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1. Full Text AB1154 Increase generation but defects of secreting ifn-[ALPHA] play a role in the pathogenesis of igg4-rd
by P. Zhang and W. Zhang
Annals of the Rheumatic Diseases, ISSN 0003-4967, 06/2018, Volume 77, p. 1681
Background IgG4 related disease (IgG4-RD) is a multi-organ involvement, fibro-inflammatory disease of unknown etiology. Both innate and adaptive immunity... 
Cell culture | CC chemokine receptors | Flow cytometry | CD86 antigen | Pathogenesis | Immunoglobulin G | CX3CR1 protein | Infections | Cell surface | Defects | CCR1 protein | α-Interferon | CCR2 protein | Etiology | Antigen presentation | CCR5 protein | Dendritic cells | CXCR2 protein | CCR7 protein | CD80 antigen | Adaptive immunity | CXCR3 protein | Patients | CCR6 protein | CD83 antigen | CCR8 protein | CCR9 protein | Surface markers | Monocyte chemoattractant protein 1
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2. Full Text CX3CR1 is expressed in differentiated human ciliated airway cells and co-localizes with respiratory syncytial virus on cilia in a G protein-dependent manner
by Jeong, Kwang-Il and Piepenhagen, Peter A and Kishko, Michael and DiNapoli, Joshua M and Groppo, Rachel P and Zhang, Linong and Almond, Jeffrey and Kleanthous, Harry and Delagrave, Simon and Parrington, Mark
PLoS ONE, ISSN 1932-6203, 06/2015, Volume 10, Issue 6, p. e0130517
Respiratory syncytial virus (RSV) is the principal cause of bronchiolitis in infants and a significant healthcare problem. The RSV Glycoprotein (G) mediates... 
MOLECULAR CHARACTERIZATION | VACCINE DEVELOPMENT | REPLICATION IN-VITRO | ATTACHMENT | EPITHELIAL-CELLS | CX3C-CX3CR1 BINDING | RSV | MULTIDISCIPLINARY SCIENCES | INFECTION | FUSION GLYCOPROTEIN | RECEPTOR CXCR1 | Respiratory Syncytial Virus, Human - metabolism | Sequence Deletion | Epithelial Cells - metabolism | Viral Fusion Proteins - metabolism | Viral Fusion Proteins - genetics | Humans | Molecular Sequence Data | Respiratory Mucosa - virology | Epitopes - immunology | Respiratory Mucosa - pathology | Respiratory Syncytial Virus, Human - genetics | Viral Fusion Proteins - chemistry | Base Sequence | Viral Envelope Proteins - metabolism | Cell Differentiation | Receptors, Chemokine - antagonists & inhibitors | Receptors, Chemokine - chemistry | Receptors, Chemokine - genetics | Binding Sites | Child | Gene Expression | Viral Envelope Proteins - genetics | Cilia - pathology | Receptors, Chemokine - metabolism | Epithelial Cells - pathology | Cilia - metabolism | Antibodies - pharmacology | Viral Envelope Proteins - antagonists & inhibitors | Epithelial Cells - virology | Viral Envelope Proteins - chemistry | Protein Binding | Epitopes - chemistry | Primary Cell Culture | Respiratory Mucosa - metabolism | CX3C Chemokine Receptor 1 | Cilia - virology | Infection | G proteins | Health aspects | Protein binding | Asthma | Health care | Cell culture | Membranes | Animal models | Epithelial cells | Bronchopneumonia | Prophylaxis | Viruses | CX3CR1 protein | Infections | Infants | Respiratory tract | Proteins | Clonal deletion | Lymphocytes | Deletion | Fusion protein | nucleolin | Cilia | Binding | Heparan sulfate | Immunization | Immunoglobulins | Glycoprotein | Gene expression | Virology | Respiratory syncytial virus | Microscopy | Lungs | Respiratory diseases | Chemokines
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3. Full Text The DRF motif of CXCR6 as chemokine receptor adaptation to adhesion
by Koenen, Andrea and Babendreyer, Aaron and Schumacher, Julian and Pasqualon, Tobias and Schwarz, Nicole and Seifert, Anke and Deupi, Xavier and Ludwig, Andreas and Dreymueller, Daniela
PLoS ONE, ISSN 1932-6203, 03/2017, Volume 12, Issue 3, pp. e0173486 - e0173486
The CXC-chemokine receptor 6 (CXCR6) is a class A GTP-binding protein-coupled receptor (GPCRs) that mediates adhesion of leukocytes by interacting with the... 
MIGRATION | SYSTEM | INTERNALIZATION | INFLAMMATION | MULTIDISCIPLINARY SCIENCES | LIGAND CXCL16 | ATHEROSCLEROSIS | PROLIFERATION | TNF-ALPHA | ACCUMULATION | T-CELLS | Usage | Phenylalanine | Metastasis | Research | G proteins | Health aspects | Chemokine receptors | Protein binding | GTP-binding protein | Phosphorylation | Calcium | Leukocyte migration | Lung cancer | Homeostasis | CX3CR1 protein | AKT protein | Adhesive bonding | Leukocytes | Chemotactic response | Kinases | Cell surface | Hydrogen bonding | Protein turnover | Cell adhesion & migration | Metastases | Proteins | Calcium signalling | Receptors | Toxicology | Hydrogen bonds | Atherosclerosis | CXCL16 protein | Adaptation | Binding | Cloning | Pharmacology | Breast cancer | Inflammation | Chemotaxis | Adhesion | White blood cells | Monocytes | Internalization | Ligands | Receptor mechanisms | Cell migration | Chemokines | Cancer | Index Medicus
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4. Full Text Selective chemokine receptor usage by central nervous system myeloid cells in CCR2-red fluorescent protein knock-in mice
by Saederup, Noah and Cardona, Astrid E and Croft, Kelsey and Mizutani, Makiko and Cotleur, Anne C and Tsou, Chia-Lin and Ransohoff, Richard M and Charo, Israel F
PLoS ONE, ISSN 1932-6203, 2010, Volume 5, Issue 10, p. e13693
Background: Monocyte subpopulations distinguished by differential expression of chemokine receptors CCR2 and CX3CR1 are difficult to track in vivo, partly due... 
IMMUNE-RESPONSE | EXPERIMENTAL AUTOIMMUNE ENCEPHALOMYELITIS | FRACTALKINE RECEPTOR | DENDRITIC CELLS | MULTIPLE-SCLEROSIS | MULTIDISCIPLINARY SCIENCES | BONE-MARROW | MONOCYTE CHEMOATTRACTANT PROTEIN-1 | SUBSETS | BLOOD MONOCYTES | MICROGLIAL CELLS | Receptors, CCR2 - genetics | Receptors, CCR2 - metabolism | Animals | Central Nervous System - metabolism | Central Nervous System - cytology | Luminescent Proteins - genetics | Mice | Fluorescence | Central nervous system | CC chemokine receptors | Flow cytometry | Neurosciences | Multiple sclerosis | Encephalomyelitis | Trafficking | CX3CR1 protein | Cardiovascular disease | Nervous system | Macrophages | Proteins | Receptors | CCR2 protein | Lymphocytes | Rodents | Localization | Subpopulations | Myeloid cells | Cytokines | Red fluorescent protein | Leukocytes (neutrophilic) | Inflammation | T cell receptors | Experimental allergic encephalomyelitis | Chemokine receptors | Microglia | Monocytes | Reagents | Stem cells | In vivo methods and tests | Chemokines | Monocyte chemoattractant protein 1
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5. Full Text Chemokine binding protein 'M3' limits atherosclerosis in apolipoprotein E-/- mice
by Ravindran, Dhanya and Ridiandries, Anisyah and Vanags, Laura Z and Henriquez, Rodney and Cartland, Siân and Tan, Joanne T.M and Bursill, Christina A
PLoS ONE, ISSN 1932-6203, 03/2017, Volume 12, Issue 3, p. e0173224
Chemokines are important in macrophage recruitment and the progression of atherosclerosis. The 'M3' chemokine binding protein inactivates key chemokines... 
RECRUITMENT | INHIBITION | E-KNOCKOUT MICE | INFLAMMATION | MULTIDISCIPLINARY SCIENCES | FRACTALKINE CX3CL1 | DISEASE | LESION FORMATION | PLAQUE-FORMATION | GENE-TRANSFER | NF-KAPPA-B | Chemokines - blood | Apolipoproteins E - deficiency | Monocytes - cytology | Humans | Actins - metabolism | Monocytes - metabolism | Aorta - metabolism | Viral Proteins - metabolism | Chemokines - antagonists & inhibitors | Diet, High-Fat | Lipids - blood | HEK293 Cells | Plasmids - genetics | Adenoviridae - genetics | Atherosclerosis - veterinary | Myocytes, Smooth Muscle - cytology | Myocytes, Smooth Muscle - metabolism | Diet, Carbohydrate-Restricted | Atherosclerosis - pathology | Liver - metabolism | Cells, Cultured | Viral Proteins - genetics | Plasmids - metabolism | Atherosclerosis - metabolism | Mice, Knockout | Aorta - pathology | Animals | Apolipoproteins E - genetics | Protein Binding | Chemokines - metabolism | Mice | Adenoviridae - metabolism | Cell Movement | Heart | CC chemokine receptors | Animal models | Leukocyte migration | Liver | Fluorescence | CX3CR1 protein | Smooth muscle | Cardiovascular disease | Promotion | Macrophages | Medical schools | Proteins | Reduction | CCR2 protein | Apolipoprotein E | Actin | Rodents | Atherosclerosis | Aorta | M3 protein | Inhibition | Binding | NF-κB protein | CCR5 protein | Muscles | Inflammation | Thorax | Apolipoproteins | Chemotaxis | Medicine | Monocytes | Arteriosclerosis | Adenoviruses | Cell migration | Chemokines | Monocyte chemoattractant protein 1
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6. Full Text Stimulation of oral fibroblast chemokine receptors identifies CCR3 and CCR4 as potential wound healing targets
by Buskermolen, Jeroen K and Roffel, Sanne and Gibbs, Susan
Journal of Cellular Physiology, ISSN 0021-9541, 11/2017, Volume 232, Issue 11, pp. 2996 - 3005
Gingiva fibroblasts express 12 chemokine receptors. Fourteen chemokines were used to study proliferation (top), migration (middle), and secretion of wound... 
cytokine | migration | chemokine | gingiva | proliferation | PERIODONTAL-DISEASES | PHYSIOLOGY | PHENOTYPIC DIFFERENCES | KERATINOCYTES | MESENCHYMAL STEM-CELLS | CELL BIOLOGY | HUMAN GINGIVAL FIBROBLASTS | GROWTH-FACTOR | ACCUMULATION | EXPRESSION | SKIN INFLAMMATION | Fibroblasts - secretion | Hepatocyte Growth Factor - secretion | Humans | Dose-Response Relationship, Drug | Receptors, CCR4 - metabolism | Tissue Inhibitor of Metalloproteinase-1 - metabolism | Gingiva - metabolism | Gingiva - pathology | Gingiva - drug effects | Gingiva - secretion | Interleukin-6 - metabolism | Wound Healing - drug effects | Fibroblasts - metabolism | Chemokines, CC - pharmacology | Fibroblasts - pathology | Interleukin-6 - secretion | Receptors, CCR4 - agonists | Hepatocyte Growth Factor - metabolism | Cell Movement - drug effects | Receptors, CCR3 - metabolism | Signal Transduction - drug effects | Fibroblasts - drug effects | Ligands | Cell Proliferation - drug effects | Tissue Inhibitor of Metalloproteinase-1 - secretion | Receptors, CCR3 - agonists | Cell Line, Transformed | Wound healing | Flow cytometry | CXCL12 protein | Leukocyte migration | Mucosa | CXCL13 protein | CX3CR1 protein | Stimulation | Tissue inhibitor of metalloproteinase 1 | Interleukin 6 | CCL22 protein | Receptors | CXCR5 protein | Fibroblasts | CCL20 protein | Activation analysis | Secretion | CXCR2 protein | Chemokine receptors | CXCR4 protein | CCR6 protein | Cytometry | Healing | CXCL11 protein | Gingiva | CCR9 protein | Chemokines | Cell migration | Index Medicus | Original | Original s
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7. Full Text CX3CL1/fractalkine shedding by human hepatic stellate cells: contribution to chronic inflammation in the liver
by Bourd‐Boittin, Katia and Basset, Laetitia and Bonnier, Dominique and L’Helgoualc’h, Annie and Samson, Michel and Théret, Nathalie
Journal of Cellular and Molecular Medicine, ISSN 1582-1838, 08/2009, Volume 13, Issue 8a, pp. 1526 - 1535
Chemokines are the inflammatory mediators that modulate liver fibrosis, a common feature of chronic inflammatory liver diseases. CX3CL1/fractalkine is a... 
liver fibrosis | hepatic stellate cells | shedding | chemokines | Shedding | Hepatic stellate cells | Liver fibrosis | Chemokines | MEDICINE, RESEARCH & EXPERIMENTAL | FRACTALKINE RECEPTOR CXCR1 | ACTIVATION | ALPHA | MATRIX METALLOPROTEINASE-2 | CLEAVAGE | CELL BIOLOGY | IV COLLAGENASE | HEPATOCELLULAR-CARCINOMA | DISINTEGRIN | REGULATED EXPRESSION | ADAM17 Protein | Solubility - drug effects | Amyloid Precursor Protein Secretases - genetics | Gene Expression Regulation, Enzymologic - drug effects | Inflammation - pathology | Liver - pathology | Chemokine CX3CL1 - metabolism | Humans | Hepatic Stellate Cells - metabolism | Liver Cirrhosis - enzymology | RNA, Messenger - metabolism | Inflammation - metabolism | Liver - drug effects | Membrane Proteins - metabolism | Hepatic Stellate Cells - pathology | Hepatic Stellate Cells - drug effects | Cell Line | Matrix Metalloproteinase 2 - metabolism | Membrane Proteins - genetics | Liver - metabolism | RNA, Messenger - genetics | Hepatic Stellate Cells - enzymology | ADAM10 Protein | Chemokine CX3CL1 - genetics | Up-Regulation - drug effects | ADAM Proteins - metabolism | Amyloid Precursor Protein Secretases - metabolism | Liver Cirrhosis - pathology | Collagen Type I - pharmacology | ADAM Proteins - genetics | Chronic Disease | Interferon-gamma - pharmacology | Stellate cells | Matrix metalloproteinases | Liver diseases | Cytokines | Peptides | Liver | Cloning | Paracrine signalling | CX3CR1 protein | Inflammation | Metastasis | Matrix metalloproteinase | Diseases | Signal transduction | Monocytes | Cell activation | Inhibitors | Medical prognosis | Fibrosis | Extracellular matrix | Fractalkine | Immune system | Interferon-gamma | Up-Regulation | Matrix Metalloproteinase 2 | Solubility | Collagen Type I | ADAM Proteins | Liver Cirrhosis | Cellular Biology | Membrane Proteins | Gene Expression Regulation, Enzymologic | Life Sciences | Chemokine CX3CL1 | Hepatic Stellate Cells | Amyloid Precursor Protein Secretases | RNA, Messenger
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8. Full Text High-level production, solubilization and purification of synthetic human GPCR chemokine receptors CCR5, CCR3, CXCR4 and CX3CR1
by Ren, Hui and Yu, Daoyong and Ge, Baosheng and Cook, Brian and Xu, Zhinan and Zhang, Shuguang
PLoS ONE, ISSN 1932-6203, 02/2009, Volume 4, Issue 2, pp. e4509 - e4509
Chemokine receptors belong to a class of integral membrane G-protein coupled receptors (GPCRs) and are responsible for transmitting signals from the... 
MULTIDISCIPLINARY SCIENCES | Humans | Receptors, G-Protein-Coupled | Receptors, CCR5 - genetics | Nucleic Acid Amplification Techniques | Receptors, CCR5 - biosynthesis | Receptors, CCR3 - biosynthesis | Receptors, CCR3 - isolation & purification | Receptors, CCR5 - isolation & purification | Receptors, CXCR4 - isolation & purification | Receptors, CXCR4 - biosynthesis | Receptors, Chemokine - isolation & purification | Escherichia coli - genetics | Receptors, CCR3 - genetics | Cloning, Molecular - methods | Polymerase Chain Reaction | Receptors, Chemokine - biosynthesis | Receptors, CXCR4 - genetics | Receptors, Chemokine - genetics | CX3C Chemokine Receptor 1 | Surface active agents | Analysis | Monoclonal antibodies | Genetic aspects | Metastasis | Health aspects | Protein binding | Asthma | Membrane proteins | Bioengineering | Size exclusion chromatography | G protein-coupled receptors | Laboratories | Toxicity | Copy number | Genes | CX3CR1 protein | Solubilization | Optimization | Monomers | Metastases | Proteins | Engineering | Lysates | Receptors | Arabinose | E coli | Human immunodeficiency virus--HIV | Dichroism | Bacteria | Biomedical engineering | Growth conditions | CCR5 protein | Level (quantity) | Life expectancy | Purification | Cloning | Detergents | Chemokine receptors | CXCR4 protein | L-Arabinose | Circular dichroism | Choline | Dimers | Biosensors | Protein structure | Chemokines | Structural analysis | Cancer | Structure-function relationships | Index Medicus | HIV | Human immunodeficiency virus
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9. Full Text Identification of primary genes in glomeruli compartment of immunoglobulin A nephropathy by bioinformatic analysis
by Miraji, MK and Cheng, YC and Ge, SW and Xu, G
PEERJ, ISSN 2167-8359, 07/2019, Volume 7, p. e7067
The current study is aimed to explore the specific genes which are responsible for the manifestation of Immunoglobulin A nephropathy (IgAN). Gene expression... 
PATHOGENESIS | PATHWAYS | Protein-protein interaction | Gene expression profiling | MULTIDISCIPLINARY SCIENCES | Down-regulation | Up-regulation | Immunoglobulin A Nephropathy | FIBRONECTIN | IGA NEPHROPATHY | PROGRESSION | Fibronectins | Genes | B cells | Kidney diseases | Immunoglobulin A | Gene expression | Protein-protein interactions | Immunoglobulins | Nephrology | Pathogenesis | CX3CR1 protein | Inflammation | Kinases | CCL4 protein | Data bases | Datasets | Proteins | Medicine | Signal transduction | Immunology | Nephropathy | CD44 antigen | Rodents | Quality control | Extracellular matrix | Protein interaction | Bioinformatics | Informatics | Protein–protein interaction
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10. Full Text Chemokines in neuron–glial cell interaction and pathogenesis of neuropathic pain
by Zhang, Zhi-Jun and Jiang, Bao-Chun and Gao, Yong-Jing
Cellular and Molecular Life Sciences, ISSN 1420-682X, 9/2017, Volume 74, Issue 18, pp. 3275 - 3291
Neuropathic pain resulting from damage or dysfunction of the nervous system is a highly debilitating chronic pain state and is often resistant to currently... 
Life Sciences | Biomedicine, general | Biochemistry, general | Spinal cord | Life Sciences, general | Astrocytes | Neuroinflammation | Chronic pain | Cell Biology | Microglia | EXPERIMENTAL AUTOIMMUNE ENCEPHALOMYELITIS | RECEPTOR ANTAGONISTS | BIOCHEMISTRY & MOLECULAR BIOLOGY | FOCAL CEREBRAL-ISCHEMIA | MONOCYTE CHEMOATTRACTANT PROTEIN-1 | DORSAL-ROOT GANGLIA | MICROGLIAL CATHEPSIN-S | BONE CANCER PAIN | CELL BIOLOGY | PERIPHERAL-NERVE INJURY | LONG-TERM POTENTIATION | SPINAL-CORD ASTROCYTES | Animals | Spinal Cord - metabolism | Signal Transduction | Receptors, Chemokine - metabolism | Neuralgia - metabolism | Neuroglia - metabolism | Chemokines - metabolism | Neurons - metabolism | Neuralgia - pathology | Medical colleges | Care and treatment | Human anatomy | Pain | Neurons | CC chemokine receptors | Regulators | Pathogenesis | Trafficking | CXCL13 protein | Central nervous system | CX3CR1 protein | Nervous system | Activation | Neuropathy | Glial cells | Neuronal-glial interactions | CCR2 protein | CXCR5 protein | Synaptic transmission | Damage | Cytokines | CXCR2 protein | Inflammation | Cells | Rope | Chemokines | Monocyte chemoattractant protein 1
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11. Full Text Correction: Characterization of kidney CD45intCD11bintF4/80+MHCII+CX3CR1+Ly6C- "intermediate mononuclear phagocytic cells"
by PLOS ONE Staff and PLOS ONE Staff and The PLOS ONE Staff
PloS one, ISSN 1932-6203, 2018, Volume 13, Issue 6, p. e0200056
[This corrects the article DOI: 10.1371/journal.pone.0198608.]. 
Phagocytes | CX3CR1 protein
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