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Clinical & Experimental Immunology, ISSN 0009-9104, 01/2012, Volume 167, Issue 1, pp. 137 - 148
The ready access to commercially available multiplex assays and the importance of inflammation in disease pathogenesis has resulted in an abundance of studies... 
hepatitis C virus | chemokines/monokines | bladder cancer | BCG | Hepatitis C virus | Bladder cancer | Chemokines/monokines | BACILLUS-CALMETTE-GUERIN | DIPEPTIDYL-PEPTIDASE-IV | LIGANDS CXCL9 | MURINE CEREBRAL MALARIA | IFN-GAMMA | CHEMOKINE RECEPTOR CXCR3 | HEPATITIS-C-VIRUS | monokines | IMMUNOLOGY | chemokines | INTERFERON-INDUCIBLE PROTEIN-10 | LEVELS CORRELATE | ADAPTIVE IMMUNE-RESPONSES | Enzyme-Linked Immunosorbent Assay - methods | Dipeptidyl Peptidase 4 - metabolism | Carcinoma, Transitional Cell - urine | Humans | Middle Aged | Culture Media, Conditioned - chemistry | Recombinant Fusion Proteins - analysis | Male | Neoplasm Proteins - urine | Chemokine CXCL10 - analysis | Peptide Fragments - immunology | Chemokine CXCL10 - immunology | Aged, 80 and over | Adult | Female | Antibodies, Monoclonal - immunology | Protein Structure, Tertiary | Urinary Bladder Neoplasms - urine | Body Fluids - chemistry | Protein Isoforms - analysis | Inflammation | Hepatitis C, Chronic - blood | Peptide Fragments - analysis | Immunoenzyme Techniques - methods | Biomarkers | Aged | Protein Processing, Post-Translational | Protein Isoforms - immunology | Universities and colleges | Dendritic cells | Hepatitis C | Atopic dermatitis | Hepatitis | Pathogenesis | Discrimination | Urine | CD26 antigen | biomarkers | Abundance | Inflammatory diseases | Asthma | Tuberculosis | CXCL10 protein | Post-translation | Immunoassays | Injuries | Cancer | Kidney transplantation | Recombinant Fusion Proteins/analysis | Chemokine CXCL10/analysis | Hepatitis C, Chronic/blood | Culture Media, Conditioned/chemistry | Carcinoma, Transitional Cell/urine | Life Sciences | Chemokine CXCL10/immunology | Antibodies, Monoclonal/immunology | Immunology | Neoplasm Proteins/urine | Protein Isoforms/immunology | Urinary Bladder Neoplasms/urine | Body Fluids/chemistry | Peptide Fragments/immunology | Protein Isoforms/analysis | Enzyme-Linked Immunosorbent Assay/methods | Dipeptidyl Peptidase 4/metabolism | Immunoenzyme Techniques/methods | Peptide Fragments/analysis | Translational Studies
Journal Article
Oncogene, ISSN 0950-9232, 03/2013, Volume 32, Issue 10, pp. 1223 - 1232
Kaposi's sarcoma-associated herpesvirus (KSHV) K13/vFLIP (viral Flice-inhibitory protein) induces transcription of numerous genes through NF-kappa B... 
AIDS | endothelial cells | KSHV | Kaposi's sarcoma | NF-kB | vFLIP/K13 | KSHV VFLIP | IKK-GAMMA | ENZYME A20 | BIOCHEMISTRY & MOLECULAR BIOLOGY | RECEPTOR | DEPENDENT GENE-EXPRESSION | A20 INTERACTS | CELL BIOLOGY | KINASE COMPLEX | UBIQUITIN CHAINS | NF-kappa B | ONCOLOGY | ENDOTHELIAL-CELLS | GENETICS & HEREDITY | ZINC-FINGER PROTEIN | Sarcoma, Kaposi - metabolism | Humans | NF-kappa B - metabolism | Intracellular Signaling Peptides and Proteins - metabolism | Viral Proteins - metabolism | DNA-Binding Proteins - metabolism | Transfection | Herpesvirus 8, Human - metabolism | Nuclear Proteins - biosynthesis | Tumor Necrosis Factor alpha-Induced Protein 3 | Nuclear Proteins - genetics | Intracellular Signaling Peptides and Proteins - genetics | NF-kappa B - antagonists & inhibitors | Transduction, Genetic | Endothelial Cells - metabolism | Viral Proteins - genetics | Herpesvirus 8, Human - genetics | Nuclear Proteins - metabolism | Viral Proteins - antagonists & inhibitors | Sarcoma, Kaposi - virology | DNA-Binding Proteins - genetics | Intracellular Signaling Peptides and Proteins - biosynthesis | Sarcoma, Kaposi - pathology | Proteins - genetics | Animals | Proteins - metabolism | NF-kappa B - genetics | Endothelial Cells - cytology | Chemokine CXCL10 - secretion | Mice | Chemokine CXCL10 - metabolism | DNA-Binding Proteins - biosynthesis | Sarcoma, Kaposi - genetics | Cell proliferation | Cytokines | Transcription | Inflammation | regulatory proteins | IKK1 protein | Endothelial cells | FLICE-inhibitory protein | NF- Kappa B protein | Transcription activation | IP-10 protein | Chemokines | Tumors
Journal Article
Journal Article
Journal Article
The Journal of infectious diseases, ISSN 0022-1899, 01/2017, Volume 215, Issue 1, pp. 42 - 51
Journal Article
Journal Article
Journal of Allergy and Clinical Immunology, The, ISSN 0091-6749, 2007, Volume 120, Issue 3, pp. 586 - 593
Background Rhinovirus-induced acute asthma is the most frequent trigger for asthma exacerbations. Objective We assessed which inflammatory mediators were... 
Allergy and Immunology | airway inflammation | rhinovirus | Asthma | BRONCHIAL EPITHELIAL-CELLS | asthma | RANDOMIZED CONTROLLED-TRIAL | IMMUNOLOGY | VIRAL-INFECTIONS | IN-VITRO | ALLERGY | INFLAMMATION | CHEMOKINE RECEPTOR | AIRWAY SMOOTH-MUSCLE | EXPRESSION | T-CELLS | VIRUS-INDUCED ASTHMA
Journal Article
Cell Death and Differentiation, ISSN 1350-9047, 11/2012, Volume 19, Issue 11, pp. 1836 - 1846
Induction of the C/EBP homologous protein (CHOP) is considered a key event for endoplasmic reticulum (ER) stress-mediated apoptosis. Type 1 diabetes (T1D) is... 
ER stress | apoptosis | CHOP | Bcl-2 proteins | inflammation | pancreatic b-cell | OXIDATIVE STRESS | BIOCHEMISTRY & MOLECULAR BIOLOGY | DOWN-REGULATION | NECROSIS-FACTOR-ALPHA | DEATH | ENDOPLASMIC-RETICULUM STRESS | INDUCTION | CELL BIOLOGY | pancreatic beta-cell | INSULIN-PRODUCING CELLS | GENE-EXPRESSION | NF-KAPPA-B | POSTTRANSLATIONAL MODIFICATION | Transcription Factor CHOP - genetics | Interleukin-1beta - pharmacology | Caspase 9 - metabolism | Apoptosis - drug effects | Caspase 3 - metabolism | Diabetes Mellitus, Type 1 - metabolism | NF-kappa B - metabolism | Proto-Oncogene Proteins c-bcl-2 - metabolism | Insulin-Secreting Cells - metabolism | Cell Nucleus - metabolism | I-kappa B Kinase - metabolism | Insulin-Secreting Cells - cytology | Cell Line | Diabetes Mellitus, Type 1 - pathology | Rats | Transcription Factor CHOP - antagonists & inhibitors | Mitochondria - metabolism | Tumor Necrosis Factor-alpha - pharmacology | Animals | Transcription Factor RelA - metabolism | Endoplasmic Reticulum Stress | Myeloid Cell Leukemia Sequence 1 Protein | Transcription Factor CHOP - metabolism | Cytokines - pharmacology | Interferon-gamma - pharmacology | Insulitis | Transcription factors | Fas antigen | Bcl-2 protein | Leukocytes (mononuclear) | CCAAT/enhancer-binding protein | Mitochondria | Pancreas | Cytokines | CD95 antigen | Diabetes mellitus | Data processing | Inflammation | Interleukin 15 | Mcl-1 protein | Nitric-oxide synthase | Nuclear transport | Stress | Beta cells | Interferon regulatory factor 7 | NF- Kappa B protein | CXCL10 protein | Caspase-9 | Islets of Langerhans | Endoplasmic reticulum | Chemokines | Apoptosis | B-cell lymphoma | Original Paper | pancreatic β-cell | Naturvetenskap | Natural Sciences
Journal Article
Journal of Investigative Dermatology, ISSN 0022-202X, 02/2007, Volume 127, Issue 2, pp. 331 - 341
Keratinocytes are continuously in contact with external stimuli and have the capacity to produce several soluble mediators. Pathogen-associated molecular... 
TOLL-LIKE-RECEPTORS | DOUBLE-STRANDED-RNA | DENDRITIC CELLS | SKIN-ASSOCIATED CHEMOKINE | MONOCYTE CHEMOATTRACTANT PROTEIN-1 | INNATE IMMUNE-RESPONSE | INFLAMMATORY PROTEIN 3-ALPHA | NECROSIS-FACTOR-ALPHA | FACTOR-KAPPA-B | HUMAN EPIDERMAL-KERATINOCYTES | DERMATOLOGY | Oligonucleotides - genetics | Phosphorylation | Toll-Like Receptor 5 - genetics | Humans | Toll-Like Receptor 9 - genetics | I-kappa B Proteins - metabolism | RNA, Messenger - metabolism | Flagellin - pharmacology | Tissue Distribution | Cell Nucleus - metabolism | Protein Isoforms - metabolism | Biological Transport | Oligonucleotides - pharmacology | Toll-Like Receptor 3 - genetics | Toll-Like Receptors - metabolism | Chemokine CXCL9 | Toll-Like Receptor 5 - metabolism | Poly I-C - pharmacology | Cytokines - metabolism | Cells, Cultured | Toll-Like Receptor 4 - genetics | Toll-Like Receptor 3 - metabolism | Toll-Like Receptor 4 - metabolism | Chemokine CXCL10 | Transcription Factor RelA - metabolism | Keratinocytes - metabolism | Toll-Like Receptors - genetics | Chemokines, CXC - metabolism | Lipopolysaccharides - pharmacology | CpG Islands | Ligands | Chemokines - metabolism | Toll-Like Receptor 9 - metabolism | CD40 antigen | Oligonucleotides | Flagellin | mRNA | intercellular adhesion molecule 1 | Immunity | Lipopolysaccharides | CXC chemokines | CCL20 protein | Toll-like receptors | CpG islands | TLR7 protein | Dermatology | Keratinocytes | Nuclear transport | Stress | TLR3 protein | TLR9 protein | TLR1 protein | External stimuli | CXCL10 protein | Histocompatibility antigen HLA | Interferon | Internet | Monocyte chemoattractant protein 1 | Tumor necrosis factor- alpha
Journal Article
BMC Microbiology, ISSN 1471-2180, 08/2017, Volume 17, Issue 1, pp. 187 - 7
Background: The bronchial epithelium serves as the first defendant line of host against respiratory inhaled pathogens, mainly through releasing chemokines... 
PepO | BEAS-2B | Il-8 | IP-10 | IMMUNE-RESPONSE | DEFENSE | INNATE | NEUTROPHILS | MICROBIOLOGY | FACTOR-KAPPA-B | COLONIZATION | PNEUMOLYSIN | INTERLEUKIN-8 | RESISTANCE | PNEUMOCOCCAL PNEUMONIA | Interleukin-8 - genetics | Phosphorylation | Epithelial Cells - metabolism | Epithelial Cells - drug effects | Humans | eIF-2 Kinase - drug effects | Phosphatidylinositol 3-Kinases | Streptococcus pneumoniae - pathogenicity | Time Factors | Streptococcus pneumoniae - metabolism | Bronchi - metabolism | Transcription, Genetic | p38 Mitogen-Activated Protein Kinases - metabolism | Interleukin-8 - metabolism | Recombinant Proteins - metabolism | Cell Line | Cytokines - metabolism | Metalloendopeptidases - administration & dosage | Toll-Like Receptor 2 - metabolism | Toll-Like Receptor 4 - metabolism | Virulence Factors | Bacterial Proteins - administration & dosage | Gene Expression Regulation, Enzymologic | Bacterial Proteins - pharmacology | Signal Transduction - drug effects | Chemokine CXCL10 - genetics | Metalloendopeptidases - pharmacology | NF-KappaB Inhibitor alpha - metabolism | Mitogen-Activated Protein Kinase Kinases - drug effects | Chemokine CXCL10 - metabolism | Polysaccharides | Pneumonia | Interleukins | Bacterial pneumonia | B cells | Biological response modifiers | Streptococcus pneumoniae | Cell culture | Pathogenesis | Epithelial cells | Virulence | Interleukin | AKT protein | Kinases | Recruitment | Streptococcus infections | Proteins | Virulence factors | Lymphocytes | Toll-like receptors | IP-10 protein | Interleukin 8 | Surface protein A | Saccharides | Enzymes | Pathogens | Cytokines | Incubation | Dendritic cells | Peptidoglycans | Neutrophils | MAP kinase | Pharmacology | Inflammation | Protein A | PspA protein | TLR4 protein | Epithelium | Chromatography | 1-Phosphatidylinositol 3-kinase | Studies | Pneumolysin | Streptococcus | Inhibitors | TLR2 protein | Endopeptidase | Ligands | Interferon | Chemokines
Journal Article