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Free Radical Biology and Medicine, ISSN 0891-5849, 2010, Volume 49, Issue 9, pp. 1406 - 1416
Journal Article
Food and Chemical Toxicology, ISSN 0278-6915, 10/2012, Volume 50, Issue 10, pp. 3413 - 3420
Journal Article
Diabetes, ISSN 0012-1797, 08/2010, Volume 59, Issue 8, pp. 2001 - 2009
Journal Article
Journal of Gastroenterology and Hepatology, ISSN 0815-9319, 02/2011, Volume 26, Issue 2, pp. 312 - 318
Background and Aim:  Antituberculosis drugs, isoniazid and rifampicin, in combination, are known to develop drug‐induced hepatotoxicity (DIH). A higher risk of... 
antituberculosis treatment | drug induced hepatitis | CYP2E1 | acetylator status | NAT2 | Acetylator status | Drug induced hepatitis | Antituberculosis treatment | S-TRANSFERASE M1 | DRUG-INDUCED HEPATOTOXICITY | INDUCED LIVER-DISEASE | RISK-FACTORS | GENOTYPE | TUBERCULOSIS | SUSCEPTIBILITY | RIFAMPIN | LUNG-CANCER | GASTROENTEROLOGY & HEPATOLOGY | CARCINOMA | Antitubercular Agents - adverse effects | Arylamine N-Acetyltransferase - metabolism | Humans | Middle Aged | Male | Cytochrome P-450 CYP2E1 - genetics | Cytochrome P-450 CYP2E1 - metabolism | Time Factors | Polymerase Chain Reaction | Adult | Female | Nutritional Status | Acetylation | Drug Therapy, Combination | Odds Ratio | Chemical and Drug Induced Liver Injury - enzymology | Promoter Regions, Genetic | Genetic Predisposition to Disease | Risk Assessment | Introns | Gene Frequency | Risk Factors | Logistic Models | Chemical and Drug Induced Liver Injury - genetics | India | Phenotype | Chemical and Drug Induced Liver Injury - ethnology | Polymorphism, Restriction Fragment Length | Arylamine N-Acetyltransferase - genetics | Polymorphism, Single Nucleotide | Genetic research | Hepatitis | Anopheles | Rifampin | Cytochrome P-450 | Index Medicus | Drugs | Lung | Population studies | Gene polymorphism | CYP2E1 gene | Risk factors | Tuberculosis | Genotyping | hepatotoxicity | Genetic factors | Isoniazid
Journal Article
Journal of Gastroenterology and Hepatology, ISSN 0815-9319, 06/2010, Volume 25, Issue 6, pp. 1136 - 1143
Background and Aim:  Reactive oxygen species produced by cytochrome P4502E1 (CYP2E1) are believed to play a role in pathophysiology of non‐alcoholic fatty... 
cytochrome P4502E1 (CYP2E1) | inducible nitric oxide synthase (iNOS) | non‐alcoholic fatty liver disease (NAFLD) | anti‐oxidant enzyme | oxidative stress | Inducible nitric oxide synthase (iNOS) | Oxidative stress | Anti-oxidant enzyme | Cytochrome P4502E1 (CYP2E1) | Non-alcoholic fatty liver disease (NAFLD) | non-alcoholic fatty liver disease (NAFLD) | PROTECTS HEPG2 CELLS | NITRIC-OXIDE SYNTHASE | GLUTATHIONE-PEROXIDASE | REACTIVE OXYGEN | DISEASE | anti-oxidant enzyme | HEME OXYGENASE-1 | CYP2E1 | GASTROENTEROLOGY & HEPATOLOGY | SUPEROXIDE-DISMUTASE | TRANSCRIPTION FACTOR | UP-REGULATION | Up-Regulation | Nitric Oxide Synthase Type II - biosynthesis | Superoxide Dismutase - genetics | Immunoprecipitation | Superoxide Dismutase - biosynthesis | Oxidative Stress - physiology | Male | Cytochrome P-450 CYP2E1 - genetics | RNA - genetics | Cytochrome P-450 CYP2E1 - biosynthesis | Heme Oxygenase-1 - genetics | Fatty Liver - enzymology | NF-E2-Related Factor 2 - genetics | Disease Models, Animal | Fatty Liver - genetics | Heme Oxygenase-1 - biosynthesis | Catalase - genetics | Electrophoresis, Polyacrylamide Gel | Reverse Transcriptase Polymerase Chain Reaction | Blotting, Western | Disease Progression | Animals | Nitric Oxide Synthase Type II - genetics | Mice | Mice, Inbred BALB C | Superoxide Dismutase-1 | Catalase - biosynthesis | NF-E2-Related Factor 2 - biosynthesis | Liver | Cytochrome P-450 | Aspartate | Superoxide | Triglycerides | Gene expression | Anopheles | Fatty liver | Analysis | Nitric oxide | Heme | Genetic engineering | Peroxidase
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