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Journal of Periodontology, ISSN 0022-3492, 09/2019, Volume 90, Issue 9, pp. 1032 - 1042
Background Metformin, a classical treatment for diabetes mellitus, has shown sound anti‐inflammatory effects. Emerging studies have focused on the mechanism... 
metformin | diabetes mellitus | cell cycle | inflammation | NIMA‐related kinases | periodontitis | EFFICACY | NLRP3 INFLAMMASOME ACTIVATION | DOWN-REGULATION | CELL-CYCLE ARREST | NIMA-related kinases | MELLITUS | PATHWAY | DENTISTRY, ORAL SURGERY & MEDICINE | GROWTH | STRESS | LIFE EXPECTANCY | INNATE IMMUNITY
Journal Article
Journal Article
Journal Article
Journal Article
FASEB JOURNAL, ISSN 0892-6638, 06/1995, Volume 9, Issue 9, pp. 726 - 735
The transmission of extracellular signals into their intracellular targets is mediated by a network of interacting proteins that regulate a large number of... 
ONCOGENESIS | MAP KINASE | BIOCHEMISTRY & MOLECULAR BIOLOGY | A431 CELLS | PROLIFERATION | SIGNALING | EPIDERMAL GROWTH-FACTOR | THREONINE RESIDUES | 3T3 CELLS | CELL BIOLOGY | V-RAF | RAF-1 KINASE | BIOLOGY | RIBOSOMAL S6 KINASE | XENOPUS OOCYTES | CELL-CYCLE | PC12 CELLS
Journal Article
PLoS ONE, ISSN 1932-6203, 02/2014, Volume 9, Issue 2, p. e90247
Background: The incidence, prevalence, and mortality of intrahepatic cholangiocarcinoma (ICC) are increasing worldwide. Protein tyrosine kinase-7 (PTK7) is... 
PATHWAYS | JNK ACTIVATION | INVASION | CANCERS | MULTIDISCIPLINARY SCIENCES | RHO-GTPASES | CELL POLARITY | Neoplasm Transplantation | RNA, Small Interfering - genetics | Cell Adhesion Molecules - genetics | Humans | Middle Aged | Gene Expression Regulation, Neoplastic | Male | Cholangiocarcinoma - metabolism | Bile Ducts, Intrahepatic - metabolism | Aged, 80 and over | Cell Cycle Proteins - genetics | Receptor Protein-Tyrosine Kinases - antagonists & inhibitors | Adult | Female | Bile Duct Neoplasms - genetics | Bile Ducts, Intrahepatic - pathology | Bile Duct Neoplasms - metabolism | Signal Transduction | Neoplasm Invasiveness | rho GTP-Binding Proteins - genetics | Cell Cycle Proteins - metabolism | Receptor Protein-Tyrosine Kinases - metabolism | Cell Adhesion Molecules - metabolism | Cholangiocarcinoma - pathology | Animals | Cell Cycle | Receptor Protein-Tyrosine Kinases - genetics | Mice, Nude | rho GTP-Binding Proteins - metabolism | Cell Line, Tumor | Cholangiocarcinoma - genetics | Aged | Mice | Bile Duct Neoplasms - pathology | Cell Movement | RNA, Small Interfering - metabolism | Proteins | Tyrosine | RNA | Analysis | Phenols | DNA synthesis | Health aspects | Cell proliferation | Membranes | Prognosis | Colorectal cancer | Digital cameras | Kinases | Cell growth | Synthesis | Surgery | Xenografts | Protein-tyrosine kinase | Chromosomes | Deoxyribonucleic acid--DNA | Cholangiocarcinoma | Invasiveness | Review boards | RhoA protein | siRNA | Gene expression | Ribonucleic acid--RNA | Patients | Survival | Bile duct | Medicine | DNA biosynthesis | Polymerase chain reaction | Pathology | Hospitals | Cell lines | Cell migration | Apoptosis | Deoxyribonucleic acid | Ribonucleic acid | DNA
Journal Article
by Jin, SF and Ma, HL and Liu, ZL and Fu, ST and Zhang, CP and He, Y
EXPERIMENTAL CELL RESEARCH, ISSN 0014-4827, 12/2015, Volume 339, Issue 2, pp. 289 - 299
Pirfenidone is an orally bioavailable synthetic compound with therapeutic potential for idiopathic pulmonary fibrosis. It is thought to act through antioxidant... 
LUNG FIBROSIS | LIVER FIBROSIS | CDC7 | Proliferation | Cell division cycle 7 | MESENCHYMAL STEM-CELLS | FIBROBLASTS | CELL BIOLOGY | Smads | REPLICATION | ONCOLOGY | Pirfenidone | Differentiation | IDIOPATHIC PULMONARY-FIBROSIS | EXPRESSION
Journal Article
Clinical Cancer Research, ISSN 1078-0432, 04/2012, Volume 18, Issue 7, pp. 1966 - 1978
Purpose: The "gate-keeper" mutations T674I platelet-derived growth factor receptor alpha (PDGFR alpha) in hypereosinophilic syndrome (HES) and T315I Bcr-Abl in... 
CHRONIC MYELOGENOUS LEUKEMIA | GASTROINTESTINAL STROMAL TUMOR | IN-VITRO | ONCOLOGY | TYROSINE KINASE | MAST-CELLS | CHRONIC LYMPHOCYTIC-LEUKEMIA | IDIOPATHIC HYPEREOSINOPHILIC SYNDROME | HARBORING T315I MUTATION | EOSINOPHILIC LEUKEMIA | CHRONIC MYELOID-LEUKEMIA | Oncogene Proteins, Fusion - metabolism | Cyclin-Dependent Kinases - metabolism | Apoptosis - drug effects | mRNA Cleavage and Polyadenylation Factors - metabolism | Humans | Leukemia, Myelogenous, Chronic, BCR-ABL Positive - drug therapy | Leukemia, Myelogenous, Chronic, BCR-ABL Positive - genetics | Male | Receptor, Platelet-Derived Growth Factor alpha - antagonists & inhibitors | Female | Cyclin-Dependent Kinases - antagonists & inhibitors | Phosphorylation - drug effects | mRNA Cleavage and Polyadenylation Factors - genetics | mRNA Cleavage and Polyadenylation Factors - antagonists & inhibitors | Cell Line | Receptor, Platelet-Derived Growth Factor alpha - metabolism | Cyclin-Dependent Kinase 9 - antagonists & inhibitors | Reverse Transcriptase Polymerase Chain Reaction | Blotting, Western | Xenograft Model Antitumor Assays | Fusion Proteins, bcr-abl - genetics | Animals | Signal Transduction - drug effects | Mice, Nude | Oncogene Proteins, Fusion - genetics | Receptor, Platelet-Derived Growth Factor alpha - genetics | Fusion Proteins, bcr-abl - antagonists & inhibitors | Cell Line, Tumor | Leukemia, Myelogenous, Chronic, BCR-ABL Positive - metabolism | Oncogene Proteins, Fusion - antagonists & inhibitors | Cell Proliferation - drug effects | Cyclin-Dependent Kinase 9 - metabolism | Mice | Mice, Inbred BALB C | Oxazoles - pharmacology | Thiazoles - pharmacology | Cell Cycle - drug effects | Fusion Proteins, bcr-abl - metabolism
Journal Article