Carcinogenesis, ISSN 0143-3334, 10/2015, Volume 37, Issue 3, pp. 290 - 300
We demonstrate that IL6 regulates the cellular behavior of human HCC through NHE1, NCX1 and CaM molecular complex-mediated signaling mechanisms, which reveals...
LIVER-CANCER | CELLS | NHE-1 | ONCOLOGY | CA2 | ROLES | INFLAMMATION | SODIUM-CALCIUM EXCHANGE | PROLIFERATION | EXPRESSION | IL-6 | Immunohistochemistry | Calmodulin - metabolism | Immunoprecipitation | Humans | Male | Blotting, Western | Gene Knockdown Techniques | Sodium-Hydrogen Exchangers - metabolism | Animals | Heterografts | Cation Transport Proteins - metabolism | Mice, Nude | Sodium-Calcium Exchanger - metabolism | Carcinoma, Hepatocellular - pathology | Cell Line, Tumor | Liver Neoplasms - pathology | Mice | Mice, Inbred BALB C | RNA, Small Interfering | Gene Expression Regulation, Neoplastic - physiology | Interleukin-6 - metabolism | Real-Time Polymerase Chain Reaction | Sodium-Hydrogen Exchanger 1 | Carcinoma, Hepatocellular - metabolism | Index Medicus
LIVER-CANCER | CELLS | NHE-1 | ONCOLOGY | CA2 | ROLES | INFLAMMATION | SODIUM-CALCIUM EXCHANGE | PROLIFERATION | EXPRESSION | IL-6 | Immunohistochemistry | Calmodulin - metabolism | Immunoprecipitation | Humans | Male | Blotting, Western | Gene Knockdown Techniques | Sodium-Hydrogen Exchangers - metabolism | Animals | Heterografts | Cation Transport Proteins - metabolism | Mice, Nude | Sodium-Calcium Exchanger - metabolism | Carcinoma, Hepatocellular - pathology | Cell Line, Tumor | Liver Neoplasms - pathology | Mice | Mice, Inbred BALB C | RNA, Small Interfering | Gene Expression Regulation, Neoplastic - physiology | Interleukin-6 - metabolism | Real-Time Polymerase Chain Reaction | Sodium-Hydrogen Exchanger 1 | Carcinoma, Hepatocellular - metabolism | Index Medicus
Journal Article
Heart Rhythm, ISSN 1547-5271, 2017, Volume 14, Issue 8, pp. 1247 - 1253
Background The cardiac sodium/calcium (Na+ /Ca2+ ) exchanger (NCX) contributes to diastolic depolarization in cardiac pacemaker cells. Increased NCX activity...
Cardiovascular | Heart rate | Heart failure | Langendorff-perfused heart | Sodium/calcium exchanger | Sinus node | Mouse | Beta-adrenergic stimulation | Atrioventricular node | CARDIAC & CARDIOVASCULAR SYSTEMS | PACEMAKER ACTIVITY | NA+-CA2+ EXCHANGER | SARCOPLASMIC-RETICULUM | VENTRICULAR MYOCYTES | RETICULUM CA2+ LEAK | SINOATRIAL NODE | NA+/CA2+ EXCHANGER | RYANODINE RECEPTOR | CHRONIC ATRIAL-FIBRILLATION | CARDIAC ELECTROPHYSIOLOGY | Calcium - metabolism | Myocardial Contraction - physiology | Sinoatrial Node - physiopathology | Adrenergic beta-Agonists - pharmacology | Mice, Transgenic | Heart Failure - metabolism | Heart Failure - drug therapy | Sodium-Calcium Exchanger - drug effects | Myocytes, Cardiac - pathology | Animals | Isoproterenol - pharmacology | Sodium-Calcium Exchanger - biosynthesis | Heart Rate - physiology | Myocytes, Cardiac - metabolism | Sinoatrial Node - metabolism | Mice | Disease Models, Animal | Heart | Heart beat | Analysis | Atrial fibrillation | Genetic engineering | Cardiovascular agents | Index Medicus
Cardiovascular | Heart rate | Heart failure | Langendorff-perfused heart | Sodium/calcium exchanger | Sinus node | Mouse | Beta-adrenergic stimulation | Atrioventricular node | CARDIAC & CARDIOVASCULAR SYSTEMS | PACEMAKER ACTIVITY | NA+-CA2+ EXCHANGER | SARCOPLASMIC-RETICULUM | VENTRICULAR MYOCYTES | RETICULUM CA2+ LEAK | SINOATRIAL NODE | NA+/CA2+ EXCHANGER | RYANODINE RECEPTOR | CHRONIC ATRIAL-FIBRILLATION | CARDIAC ELECTROPHYSIOLOGY | Calcium - metabolism | Myocardial Contraction - physiology | Sinoatrial Node - physiopathology | Adrenergic beta-Agonists - pharmacology | Mice, Transgenic | Heart Failure - metabolism | Heart Failure - drug therapy | Sodium-Calcium Exchanger - drug effects | Myocytes, Cardiac - pathology | Animals | Isoproterenol - pharmacology | Sodium-Calcium Exchanger - biosynthesis | Heart Rate - physiology | Myocytes, Cardiac - metabolism | Sinoatrial Node - metabolism | Mice | Disease Models, Animal | Heart | Heart beat | Analysis | Atrial fibrillation | Genetic engineering | Cardiovascular agents | Index Medicus
Journal Article
JOURNAL OF GENERAL PHYSIOLOGY, ISSN 0022-1295, 12/2005, Volume 126, Issue 6, pp. 563 - 570
CLC-ec1 is a prokaryotic CLC-type Cl-/H+ exchange transporter. Little is known about the mechanism of H+ coupling to Cl-. A critical glutamate residue, E148,...
CLC CHLORIDE CHANNELS | SITE | PHYSIOLOGY | TRANSPORTERS | ESCHERICHIA-COLI | PROKARYOTIC HOMOLOG | CARDIAC NA+/CA2+ EXCHANGER | GATE | PROTEINS | SELECTIVITY | Amino Acid Sequence | Chloride Channels - chemistry | Proton Pumps - chemistry | Molecular Sequence Data | Crystallography | Proton Pumps - metabolism | Chloride Channels - metabolism | Escherichia coli - genetics | Chlorides - metabolism | Protein Conformation | Ion Transport | Mutation | Hydrogen-Ion Concentration
CLC CHLORIDE CHANNELS | SITE | PHYSIOLOGY | TRANSPORTERS | ESCHERICHIA-COLI | PROKARYOTIC HOMOLOG | CARDIAC NA+/CA2+ EXCHANGER | GATE | PROTEINS | SELECTIVITY | Amino Acid Sequence | Chloride Channels - chemistry | Proton Pumps - chemistry | Molecular Sequence Data | Crystallography | Proton Pumps - metabolism | Chloride Channels - metabolism | Escherichia coli - genetics | Chlorides - metabolism | Protein Conformation | Ion Transport | Mutation | Hydrogen-Ion Concentration
Journal Article
Pflügers Archiv - European Journal of Physiology, ISSN 0031-6768, 6/2015, Volume 467, Issue 6, pp. 1277 - 1290
Bile acids play important physiological role in the solubilisation and absorption of dietary lipids. However, under pathophysiological conditions, such as...
Biomedicine | Bile acids | Human Physiology | Intracellular Ca 2 | Colonic epithelial cells | Ion transporters | Intracellular Ca | CONGENITAL CHLORIDE DIARRHEA | PHYSIOLOGY | INOSITOL TRISPHOSPHATE RECEPTORS | INTRACELLULAR CALCIUM | ULCERATIVE-COLITIS | GUINEA-PIG | ENDOPLASMIC-RETICULUM | PANCREATIC-DUCT CELLS | Intracellular Ca2 | POSTCHOLECYSTECTOMY DIARRHEA | BICARBONATE SECRETION | Intestinal Mucosa - metabolism | Humans | Middle Aged | Cells, Cultured | Ileum - metabolism | Colon - metabolism | Intestinal Mucosa - drug effects | Sodium-Hydrogen Exchangers - metabolism | Chloride-Bicarbonate Antiporters - metabolism | Gastrointestinal Agents - pharmacology | Adenosine Triphosphate - metabolism | Membrane Potential, Mitochondrial | Chenodeoxycholic Acid - pharmacology | Adult | Calcium Signaling
Biomedicine | Bile acids | Human Physiology | Intracellular Ca 2 | Colonic epithelial cells | Ion transporters | Intracellular Ca | CONGENITAL CHLORIDE DIARRHEA | PHYSIOLOGY | INOSITOL TRISPHOSPHATE RECEPTORS | INTRACELLULAR CALCIUM | ULCERATIVE-COLITIS | GUINEA-PIG | ENDOPLASMIC-RETICULUM | PANCREATIC-DUCT CELLS | Intracellular Ca2 | POSTCHOLECYSTECTOMY DIARRHEA | BICARBONATE SECRETION | Intestinal Mucosa - metabolism | Humans | Middle Aged | Cells, Cultured | Ileum - metabolism | Colon - metabolism | Intestinal Mucosa - drug effects | Sodium-Hydrogen Exchangers - metabolism | Chloride-Bicarbonate Antiporters - metabolism | Gastrointestinal Agents - pharmacology | Adenosine Triphosphate - metabolism | Membrane Potential, Mitochondrial | Chenodeoxycholic Acid - pharmacology | Adult | Calcium Signaling
Journal Article
Glia, ISSN 0894-1491, 11/2018, Volume 66, Issue 11, pp. 2279 - 2298
Na+/H+ exchanger (NHE1) activation is required for multiple microglial functions. We investigated effects of selective deletion of microglial Nhe1 in...
phagocytosis | macrophages | inflammation | microglia | white matter tissue repair | SURVIVAL | BRAIN-INJURY | FOCAL CEREBRAL-ISCHEMIA | RECEPTOR | CNS | NEUROSCIENCES | NEURONAL DEATH | GROWTH | MICE | EXPRESSION | Microglia - metabolism | Somatosensory Disorders - etiology | Brain - diagnostic imaging | Recovery of Function - drug effects | Male | CX3C Chemokine Receptor 1 - metabolism | CX3C Chemokine Receptor 1 - genetics | Infarction, Middle Cerebral Artery - complications | Recovery of Function - physiology | Female | Demyelinating Diseases - etiology | Infarction, Middle Cerebral Artery - drug therapy | Microfilament Proteins - metabolism | Infarction, Middle Cerebral Artery - diagnostic imaging | Demyelinating Diseases - drug therapy | Disease Models, Animal | Calcium-Binding Proteins - metabolism | Macrophages - pathology | Microglia - drug effects | Gene Expression Regulation - genetics | Mice, Inbred C57BL | Sodium-Hydrogen Exchanger 1 - genetics | Mice, Transgenic | Nerve Tissue Proteins - genetics | White Matter - pathology | Brain - drug effects | Gene Expression Regulation - drug effects | Nerve Tissue Proteins - metabolism | Macrophages - metabolism | White Matter - diagnostic imaging | Animals | Tamoxifen - pharmacology | Sodium-Hydrogen Exchanger 1 - metabolism | Mice | Stroke (Disease) | Neurons | Analysis | Brain damage | Bone morphogenetic proteins | Inflammation | Macrophages | Transforming growth factors | Brain | CD86 antigen | Hydrogen | CX3CR1 protein | Activation | Recovery | Proteins | Cell activation | Corn oil | Clonal deletion | Ischemia | Demyelination | Rodents | Animal tissues | Deletion | Lesions | Repair | Stroke | CD11b antigen | Cell survival | Therapeutic applications | Astrocytes | Na+/H+-exchanging ATPase | Glial fibrillary acidic protein | Tamoxifen | Substantia alba | Survival | Microglia | Myelination | Brain injury | Ca2+/calmodulin-dependent protein kinase II
phagocytosis | macrophages | inflammation | microglia | white matter tissue repair | SURVIVAL | BRAIN-INJURY | FOCAL CEREBRAL-ISCHEMIA | RECEPTOR | CNS | NEUROSCIENCES | NEURONAL DEATH | GROWTH | MICE | EXPRESSION | Microglia - metabolism | Somatosensory Disorders - etiology | Brain - diagnostic imaging | Recovery of Function - drug effects | Male | CX3C Chemokine Receptor 1 - metabolism | CX3C Chemokine Receptor 1 - genetics | Infarction, Middle Cerebral Artery - complications | Recovery of Function - physiology | Female | Demyelinating Diseases - etiology | Infarction, Middle Cerebral Artery - drug therapy | Microfilament Proteins - metabolism | Infarction, Middle Cerebral Artery - diagnostic imaging | Demyelinating Diseases - drug therapy | Disease Models, Animal | Calcium-Binding Proteins - metabolism | Macrophages - pathology | Microglia - drug effects | Gene Expression Regulation - genetics | Mice, Inbred C57BL | Sodium-Hydrogen Exchanger 1 - genetics | Mice, Transgenic | Nerve Tissue Proteins - genetics | White Matter - pathology | Brain - drug effects | Gene Expression Regulation - drug effects | Nerve Tissue Proteins - metabolism | Macrophages - metabolism | White Matter - diagnostic imaging | Animals | Tamoxifen - pharmacology | Sodium-Hydrogen Exchanger 1 - metabolism | Mice | Stroke (Disease) | Neurons | Analysis | Brain damage | Bone morphogenetic proteins | Inflammation | Macrophages | Transforming growth factors | Brain | CD86 antigen | Hydrogen | CX3CR1 protein | Activation | Recovery | Proteins | Cell activation | Corn oil | Clonal deletion | Ischemia | Demyelination | Rodents | Animal tissues | Deletion | Lesions | Repair | Stroke | CD11b antigen | Cell survival | Therapeutic applications | Astrocytes | Na+/H+-exchanging ATPase | Glial fibrillary acidic protein | Tamoxifen | Substantia alba | Survival | Microglia | Myelination | Brain injury | Ca2+/calmodulin-dependent protein kinase II
Journal Article
Hypertension, ISSN 0194-911X, 02/2009, Volume 53, Issue 2, Part 2 Suppl, pp. 291 - 298
RENIN-ANGIOTENSIN SYSTEM | PHYSIOLOGICAL IMPLICATIONS | NA+/CA2+ EXCHANGERS | SENSITIVE HYPERTENSION | PERIPHERAL VASCULAR DISEASE | SODIUM-CALCIUM EXCHANGE | NA+-CA2+ EXCHANGER | BLOOD-PRESSURE REGULATION | MINERALOCORTICOID-INDUCED HYPERTENSION | VASCULAR SMOOTH-MUSCLE | NA+-K+-ATPASE | Sodium-Potassium-Exchanging ATPase - metabolism | Animals | Sodium-Calcium Exchanger - metabolism | Humans | Signal Transduction - physiology | Sodium Chloride - metabolism | Disease Models, Animal | Hypertension - metabolism | Ouabain - metabolism | Sodium Pump | Myogenic Tone | Calcium | Sodium | Salt-dependent hypertension | Calcium Exchanger
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 1/2011, Volume 108, Issue 4, pp. 1699 - 1704
Cytoplasmic Ca²⺠is known to regulate NaāŗāCa²⺠exchanger (NCX) activity by binding to two adjacent Ca²āŗ-binding domains (CBD1 and CBD2) located in the large...
Oligomers | Ratios | Fluorescence | Dimers | Cell membranes | Physiological regulation | Kinetics | Energy transfer | Oocytes | Crystal structure | DYNAMIC PROPERTIES | CONFORMATIONAL-CHANGES | NA+/CA2+ EXCHANGER | MULTIDISCIPLINARY SCIENCES | RESONANCE ENERGY-TRANSFER | CA2+ REGULATION | SODIUM-CALCIUM EXCHANGE | STEADY-STATE | CA2+-BINDING DOMAIN | PLASMA-MEMBRANE | MUTATIONAL ANALYSIS | Calcium - pharmacology | Calcium - metabolism | Oocytes - metabolism | Humans | Protein Conformation - drug effects | Protein Multimerization | Sodium-Calcium Exchanger - chemistry | Xenopus laevis | Cytoplasm - metabolism | Sodium-Calcium Exchanger - genetics | Binding Sites - genetics | Animals | Oocytes - physiology | Membrane Potentials | Sodium-Calcium Exchanger - metabolism | Dogs | HEK293 Cells | Fluorescence Resonance Energy Transfer | Female | Luminescent Proteins - genetics | Cell Membrane - metabolism | Mutation | Luminescent Proteins - metabolism | Biological Sciences
Oligomers | Ratios | Fluorescence | Dimers | Cell membranes | Physiological regulation | Kinetics | Energy transfer | Oocytes | Crystal structure | DYNAMIC PROPERTIES | CONFORMATIONAL-CHANGES | NA+/CA2+ EXCHANGER | MULTIDISCIPLINARY SCIENCES | RESONANCE ENERGY-TRANSFER | CA2+ REGULATION | SODIUM-CALCIUM EXCHANGE | STEADY-STATE | CA2+-BINDING DOMAIN | PLASMA-MEMBRANE | MUTATIONAL ANALYSIS | Calcium - pharmacology | Calcium - metabolism | Oocytes - metabolism | Humans | Protein Conformation - drug effects | Protein Multimerization | Sodium-Calcium Exchanger - chemistry | Xenopus laevis | Cytoplasm - metabolism | Sodium-Calcium Exchanger - genetics | Binding Sites - genetics | Animals | Oocytes - physiology | Membrane Potentials | Sodium-Calcium Exchanger - metabolism | Dogs | HEK293 Cells | Fluorescence Resonance Energy Transfer | Female | Luminescent Proteins - genetics | Cell Membrane - metabolism | Mutation | Luminescent Proteins - metabolism | Biological Sciences
Journal Article
The Journal of Physiology, ISSN 0022-3751, 04/2013, Volume 591, Issue 8, pp. 2067 - 2086
Key points ā¢ā Calciumādependent Na+āCa2+ exchange (NCX) activation undergoes bidirectional up/down control in intact rabbit cardiomyocytes. ā¢ā In rested cells...
PHYSIOLOGY | HAMSTER OVARY CELLS | NA+/CA2+ EXCHANGER | INTRACELLULAR CALCIUM | SODIUM-CALCIUM EXCHANGE | NA+-CA2+ EXCHANGER | NA-CA EXCHANGE | SARCOPLASMIC-RETICULUM | RAT VENTRICULAR MYOCYTES | CA2+-BINDING DOMAIN | NEUROSCIENCES | ISOLATED HEART-CELLS | Rabbits | Animals | Calcium - physiology | Models, Biological | Myocytes, Cardiac - physiology | Sarcoplasmic Reticulum - physiology | Mice | Sodium-Calcium Exchanger - physiology | In Vitro Techniques | Sodium - physiology | Molecular and Cellular
PHYSIOLOGY | HAMSTER OVARY CELLS | NA+/CA2+ EXCHANGER | INTRACELLULAR CALCIUM | SODIUM-CALCIUM EXCHANGE | NA+-CA2+ EXCHANGER | NA-CA EXCHANGE | SARCOPLASMIC-RETICULUM | RAT VENTRICULAR MYOCYTES | CA2+-BINDING DOMAIN | NEUROSCIENCES | ISOLATED HEART-CELLS | Rabbits | Animals | Calcium - physiology | Models, Biological | Myocytes, Cardiac - physiology | Sarcoplasmic Reticulum - physiology | Mice | Sodium-Calcium Exchanger - physiology | In Vitro Techniques | Sodium - physiology | Molecular and Cellular
Journal Article
British journal of pharmacology, ISSN 0007-1188, 2008, Volume 154, Issue 6, pp. 1266 - 1275
Background and purpose:Increased activity of the Na(+)/H(+)-exchanger (NHE-1) in heart failure underlies raised [Na(+)](i) causing disturbances of calcium...
SR function | heart failure | sodium | arrhythmias | Na/H exchanger | regression | Na/Ca exchanger | remodelling | Na channel | calcium (cellular) | Heart failure | Remodelling | Calcium (cellular) | Sodium | Regression | Arrhythmias | OVERLOAD | NHE-1 | MYOCARDIAL-INFARCTION | CARDIAC MYOCYTES | ANGIOTENSIN-CONVERTING ENZYME | SODIUM-HYDROGEN EXCHANGER | NA+/CA2+ EXCHANGER | RABBIT VENTRICULAR MYOCYTES | CALCIUM | PHARMACOLOGY & PHARMACY | SMOOTH-MUSCLE-CELLS | Cardiomyopathy, Dilated - pathology | Calcium - metabolism | Sarcoplasmic Reticulum - drug effects | Anti-Arrhythmia Agents - pharmacology | Electrophysiology | Cardiomegaly - pathology | Guanidines - pharmacology | Ion Channels - drug effects | Male | Sodium - metabolism | Sulfones - pharmacology | Cardiomyopathy, Dilated - drug therapy | Sodium Channels - metabolism | Sarcoplasmic Reticulum - metabolism | Action Potentials - drug effects | Sodium-Hydrogen Exchangers - antagonists & inhibitors | Rabbits | Cardiomegaly - drug therapy | Enzyme Inhibitors - pharmacology | Cardiomegaly - physiopathology | Sodium Channels - drug effects | Animals | Cardiomyopathy, Dilated - physiopathology | Ion Channels - metabolism | Calcium Signaling - drug effects | In Vitro Techniques | Ca exchanger | Research Papers | H exchanger
SR function | heart failure | sodium | arrhythmias | Na/H exchanger | regression | Na/Ca exchanger | remodelling | Na channel | calcium (cellular) | Heart failure | Remodelling | Calcium (cellular) | Sodium | Regression | Arrhythmias | OVERLOAD | NHE-1 | MYOCARDIAL-INFARCTION | CARDIAC MYOCYTES | ANGIOTENSIN-CONVERTING ENZYME | SODIUM-HYDROGEN EXCHANGER | NA+/CA2+ EXCHANGER | RABBIT VENTRICULAR MYOCYTES | CALCIUM | PHARMACOLOGY & PHARMACY | SMOOTH-MUSCLE-CELLS | Cardiomyopathy, Dilated - pathology | Calcium - metabolism | Sarcoplasmic Reticulum - drug effects | Anti-Arrhythmia Agents - pharmacology | Electrophysiology | Cardiomegaly - pathology | Guanidines - pharmacology | Ion Channels - drug effects | Male | Sodium - metabolism | Sulfones - pharmacology | Cardiomyopathy, Dilated - drug therapy | Sodium Channels - metabolism | Sarcoplasmic Reticulum - metabolism | Action Potentials - drug effects | Sodium-Hydrogen Exchangers - antagonists & inhibitors | Rabbits | Cardiomegaly - drug therapy | Enzyme Inhibitors - pharmacology | Cardiomegaly - physiopathology | Sodium Channels - drug effects | Animals | Cardiomyopathy, Dilated - physiopathology | Ion Channels - metabolism | Calcium Signaling - drug effects | In Vitro Techniques | Ca exchanger | Research Papers | H exchanger
Journal Article
Clinical and Experimental Pharmacology and Physiology, ISSN 0305-1870, 08/2008, Volume 35, Issue 8, pp. 863 - 871
SUMMARY ⢠The Na+/H+ exchanger NHE3 associates with the actin cytoskeleton by binding ezrin both directly and indirectly. Both types of interaction are...
Na absorption | Na+/H+ exchanger isoform 3 (NHE3)PDZ domains | epithelial cells | ezrin | intestine | exchanger isoform 3 (NHE3)PDZ domains | Epithelial cells | Intestine | Ezrin | CYTOPLASMIC DOMAIN | PHYSIOLOGY | CA2+-DEPENDENT INHIBITION | PHOSPHATIDYLINOSITOL 3-KINASE | CRYSTAL-STRUCTURE | MICROVILLI-LIKE STRUCTURES | GLUCOSE COTRANSPORT | PLASMA-MEMBRANE | TRANSMEMBRANE CONDUCTANCE REGULATOR | EZRIN/RADIXIN/MOESIN ERM PROTEINS | Na+/H+ exchanger isoform 3 (NHE3) PDZ domains | ENDOTHELIAL-CELLS | PHARMACOLOGY & PHARMACY | Epithelial Cells - metabolism | Microvilli - metabolism | Cytoskeleton - metabolism | Epithelial Cells - ultrastructure | PDZ Domains | Protein Binding | Cytoskeletal Proteins - metabolism | Phosphoproteins - metabolism | Sodium-Hydrogen Exchangers - metabolism | Cytoskeletal Proteins, metabolism | Epithelial Cells, ultrastructure | Phosphoproteins, metabolism | Microvilli, metabolism | Cytoskeleton, metabolism | Epithelial Cells, metabolism | Sodium-Hydrogen Antiporter, metabolism
Na absorption | Na+/H+ exchanger isoform 3 (NHE3)PDZ domains | epithelial cells | ezrin | intestine | exchanger isoform 3 (NHE3)PDZ domains | Epithelial cells | Intestine | Ezrin | CYTOPLASMIC DOMAIN | PHYSIOLOGY | CA2+-DEPENDENT INHIBITION | PHOSPHATIDYLINOSITOL 3-KINASE | CRYSTAL-STRUCTURE | MICROVILLI-LIKE STRUCTURES | GLUCOSE COTRANSPORT | PLASMA-MEMBRANE | TRANSMEMBRANE CONDUCTANCE REGULATOR | EZRIN/RADIXIN/MOESIN ERM PROTEINS | Na+/H+ exchanger isoform 3 (NHE3) PDZ domains | ENDOTHELIAL-CELLS | PHARMACOLOGY & PHARMACY | Epithelial Cells - metabolism | Microvilli - metabolism | Cytoskeleton - metabolism | Epithelial Cells - ultrastructure | PDZ Domains | Protein Binding | Cytoskeletal Proteins - metabolism | Phosphoproteins - metabolism | Sodium-Hydrogen Exchangers - metabolism | Cytoskeletal Proteins, metabolism | Epithelial Cells, ultrastructure | Phosphoproteins, metabolism | Microvilli, metabolism | Cytoskeleton, metabolism | Epithelial Cells, metabolism | Sodium-Hydrogen Antiporter, metabolism
Journal Article
Fundamental & Clinical Pharmacology, ISSN 0767-3981, 02/2019, Volume 33, Issue 1, pp. 41 - 42
Journal Article
American Journal of Physiology - Cell Physiology, ISSN 0363-6143, 04/2011, Volume 300, Issue 4, pp. C771 - C782
Sarker R, Valkhoff VE, Zachos NC, Lin R, Cha B, Chen T, Guggino S, Zizak M, de Jonge H, Hogema B, Donowitz M. NHERF1 and NHERF2 are necessary for multiple but...
Knockdown | exchanger regulatory factor 2 | exchanger regulatory factor 1 | Intestinal sodium absorption | exchanger 3 | CAMP-MEDIATED INHIBITION | ILEAL BRUSH-BORDER | PDZ-DOMAIN | PHYSIOLOGY | CA2+-DEPENDENT INHIBITION | knockdown | Na+/H+ exchanger 3 | PROTEIN-COUPLED RECEPTORS | CELL BIOLOGY | TRANSMEMBRANE CONDUCTANCE REGULATOR | RENAL PROXIMAL TUBULE | intestinal sodium absorption | C-TERMINAL DOMAINS | Na+/H+ exchanger regulatory factor 1 | Na+/H+ exchanger regulatory factor 2 | CYSTIC-FIBROSIS | BORDER NA+/H+ EXCHANGER | Caco-2 Cells | RNA, Small Interfering - genetics | Epithelial Cells - metabolism | Humans | Epidermal Growth Factor - metabolism | Cholinergic Agonists - metabolism | Phosphoproteins - genetics | Phosphoproteins - metabolism | Sodium-Hydrogen Exchangers - metabolism | Animals | Cyclic GMP - metabolism | Microvilli - metabolism | Carbachol - metabolism | Adenoviridae - genetics | Sodium-Hydrogen Exchangers - genetics | Mice | Adenoviridae - metabolism | Epithelial Cells - cytology | Genetic Vectors | Cyclic AMP - metabolism | Sodium-Hydrogen Exchangers - antagonists & inhibitors | RNA, Small Interfering - metabolism | Sodium-Hydrogen Exchanger 3 | Proteins | Physiological aspects | Epithelial cells | Health aspects | Na+ | H+ exchanger 3 | H+ exchanger regulatory factor 1 | Membrane Transporters, Ion Channels and Pumps | H+ exchanger regulatory factor 2
Knockdown | exchanger regulatory factor 2 | exchanger regulatory factor 1 | Intestinal sodium absorption | exchanger 3 | CAMP-MEDIATED INHIBITION | ILEAL BRUSH-BORDER | PDZ-DOMAIN | PHYSIOLOGY | CA2+-DEPENDENT INHIBITION | knockdown | Na+/H+ exchanger 3 | PROTEIN-COUPLED RECEPTORS | CELL BIOLOGY | TRANSMEMBRANE CONDUCTANCE REGULATOR | RENAL PROXIMAL TUBULE | intestinal sodium absorption | C-TERMINAL DOMAINS | Na+/H+ exchanger regulatory factor 1 | Na+/H+ exchanger regulatory factor 2 | CYSTIC-FIBROSIS | BORDER NA+/H+ EXCHANGER | Caco-2 Cells | RNA, Small Interfering - genetics | Epithelial Cells - metabolism | Humans | Epidermal Growth Factor - metabolism | Cholinergic Agonists - metabolism | Phosphoproteins - genetics | Phosphoproteins - metabolism | Sodium-Hydrogen Exchangers - metabolism | Animals | Cyclic GMP - metabolism | Microvilli - metabolism | Carbachol - metabolism | Adenoviridae - genetics | Sodium-Hydrogen Exchangers - genetics | Mice | Adenoviridae - metabolism | Epithelial Cells - cytology | Genetic Vectors | Cyclic AMP - metabolism | Sodium-Hydrogen Exchangers - antagonists & inhibitors | RNA, Small Interfering - metabolism | Sodium-Hydrogen Exchanger 3 | Proteins | Physiological aspects | Epithelial cells | Health aspects | Na+ | H+ exchanger 3 | H+ exchanger regulatory factor 1 | Membrane Transporters, Ion Channels and Pumps | H+ exchanger regulatory factor 2
Journal Article