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BBA - Molecular and Cell Biology of Lipids, ISSN 1388-1981, 06/2015, Volume 1851, Issue 6, pp. 844 - 856
Journal Article
New Phytologist, ISSN 0028-646X, 1/2013, Volume 197, Issue 1, pp. 88 - 98
The Arabidopsis guard cell anion channel SLAC1 is essential for stomatal closure in response to various endogenous and environmental stimuli. Interestingly,... 
Full papers | Electric potential | Anions | Calcium | Humidity | Stomatal conductance | Guard cells | Cell membranes | Plants | Pipettes | Plant cells | ABA | stomatal opening | signaling | guard cells | potassium uptake channel | anion channel | abscisic acid | concentration | SLAC | cytosolic Ca | Cytosolic Ca | Potassium uptake channel | SLAC1 | Abscisic acid (ABA) | Anion channel | Stomatal opening | S-TYPE ANION | ARABIDOPSIS-THALIANA | ABSCISIC-ACID ACTIVATION | cytosolic Ca2+concentration | POTASSIUM CHANNELS | FREE CALCIUM | PLASMA-MEMBRANE | BLUE-LIGHT | PLANT SCIENCES | abscisic acid (ABA) | VICIA-FABA | Ca2+signaling | GUARD-CELL PROTOPLASTS | KINASE-PHOSPHATASE PAIR | Arabidopsis Proteins - genetics | Arabidopsis - drug effects | Carbon Dioxide - metabolism | Plant Cells - metabolism | Calcium - metabolism | Membrane Proteins - genetics | Plant Epidermis - drug effects | Plant Stomata - metabolism | Plant Epidermis - metabolism | Arabidopsis - metabolism | Arabidopsis - genetics | Arabidopsis Proteins - metabolism | Abscisic Acid - pharmacology | Cell Membrane - enzymology | Protoplasts - metabolism | Patch-Clamp Techniques | Light | Alleles | Gene Expression Regulation, Plant | Plant Stomata - drug effects | Cytosol - metabolism | Membrane Proteins - metabolism | Mutation | Potassium Channels, Inwardly Rectifying - metabolism | Arabidopsis thaliana | Abscisic acid | Genetic aspects | Ca2+ signaling | cytosolic Ca2+ concentration
Journal Article
Circulation Research, ISSN 0009-7330, 02/2007, Volume 100, Issue 3, pp. 342 - 353
A large body of evidence has accrued indicating that voltage-gated Ca channel subtypes, including L-, T-, N-, and P/Q-type, are present within renal vascular... 
Renal microcirculation | Mibefradil | channels | Afferent arteriole | channel blockers | Efferent arteriole | Renal disease | Voltage-dependent Ca | Efonidipine | Ca2+ channel blockers | N-TYPE | CARDIAC & CARDIOVASCULAR SYSTEMS | EFFERENT ARTERIOLES | efonidipine | SPONTANEOUSLY HYPERTENSIVE-RATS | RHO-KINASE INHIBITOR | renal microcirculation | ANGIOTENSIN-II | T-TYPE | renal disease | mibefradil | DEPENDENT CALCIUM-CHANNELS | CARDIAC L-TYPE | RENAL MICROVASCULAR CONSTRICTION | efferent arteriole | voltage-dependent Ca2+ channels | PERIPHERAL VASCULAR DISEASE | afferent arteriole | HEMATOLOGY | IN-VIVO VISUALIZATION | Arterioles - physiology | Kidney - physiology | Protein Subunits | Antihypertensive Agents - pharmacology | Kidney - blood supply | Humans | Calcium Channel Blockers - therapeutic use | Hypertension - drug therapy | Antihypertensive Agents - classification | Calcium Channels - physiology | Calcium Channels, T-Type - chemistry | Neurotransmitter Agents - secretion | Calcium Channels - drug effects | Calcium Channels, N-Type - drug effects | Cardiovascular Diseases - physiopathology | Kidney - drug effects | Calcium Channels - classification | Rats | Calcium Channels, L-Type - physiology | Antihypertensive Agents - therapeutic use | Arterioles - drug effects | Disease Progression | Antihypertensive Agents - adverse effects | Mice, Knockout | Calcium Signaling - drug effects | Diabetes Mellitus - physiopathology | Models, Biological | Calcium Channels - chemistry | Calcium Channels, T-Type - drug effects | Mice | Vasodilation - drug effects | Hydronephrosis - physiopathology | Calcium Channel Blockers - adverse effects | Calcium Signaling - physiology | Cardiovascular Diseases - drug therapy | Renal Circulation - physiology | Microcirculation - drug effects | Microcirculation - physiology | Renal Circulation - drug effects | Blood Pressure - drug effects | Calcium Channels, L-Type - chemistry | Kidney Diseases - metabolism | Calcium Channels, T-Type - physiology | Renin - secretion | Aldosterone - physiology | Kidney Diseases - drug therapy | Renin-Angiotensin System - physiology | Calcium Channel Blockers - pharmacology | Hypertension - physiopathology | Animals | Calcium Channels, L-Type - drug effects | Calcium Channels, N-Type - physiology | Calcium Channels, N-Type - chemistry
Journal Article
Journal of Neurochemistry, ISSN 0022-3042, 10/2016, Volume 139, Issue S1, pp. 156 - 178
We propose that SN DA neurons possess several feedback and feed‐forward mechanisms to protect and adapt their activity‐pattern and calcium‐homeostasis within a... 
Kir6.2 | SUR | 2 channels | hip3 potassium channels | 1 potassium channels | D2‐autoreceptor‐coupled | GIRK | neuronal calcium sensor | Cav1.3 L‐type/Cav3.1 T‐type calcium channels | ambroxol | A‐type Kv4.3 | NCS | Cav1.3 L-type/Cav3.1 T-type calcium channels | neuronal calcium sensor NCS-1 | A-type Kv4.3/KChip3 potassium channels | Kir6.2/SUR1 potassium channels | D2-autoreceptor-coupled GIRK2 channels | SENSITIVE POTASSIUM CHANNELS | Kir6 | Cav3 | OXIDANT STRESS | Cav1 | K-ATP CHANNELS | BIOCHEMISTRY & MOLECULAR BIOLOGY | CA2+ CHANNELS | A-type Kv4 | GLUCOCEREBROSIDASE MUTATIONS | NEUROSCIENCES | KChip3 potassium channels | 3L-type | D2-AUTORECEPTOR RESPONSES | ALPHA-SYNUCLEIN FORM | SUR1 potassium channels | LOCUS-COERULEUS | MPTP MOUSE MODEL | MITOCHONDRIAL-DNA DELETIONS | 1 T-type calcium channels | Stress, Physiological - physiology | Parkinson Disease - pathology | Animals | Calcium - physiology | Substantia Nigra - pathology | Dopaminergic Neurons - pathology | Dopaminergic Neurons - metabolism | Humans | Ion Channels - physiology | Parkinson Disease - metabolism | Substantia Nigra - metabolism | Health Status | Calcium channels | Stress (Physiology) | Parkinson's disease | Potassium channels | Neurons | Dopamine | Calcium | Animal behavior | Rodents | Bandwidths | Parkinsons disease | Homeostasis | Physiology | Metabolism | Cav3.1 T‐type calcium channels | D2‐autoreceptor‐coupled GIRK2 channels | Cav1.3 L‐type | Jörg B. Schulz and John Hardy | Review | neuronal calcium sensor NCS‐1 | Parkinson Disease. Guest Editors
Journal Article