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1. Full Text A novel CXCR3-B Chemokine Receptor-induced Growth-inhibitory signal in cancer cells is Mediated through the Regulation of Bach-1 Protein and Nrf2 Protein Nuclear Translocation
by Balan, Murugabaskar and Pal, Soumitro
Journal of Biological Chemistry, ISSN 0021-9258, 02/2014, Volume 289, Issue 6, pp. 3126 - 3137
Background: The chemokine receptor CXCR3-B initiates inhibitory signals. Results: CXCR3-B-mediated signal induced apoptosis and inhibited autophagy of breast... 
Signal Transduction | SPLICE VARIANT | BIOCHEMISTRY & MOLECULAR BIOLOGY | I-TAC | Heme Oxygenase | HEME OXYGENASE-1 GENE | CXC CHEMOKINES | HUMAN BREAST-CANCER | Signaling | MURINE MODEL | EPITHELIAL-CELLS | Nrf2 | Nuclear Transport | TRANSCRIPTION FACTOR | EXPRESSION | Chemokines | Apoptosis | Basic-Leucine Zipper Transcription Factors - metabolism | Microtubule-Associated Proteins - genetics | Microtubule-Associated Proteins - metabolism | Humans | bcl-X Protein - genetics | Apoptosis - genetics | Breast Neoplasms - metabolism | MAP Kinase Signaling System | Heme Oxygenase-1 - genetics | Fanconi Anemia Complementation Group Proteins - metabolism | Cell Nucleus - metabolism | Protein Isoforms - metabolism | Cell Nucleus - pathology | Mitogen-Activated Protein Kinase 1 - genetics | Apoptosis Regulatory Proteins - genetics | Female | NF-E2-Related Factor 2 - genetics | Membrane Proteins - metabolism | p38 Mitogen-Activated Protein Kinases - metabolism | Active Transport, Cell Nucleus - genetics | Beclin-1 | Receptors, CXCR3 - metabolism | Heme Oxygenase-1 - biosynthesis | Mitogen-Activated Protein Kinase 3 - genetics | Membrane Proteins - genetics | Alternative Splicing - genetics | Fanconi Anemia Complementation Group Proteins - genetics | p38 Mitogen-Activated Protein Kinases - genetics | Basic-Leucine Zipper Transcription Factors - genetics | Apoptosis Regulatory Proteins - metabolism | Breast Neoplasms - genetics | Cell Nucleus - genetics | Mitogen-Activated Protein Kinase 3 - metabolism | Breast Neoplasms - pathology | NF-E2-Related Factor 2 - metabolism | Receptors, CXCR3 - genetics | Cell Line, Tumor | bcl-X Protein - metabolism | Mitogen-Activated Protein Kinase 1 - metabolism | Protein Isoforms - genetics
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2. Full Text Codelivery of envelope protein in alum with MVA vaccine induces CXCR3- biased CXCR5 + and CXCR5 - CD4 T cell responses in rhesus macaques
by Iyer, Smita S and Gangadhara, Sailaja and Victor, Blandine and Gomez, Rosy and Basu, Rahul and Hong, Jung Joo and Labranche, Celia and Montefiori, David C and Villinger, Francois and Moss, Bernard and Amara, Rama Rao
Journal of Immunology, ISSN 0022-1767, 08/2015, Volume 195, Issue 3, pp. 994 - 1005
The goal of an HIV vaccine is to generate robust and durable protective Ab. Vital to this goal is the induction of CD4(+) T follicular helper (T-FH) cells.... 
B-CELLS | ACUTE VIRAL-INFECTION | IMMUNIZATION | PLASMABLAST RESPONSES | VIRUS | IMMUNOLOGY | MONOCLONAL-ANTIBODIES | FOLLICULAR HELPER | MUCOSAL CHALLENGE | SIV INFECTION | EXPRESSION | Adjuvants, Immunologic - administration & dosage | Receptors, CXCR3 - biosynthesis | Male | Macaca mulatta | Vaccination - veterinary | Lymph Nodes - immunology | Lymph Nodes - cytology | Antibodies, Viral - blood | Viral Load - immunology | Inducible T-Cell Co-Stimulator Protein - biosynthesis | Receptors, CCR5 - biosynthesis | Glycoproteins - immunology | Simian Acquired Immunodeficiency Syndrome - virology | Animals | SAIDS Vaccines - immunology | Simian Acquired Immunodeficiency Syndrome - immunology | Viremia - virology | T-Lymphocytes, Helper-Inducer - immunology | Programmed Cell Death 1 Receptor - biosynthesis | Receptors, CXCR5 - biosynthesis | Viral Envelope Proteins - immunology | Viremia - immunology | Alum Compounds - administration & dosage | Viral Vaccines - immunology
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3. Full Text Identification of Potential Key lncRNAs and Genes Associated with Aging Based on Microarray Data of Adipocytes from Mice
by Yang, Yi and Teng, Zongyan and Meng, Songyan and Yu, Weigang
BioMed Research International, ISSN 2314-6133, 2016, Volume 2016, pp. 9181702 - 11
Objective. This study aimed to screen potential crucial lncRNAs and genes involved in aging. Methods. The data of 9 peripheral white adipocytes, respectively,... 
DENSITY | MEDICINE, RESEARCH & EXPERIMENTAL | PROTEIN | EVOLUTION | LONG NONCODING RNAS | MESSENGER-RNA | METABOLISM | BIOTECHNOLOGY & APPLIED MICROBIOLOGY | TISSUE | SENESCENCE | BONE | EXPRESSION | Gene Regulatory Networks - genetics | Oligonucleotide Array Sequence Analysis | Humans | RNA, Messenger - genetics | Gene Expression Profiling | RNA, Long Noncoding - genetics | Transcriptome - genetics | Adipocytes - pathology | RNA, Long Noncoding - isolation & purification | Aging - pathology | RNA, Messenger - biosynthesis | Animals | Aging - genetics | Adipocytes - metabolism | RNA, Long Noncoding - biosynthesis | Mice | Cluster Analysis | RNA sequencing | Adipose tissues | Aging | Antisense RNA | Genetic aspects | Health aspects | Methods | Cluster analysis | Senescence | Time series | Biosynthesis | Adipocytes | Metabolism | Gene expression | Cholesterol | Studies | Proteins | Algorithms | Protein synthesis | Bone marrow | Age
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4. Full Text Elevated interferon‐γ–induced protein 10 and its receptor CXCR3 impair NK cell function during HIV infection
by Wang, Zhuo and Wu, Tong and Ma, Meichen and Zhang, Zining and Fu, Yajing and Liu, Jing and Xu, Junjie and Ding, Haibo and Han, Xiaoxu and Chu, Zhenxing and Wu, Yuntao and Shang, Hong and Jiang, Yongjun
Journal of Leukocyte Biology, ISSN 0741-5400, 07/2017, Volume 102, Issue 1, pp. 163 - 170
IP‐10 negatively regulates NK cell function during HIV infection, making its blockage a potential novel immunotherapy target to increase NK‐mediated clearance... 
IP‐10 | natural killer cell | chemokine receptor | dysfunction | CYTOKINE | IMMUNOLOGY | IP-10 | CELL BIOLOGY | IFN | EPITHELIAL-CELLS | MESSENGER-RNA | CXCL10/IP-10 | PLASMA IP-10 | IP-10 EXPRESSION | DISEASE | PULMONARY TUBERCULOSIS | HEPATITIS-B | HEMATOLOGY | HIV Infections - immunology | HIV-1 - immunology | HIV Infections - pathology | Humans | Chemokine CXCL10 - immunology | K562 Cells | Receptors, CXCR3 - immunology | HIV Infections - therapy | Killer Cells, Natural - immunology | Female | Male | Killer Cells, Natural - pathology | Immune clearance | Recovery of function | Viruses | Infections | Inflammation | CXCR3 protein | Blockage | Hepatitis | Tuberculosis | Immunotherapy | Pancreatic cancer | Human immunodeficiency virus--HIV | γ-Interferon | IP-10 protein | Tumorigenesis | Interferon | Natural killer cells | Hepatitis B virus | Hepatitis B | Cancer | Immune system
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5. Full Text Discrimination of agonist and antagonist forms of CXCL10 in biological samples
by Casrouge, A and Bisiaux, A and Stephen, L and Schmolz, M and Mapes, J and Pfister, C and Pol, S and Mallet, V and Albert, M. L
Clinical & Experimental Immunology, ISSN 0009-9104, 01/2012, Volume 167, Issue 1, pp. 137 - 148
Summary The ready access to commercially available multiplex assays and the importance of inflammation in disease pathogenesis has resulted in an abundance of... 
hepatitis C virus | chemokines/monokines | bladder cancer | BCG | Hepatitis C virus | Bladder cancer | Chemokines/monokines | BACILLUS-CALMETTE-GUERIN | DIPEPTIDYL-PEPTIDASE-IV | LIGANDS CXCL9 | MURINE CEREBRAL MALARIA | IFN-GAMMA | CHEMOKINE RECEPTOR CXCR3 | HEPATITIS-C-VIRUS | monokines | IMMUNOLOGY | chemokines | INTERFERON-INDUCIBLE PROTEIN-10 | LEVELS CORRELATE | ADAPTIVE IMMUNE-RESPONSES | Enzyme-Linked Immunosorbent Assay - methods | Dipeptidyl Peptidase 4 - metabolism | Carcinoma, Transitional Cell - urine | Humans | Middle Aged | Culture Media, Conditioned - chemistry | Recombinant Fusion Proteins - analysis | Male | Neoplasm Proteins - urine | Chemokine CXCL10 - analysis | Peptide Fragments - immunology | Chemokine CXCL10 - immunology | Aged, 80 and over | Adult | Female | Antibodies, Monoclonal - immunology | Protein Structure, Tertiary | Urinary Bladder Neoplasms - urine | Body Fluids - chemistry | Protein Isoforms - analysis | Inflammation | Hepatitis C, Chronic - blood | Peptide Fragments - analysis | Immunoenzyme Techniques - methods | Biomarkers | Aged | Protein Processing, Post-Translational | Protein Isoforms - immunology | Universities and colleges | Dendritic cells | Hepatitis C | Atopic dermatitis | Hepatitis | Pathogenesis | Discrimination | Urine | CD26 antigen | biomarkers | Abundance | Inflammatory diseases | Asthma | Tuberculosis | CXCL10 protein | Post-translation | Immunoassays | Injuries | Cancer | Kidney transplantation | Recombinant Fusion Proteins/analysis | Chemokine CXCL10/analysis | Hepatitis C, Chronic/blood | Culture Media, Conditioned/chemistry | Carcinoma, Transitional Cell/urine | Life Sciences | Chemokine CXCL10/immunology | Antibodies, Monoclonal/immunology | Immunology | Neoplasm Proteins/urine | Protein Isoforms/immunology | Urinary Bladder Neoplasms/urine | Body Fluids/chemistry | Peptide Fragments/immunology | Protein Isoforms/analysis | Enzyme-Linked Immunosorbent Assay/methods | Dipeptidyl Peptidase 4/metabolism | Immunoenzyme Techniques/methods | Peptide Fragments/analysis | Translational Studies
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6. Full Text Chemokines as the modulators of endometrial epithelial cells remodelling
by Zlotkowska, A and Andronowska, A
SCIENTIFIC REPORTS, ISSN 2045-2322, 09/2019, Volume 9, Issue 1, pp. 1 - 9
Previous studies highlighted chemokines as potential factors regulating changes in the endometrium during early pregnancy. The current study aimed to screen... 
EXTRACELLULAR-MATRIX PROTEINS | MATERNAL-FETAL INTERFACE | PORCINE ENDOMETRIUM | IMMUNE CELLS | EARLY-PREGNANCY | IMPLANTATION | INTEGRINS | MULTIDISCIPLINARY SCIENCES | TROPHOBLAST MIGRATION | RECEPTORS | EXPRESSION | Cell proliferation | CC chemokine receptors | CCR5 protein | CXCL12 protein | Motility | CXCR2 protein | Epithelial cells | Implantation | Leukocytes (neutrophilic) | CXCR3 protein | Gene expression | CCL4 protein | CXCR4 protein | Pregnancy | CCR1 protein | CCR2 protein | CXCL10 protein | Cell adhesion | Chemokines | Monocyte chemoattractant protein 1 | Cell migration | Endometrium
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7. Full Text AB1154 Increase generation but defects of secreting ifn-[ALPHA] play a role in the pathogenesis of igg4-rd
by P. Zhang and W. Zhang
Annals of the Rheumatic Diseases, ISSN 0003-4967, 06/2018, Volume 77, p. 1681
Background IgG4 related disease (IgG4-RD) is a multi-organ involvement, fibro-inflammatory disease of unknown etiology. Both innate and adaptive immunity... 
Cell culture | CC chemokine receptors | Flow cytometry | CD86 antigen | Pathogenesis | Immunoglobulin G | CX3CR1 protein | Infections | Cell surface | Defects | CCR1 protein | α-Interferon | CCR2 protein | Etiology | Antigen presentation | CCR5 protein | Dendritic cells | CXCR2 protein | CCR7 protein | CD80 antigen | Adaptive immunity | CXCR3 protein | Patients | CCR6 protein | CD83 antigen | CCR8 protein | CCR9 protein | Surface markers | Monocyte chemoattractant protein 1
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8. Full Text Induction of the CXC Chemokine Interferon-γ–Inducible Protein 10 Regulates the Reparative Response Following Myocardial Infarction
by Bujak, Marcin and Dobaczewski, Marcin and Gonzalez-Quesada, Carlos and Xia, Ying and Leucker, Thorsten and Zymek, Pawel and Veeranna, Vikas and Tager, Andrew M and Luster, Andrew D and Frangogiannis, Nikolaos G
Circulation Research, ISSN 0009-7330, 11/2009, Volume 105, Issue 10, pp. 973 - 983
RATIONALE:Interferon-γ–inducible protein (IP)-10/CXCL10, an angiostatic and antifibrotic chemokine with an important role in T-cell trafficking, is markedly... 
Infarction | Wound healing | Remodeling | Chemokines | Fibrosis | remodeling | wound healing | CARDIAC & CARDIOVASCULAR SYSTEMS | ANGIOGENESIS | infarction | RECEPTOR | REPERFUSION | ISCHEMIA | chemokines | EFFECTOR | PERIPHERAL VASCULAR DISEASE | fibrosis | IP-10 CHEMOKINE | MICE | INHIBITOR | HEMATOLOGY | EXPRESSION | PULMONARY-FIBROSIS | Myocardial Infarction - genetics | Cell Proliferation | Leukocyte Common Antigens - metabolism | Actins - metabolism | Galectin 3 - metabolism | Actins - genetics | Chemokine CXCL10 - biosynthesis | T-Lymphocytes - metabolism | Myocardial Infarction - pathology | T-Lymphocytes - pathology | Fibroblasts - metabolism | Receptors, CXCR3 - metabolism | Macrophages - pathology | Signal Transduction | Gene Expression Regulation - genetics | Myocardial Infarction - metabolism | Galectin 3 - genetics | Mice, Knockout | Regeneration | Macrophages - metabolism | Animals | Chemokine CXCL10 - genetics | Receptors, CXCR3 - genetics | Mice | Cell Movement
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9. Full Text Loss of epidermal AP1 transcription factor function reduces filaggrin level, alters chemokine expression and produces an ichthyosis-related phenotype
by Young, Christina A and Rorke, Ellen A and Adhikary, Gautam and Xu, Wen and Eckert, Richard L
Cell death & disease, ISSN 2041-4889, 06/2017, Volume 8, Issue 6, pp. e2840 - e2840
AP1 transcription factors are important controllers of epidermal differentiation. Multiple family members are expressed in the epidermis in a... 
ATOPIC-DERMATITIS | LORICRIN KERATODERMA | GENE | CXCR3 | SKIN-DISEASE | OF-FUNCTION MUTATIONS | HUMAN INVOLUCRIN PROMOTER | C-JUN | MICE LACKING | T-CELLS | CELL BIOLOGY | Calgranulin B - metabolism | Ichthyosis - metabolism | Calgranulin A - metabolism | Transcription Factor AP-1 - genetics | Humans | Male | Calgranulin A - genetics | Interferon-gamma - metabolism | Ichthyosis - pathology | Transcription Factor AP-1 - deficiency | Ichthyosis - genetics | Intermediate Filament Proteins - genetics | Female | Calgranulin B - genetics | Cell Differentiation | Membrane Proteins - metabolism | Interferon-gamma - genetics | Peptide Fragments - genetics | Disease Models, Animal | Epidermis - metabolism | Epidermis - pathology | Peptide Fragments - metabolism | Signal Transduction | Membrane Proteins - genetics | Proto-Oncogene Proteins c-jun - genetics | Gene Expression Regulation | Mice, Transgenic | Th1-Th2 Balance - genetics | Chemokines, CXC - genetics | Keratinocytes - pathology | Phenotype | Animals | Keratinocytes - metabolism | Proto-Oncogene Proteins c-jun - metabolism | Chemokines, CC - genetics | Chemokines, CXC - metabolism | Mice | Intermediate Filament Proteins - metabolism | Chemokines, CC - metabolism | Transcription factors | Activator protein 1 | c-Jun protein | CCL3 protein | Epidermis | Lymphocytes T | Filaggrin | CXCR3 protein | Gene expression | Vasodilation | Ichthyosis | Proteins | Genotype & phenotype | Signal transduction | S100A9 protein | CXCL10 protein | CXCL11 protein | Localization | Chemokines | Monocyte chemoattractant protein 1 | Original
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10. Full Text LIM and SH3 protein -1 modulates CXCR2-mediated cell migration
by Raman, Dayanidhi and Sai, Jiqing and Neel, Nicole F and Chew, Catherine S and Richmond, Ann
PLoS ONE, ISSN 1932-6203, 2010, Volume 5, Issue 4, p. e10050
Background: The chemokine receptor CXCR2 plays a pivotal role in migration of neutrophils, macrophages and endothelial cells, modulating several biological... 
BREAST-CANCER | II INTERLEUKIN-8 RECEPTOR | IN-VITRO | CAMP SIGNALING PATHWAY | CHEMOTAXIS | LLKIL MOTIF | MULTIDISCIPLINARY SCIENCES | CXCR2 | TUMOR-GROWTH | LASP-1 | INFLUENCES ZYXIN LOCALIZATION | Receptors, Interleukin-8B - physiology | Cell Line | Immunoprecipitation | LIM Domain Proteins | Humans | Focal Adhesions | Chemotaxis | Protein Interaction Mapping | Adaptor Proteins, Signal Transducing - physiology | Receptors, Interleukin-8B - metabolism | HL-60 Cells | Protein Binding | Cytoskeletal Proteins - metabolism | Cytoskeletal Proteins - physiology | Adaptor Proteins, Signal Transducing - metabolism | Binding Sites | Information management | Muscle proteins | Binding proteins | Analysis | Atherosclerosis | Protein binding | Motility | Leukocyte migration | Pathogenesis | Biology | Metastasis | Kinases | Macrophages | Cell adhesion & migration | Proteins | Angiogenesis | Signal transduction | Receptors | Clonal deletion | Actin | Rodents | Penicillin | Cell cycle | Deletion | Extracellular matrix | Inhibition | Growth factors | Wound healing | CXCR2 protein | Extracellular signal-regulated kinase | Leukocytes (neutrophilic) | Breast cancer | CXCR3 protein | Chemokine receptors | CXCR4 protein | Endothelial cells | Medicine | Mutagenesis | Arteriosclerosis | Cytoskeleton | Ligands | Sepsis | Paxillin | Cell migration | Chemokines | Binding sites
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11. Full Text THU0241 Decreased circulating CXCR3+CCR9+ th cells are associated with elevated levels of their ligands CXCL10 and CCL25 in the salivary gland of patients with SJÖGREN'S syndrome to potentially facilitate concerted migration
by Blokland, SL and Hillen, MR and Meller, S and Homey, B and Smithson, GM and Kruize, AA and Radstake, TR and Roon, JA van
Annals of the Rheumatic Diseases, ISSN 0003-4967, 06/2017, Volume 76, Issue Suppl 2, p. 295
BackgroundPrimary Sjögren's syndrome (pSS) is characterized by dryness and lymphocytic infiltration in the salivary glands. Both CXCR5+ T follicular helper... 
Flow cytometry | Sjogren's syndrome | Mucosal immunity | Leukocyte migration | Mucosa | Immunoglobulin G | Helper cells | CXCL13 protein | Lymphocytes T | mRNA | Proteins | CXCR5 protein | CCL20 protein | CCL17 protein | Salivary gland | CXCR3 protein | Gene expression | Chemotaxis | Chemokine receptors | CCR6 protein | Exocrine glands | γ-Interferon | CXCL10 protein | Ligands | CCR9 protein | Chemokines | Cell migration
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12. Full Text Reassessing Th1 versus Th17.1 in sarcoidosis: New tricks for old dogma
by Chen, Edward S
European Respiratory Journal, ISSN 0903-1936, 03/2018, Volume 51, Issue 3, pp. 1800010 - 1800010
Immunologically, sarcoidosis has been classically defined by the presence of a Th1-dominant environment localised to sites of disease, characterised by... 
GAMMA | PULMONARY SARCOIDOSIS | DISORDERS | INTERFERON | RESPIRATORY SYSTEM | T-CELLS | CCR5 protein | Sarcoidosis | RANTES | Helper cells | CCL3 protein | Lymphocytes T | CXCR3 protein | CCL4 protein | Interleukin 18 | Interleukin 1 | CXCL10 protein | CXCL11 protein | Interferon | Chemokines | Monocyte chemoattractant protein 1
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13. Full Text An integrated method for the identification of novel genes related to oral cancer
by Chen, Lei and Yang, Jing and Xing, Zhihao and Yuan, Fei and Shu, Yang and Zhang, YunHua and Kong, XiangYin and Huang, Tao and Li, HaiPeng and Cai, Yu-Dong
PLoS ONE, ISSN 1932-6203, 04/2017, Volume 12, Issue 4, p. e0175185
Cancer is a significant public health problem worldwide. Complete identification of genes related to one type of cancer facilitates earlier diagnosis and... 
BREAST-CANCER | TUMOR-NECROSIS-FACTOR | PROTEIN-INTERACTION NETWORKS | SHORTEST-PATH | GASTRIC-CANCER | MULTIDISCIPLINARY SCIENCES | SQUAMOUS-CELL CARCINOMA | C-MYC | GROWTH-FACTOR | NF-KAPPA-B | CLINICOPATHOLOGICAL FEATURES | Mouth Neoplasms - genetics | Humans | Algorithms | Genetic aspects | Research | Mouth cancer | Oncogenes | Health sciences | Disease | Oral cancer | Laboratories | Identification methods | Genes | Biology | Kinases | Proteins | Immunology | Cell cycle | Life sciences | Growth factors | Public health | Fibroblast growth factor receptor 2 | NF-κB protein | Bone morphogenetic protein 4 | Random walk | Shortest-path problems | CXCR3 protein | Gene expression | Tuberculosis | Medical prognosis | Construction methods | Computer applications | MyD88 protein | Methods | Cancer
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14. Full Text Serum IFN-γ–induced protein 10 (IP-10) as a biomarker for severity of acute respiratory infection in healthy adults
by Hayney, Mary S and Henriquez, Kelsey M and Barnet, Jodi H and Ewers, Tola and Champion, Heather M and Flannery, Sean and Barrett, Bruce
Journal of Clinical Virology, ISSN 1386-6532, 2017, Volume 90, pp. 32 - 37
Highlights • Serum IP-10 concentrations from 225 ARI episodes correlated with ARI severity on the day that it was measured and overall ARI severity for the... 
Infectious Disease | Allergy and Immunology | Healthy adults | Biomarker | Acute respiratory infection | IP-10 | Viral pathogens | CELLS | INDUCIBLE PROTEIN-10 | RHINOVIRUS | PROLIFERATION | RESPONSES | VIROLOGY | ASTHMA EXACERBATIONS | CXCR3 | DISEASE | SYMPTOM SURVEY | EXPRESSION | Severity of Illness Index | Chemokine CXCL10 - blood | Enzyme-Linked Immunosorbent Assay | Humans | Middle Aged | Male | Biomarkers - blood | Respiratory Tract Infections - diagnosis | Randomized Controlled Trials as Topic | Virus Diseases - diagnosis | Virus Diseases - pathology | Adult | Female | Aged | Cohort Studies | Respiratory Tract Infections - pathology | Serum - chemistry | viral pathogens | healthy adults | acute respiratory infection | biomarker
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