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Journal of Experimental Medicine, ISSN 0022-1007, 06/2013, Volume 210, Issue 6, pp. 1137 - 1151
Journal Article
Nature Medicine, ISSN 1078-8956, 11/2015, Volume 21, Issue 11, pp. 1262 - 1271
Cancer-associated muscle weakness is a poorly understood phenomenon, and there is no effective treatment. Here we find that seven different mouse models of... 
MEDICINE, RESEARCH & EXPERIMENTAL | CANCER CACHEXIA | CELLS | INTRACELLULAR CALCIUM | BIOCHEMISTRY & MOLECULAR BIOLOGY | RELEASE | CELL BIOLOGY | DYSTROPHIC MUSCLE | GROWTH-FACTOR-BETA | HORMONE-RELATED PROTEIN | RYANODINE RECEPTOR | CAMURATI-ENGELMANN-DISEASE | IN-VIVO | Neoplasms - metabolism | Prostatic Neoplasms - metabolism | Up-Regulation | Calcium - metabolism | Humans | Lung Neoplasms - metabolism | NADPH Oxidases - metabolism | Bone Neoplasms - secondary | Lung Neoplasms - pathology | Male | Muscle, Skeletal - metabolism | Osteolysis - etiology | Ryanodine Receptor Calcium Release Channel - metabolism | X-Ray Microtomography | Bone Neoplasms - metabolism | Breast Neoplasms - metabolism | Neoplasms - complications | MCF-7 Cells | Osteolysis - diagnostic imaging | Muscle Proteins - metabolism | NADPH Oxidases - genetics | Female | Camurati-Engelmann Syndrome - metabolism | Muscle Strength | Calcium Signaling | Disease Models, Animal | Muscle Weakness - etiology | Prostatic Neoplasms - pathology | Oxidation-Reduction | Bone Neoplasms - diagnostic imaging | NADPH Oxidase 4 | Mice, SCID | Multiple Myeloma - metabolism | Absorptiometry, Photon | Osteolysis - metabolism | Multiple Myeloma - pathology | Animals | Muscle Contraction | Breast Neoplasms - pathology | Mice, Nude | Muscle Weakness - metabolism | Cell Line, Tumor | Mice | Neoplasms - pathology | Transforming Growth Factor beta - metabolism
Journal Article
Developmental Cell, ISSN 1534-5807, 04/2015, Volume 33, Issue 1, pp. 36 - 46
Organ wasting, related to changes in nutrition and metabolic activity of cells and tissues, is observed under conditions of starvation and in the context... 
CANCER CACHEXIA | GENE | RNA-SEQ | MUSCLE ATROPHY | IN-VIVO | GROWTH | HIPPO PATHWAY | TUMOR-SUPPRESSOR | STEM-CELL PROLIFERATION | DEVELOPMENTAL BIOLOGY | DROSOPHILA | CELL BIOLOGY | Metabolomics | Wasting Syndrome - metabolism | Oligonucleotide Array Sequence Analysis | Male | Muscle, Skeletal - metabolism | Gene Expression Profiling | Stem Cells - cytology | Drosophila Proteins - metabolism | Muscle, Skeletal - cytology | Ovary - cytology | Stem Cells - metabolism | Drosophila melanogaster - genetics | Drosophila melanogaster - metabolism | Fat Body - cytology | Hyperglycemia - pathology | Trans-Activators - genetics | Female | Nuclear Proteins - genetics | Fat Body - metabolism | Insulin Secretion | Real-Time Polymerase Chain Reaction | Ovary - metabolism | Biomarkers - metabolism | RNA, Messenger - genetics | Cells, Cultured | Hemolymph - metabolism | Nuclear Proteins - metabolism | Reverse Transcriptase Polymerase Chain Reaction | Animals, Genetically Modified - metabolism | Blotting, Western | Gastrointestinal Tract - metabolism | Hyperglycemia - metabolism | Insulin - metabolism | Animals | Animals, Genetically Modified - growth & development | Animals, Genetically Modified - genetics | Insulin - chemistry | Drosophila melanogaster - growth & development | Trans-Activators - metabolism | Gastrointestinal Tract - cytology | Drosophila Proteins - genetics | Insulin-Like Growth Factor I - antagonists & inhibitors | Insulin-Like Growth Factor I - metabolism | Wasting Syndrome - pathology | Enzymes | Starvation | Analysis | Stem cells | Physiological aspects | Development and progression | Cellular signal transduction | Ovarian cancer | Medical colleges | Resveratrol
Journal Article
Journal of Biological Chemistry, ISSN 0021-9258, 04/2015, Volume 290, Issue 17, pp. 11177 - 11187
Cachexia occurs in patients with advanced cancers. Despite the adverse clinical impact of cancer-induced muscle wasting, pathways causing cachexia are... 
TRANSCRIPTION FACTORS | SKELETAL-MUSCLE | PROTEIN-DEGRADATION | MYOSTATIN GENE | PATHWAY | ATROPHY | BIOCHEMISTRY & MOLECULAR BIOLOGY | KAPPA-B | UP-REGULATION | EXPRESSION | CATABOLIC CONDITIONS | Colonic Neoplasms - genetics | SKP Cullin F-Box Protein Ligases - genetics | Caspase 3 - metabolism | Ubiquitin - metabolism | Muscle, Skeletal - metabolism | Colonic Neoplasms - metabolism | Tripartite Motif Proteins | Proteolysis | Caspase 3 - genetics | Muscle Proteins - metabolism | Myostatin - genetics | CCAAT-Enhancer-Binding Protein-delta - metabolism | Myostatin - metabolism | Cachexia - metabolism | STAT3 Transcription Factor - genetics | STAT3 Transcription Factor - metabolism | Cachexia - genetics | Ubiquitin-Protein Ligases - metabolism | SKP Cullin F-Box Protein Ligases - metabolism | Ubiquitin - genetics | Carcinoma, Lewis Lung - metabolism | Mice, Knockout | Muscle Proteins - genetics | Proteasome Endopeptidase Complex - genetics | Animals | Carcinoma, Lewis Lung - genetics | CCAAT-Enhancer-Binding Protein-delta - genetics | Cachexia - pathology | Colonic Neoplasms - pathology | Carcinoma, Lewis Lung - pathology | Cell Line, Tumor | Mice | Muscle, Skeletal - pathology | Proteasome Endopeptidase Complex - metabolism | Ubiquitin-Protein Ligases - genetics | Ubiquitin | Signal Transduction | STAT3 | Caspase | Cancer Cachexia | Cancer Biology | Muscle Atrophy | Caspase-3
Journal Article
The EMBO Journal, ISSN 0261-4189, 10/2011, Volume 30, Issue 20, pp. 4323 - 4335
Journal Article
Journal of the National Cancer Institute, ISSN 0027-8874, 12/2015, Volume 107, Issue 12, p. djv274
Background: Cancer cachexia is a debilitating condition that impacts patient morbidity, mortality, and quality of life and for which effective therapies are... 
SKELETAL-MUSCLE ATROPHY | MURINE MODEL | FOXO | OXIDATIVE STRESS | TUMOR-BEARING MICE | ONCOLOGY | INSULIN-RESISTANCE | ADENOCARCINOMA | BIOCHEMISTRY | HDAC COMPLEXES | MECHANISMS | Cachexia - drug therapy | Histone Deacetylase Inhibitors - administration & dosage | Muscle, Skeletal - metabolism | Weight Loss - drug effects | Leukemia Inhibitory Factor - metabolism | Tripartite Motif Proteins | Carcinoma, Lewis Lung - complications | Forkhead Transcription Factors - metabolism | Mitochondrial Proteins - metabolism | Muscle, Skeletal - drug effects | Phenylbutyrates - administration & dosage | Cachexia - etiology | Muscle Proteins - metabolism | Phenylbutyrates - pharmacology | Cachexia - metabolism | Interleukin-6 - metabolism | Cytokines - metabolism | Neoplasms, Experimental - complications | Administration, Oral | Ubiquitin-Protein Ligases - metabolism | SKP Cullin F-Box Protein Ligases - metabolism | Lipase - metabolism | Adenocarcinoma - complications | Cachexia - prevention & control | Colonic Neoplasms - complications | Gene Expression Regulation - drug effects | Animals | Cytokines - drug effects | Ion Channels - metabolism | Survival Analysis | MEF2 Transcription Factors - metabolism | Histone Deacetylase Inhibitors - pharmacology | Forkhead Box Protein O1 | Mice | Muscle Strength - drug effects | Adipose Tissue - drug effects | Uncoupling Protein 3 | Cytokines - biosynthesis | Receptors, Interleukin-6 - metabolism | Care and treatment | Synthesis | Proteolysis | Glycogen | Patient outcomes | Colorectal cancer | Development and progression | Research
Journal Article
American Journal of Physiology - Regulatory Integrative and Comparative Physiology, ISSN 0363-6119, 02/2011, Volume 300, Issue 2, pp. R201 - R211
White JP, Baltgalvis KA, Puppa MJ, Sato S, Baynes JW, Carson JA. Muscle oxidative capacity during IL-6-dependent cancer cachexia. Am J Physiol Regul Integr... 
Wasting | Mitochondria | Fission 1 protein | Mitofusin-2 protein | 2A GENE | wasting | TUMOR-BEARING RATS | PHYSIOLOGY | mitochondria | fission 1 protein | PGC-1-ALPHA | HEART-FAILURE | APC(MIN/+) MICE | EXERCISE CAPACITY | HUMAN SKELETAL-MUSCLE | GENE-EXPRESSION | mitofusin-2 protein | MAMMALIAN MITOCHONDRIAL FISSION | STRESS | Superoxide Dismutase - genetics | Body Weight | Gene Expression - genetics | Mitochondria, Muscle - metabolism | Muscle Fibers, Fast-Twitch - metabolism | Oxidative Stress - physiology | Muscle, Skeletal - metabolism | Mitochondrial Proteins - genetics | Muscle Fibers, Slow-Twitch - metabolism | Aldehydes - metabolism | Sirtuin 1 - genetics | Electron Transport Complex IV - metabolism | Genes, APC | Hindlimb - physiopathology | Interleukin-6 - blood | Mitochondrial Proteins - metabolism | Peroxisome Proliferator-Activated Receptor Gamma Coactivator 1-alpha | Cachexia - etiology | Trans-Activators - genetics | Cachexia - metabolism | STAT3 Transcription Factor - metabolism | DNA, Mitochondrial - metabolism | Interleukin-6 - genetics | Catalase - genetics | Cytochromes c - metabolism | Mice, Inbred C57BL | Adipose Tissue - pathology | Oxidative Phosphorylation | Mice, Transgenic | Colonic Neoplasms - complications | Animals | Ion Channels - metabolism | GTP Phosphohydrolases - genetics | Cachexia - pathology | Muscle, Skeletal - physiopathology | Hindlimb - pathology | Succinate Dehydrogenase - metabolism | Trans-Activators - metabolism | Mice | Transcription Factors | Muscle, Skeletal - pathology | Hindlimb - metabolism | Uncoupling Protein 3 | Cachexia - physiopathology | Myosin | Physiological aspects | Cachexia | Research | Risk factors | Interleukin-6 | Call for Papers
Journal Article
American Journal of Physiology - Endocrinology and Metabolism, ISSN 0193-1849, 08/2016, Volume 311, Issue 2, pp. E436 - E448
Burn trauma results in prolonged hypermetabolism and skeletal muscle wasting. How hypermetabolism contributes to muscle wasting in burn patients remains... 
Hypermetabolism-induced oxidative stress | Mitochondria proteases | Cachexia | Burn injury | Mitochondrial unfolded protein response | Up-Regulation | ATP-Dependent Proteases - genetics | Oxidative Stress | TOR Serine-Threonine Kinases - metabolism | Humans | Middle Aged | Mitochondria, Muscle - metabolism | Male | Multiprotein Complexes - genetics | Muscle, Skeletal - metabolism | Mitochondrial Proteins - genetics | RNA, Messenger - metabolism | Case-Control Studies | Mechanistic Target of Rapamycin Complex 1 | Mitochondrial Membrane Transport Proteins - genetics | Young Adult | Multiprotein Complexes - metabolism | TOR Serine-Threonine Kinases - genetics | Mitochondrial Proteins - metabolism | Proteolysis | Cachexia - etiology | Muscle Proteins - metabolism | Adult | Burns - metabolism | Female | Body Surface Area | Burns - complications | Cachexia - metabolism | Real-Time Polymerase Chain Reaction | Mitochondrial Membrane Transport Proteins - metabolism | Cachexia - genetics | Oxygen Consumption | Endopeptidase Clp - genetics | Endopeptidase Clp - metabolism | Mitochondria - metabolism | Metabolism | Blotting, Western | Proteasome Endopeptidase Complex - genetics | Adolescent | Proteasome Endopeptidase Complex - metabolism | ATP-Dependent Proteases - metabolism | Burns - genetics | Physiological aspects | Muscles | Burns and scalds | Mitochondria | Research | Call for Papers | mitochondria proteases | hypermetabolism-induced oxidative stress | burn injury | mitochondrial unfolded protein response | cachexia
Journal Article
International Journal of Biochemistry and Cell Biology, ISSN 1357-2725, 2005, Volume 37, Issue 10, pp. 1974 - 1984
Journal Article