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Blood, ISSN 0006-4971, 2009, Volume 114, Issue 13, pp. 2667 - 2677
Journal Article
Environmental Health Perspectives, ISSN 0091-6765, 8/2011, Volume 119, Issue 8, pp. 1142 - 1148
Background: The large and increasing number of chemicals released into the environment demands more efficient and cost-effective approaches for assessing... 
Chemical hazards | Receptors | Genomics | Environmental health | Agonists | Ligands | Cytotoxicity | Libraries | Environmental agencies | Research | Nuclear receptors | Chemical genomics | qHTS | Assay performance | Tox21 | assay performance | TOXICITY | ANTAGONISM | IDENTIFICATION | ESTROGEN-RECEPTOR | nuclear receptors | ENVIRONMENTAL SCIENCES | IN-VITRO | PUBLIC, ENVIRONMENTAL & OCCUPATIONAL HEALTH | EVOLUTION | DISEASE | ASSAYS | SUPERFAMILY | HEALTH | chemical genomics | cytotoxicity | Peroxisome Proliferator-Activated Receptors - agonists | Cell Line | Retinoid X Receptor alpha - metabolism | Peroxisome Proliferator-Activated Receptors - antagonists & inhibitors | Humans | Orphan Nuclear Receptors - agonists | Receptors, Androgen - metabolism | Receptors, Cytoplasmic and Nuclear - agonists | Orphan Nuclear Receptors - metabolism | PPAR gamma - metabolism | Receptors, Thrombin - antagonists & inhibitors | Retinoid X Receptor alpha - agonists | Fragile X Mental Retardation Protein | Orphan Nuclear Receptors - antagonists & inhibitors | Retinoid X Receptor alpha - antagonists & inhibitors | PPAR gamma - antagonists & inhibitors | Receptors, Thrombin - agonists | Liver X Receptors | Receptors, Cytoplasmic and Nuclear - antagonists & inhibitors | Peroxisome Proliferator-Activated Receptors - metabolism | Receptors, Thrombin - metabolism | Estrogen Receptor alpha - metabolism | Environmental Pollutants - toxicity | Receptors, Cytoplasmic and Nuclear - metabolism | Environmental aspects | Physiological aspects | Cell receptors | Health aspects | Chemicals | Index Medicus
Journal Article
Cancer Science, ISSN 1347-9032, 07/2016, Volume 107, Issue 7, pp. 1039 - 1046
Antibody–drug conjugates deliver anticancer agents selectively and efficiently to tumor tissue and have significant antitumor efficacy with a wide therapeutic... 
topoisomerase I inhibitor | HER | bystander killing | Antibody‐drug conjugate | T-DM | HER2 | Antibody-drug conjugate | HODGKINS LYMPHOMA | GEMTUZUMAB OZOGAMICIN | ADVANCED BREAST-CANCER | BRENTUXIMAB VEDOTIN | ACUTE MYELOID-LEUKEMIA | TRASTUZUMAB EMTANSINE | CHALLENGES | THERAPY | ONCOLOGY | EXPRESSION | CATHEPSIN-B | Receptor, ErbB-2 - genetics | Breast Neoplasms - immunology | Humans | Stomach Neoplasms - pathology | Maytansine - pharmacology | Immunoconjugates - immunology | Breast Neoplasms - enzymology | Immunoconjugates - pharmacology | Topoisomerase I Inhibitors - pharmacology | Neoplasms - genetics | Antibodies, Monoclonal, Humanized - pharmacology | Female | Camptothecin - immunology | Receptor, ErbB-2 - immunology | Camptothecin - analogs & derivatives | Stomach Neoplasms - enzymology | Stomach Neoplasms - genetics | Maytansine - analogs & derivatives | Neoplasms - enzymology | Stomach Neoplasms - immunology | Animals | Breast Neoplasms - genetics | Bystander Effect - drug effects | Breast Neoplasms - pathology | Mice, Nude | Neoplasms - immunology | Cell Membrane Permeability - drug effects | Cell Line, Tumor | Cell Proliferation - drug effects | Mice | Mice, Inbred BALB C | Antibodies, Monoclonal, Humanized - immunology | Neoplasms - pathology | Camptothecin - pharmacology | Maytansine - immunology | Trastuzumab | Viral antibodies | Epidermal growth factor | Imaging systems | Antibodies | Permeability | Drug therapy | Biopharmaceutics | Tumors | Antigens | Hematology | Laboratories | Toxicity | DNA topoisomerase | Polymerization | Clinical trials | Cytotoxicity | Breast cancer | ErbB-2 protein | Stomach cancer | Hypotheses | Antitumor agents | Xenografts | Antitumor activity | Lymphomas | Drug dosages | Payloads | Index Medicus | T‐DM | Original
Journal Article
PLoS ONE, ISSN 1932-6203, 06/2013, Volume 8, Issue 6, p. e65860
Dopamine (DA), a neurotransmitter in the nervous system, has been shown to modulate immune function. We have previously reported that five subtypes of DA... 
PROTEIN-KINASE-A | ACTIVATED T-CELLS | NUCLEAR-FACTOR | MULTIDISCIPLINARY SCIENCES | FACTOR-KAPPA-B | PROLIFERATION | SECRETION | MEDIATED MECHANISM | EXPRESSION | D2 | LYMPHOCYTES | Enzyme Activators - pharmacology | Phosphorylation | Receptors, Dopamine D3 - metabolism | Receptors, Dopamine D4 - genetics | Receptors, Dopamine D3 - genetics | Isoquinolines - pharmacology | Killer Cells, Natural - immunology | Cyclic AMP-Dependent Protein Kinases - antagonists & inhibitors | Cyclic AMP - metabolism | Second Messenger Systems | Cyclic AMP-Dependent Protein Kinases - metabolism | Dopamine Agonists - pharmacology | Gene Expression | Colforsin - pharmacology | Adenylyl Cyclases - metabolism | Benzazepines - pharmacology | Sulfonamides - pharmacology | Mice, Inbred ICR | Animals | Dopamine Antagonists - pharmacology | Cyclic AMP Response Element-Binding Protein - metabolism | 2,3,4,5-Tetrahydro-7,8-dihydroxy-1-phenyl-1H-3-benzazepine - pharmacology | Cell Line, Tumor | Receptors, Dopamine D5 - metabolism | Receptors, Dopamine D4 - metabolism | Protein Processing, Post-Translational | Quinpirole - pharmacology | Receptors, Dopamine D5 - genetics | Killer Cells, Natural - metabolism | Cytotoxicity, Immunologic | Phenols | Cyclic adenylic acid | Dopamine | Lymphocytes | Dopamine receptors | Drugs | Flow cytometry | Toxicity | Cytotoxicity | Nervous system | Lymphocytes T | mRNA | Activation | Haloperidol | Kinases | Dopamine D4 receptors | Immunosuppressive agents | Proteins | Signal transduction | Receptors | Cell activation | Modulation | Clonal selection | Physiology | Natural killer cells | Dopamine D2 receptors | Spleen | Dopamine D5 receptors | Adenosine | Immune response | Cytokines | Dopamine D3 receptors | Cyclic AMP | T cell receptors | Cyclic AMP response element-binding protein | Gene expression | Lymphoma | Medicine | Cytometry | Signaling | Dopamine D1 receptors | Forskolin | Receptor mechanisms | Laboratory animals | Adenylate cyclase
Journal Article
Cancer Cell, ISSN 1535-6108, 2011, Volume 19, Issue 1, pp. 101 - 113
Antibodies to cell-surface antigens trigger activatory Fcγ receptor (FcγR)-mediated retrograde signals in leukocytes to control immune effector functions.... 
GROWTH-FACTOR RECEPTOR | IMMUNE-RESPONSE | ANTI-CD20 MONOCLONAL-ANTIBODY | ONCOLOGY | IN-VIVO | CELLULAR CYTOTOXICITY | THERAPEUTIC ANTIBODIES | TUMOR-CELLS | FOLLICULAR LYMPHOMA | DEFICIENT MICE | ESTABLISHED TUMORS | CELL BIOLOGY | Neoplasms - metabolism | Protein Binding - genetics | Receptors, TNF-Related Apoptosis-Inducing Ligand - agonists | Humans | Antibodies, Monoclonal - therapeutic use | NF-kappa B - metabolism | Receptors, TNF-Related Apoptosis-Inducing Ligand - metabolism | Mutation - immunology | Receptors, IgG - metabolism | Receptors, TNF-Related Apoptosis-Inducing Ligand - immunology | Leukocytes - immunology | Signal Transduction - immunology | Receptors, IgG - genetics | Immunoglobulin G - immunology | Myeloid Cells - immunology | Immunoglobulin G - pharmacology | Killer Cells, Natural - immunology | Female | Receptor Aggregation - immunology | Polymorphism, Single Nucleotide - immunology | Antibodies, Monoclonal - immunology | B-Lymphocytes - metabolism | Antibodies, Monoclonal - pharmacology | HCT116 Cells | Mice, Inbred C57BL | CD40 Antigens - agonists | Mutation - genetics | Protein Binding - immunology | Mice, Knockout | Neoplasms - drug therapy | Xenograft Model Antitumor Assays | Antibodies, Monoclonal - genetics | B-Lymphocytes - drug effects | Animals | Apoptosis - immunology | Immunoglobulin G - genetics | Cell Line, Tumor | Polymorphism, Single Nucleotide - genetics | Mice | Mice, Inbred BALB C | Antibodies, Monoclonal - metabolism | Neoplasms - pathology | Immunoglobulin Fc Fragments - genetics | Leukocytes - metabolism | CD40 Antigens - immunology
Journal Article
Journal of Allergy and Clinical Immunology, The, ISSN 0091-6749, 2013, Volume 132, Issue 2, pp. 264 - 286
Nuclear receptors are ligand-activated transcription factors linking lipid signaling to the expression of the genome. There is increasing appreciation of the... 
Allergy and Immunology | retinoid X receptor | asthma | lipid | rheumatoid arthritis | inflammatory bowel disease | retinoic acid receptor | systemic lupus erythematosus | nuclear receptor | vitamin D receptor | peroxisome proliferator-associated receptor | liver X receptor | dendritic cell | Macrophage | systemic lupus erythematosus asthma | SYSTEMIC-LUPUS-ERYTHEMATOSUS | RANDOMIZED CONTROLLED-TRIAL | IMMUNOLOGY | ALLERGY | REGULATORY T-CELLS | NF-KAPPA-B | CULTURED ALVEOLAR MACROPHAGES | COLLAGEN-INDUCED ARTHRITIS | VITAMIN-D-RECEPTOR | GAMMA AGONIST ROSIGLITAZONE | INFLAMMATORY-BOWEL-DISEASE | RETINOID-X-RECEPTOR | Autoimmune Diseases - physiopathology | Macrophages - pathology | Receptors, Cytoplasmic and Nuclear - drug effects | Dendritic Cells - immunology | Humans | Autoimmune Diseases - immunology | Dendritic Cells - pathology | Receptors, Retinoic Acid - metabolism | Macrophages - cytology | Orphan Nuclear Receptors - metabolism | PPAR gamma - metabolism | Macrophages - metabolism | Animals | Lipids - pharmacology | Liver X Receptors | Receptors, Calcitriol | Dendritic Cells - cytology | Mice | Retinoid X Receptors - metabolism | Dendritic Cells - metabolism | Macrophages - immunology | Receptors, Cytoplasmic and Nuclear - metabolism | Rheumatoid factor | Vitamin D | Dendritic cells | Analysis | Gastrointestinal diseases | Genomics | Physiological aspects | Calcifediol | Alfacalcidol | Genetic transcription | Macrophages | Transcription factors | Lipids | Cytotoxicity | Fatty acids | Asthma | Proteins | Vitamin A | Pathology | Body mass index | Lymphocytes | Atherosclerosis | Insulin resistance | Deoxyribonucleic acid--DNA | Metabolic disorders | Immune system
Journal Article
Journal Article