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Journal of Clinical Investigation, ISSN 0021-9738, 01/2011, Volume 121, Issue 1, pp. 308 - 317
Chronic infection with hepatitis C virus (HCV) is a major public health problem, with nearly 170 million infected individuals worldwide Current treatment for... 
MEDICINE, RESEARCH & EXPERIMENTAL | LIVER INFLAMMATION | GAMMA-INDUCIBLE PROTEIN-10 | VIRUS-INFECTION | DIPEPTIDYL-PEPTIDASE-IV | ANTIVIRAL THERAPY | INSULIN-RESISTANCE | IMMUNE-RESPONSES | CHRONIC HEPATITIS-C | LEVELS CORRELATE | PEGINTERFERON PLUS RIBAVIRIN | Chemokine CXCL10 - blood | Prognosis | Antiviral Agents - therapeutic use | Receptors, CXCR3 - blood | Humans | Ribavirin - therapeutic use | Interferon-alpha - therapeutic use | Chemokine CXCL10 - antagonists & inhibitors | Dipeptidyl Peptidase 4 - blood | Hepatitis C, Chronic - virology | Hepatitis C, Chronic - therapy | Recombinant Proteins | Hepatitis C, Chronic - blood | Polyethylene Glycols - therapeutic use | T-Lymphocytes - virology | Treatment Failure | Hepatitis C, Chronic - immunology | Peptide Fragments - blood | Protein Array Analysis | T-Lymphocytes - immunology | Proteases | Physiological aspects | Research | Diagnosis | Hepatitis C | Drug therapy | Chemokines | Hepatitis C, Chronic/therapy | T-Lymphocytes/immunology | Polyethylene Glycols/therapeutic use | Dipeptidyl Peptidase 4/blood | Receptors, CXCR3/blood | Chemokine CXCL10/blood | T-Lymphocytes/virology | Hepatitis C, Chronic/blood | Interferon-alpha/therapeutic use | Chemokine CXCL10/antagonists & inhibitors | Life Sciences | Immunology | Antiviral Agents/therapeutic use | Hepatitis C, Chronic/immunology | Peptide Fragments/blood | Ribavirin/therapeutic use | Hepatitis C, Chronic/virology
Journal Article
Nature Immunology, ISSN 1529-2908, 07/2015, Volume 16, Issue 8, pp. 850 - 858
The success of antitumor immune responses depends on the infiltration of solid tumors by effector T cells, a process guided by chemokines. Here we show that in... 
CD26 EXPRESSION | CELLS | CXCL10 | CHEMOKINE ACTIVITY | IN-VIVO | RECEPTOR | IMMUNOLOGY | CD26/DIPEPTIDYL PEPTIDASE-IV | PROTEINS | CANCER | POSTTRANSLATIONAL MODIFICATION | Dipeptidyl Peptidase 4 - metabolism | Immunotherapy - methods | Male | Adoptive Transfer | Dipeptidyl-Peptidase IV Inhibitors - pharmacology | Cell Movement - immunology | Flow Cytometry | Lymphocytes - immunology | Neoplasms, Experimental - immunology | Chemokine CXCL10 - immunology | Neoplasms, Experimental - genetics | Female | Sitagliptin Phosphate | Dipeptidyl Peptidase 4 - immunology | Chemokines - immunology | Lymphocytes - metabolism | Receptors, CXCR3 - metabolism | Mice, Inbred C57BL | Neoplasms, Experimental - therapy | Dipeptidyl Peptidase 4 - genetics | Mice, Transgenic | Mice, Knockout | Triazoles - pharmacology | Cell Movement - drug effects | Animals | Receptors, CXCR3 - immunology | Cell Line, Tumor | Chemokines - metabolism | Mice, Inbred BALB C | Pyrazines - pharmacology | Chemokine CXCL10 - metabolism | Care and treatment | Usage | Immunotherapy | Development and progression | Inflammation | Health aspects | Chemokines | Tumors | Neoplasms, Experimental/genetics | Immunotherapy/methods | Cell Movement/drug effects | Neoplasms, Experimental/therapy | Lymphocytes/metabolism | Lymphocytes/immunology | Dipeptidyl Peptidase 4/genetics | Life Sciences | Chemokine CXCL10/immunology | Immunology | Pyrazines/pharmacology | Dipeptidyl Peptidase 4/immunology | Dipeptidyl-Peptidase IV Inhibitors/pharmacology | Chemokines/metabolism | Receptors, CXCR3/immunology | Receptors, CXCR3/metabolism | Neoplasms, Experimental/immunology | Triazoles/pharmacology | Cell Movement/immunology | Chemokines/immunology | Dipeptidyl Peptidase 4/metabolism | Chemokine CXCL10/metabolism
Journal Article
PLoS ONE, ISSN 1932-6203, 07/2015, Volume 10, Issue 7, p. e0133236
Serum levels of the interferon (IFN)-stimulated chemokine CXCL10 are increased during chronic HCV infection and associate with outcome of IFN-based therapy.... 
VIRUS-INFECTION | CHEMOKINES | MULTIDISCIPLINARY SCIENCES | CHRONIC HEPATITIS-C | PROTEIN-10 | GENOTYPE 1 INFECTION | PREDICTIVE-VALUE | RIBAVIRIN | INDUCTION | ASSOCIATION | SPONTANEOUS CLEARANCE | Recurrence | Interferons | Humans | Fluorenes - therapeutic use | Dipeptidyl Peptidase 4 - blood | Hepatitis C, Chronic - virology | Male | Adult | Female | Hepacivirus - physiology | Drug Therapy, Combination | Benzimidazoles - therapeutic use | Hepacivirus - drug effects | Sofosbuvir - therapeutic use | Virus Replication - drug effects | Chemokine CXCL10 - blood | Antiviral Agents - therapeutic use | Ribavirin - therapeutic use | Dipeptidyl Peptidase 4 - genetics | Treatment Outcome | Hepatitis C, Chronic - drug therapy | Hepatitis C, Chronic - blood | Convalescence | Chemokine CXCL10 - genetics | Protein Processing, Post-Translational | Hepatitis C, Chronic - genetics | Viral Load - drug effects | Infection | Care and treatment | Analysis | Interferon | Biological response modifiers | Hepatitis C virus | Health aspects | Therapy | Laboratories | Chronic infection | Critical care | Amino acids | Infections | Ribavirin | Clinical outcomes | Proteins | Genotype & phenotype | Hepatitis | Immunology | Hepatology | Post-translation | Pretreatment | Drug dosages | Genotypes | Statistical analysis | Liver diseases | Dendritic cells | Serum levels | Studies | Infectious diseases | CXCL10 protein | Biomarkers | Chemokines | Virus Replication/drug effects | Viral Load/drug effects | Dipeptidyl Peptidase 4/blood | Dipeptidyl Peptidase 4/genetics | Chemokine CXCL10/blood | Hepatitis C, Chronic/blood | Life Sciences | Hepatitis C, Chronic/drug therapy | Hepatitis C, Chronic/virology | Hepatitis C, Chronic/genetics | Chemokine CXCL10/genetics | Benzimidazoles/therapeutic use | Hepacivirus/physiology | Fluorenes/therapeutic use | Antiviral Agents/therapeutic use | Sofosbuvir/therapeutic use | Hepacivirus/drug effects | Ribavirin/therapeutic use
Journal Article
Nature, ISSN 0028-0836, 11/2015, Volume 527, Issue 7577, pp. 249 - 253
Epigenetic silencing including histone modifications and DNA methylation is an important tumorigenic mechanism. However, its role in cancer immunopathology and... 
Polycomb Repressive Complex 2 - antagonists & inhibitors | CD8-Positive T-Lymphocytes - cytology | Immunotherapy - methods | Prognosis | Histones - chemistry | Chemokine CXCL9 - biosynthesis | Humans | Ovarian Neoplasms - pathology | DNA (Cytosine-5-)-Methyltransferases - antagonists & inhibitors | Th1 Cells - immunology | DNA (Cytosine-5-)-Methyltransferases - metabolism | Th1 Cells - metabolism | Chemokine CXCL10 - biosynthesis | Chemokine CXCL10 - immunology | Female | Epigenesis, Genetic - drug effects | Lysine - metabolism | Tumor Cells, Cultured | Chemokines - immunology | Tumor Escape - immunology | Chemokines - biosynthesis | DNA (Cytosine-5-)-Methyltransferase 1 | Gene Silencing | Chemokine CXCL9 - immunology | Chemokine CXCL9 - genetics | Chemokines - genetics | Enhancer of Zeste Homolog 2 Protein | Ovarian Neoplasms - enzymology | Xenograft Model Antitumor Assays | B7-H1 Antigen - metabolism | Animals | Chemokine CXCL10 - genetics | Ovarian Neoplasms - therapy | Mice | Histones - metabolism | CD8-Positive T-Lymphocytes - immunology | Polycomb Repressive Complex 2 - metabolism | DNA Methylation - drug effects | Lymphocytes, Tumor-Infiltrating - immunology | Ovarian Neoplasms - immunology | checkpoint | CXCL9 | histone modification | chemotherapy | epigenetics | Chemokine | CXCL10 | DNMT | T cell therapy | DNA methylation | PD-L1 | B7-H1 | cancer | trafficking | PD-1 | EZH2
Journal Article
Clinical & Experimental Immunology, ISSN 0009-9104, 01/2012, Volume 167, Issue 1, pp. 137 - 148
The ready access to commercially available multiplex assays and the importance of inflammation in disease pathogenesis has resulted in an abundance of studies... 
hepatitis C virus | chemokines/monokines | bladder cancer | BCG | Hepatitis C virus | Bladder cancer | Chemokines/monokines | BACILLUS-CALMETTE-GUERIN | DIPEPTIDYL-PEPTIDASE-IV | LIGANDS CXCL9 | MURINE CEREBRAL MALARIA | IFN-GAMMA | CHEMOKINE RECEPTOR CXCR3 | HEPATITIS-C-VIRUS | monokines | IMMUNOLOGY | chemokines | INTERFERON-INDUCIBLE PROTEIN-10 | LEVELS CORRELATE | ADAPTIVE IMMUNE-RESPONSES | Enzyme-Linked Immunosorbent Assay - methods | Dipeptidyl Peptidase 4 - metabolism | Carcinoma, Transitional Cell - urine | Humans | Middle Aged | Culture Media, Conditioned - chemistry | Recombinant Fusion Proteins - analysis | Male | Neoplasm Proteins - urine | Chemokine CXCL10 - analysis | Peptide Fragments - immunology | Chemokine CXCL10 - immunology | Aged, 80 and over | Adult | Female | Antibodies, Monoclonal - immunology | Protein Structure, Tertiary | Urinary Bladder Neoplasms - urine | Body Fluids - chemistry | Protein Isoforms - analysis | Inflammation | Hepatitis C, Chronic - blood | Peptide Fragments - analysis | Immunoenzyme Techniques - methods | Biomarkers | Aged | Protein Processing, Post-Translational | Protein Isoforms - immunology | Universities and colleges | Dendritic cells | Hepatitis C | Atopic dermatitis | Hepatitis | Pathogenesis | Discrimination | Urine | CD26 antigen | biomarkers | Abundance | Inflammatory diseases | Asthma | Tuberculosis | CXCL10 protein | Post-translation | Immunoassays | Injuries | Cancer | Kidney transplantation | Recombinant Fusion Proteins/analysis | Chemokine CXCL10/analysis | Hepatitis C, Chronic/blood | Culture Media, Conditioned/chemistry | Carcinoma, Transitional Cell/urine | Life Sciences | Chemokine CXCL10/immunology | Antibodies, Monoclonal/immunology | Immunology | Neoplasm Proteins/urine | Protein Isoforms/immunology | Urinary Bladder Neoplasms/urine | Body Fluids/chemistry | Peptide Fragments/immunology | Protein Isoforms/analysis | Enzyme-Linked Immunosorbent Assay/methods | Dipeptidyl Peptidase 4/metabolism | Immunoenzyme Techniques/methods | Peptide Fragments/analysis | Translational Studies
Journal Article
Arteriosclerosis, Thrombosis, and Vascular Biology, ISSN 1079-5642, 03/2014, Volume 34, Issue 3, pp. 594 - 602
OBJECTIVE—In arteriogenesis, pre-existing anastomoses undergo enlargement to restore blood flow in ischemic tissues. Chemokine (C-X-C motif) ligand 10 (CXCL10)... 
Cell movement | Inflammation | interferon-γ-inducible protein of 10 kDa | Myocytes, smooth muscle | Chemokine CXCL10 | chemokine CXCL10 | ANGIOGENESIS | smooth muscle | MECHANISMS | myocytes | COLLATERAL GROWTH | ARTERIOGENESIS | INTERFERON-INDUCIBLE PROTEIN-10 | cell movement | inflammation | MARROW-DERIVED CELLS | PERIPHERAL VASCULAR DISEASE | HEMATOLOGY | interferon--inducible protein of 10 kDa | EXPRESSION | PROMOTES | T-LYMPHOCYTES | RECEPTOR-3 | Human Umbilical Vein Endothelial Cells | Humans | Chemokine CXCL10 - physiology | Male | Muscle, Skeletal - metabolism | Chemokine CXCL10 - pharmacology | Ligation | RNA Interference | Bone Marrow - metabolism | Female | Femoral Artery | Myocytes, Smooth Muscle - metabolism | Muscle, Skeletal - blood supply | Chemokine CXCL10 - blood | Collateral Circulation - physiology | Chemokine CXCL10 - deficiency | Mice, Inbred C57BL | RNA, Small Interfering - pharmacology | Cells, Cultured | Chemokine CXCL10 - antagonists & inhibitors | Radiation Chimera | Ischemia - physiopathology | Laser-Doppler Flowmetry | Recombinant Proteins - pharmacology | Mice, Knockout | Neovascularization, Physiologic - physiology | Muscle, Smooth, Vascular - pathology | Animals | Chemokine CXCL10 - genetics | Hindlimb - blood supply | Reperfusion Injury - physiopathology | Mice | Aorta - cytology
Journal Article
Journal of Leukocyte Biology, ISSN 0741-5400, 12/2014, Volume 96, Issue 6, pp. 1055 - 1063
IP‐10 can inhibit T cell responses in HIV‐1 infected subjects on ART. HIV‐1‐infected subjects, despite control of viral replication with ART, have an altered... 
CXCL10 | infection | chemokines | antiretroviral therapy | immune responses | Infection | Immune responses | Antiretroviral therapy | Chemokines | INDUCIBLE PROTEIN-10 | DOWN-REGULATION | ANTAGONISM | CYTOKINE | IMMUNOLOGY | CELL BIOLOGY | HIV-INFECTION | INFLAMMATION | CXCR3 | CHEMOATTRACTANT | HEMATOLOGY | EXPRESSION | HIV-1 | Leukocytes, Mononuclear - metabolism | Phosphorylation | Humans | Middle Aged | Chemokine CXCL10 - physiology | Young Adult | HIV Infections - immunology | Antibodies, Neutralizing - immunology | Chemokine CXCL10 - pharmacology | T-Lymphocytes - drug effects | Leukocytes, Mononuclear - immunology | Chemokine CXCL10 - immunology | Adult | Anti-HIV Agents - therapeutic use | p38 Mitogen-Activated Protein Kinases - metabolism | HLA Antigens - biosynthesis | Cytokines - blood | HIV Infections - blood | Chemokine CXCL10 - blood | Receptors, CXCR3 - metabolism | Interferon-gamma Release Tests | Lymphocyte Activation | Cells, Cultured | Chemokine CXCL10 - antagonists & inhibitors | Antibodies, Neutralizing - pharmacology | Recombinant Proteins - pharmacology | Calcium Signaling - drug effects | Antigens - immunology | HIV Infections - drug therapy | T-Lymphocytes - immunology | Protein Processing, Post-Translational | Cytokines - biosynthesis | Cytotoxicity, Immunologic | Inflammation, Extracellular Mediators, & Effector Molecules
Journal Article
PLoS ONE, ISSN 1932-6203, 03/2011, Volume 6, Issue 2, p. e17232
textabstractBackground: High baseline levels of IP-10 predict a slower first phase decline in HCV RNA and a poor outcome following interferon/ribavirin therapy... 
GENETIC-VARIATION | IL28B | CLEARANCE | PLUS RIBAVIRIN | VIRUS-INFECTION | THERAPY | PEGINTERFERON ALPHA-2A | PROTEIN-10 | BIOLOGY | VIRAL RESPONSE | ASSOCIATION | Prognosis | Humans | Middle Aged | Polymorphism, Single Nucleotide - physiology | Male | Recombinant Proteins | Interleukins - analysis | Interleukins - metabolism | Interleukins - genetics | Chemokine CXCL10 - analysis | RNA, Viral - genetics | Adult | Female | Biomarkers, Pharmacological - analysis | Pharmacogenetics | Drug Administration Schedule | Antiviral Agents - therapeutic use | Interferon-alpha - therapeutic use | RNA, Viral - analysis | Hepatitis C, Chronic - diagnosis | Hepatitis C, Chronic - drug therapy | Antiviral Agents - administration & dosage | Interferon-alpha - administration & dosage | Chemokine CXCL10 - genetics | Interferon alpha-2 | Hepatitis C, Chronic - genetics | Biomarkers, Pharmacological - metabolism | Chemokine CXCL10 - metabolism | Medical research | Care and treatment | RNA | Infection | Analysis | Medicine, Experimental | Genetic aspects | Interferon | Single nucleotide polymorphisms | Hepatitis C virus | Hepatitis C | Chromosomes | Health aspects | Therapy | Liver | Infections | Genomes | Single-nucleotide polymorphism | Ribonucleic acid--RNA | Patients | Ribavirin | Virology | Carriers | Hepatitis | Ethics | Infectious diseases | Hospitals | Correlation analysis | Gastroenterology | Predictions | IP-10 protein | Pretreatment | Genotypes | Pharmacological | Viral | Mikrobiologi inom det medicinska området | Single Nucleotide | genetics | administration & dosage | Interleukins | drug therapy | Interferon-alpha | physiology | diagnosis | Antiviral Agents | analysis | Chemokine CXCL10 | Chronic | Microbiology in the medical area | Biomarkers | metabolism | therapeutic use | Polymorphism | Ribonucleic acid
Journal Article
Hepatology, ISSN 0270-9139, 08/2014, Volume 60, Issue 2, pp. 487 - 496
The pathogenesis of hepatitis C virus (HCV) infection is strongly influenced by the nature of the host's antiviral immunity. Counterintuitively, elevated serum... 
NATURAL-KILLER-CELLS | INJECTION-DRUG USERS | INTERFERON | VIRUS-INFECTION | NK CELLS | APPARENT RESISTANCE | PROTEIN-10 | REACTIVITY | RIBAVIRIN | GASTROENTEROLOGY & HEPATOLOGY | HCV INFECTION | Dipeptidyl Peptidase 4 - metabolism | Prospective Studies | Follow-Up Studies | Humans | Middle Aged | Hepatitis C, Chronic - virology | Male | Interferon-gamma - metabolism | T-Lymphocytes - virology | Young Adult | T-Lymphocytes - metabolism | Chemokine CXCL10 - immunology | Interleukin-10 - metabolism | Killer Cells, Natural - immunology | Adult | Female | Adaptive Immunity - immunology | Dipeptidyl Peptidase 4 - immunology | Chemokine CXCL10 - blood | Killer Cells, Natural - virology | Immunity, Innate - immunology | Interferon-gamma - immunology | Hepatitis C, Chronic - immunology | T-Lymphocytes - immunology | Killer Cells, Natural - metabolism | Interleukin-10 - immunology | Longitudinal Studies | Hepatitis | Infections | Drug therapy | Hepatology | Chemokines | Immune system | Killer Cells, Natural/immunology | Interferon-gamma/immunology | Chemokine CXCL10/blood | Life Sciences | Chemokine CXCL10/immunology | Immunology | Hepatitis C, Chronic/immunology | Interleukin-10/immunology | T-Lymphocytes/metabolism | Dipeptidyl Peptidase 4/immunology | Hepatitis C, Chronic/virology | Immunity, Innate/immunology | Adaptive Immunity/immunology | T-Lymphocytes/immunology | Interferon-gamma/metabolism | T-Lymphocytes/virology | Killer Cells, Natural/metabolism | Killer Cells, Natural/virology | Dipeptidyl Peptidase 4/metabolism | Interleukin-10/metabolism
Journal Article