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Science, ISSN 0036-8075, 09/2014, Volume 345, Issue 6203, pp. 1466 - 1466
Journal Article
CELL CYCLE, ISSN 1538-4101, 02/2013, Volume 12, Issue 4, pp. 674 - 683
The term "mitochondrial permeability transition" (MPT) refers to an abrupt increase in the permeability of the inner mitochondrial membrane to low molecular... 
mitochondrial respiratory chain | CANCER-CELL DEATH | INDUCED APOPTOSIS | PORE COMPLEX | apoptosis | PHOSPHATE CARRIER | ATP5G1 | OUTER-MEMBRANE PERMEABILIZATION | p53 | CELL BIOLOGY | CYTOCHROME-C | CYCLOPHILIN-D | CYCLOSPORINE-A | permeability transition pore (PTP) | ENDOPLASMIC-RETICULUM | cytochrome c | caspases | GLUTAMATE EXCITOTOXICITY
Journal Article
Developmental Cell, ISSN 1534-5807, 09/2011, Volume 21, Issue 3, pp. 469 - 478
Although mature myocytes rely on mitochondria as the primary source of energy, the role of mitochondria in the developing heart is not well known. Here, we... 
PATHOGENESIS | APOPTOSIS | CYCLOPHILIN-D | CYCLOSPORINE-A | CALCIUM | DISEASE | NEURONS | DEVELOPMENTAL BIOLOGY | DYSFUNCTION | FISSION | CELL-DEATH | CELL BIOLOGY | Antioxidants | Medical colleges | Mitochondrial DNA | Permeability
Journal Article
Cell Calcium, ISSN 0143-4160, 2011, Volume 50, Issue 3, pp. 222 - 233
Abstract A variety of stimuli utilize an increase of cytosolic free Ca2+ concentration as a second messenger to transmit signals, through Ca2+ release from the... 
Advanced Basic Science | Mitochondria | Permeability transition pore | Apoptosis | Necrosis | AMYOTROPHIC-LATERAL-SCLEROSIS | WOLF-HIRSCHHORN-SYNDROME | CELL-DEATH | CELL BIOLOGY | HEXOKINASE-II | CYCLOPHILIN-D | AMYLOID BETA-PEPTIDE | CYCLOSPORINE-A | Ca2 | BCL-2 PROTEIN FAMILY | ENDOPLASMIC-RETICULUM | PARKINSONS-DISEASE
Journal Article
Journal Article
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 7/2013, Volume 110, Issue 29, pp. 12024 - 12029
Journal Article
Circulation, ISSN 0009-7322, 05/2008, Volume 117, Issue 21, pp. 2761 - 2768
Background-Opening of the mitochondrial permeability transition pore (mPTP) is a crucial event in lethal reperfusion injury. Phosphorylation (inhibition) of... 
Myocardial infarction | Ischemia | Reperfusion | CARDIAC & CARDIOVASCULAR SYSTEMS | myocardial infarction | INJURY | MYOCARDIAL REPERFUSION | ischemia | GLYCOGEN-SYNTHASE KINASE-3-BETA | OPIOID-INDUCED CARDIOPROTECTION | CARDIOMYOCYTE DEATH | HEART | CYCLOPHILIN-D | CARDIAC-HYPERTROPHY | IN-VIVO | PERIPHERAL VASCULAR DISEASE | ISCHEMIA-REPERFUSION | reperfusion
Journal Article
CARDIOVASCULAR RESEARCH, ISSN 0008-6363, 08/2007, Volume 75, Issue 3, pp. 530 - 535
Objective: The opening of the mitochondrial permeability transition pore (mPTP) at the time of myocardial reperfusion is a critical determinant of cell death.... 
OXIDATIVE STRESS | CARDIAC & CARDIOVASCULAR SYSTEMS | PROTECTION | mitochondria | REPERFUSION INJURY | ischemia | preconditioning | CELL-DEATH | CA-2+-DEPENDENT PORE | CYCLOPHILIN-D | reperfusion | HEART-MITOCHONDRIA | INHIBIT
Journal Article