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1. Full Text Sensory lesioning induces microglial synapse elimination via ADAM10 and fractalkine signaling
by Gunner, Georgia and Cheadle, Lucas and Johnson, Kasey M and Ayata, Pinar and Badimon, Ana and Mondo, Erica and Nagy, M. Aurel and Liu, Liwang and Bemiller, Shane M and Kim, Ki-Wook and Lira, Sergio A and Lamb, Bruce T and Tapper, Andrew R and Ransohoff, Richard M and Greenberg, Michael E and Schaefer, Anne and Schafer, Dorothy P
Nature neuroscience, ISSN 1097-6256, 07/2019, Volume 22, Issue 7, pp. 1075 - 1088
Neurosciences | Neurosciences & Neurology | Life Sciences & Biomedicine | Science & Technology | Amyloid Precursor Protein Secretases - genetics | Vibrissae - injuries | Amyloid Precursor Protein Secretases - physiology | ADAM10 Protein - genetics | CX3C Chemokine Receptor 1 - physiology | Cell Count | Transcriptome | Male | Chemokine CX3CL1 - physiology | RNA, Messenger - biosynthesis | ADAM10 Protein - antagonists & inhibitors | Microglia - physiology | CX3C Chemokine Receptor 1 - genetics | Membrane Proteins - physiology | Female | Single-Cell Analysis | Microfluidic Analytical Techniques | Membrane Proteins - genetics | Mice, Inbred C57BL | RNA, Messenger - genetics | Gene Expression Regulation | CX3C Chemokine Receptor 1 - deficiency | Sensorimotor Cortex - physiopathology | Mice, Knockout | Animals | Membrane Proteins - antagonists & inhibitors | Vibrissae - physiology | Signal Transduction - physiology | ADAM10 Protein - physiology | Mice | Amyloid Precursor Protein Secretases - antagonists & inhibitors | Touch - physiology | Sensorimotor Cortex - metabolism | Sensorimotor Cortex - pathology | Physiological aspects | Neural transmission | Control | Neurons | Analysis | Synapses | Brain | Complement receptor 3 | CX3CR1 protein | Ribonucleic acid--RNA | Microglia | Gene sequencing | Defects | Signaling | Neural networks | Metalloproteinase | Fractalkine | Synapse elimination | Index Medicus
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2. Full Text Neurotoxic microglia promote TDP-43 proteinopathy in progranulin deficiency
by Zhang, Jiasheng and Velmeshev, Dmitry and Hashimoto, Kei and Huang, Yu-Hsin and Hofmann, Jeffrey W and Shi, Xiaoyu and Chen, Jiapei and Leidal, Andrew M and Dishart, Julian G and Cahill, Michelle K and Kelley, Kevin W and Liddelow, Shane A and Seeley, William W and Miller, Bruce L and Walther, Tobias C and Farese, Robert V and Taylor, J. Paul and Ullian, Erik M and Huang, Bo and Debnath, Jayanta and Wittmann, Torsten and Kriegstein, Arnold R and Huang, Eric J
Nature (London), ISSN 0028-0836, 12/2020, Volume 588, Issue 7838, pp. 459 - 3
Science & Technology - Other Topics | Multidisciplinary Sciences | Science & Technology | Microglia - metabolism | Neurons - pathology | Thalamus - metabolism | Complement C3b - antagonists & inhibitors | Complement C1q - immunology | Culture Media, Conditioned - chemistry | Transcriptome | Culture Media, Conditioned - pharmacology | Male | Progranulins - deficiency | Thalamus - pathology | Complement Activation - immunology | DNA-Binding Proteins - metabolism | TDP-43 Proteinopathies - genetics | Aging - genetics | Cell Nucleus - pathology | Microglia - pathology | Progranulins - genetics | Female | Neurons - metabolism | TDP-43 Proteinopathies - drug therapy | TDP-43 Proteinopathies - pathology | Single-Cell Analysis | Complement Activation - drug effects | Disease Models, Animal | Nuclear Pore - pathology | TDP-43 Proteinopathies - metabolism | Nuclear Pore - metabolism | Aging - pathology | Animals | Cell Nucleus - genetics | Complement C1q - antagonists & inhibitors | RNA-Seq | Mice | Complement C3b - immunology | Physiological aspects | Causes of | Glycoproteins | Diagnosis | Protein deficiency | Binding proteins | Health aspects | Neurotoxicity syndromes | RNA sequencing | RNA | Cell death | Neurons | Genes | Protein binding | Normalizing | Protein kinase C | Membranes | Coagulation | Cysteine proteinase | Homeostasis | CX3CR1 protein | Kinases | Exocytosis | Cell adhesion & migration | Proteins | Signal transduction | Neurotoxicity | Annotations | Neurodegeneration | Thalamus | Protein-tyrosine kinase receptors | Protein transport | Localization | Protein-tyrosine kinase | Age | Tyrosine | MAP kinase | Amyotrophic lateral sclerosis | Proteinase inhibitors | Ras protein | Gene expression | Microglia | Membrane proteins | Protease inhibitors | Alzheimers disease | Dendritic structure | Cell migration | Phagocytosis | Index Medicus
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3. Full Text AB1154 Increase generation but defects of secreting ifn-[ALPHA] play a role in the pathogenesis of igg4-rd
by P. Zhang and W. Zhang
Annals of the rheumatic diseases, ISSN 0003-4967, 06/2018, Volume 77, p. 1681
Cell culture | CC chemokine receptors | Flow cytometry | CD86 antigen | Pathogenesis | Immunoglobulin G | CX3CR1 protein | Infections | Cell surface | Defects | CCR1 protein | α-Interferon | CCR2 protein | Etiology | Antigen presentation | CCR5 protein | Dendritic cells | CXCR2 protein | CCR7 protein | CD80 antigen | Adaptive immunity | CXCR3 protein | Patients | CCR6 protein | CD83 antigen | CCR8 protein | CCR9 protein | Surface markers | Monocyte chemoattractant protein 1
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4. Full Text Stimulation of oral fibroblast chemokine receptors identifies CCR3 and CCR4 as potential wound healing targets
by Buskermolen, J.K and Roffel, S and Gibbs, S
Journal of cellular physiology, ISSN 0021-9541, 11/2017, Volume 232, Issue 11, pp. 2996 - 3005
cytokine | chemokine | gingiva | proliferation | migration | Clinical Biochemistry | Physiology | Cell Biology | Life Sciences & Biomedicine | Science & Technology | Chemokines, CC - pharmacology | Humans | Fibroblasts - pathology | Receptors, CCR4 - agonists | Hepatocyte Growth Factor - metabolism | Dose-Response Relationship, Drug | Cell Movement - drug effects | Receptors, CCR3 - metabolism | Receptors, CCR4 - metabolism | Tissue Inhibitor of Metalloproteinase-1 - metabolism | Gingiva - metabolism | Gingiva - pathology | Gingiva - drug effects | Signal Transduction - drug effects | Fibroblasts - drug effects | Ligands | Cell Proliferation - drug effects | Interleukin-6 - metabolism | Receptors, CCR3 - agonists | Wound Healing - drug effects | Cell Line, Transformed | Fibroblasts - metabolism | Wound healing | Flow cytometry | CXCL12 protein | Leukocyte migration | Mucosa | CXCL13 protein | CX3CR1 protein | Stimulation | Tissue inhibitor of metalloproteinase 1 | Interleukin 6 | CCL22 protein | Receptors | CXCR5 protein | Fibroblasts | CCL20 protein | Activation analysis | CXCR2 protein | Chemokine receptors | CXCR4 protein | CCR6 protein | Cytometry | CXCL11 protein | Gingiva | CCR9 protein | Chemokines | Cell migration | Index Medicus | Original | Original s
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5. Full Text Chemokines in neuron–glial cell interaction and pathogenesis of neuropathic pain
by Zhang, Zhi-Jun and Jiang, Bao-Chun and Gao, Yong-Jing
Cellular and molecular life sciences : CMLS, ISSN 1420-682X, 9/2017, Volume 74, Issue 18, pp. 3275 - 3291
Life Sciences | Biomedicine, general | Biochemistry, general | Spinal cord | Life Sciences, general | Astrocytes | Neuroinflammation | Chronic pain | Cell Biology | Microglia | Biochemistry & Molecular Biology | Life Sciences & Biomedicine | Science & Technology | Animals | Spinal Cord - metabolism | Signal Transduction | Receptors, Chemokine - metabolism | Neuralgia - metabolism | Neuroglia - metabolism | Chemokines - metabolism | Neurons - metabolism | Neuralgia - pathology | Medical colleges | Care and treatment | Human anatomy | Pain | Neurons | CC chemokine receptors | Regulators | Pathogenesis | CXCL13 protein | Central nervous system | CX3CR1 protein | Nervous system | Activation | Neuropathy | Glial cells | Neuronal-glial interactions | CCR2 protein | CXCR5 protein | Synaptic transmission | Damage | Cytokines | CXCR2 protein | Inflammation | Cells | Rope | Chemokines | Monocyte chemoattractant protein 1 | Index Medicus
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6. Full Text The role of the microglial Cx3cr1 pathway in the postnatal maturation of retinal photoreceptors
by Jobling, Andrew I and Waugh, Michelle and Vessey, Kirstan A and Phipps, Joanna A and Trogrlic, Lidia and Greferath, Una and Mills, Samuel A and Tan, Zhi L and Ward, Michelle M and Fletcher, Erica L
The Journal of neuroscience, ISSN 0270-6474, 05/2018, Volume 38, Issue 20, pp. 4708 - 4723
Retina | Fractalkine | Cilium | Cx3cr1 | Photoreceptor | Microglia | Cytoskeletal Proteins | Retina - growth & development | CX3C Chemokine Receptor 1 - physiology | Male | Photoreceptor Connecting Cilium - physiology | Photoreceptor Cells, Vertebrate - physiology | Light | Female | Eye Proteins - genetics | Carrier Proteins - physiology | Proteins - physiology | Mice, Inbred C57BL | Retinal Cone Photoreceptor Cells - physiology | Eye - growth & development | Mice, Transgenic | Mice, Knockout | Proteins - genetics | Retina - physiology | Carrier Proteins - genetics | Retinal Photoreceptor Cell Outer Segment - physiology | Animals | Signal Transduction - physiology | Eye Proteins - physiology | Mice | Photoreception | Maturation | Genes | Gene regulation | Central nervous system | CX3CR1 protein | Gene expression | Proteins | Eye | Signal transduction | Signaling | Elongated structure | Eye injuries | Photoreceptors | Elongation | Immune system | Index Medicus | cilium | s | fractalkine | retina | photoreceptor | microglia
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7. Full Text The macrophage phenotype and inflammasome component NLRP3 contributes to nephrocalcinosis-related chronic kidney disease independent from IL-1–mediated tissue injury
by Anders, Hans-Joachim and Suarez-Alvarez, Beatriz and Grigorescu, Melissa and Foresto-Neto, Orestes and Steiger, Stefanie and Desai, Jyaysi and Marschner, Julian A and Honarpisheh, Mohsen and Shi, Chongxu and Jordan, Jutta and Müller, Lisa and Burzlaff, Nicolai and Bäuerle, Tobias and Mulay, Shrikant R
Kidney international, ISSN 0085-2538, 03/2018, Volume 93, Issue 3, pp. 656 - 669
beta-hydroxybutyrate | necroinflammation | fibrosis | crystal nephropathy | NALP3 | Life Sciences & Biomedicine | Urology & Nephrology | Science & Technology | Cell Plasticity - drug effects | Inflammasomes - metabolism | Kidney - pathology | Nephrocalcinosis - immunology | Kidney - immunology | Hyperoxaluria - metabolism | Male | CARD Signaling Adaptor Proteins - genetics | Hyperoxaluria - drug therapy | NLR Family, Pyrin Domain-Containing 3 Protein - genetics | Renal Insufficiency, Chronic - metabolism | CARD Signaling Adaptor Proteins - metabolism | Kidney - metabolism | Nephrocalcinosis - metabolism | Butylene Glycols - pharmacology | Inflammasomes - drug effects | Interleukin-1 - immunology | Nephrocalcinosis - prevention & control | Female | Macrophages - immunology | Disease Models, Animal | Fibroblasts - metabolism | Interleukin-1 - metabolism | Renal Insufficiency, Chronic - immunology | Macrophages - pathology | Signal Transduction | NLR Family, Pyrin Domain-Containing 3 Protein - metabolism | Mice, Inbred C57BL | Cells, Cultured | NLR Family, Pyrin Domain-Containing 3 Protein - antagonists & inhibitors | Fibroblasts - pathology | Inflammasomes - genetics | Hyperoxaluria - pathology | Mice, Knockout | Renal Insufficiency, Chronic - pathology | Renal Insufficiency, Chronic - prevention & control | Macrophages - metabolism | Nephrocalcinosis - pathology | Phenotype | Animals | NLR Family, Pyrin Domain-Containing 3 Protein - immunology | Receptors, Transforming Growth Factor beta - metabolism | Inflammasomes - immunology | Fibroblasts - immunology | Macrophages - drug effects | Hyperoxaluria - immunology | End-stage renal disease | Phenotypes | CD11b antigen | Kidneys | Interleukin 1 receptor antagonist | Therapeutic applications | CX3CR1 protein | Inflammation | Macrophages | CD45 antigen | Atrophy | Signal transduction | Calcium oxalate | Oxalic acid | Calcinosis | Interleukin 1 | Fibrosis | Fibroblasts | Hyperoxaluria | Kidney diseases | Index Medicus
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8. Full Text Self-renewing resident cardiac macrophages limit adverse remodeling following myocardial infarction
by Dick, Sarah A and Macklin, Jillian A and Nejat, Sara and Momen, Abdul and Clemente-Casares, Xavier and Althagafi, Marwan G and Chen, Jinmiao and Kantores, Crystal and Hosseinzadeh, Siyavash and Aronoff, Laura and Wong, Anthony and Zaman, Rysa and Barbu, Iulia and Besla, Rickvinder and Lavine, Kory J and Razani, Babak and Ginhoux, Florent and Husain, Mansoor and Cybulsky, Myron I and Robbins, Clinton S and Epelman, Slava
Nature immunology, ISSN 1529-2908, 01/2019, Volume 20, Issue 1, pp. 29 - 39
Life Sciences & Biomedicine | Immunology | Science & Technology | Macrophages - physiology | Receptors, CCR2 - genetics | Vesicular Transport Proteins - metabolism | Mice, Transgenic | Myocardium - pathology | Gene Expression Profiling | Myocardial Infarction - immunology | Parabiosis | Cell Lineage | Cell Self Renewal | Receptors, CCR2 - metabolism | Ventricular Remodeling | Animals | CX3C Chemokine Receptor 1 - metabolism | Histocompatibility Antigens Class II - metabolism | Cell Differentiation | Membrane Proteins - metabolism | Mice | Single-Cell Analysis | RNA sequencing | Usage | Genetic aspects | Research | Gene expression | Macrophages | Heart attack | Heart | Myocardial infarction | CC chemokine receptors | Fate maps | Heart attacks | CX3CR1 protein | Mapping | Remodeling | Monocytes | Injury prevention | Depletion | Major histocompatibility complex | Cell fate | Ischemia | CCR2 protein | Infarction | Monocyte chemoattractant protein 1 | Index Medicus
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9. Full Text Inflammation shapes pathogenesis of murine arrhythmogenic cardiomyopathy
by Lubos, Nadine and van der Gaag, Svenja and Gercek, Muhammed and Kant, Sebastian and Leube, Rudolf E and Krusche, Claudia A
Basic research in cardiology, ISSN 0300-8428, 06/2020, Volume 115, Issue 4, pp. 42 - 42
Cardiac & Cardiovascular Systems | Life Sciences & Biomedicine | Cardiovascular System & Cardiology | Science & Technology | CC chemokine receptors | Animal models | Arrhythmia | Cardiomyopathy | Pathogenesis | Antibodies | CX3CR1 protein | Inflammatory response | Lymphocytes T | mRNA | Macrophages | Necrosis | CCR2 protein | Lymphocytes | Heart diseases | Immune system | CCR5 protein | Autoantibodies | CXCR2 protein | CCL3 protein | Cardiomyocytes | Inflammation | CXCR3 protein | Scars | Collagen | CXCL10 protein | Fibrosis | Mutation | Monocyte chemoattractant protein 1 | Chemokines | Index Medicus | Original Contribution | Desmoglein | Chronic disease progression | Desmosome | Immune cells
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