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Annals of the Rheumatic Diseases, ISSN 0003-4967, 06/2018, Volume 77, p. 1681
Journal Article
PLoS ONE, ISSN 1932-6203, 06/2015, Volume 10, Issue 6, p. e0130517
Respiratory syncytial virus (RSV) is the principal cause of bronchiolitis in infants and a significant healthcare problem. The RSV Glycoprotein (G) mediates... 
MOLECULAR CHARACTERIZATION | VACCINE DEVELOPMENT | REPLICATION IN-VITRO | ATTACHMENT | EPITHELIAL-CELLS | CX3C-CX3CR1 BINDING | RSV | MULTIDISCIPLINARY SCIENCES | INFECTION | FUSION GLYCOPROTEIN | RECEPTOR CXCR1 | Respiratory Syncytial Virus, Human - metabolism | Sequence Deletion | Epithelial Cells - metabolism | Viral Fusion Proteins - metabolism | Viral Fusion Proteins - genetics | Humans | Molecular Sequence Data | Respiratory Mucosa - virology | Epitopes - immunology | Respiratory Mucosa - pathology | Respiratory Syncytial Virus, Human - genetics | Viral Fusion Proteins - chemistry | Base Sequence | Viral Envelope Proteins - metabolism | Cell Differentiation | Receptors, Chemokine - antagonists & inhibitors | Receptors, Chemokine - chemistry | Receptors, Chemokine - genetics | Binding Sites | Child | Gene Expression | Viral Envelope Proteins - genetics | Cilia - pathology | Receptors, Chemokine - metabolism | Epithelial Cells - pathology | Cilia - metabolism | Antibodies - pharmacology | Viral Envelope Proteins - antagonists & inhibitors | Epithelial Cells - virology | Viral Envelope Proteins - chemistry | Protein Binding | Epitopes - chemistry | Primary Cell Culture | Respiratory Mucosa - metabolism | CX3C Chemokine Receptor 1 | Cilia - virology | Infection | G proteins | Health aspects | Protein binding | Asthma | Health care | Cell culture | Membranes | Animal models | Epithelial cells | Bronchopneumonia | Prophylaxis | Viruses | CX3CR1 protein | Infections | Infants | Respiratory tract | Proteins | Clonal deletion | Lymphocytes | Deletion | Fusion protein | nucleolin | Cilia | Binding | Heparan sulfate | Immunization | Immunoglobulins | Glycoprotein | Gene expression | Virology | Respiratory syncytial virus | Microscopy | Lungs | Respiratory diseases | Chemokines
Journal Article
Journal Article
PLoS ONE, ISSN 1932-6203, 03/2017, Volume 12, Issue 3, p. e0173224
Chemokines are important in macrophage recruitment and the progression of atherosclerosis. The 'M3' chemokine binding protein inactivates key chemokines... 
RECRUITMENT | INHIBITION | E-KNOCKOUT MICE | INFLAMMATION | MULTIDISCIPLINARY SCIENCES | FRACTALKINE CX3CL1 | DISEASE | LESION FORMATION | PLAQUE-FORMATION | GENE-TRANSFER | NF-KAPPA-B | Chemokines - blood | Apolipoproteins E - deficiency | Monocytes - cytology | Humans | Actins - metabolism | Monocytes - metabolism | Aorta - metabolism | Viral Proteins - metabolism | Chemokines - antagonists & inhibitors | Diet, High-Fat | Lipids - blood | HEK293 Cells | Plasmids - genetics | Adenoviridae - genetics | Atherosclerosis - veterinary | Myocytes, Smooth Muscle - cytology | Myocytes, Smooth Muscle - metabolism | Diet, Carbohydrate-Restricted | Atherosclerosis - pathology | Liver - metabolism | Cells, Cultured | Viral Proteins - genetics | Plasmids - metabolism | Atherosclerosis - metabolism | Mice, Knockout | Aorta - pathology | Animals | Apolipoproteins E - genetics | Protein Binding | Chemokines - metabolism | Mice | Adenoviridae - metabolism | Cell Movement | Heart | CC chemokine receptors | Animal models | Leukocyte migration | Liver | Fluorescence | CX3CR1 protein | Smooth muscle | Cardiovascular disease | Promotion | Macrophages | Medical schools | Proteins | Reduction | CCR2 protein | Apolipoprotein E | Actin | Rodents | Atherosclerosis | Aorta | M3 protein | Inhibition | Binding | NF-κB protein | CCR5 protein | Muscles | Inflammation | Thorax | Apolipoproteins | Chemotaxis | Medicine | Monocytes | Arteriosclerosis | Adenoviruses | Cell migration | Chemokines | Monocyte chemoattractant protein 1
Journal Article
Journal of Cellular Physiology, ISSN 0021-9541, 11/2017, Volume 232, Issue 11, pp. 2996 - 3005
Gingiva fibroblasts express 12 chemokine receptors. Fourteen chemokines were used to study proliferation (top), migration (middle), and secretion of wound... 
cytokine | migration | chemokine | gingiva | proliferation | PERIODONTAL-DISEASES | PHYSIOLOGY | PHENOTYPIC DIFFERENCES | KERATINOCYTES | MESENCHYMAL STEM-CELLS | CELL BIOLOGY | HUMAN GINGIVAL FIBROBLASTS | GROWTH-FACTOR | ACCUMULATION | EXPRESSION | SKIN INFLAMMATION | Fibroblasts - secretion | Hepatocyte Growth Factor - secretion | Humans | Dose-Response Relationship, Drug | Receptors, CCR4 - metabolism | Tissue Inhibitor of Metalloproteinase-1 - metabolism | Gingiva - metabolism | Gingiva - pathology | Gingiva - drug effects | Gingiva - secretion | Interleukin-6 - metabolism | Wound Healing - drug effects | Fibroblasts - metabolism | Chemokines, CC - pharmacology | Fibroblasts - pathology | Interleukin-6 - secretion | Receptors, CCR4 - agonists | Hepatocyte Growth Factor - metabolism | Cell Movement - drug effects | Receptors, CCR3 - metabolism | Signal Transduction - drug effects | Fibroblasts - drug effects | Ligands | Cell Proliferation - drug effects | Tissue Inhibitor of Metalloproteinase-1 - secretion | Receptors, CCR3 - agonists | Cell Line, Transformed | Wound healing | Flow cytometry | CXCL12 protein | Leukocyte migration | Mucosa | CXCL13 protein | CX3CR1 protein | Stimulation | Tissue inhibitor of metalloproteinase 1 | Interleukin 6 | CCL22 protein | Receptors | CXCR5 protein | Fibroblasts | CCL20 protein | Activation analysis | Secretion | CXCR2 protein | Chemokine receptors | CXCR4 protein | CCR6 protein | Cytometry | Healing | CXCL11 protein | Gingiva | CCR9 protein | Chemokines | Cell migration | Index Medicus | Original | Original s
Journal Article
Journal of Cellular and Molecular Medicine, ISSN 1582-1838, 08/2009, Volume 13, Issue 8a, pp. 1526 - 1535
Chemokines are the inflammatory mediators that modulate liver fibrosis, a common feature of chronic inflammatory liver diseases. CX3CL1/fractalkine is a... 
liver fibrosis | hepatic stellate cells | shedding | chemokines | Shedding | Hepatic stellate cells | Liver fibrosis | Chemokines | MEDICINE, RESEARCH & EXPERIMENTAL | FRACTALKINE RECEPTOR CXCR1 | ACTIVATION | ALPHA | MATRIX METALLOPROTEINASE-2 | CLEAVAGE | CELL BIOLOGY | IV COLLAGENASE | HEPATOCELLULAR-CARCINOMA | DISINTEGRIN | REGULATED EXPRESSION | ADAM17 Protein | Solubility - drug effects | Amyloid Precursor Protein Secretases - genetics | Gene Expression Regulation, Enzymologic - drug effects | Inflammation - pathology | Liver - pathology | Chemokine CX3CL1 - metabolism | Humans | Hepatic Stellate Cells - metabolism | Liver Cirrhosis - enzymology | RNA, Messenger - metabolism | Inflammation - metabolism | Liver - drug effects | Membrane Proteins - metabolism | Hepatic Stellate Cells - pathology | Hepatic Stellate Cells - drug effects | Cell Line | Matrix Metalloproteinase 2 - metabolism | Membrane Proteins - genetics | Liver - metabolism | RNA, Messenger - genetics | Hepatic Stellate Cells - enzymology | ADAM10 Protein | Chemokine CX3CL1 - genetics | Up-Regulation - drug effects | ADAM Proteins - metabolism | Amyloid Precursor Protein Secretases - metabolism | Liver Cirrhosis - pathology | Collagen Type I - pharmacology | ADAM Proteins - genetics | Chronic Disease | Interferon-gamma - pharmacology | Stellate cells | Matrix metalloproteinases | Liver diseases | Cytokines | Peptides | Liver | Cloning | Paracrine signalling | CX3CR1 protein | Inflammation | Metastasis | Matrix metalloproteinase | Diseases | Signal transduction | Monocytes | Cell activation | Inhibitors | Medical prognosis | Fibrosis | Extracellular matrix | Fractalkine | Immune system | Interferon-gamma | Up-Regulation | Matrix Metalloproteinase 2 | Solubility | Collagen Type I | ADAM Proteins | Liver Cirrhosis | Cellular Biology | Membrane Proteins | Gene Expression Regulation, Enzymologic | Life Sciences | Chemokine CX3CL1 | Hepatic Stellate Cells | Amyloid Precursor Protein Secretases | RNA, Messenger
Journal Article
PLoS ONE, ISSN 1932-6203, 02/2009, Volume 4, Issue 2, pp. e4509 - e4509
Chemokine receptors belong to a class of integral membrane G-protein coupled receptors (GPCRs) and are responsible for transmitting signals from the... 
MULTIDISCIPLINARY SCIENCES | Humans | Receptors, G-Protein-Coupled | Receptors, CCR5 - genetics | Nucleic Acid Amplification Techniques | Receptors, CCR5 - biosynthesis | Receptors, CCR3 - biosynthesis | Receptors, CCR3 - isolation & purification | Receptors, CCR5 - isolation & purification | Receptors, CXCR4 - isolation & purification | Receptors, CXCR4 - biosynthesis | Receptors, Chemokine - isolation & purification | Escherichia coli - genetics | Receptors, CCR3 - genetics | Cloning, Molecular - methods | Polymerase Chain Reaction | Receptors, Chemokine - biosynthesis | Receptors, CXCR4 - genetics | Receptors, Chemokine - genetics | CX3C Chemokine Receptor 1 | Surface active agents | Analysis | Monoclonal antibodies | Genetic aspects | Metastasis | Health aspects | Protein binding | Asthma | Membrane proteins | Bioengineering | Size exclusion chromatography | G protein-coupled receptors | Laboratories | Toxicity | Copy number | Genes | CX3CR1 protein | Solubilization | Optimization | Monomers | Metastases | Proteins | Engineering | Lysates | Receptors | Arabinose | E coli | Human immunodeficiency virus--HIV | Dichroism | Bacteria | Biomedical engineering | Growth conditions | CCR5 protein | Level (quantity) | Life expectancy | Purification | Cloning | Detergents | Chemokine receptors | CXCR4 protein | L-Arabinose | Circular dichroism | Choline | Dimers | Biosensors | Protein structure | Chemokines | Structural analysis | Cancer | Structure-function relationships | Index Medicus | HIV | Human immunodeficiency virus
Journal Article
Journal Article
PloS one, ISSN 1932-6203, 2018, Volume 13, Issue 6, p. e0200056
[This corrects the article DOI: 10.1371/journal.pone.0198608.]. 
Phagocytes | CX3CR1 protein
Journal Article