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Circulation Research, ISSN 0009-7330, 02/2010, Volume 106, Issue 2, pp. 363 - 372
Journal Article
Circulation research, ISSN 1524-4571, 2012, Volume 111, Issue 5, pp. 543 - 552
Journal Article
Journal Article
Arteriosclerosis, thrombosis, and vascular biology, ISSN 1524-4636, 2016, Volume 36, Issue 8, pp. 1496 - 1506
...–producing smooth muscle cells. Next, we generated Il17cApoe mice and demonstrated that atherosclerotic lesion and collagen content was diminished within Western diet... 
atherosclerosis | smooth muscle cells | leukocytes | cytokines | inflammation | IL-17C | RECEPTOR | IMMUNE | HOST-DEFENSE | PERIPHERAL VASCULAR DISEASE | INFECTION | ATHEROSCLEROTIC PLAQUE | MANNER | HEMATOLOGY | E-DEFICIENT MICE | NF-KAPPA-B | Chemotaxis, Leukocyte | Inflammation - pathology | T-Lymphocytes, Regulatory - metabolism | Apolipoproteins E - deficiency | Muscle, Smooth, Vascular - metabolism | Atherosclerosis - genetics | Myocytes, Smooth Muscle - pathology | Male | Aorta - metabolism | Aortic Diseases - metabolism | Inflammation - metabolism | Th17 Cells - metabolism | Diet, High-Fat | Aortic Diseases - prevention & control | Female | Myocytes, Smooth Muscle - metabolism | Aortic Diseases - pathology | Disease Models, Animal | Fibroblasts - metabolism | Th17 Cells - pathology | Atherosclerosis - pathology | Genetic Predisposition to Disease | Collagen Type I - metabolism | Paracrine Communication | Signal Transduction | Mice, Inbred C57BL | Cells, Cultured | Interleukin-17 - genetics | Interleukin-17 - deficiency | Plaque, Atherosclerotic | Atherosclerosis - metabolism | Mice, Knockout | Aorta - pathology | Interleukin-17 - metabolism | Aortic Diseases - genetics | Muscle, Smooth, Vascular - pathology | Phenotype | Animals | Apolipoproteins E - genetics | Inflammation - genetics | Inflammation - prevention & control | Atherosclerosis - prevention & control
Journal Article
Circulation (New York, N.Y.), ISSN 1524-4539, 2009, Volume 119, Issue 10, pp. 1424 - 1432
Background - Atherosclerosis is an inflammatory disease in which interferon (IFN)-gamma, the signature cytokine of Th1 cells, plays a central role. We... 
Smooth | Inflammation | Muscle | Interleukins | Lymphocytes | Coronary disease | CYTOKINES | CARDIAC & CARDIOVASCULAR SYSTEMS | lymphocytes | muscle, smooth | AUTOIMMUNE INFLAMMATION | HELPER-CELLS | ATHEROSCLEROSIS | IL-23 | interleukins | coronary disease | GROWTH-FACTOR-BETA | inflammation | DISEASE | IL-17 | PERIPHERAL VASCULAR DISEASE | DIFFERENTIATION | HEMATOLOGY | EXPRESSION | Vasculitis - physiopathology | Humans | Middle Aged | Receptors, Interleukin-17 - immunology | Vasculitis - etiology | Interleukin-17 - physiology | Myocytes, Smooth Muscle - pathology | Male | Interferon-gamma - metabolism | Chemokine CXCL10 - biosynthesis | Coronary Artery Disease - pathology | Inflammation Mediators - metabolism | Interferon-gamma - antagonists & inhibitors | Adult | Female | Interleukin-8 - metabolism | Interleukins - pharmacology | Receptors, Interferon - antagonists & inhibitors | Interleukin-6 - metabolism | Myocytes, Smooth Muscle - metabolism | Transforming Growth Factor beta1 - pharmacology | Cells, Cultured - drug effects | Coronary Vessels - drug effects | Interleukin-8 - biosynthesis | CD4-Positive T-Lymphocytes - metabolism | Interferon-gamma - physiology | Receptors, Interleukin-17 - antagonists & inhibitors | Interleukin-17 - biosynthesis | Gene Expression Regulation - drug effects | Interleukin-17 - metabolism | Muscle, Smooth, Vascular - pathology | Receptors, Interferon - immunology | Signal Transduction - drug effects | T-Lymphocyte Subsets - metabolism | Coronary Artery Disease - complications | Interleukin-6 - biosynthesis | Aged | Cells, Cultured - metabolism | Coronary Artery Disease - immunology | Chemokine CXCL10 - metabolism | Interferon-gamma - biosynthesis | muscle | smooth
Journal Article
Arteriosclerosis, Thrombosis, and Vascular Biology, ISSN 1079-5642, 11/2016, Volume 36, Issue 11, pp. 2158 - 2162
OBJECTIVE—The purpose of this study was to determine the role of smooth muscle cell... 
aneurysm | hypoxia | angiotensin II | smooth muscle | vascular remodeling | elastin | extracellular matrix | WALL | ANGIOTENSIN-II | INACTIVATION | GENE | DISSECTION | LEADS | PERIPHERAL VASCULAR DISEASE | MICE | HIF-1-ALPHA | HEMATOLOGY | Aminopropionitrile | Aortic Aneurysm, Abdominal - chemically induced | Vascular Remodeling | Aortic Aneurysm, Abdominal - prevention & control | Muscle, Smooth, Vascular - metabolism | Aortic Aneurysm, Thoracic - prevention & control | Myocytes, Smooth Muscle - pathology | Male | Aortic Aneurysm, Thoracic - chemically induced | Aortic Aneurysm, Abdominal - metabolism | Hypoxia-Inducible Factor 1, alpha Subunit - deficiency | Elastic Tissue - metabolism | Aortic Aneurysm, Thoracic - pathology | Hypoxia-Inducible Factor 1, alpha Subunit - metabolism | Tropoelastin - metabolism | Aorta, Abdominal - pathology | Angiotensin II | Aorta, Abdominal - metabolism | Dilatation, Pathologic | Myocytes, Smooth Muscle - metabolism | Aorta, Thoracic - pathology | Aortic Aneurysm, Abdominal - pathology | Disease Models, Animal | Genetic Predisposition to Disease | Hypoxia-Inducible Factor 1, alpha Subunit - genetics | Cells, Cultured | Aorta, Thoracic - metabolism | Mice, Knockout | Elastic Tissue - pathology | Muscle, Smooth, Vascular - pathology | Phenotype | Animals | Tropoelastin - genetics | Aortic Aneurysm, Thoracic - metabolism | Protein-Lysine 6-Oxidase - metabolism
Journal Article
PloS one, ISSN 1932-6203, 2018, Volume 13, Issue 4, p. e0195116
Over expression of Tissue Inhibitor of Metalloproteinases-3 (TIMP-3) in vascular smooth muscle cells (VSMCs... 
MATRIX METALLOPROTEINASES | DEATH DOMAIN | OVEREXPRESSION | LEUKEMIC-CELLS | CD95 | INVASION | MECHANISM | REDISTRIBUTION | MULTIDISCIPLINARY SCIENCES | LIPID RAFTS | PROLIFERATION | Disintegrins - metabolism | Muscle, Smooth, Vascular - metabolism | Apoptosis - drug effects | Humans | Cells, Cultured | fas Receptor - antagonists & inhibitors | Membrane Potential, Mitochondrial - drug effects | Muscle, Smooth, Vascular - cytology | fas Receptor - metabolism | ADAM17 Protein - metabolism | Microscopy, Confocal | ADAM17 Protein - genetics | Caspases - metabolism | ADAM17 Protein - antagonists & inhibitors | RNA Interference | Tissue Inhibitor of Metalloproteinase-3 - metabolism | fas Receptor - genetics | Disintegrins - antagonists & inhibitors | CASP8 and FADD-Like Apoptosis Regulating Protein - metabolism | Staurosporine - pharmacology | RNA, Small Interfering - metabolism | Tissue Inhibitor of Metalloproteinase-3 - genetics | Medical research | Tumor necrosis factor | Physiological aspects | Medicine, Experimental | Research | Vascular smooth muscle | Health aspects | Apoptosis | Shedding | Immunoprecipitation | Smooth muscle | Lipids | Infections | Activation | Tissue inhibitor of metalloproteinase 3 | Caspase-3 | Cell surface | Proteins | Fas-associated protein | FADD protein | Depolarization | Signal transduction | Mitochondria | Rodents | FLIP protein | Interleukin 1 | Inhibition | Enzymes | ADAM protein | Cytokines | Mortality | Muscles | Caspase | c-FLIP protein | Clustering | Lymphoma | Caspase-8 | Signaling | Depletion | Overexpression | Inhibitors | Lymphocytes B | Adenoviruses | Caspase-9 | Death | Position (location) | Immunofluorescence | Cancer | B-cell lymphoma
Journal Article
Arteriosclerosis, Thrombosis, and Vascular Biology, ISSN 1079-5642, 11/2013, Volume 33, Issue 11, pp. 2625 - 2632
Journal Article
Circulation, ISSN 0009-7322, 02/2014, Volume 129, Issue 8, pp. 864 - 874
Journal Article
Journal of Cerebral Blood Flow & Metabolism, ISSN 0271-678X, 10/2013, Volume 33, Issue 10, pp. 1564 - 1573
Little is known about vascular smooth muscle cell (SMC) phenotypic modulation in the cerebral circulation or pathogenesis of intracranial aneurysms... 
Smooth muscle cell phenotypic modulation | Cerebral aneurysm | TNF-alpha | ATHEROSCLEROSIS | RATS | NECROSIS-FACTOR-ALPHA | NEUROSCIENCES | INTRACRANIAL ANEURYSMS | NEUROINFLAMMATION | INFLAMMATION | Cerebral vascular smooth muscle cells | IN-VIVO | DISEASE | ENDOCRINOLOGY & METABOLISM | GENE-EXPRESSION | DIFFERENTIATION | HEMATOLOGY | Carotid Arteries - drug effects | Muscle, Smooth, Vascular - metabolism | Apoptosis - drug effects | Epigenesis, Genetic | Transcriptome | Apoptosis - genetics | Thalidomide - pharmacology | Dose-Response Relationship, Drug | Cell Differentiation - genetics | Thalidomide - analogs & derivatives | Trans-Activators - genetics | Nuclear Proteins - genetics | Circle of Willis - metabolism | Disease Models, Animal | Muscle, Smooth, Vascular - drug effects | Promoter Regions, Genetic | Intracranial Aneurysm - genetics | Cells, Cultured | Kruppel-Like Transcription Factors - physiology | Rats | Genetic Markers - drug effects | Intracranial Aneurysm - immunology | Tumor Necrosis Factor-alpha - pharmacology | Muscle, Smooth, Vascular - pathology | Animals | Circle of Willis - pathology | Cell Differentiation - drug effects | Tumor Necrosis Factor-alpha - physiology | Carotid Arteries - pathology | Intracranial Aneurysm - pathology | Circle of Willis - drug effects | Kruppel-Like Transcription Factors - genetics | Tumor Necrosis Factor-alpha - antagonists & inhibitors | Original
Journal Article
Circulation research, ISSN 1524-4571, 2016, Volume 118, Issue 2, pp. 230 - 240
RATIONALE:Genetic variation at the chromosome 9p21 cardiovascular risk locus has been associated with peripheral artery disease, but its mechanism remains... 
atherosclerosis | smooth muscle cells | genetic variation | peripheral artery disease | cyclin-dependent kinase inhibitor p15 | pathologic angiogenesis | CARDIAC & CARDIOVASCULAR SYSTEMS | PROTEIN | MYOCARDIAL-INFARCTION | AORTIC-ANEURYSM | RISK INTERVAL | CHROMOSOME 9P21 VARIANTS | HIC-5 | GENE | PERIPHERAL ARTERY-DISEASE | TUMOR-SUPPRESSOR | PERIPHERAL VASCULAR DISEASE | HEMATOLOGY | EXPRESSION | Hindlimb | Atherosclerosis - genetics | Humans | Transforming Growth Factor beta1 - metabolism | Neovascularization, Pathologic | Male | Muscle, Smooth, Vascular - physiopathology | Cell Hypoxia | Atherosclerosis - enzymology | Transfection | RNA Interference | Time Factors | Cyclin-Dependent Kinase Inhibitor p15 - genetics | Carotid Arteries - enzymology | Female | Disease Models, Animal | Muscle, Skeletal - blood supply | Smad7 Protein - metabolism | Atherosclerosis - pathology | Coronary Vessels - pathology | Genetic Predisposition to Disease | Coronary Vessels - enzymology | Signal Transduction | Cyclin-Dependent Kinase Inhibitor p15 - deficiency | Myocytes, Smooth Muscle - enzymology | Mice, Inbred C57BL | Cells, Cultured | Atherosclerosis - mortality | Transforming Growth Factor beta1 - genetics | Chromosomes, Human, Pair 9 | Mice, Knockout | Phenotype | Animals | Carotid Arteries - pathology | Cyclin-Dependent Kinase Inhibitor p15 - metabolism | Muscle, Smooth, Vascular - enzymology | Neovascularization, Physiologic | cardiovascular disease CDKN2B | Pathophysiology | Risk Factors | angiogenesis | Atherosclerosis | TGFβ | Peripheral Vascular Disease | 9p21 | Gene Expression and Regulation
Journal Article
Acta pharmacologica Sinica, ISSN 1671-4083, 2013, Volume 34, Issue 7, pp. 901 - 911
Journal Article