SYNTHASE KINASE-3 | GSK-3 | CRMP-2 | CANCER | Colonic Neoplasms - genetics | Protein Binding - genetics | Humans | Neoplasm Proteins - metabolism | RNA, Messenger - metabolism | RNA, Neoplasm - metabolism | Colonic Neoplasms - metabolism | Breast Neoplasms - metabolism | Intercellular Signaling Peptides and Proteins - metabolism | Phosphorylation - genetics | RNA Splicing | rho-Associated Kinases - metabolism | Female | Carcinoma - pathology | Neoplasm Proteins - genetics | Protein Structure, Tertiary | Neoplasm Invasiveness | RNA, Messenger - genetics | Intercellular Signaling Peptides and Proteins - genetics | Rats | rho-Associated Kinases - genetics | Glycogen Synthase Kinase 3 - metabolism | Nerve Tissue Proteins - genetics | Nerve Tissue Proteins - metabolism | Animals | Breast Neoplasms - genetics | Glycogen Synthase Kinase 3 - genetics | Breast Neoplasms - pathology | Colonic Neoplasms - pathology | Cell Line, Tumor | RNA, Neoplasm - genetics | Carcinoma - genetics | Carcinoma - metabolism | Cell Movement | Cell Migration | threonine Kinase | Cell Invasion | Rho GTPases | CRMP-2 Phosphorylation | Carcinoma | Rho-associated Protein Kinase II | Glycogen Synthase Kinase 3 | Collapsin Response Mediator Protein-2 | Protein-serine | Cell Biology
Journal Article