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Philosophical Transactions of the Royal Society B: Biological Sciences, ISSN 0962-8436, 02/2015, Volume 370, Issue 1661, p. 20140039
Journal Article
Gastroenterology, ISSN 0016-5085, 2009, Volume 137, Issue 6, pp. 2136 - 2145.e7
Background & Aims The infiltrating ductal adenocarcinoma of the pancreas is among the most lethal of all solid malignancies, largely owing to a high frequency... 
Gastroenterology and Hepatology | BREAST-CANCER | ZEBRAFISH EMBRYOS | TUMOR INVASION | ANGIOGENESIS | CLUSTERS | ADENOCARCINOMA | GENE-EXPRESSION | E-CADHERIN | MICRORNAS | CELL-LINES | GASTROENTEROLOGY & HEPATOLOGY | Up-Regulation | Pancreatic Neoplasms - metabolism | Cadherins - metabolism | alpha Catenin - metabolism | Homeodomain Proteins - metabolism | Humans | Oligonucleotides, Antisense - metabolism | MicroRNAs - metabolism | Zebrafish - embryology | Receptors, Retinoic Acid - genetics | Pancreatic Neoplasms - secondary | Adenocarcinoma - metabolism | Transfection | Chromans - pharmacology | RNA Interference | Adenocarcinoma - genetics | Gene Expression Regulation, Neoplastic - drug effects | Neoplasm Invasiveness | Receptors, Retinoic Acid - antagonists & inhibitors | Morpholines - metabolism | Pancreatic Neoplasms - genetics | Receptors, Retinoic Acid - metabolism | beta Catenin - metabolism | Adenocarcinoma - secondary | Homeodomain Proteins - genetics | Xenograft Model Antitumor Assays | Blotting, Northern | Animals | Adenocarcinoma - therapy | Benzoates - pharmacology | Cell Line, Tumor | Retinoids - pharmacology | Genetic Therapy - methods | Pancreatic Neoplasms - therapy | RNA, Small Interfering - metabolism | Polymerase chain reaction | Phosphates | Espionage, German | Pancreatic cancer | Dimethyl sulfoxide | Fluorescence | Metastasis
Journal Article
International Journal of Cancer, ISSN 0020-7136, 06/2018, Volume 142, Issue 11, pp. 2394 - 2404
We investigated the effects of novel antagonists of growth hormone releasing hormone (GHRH)—MIA602 and MIA690—on three human small cell lung cancer (SCLC)... 
SCLC | GHRH receptors | signaling pathways | NSCLC | GHRH antagonists | RECEPTOR | PROLIFERATION | SUPPRESSION | BOMBESIN | ONCOLOGY | GHRH | IN-VIVO | PROSTATE-CANCER | TUMOR-GROWTH | CARCINOMA | EXPRESSION | Apoptosis - drug effects | Humans | Small Cell Lung Carcinoma - drug therapy | Small Cell Lung Carcinoma - metabolism | Sermorelin - pharmacology | Sermorelin - analogs & derivatives | Female | Antineoplastic Agents - pharmacology | Cyclic AMP - metabolism | STAT3 Transcription Factor - metabolism | Disease Models, Animal | Cell Survival - drug effects | Gene Expression | Carcinoma, Non-Small-Cell Lung - genetics | Carcinoma, Non-Small-Cell Lung - metabolism | Small Cell Lung Carcinoma - genetics | Growth Hormone-Releasing Hormone - genetics | Xenograft Model Antitumor Assays | Growth Hormone-Releasing Hormone - metabolism | Animals | Signal Transduction - drug effects | Mice, Nude | Cyclic AMP Response Element-Binding Protein - metabolism | Cell Line, Tumor | Cell Proliferation - drug effects | Mice | Carcinoma, Non-Small-Cell Lung - drug therapy | Cell Cycle - drug effects | Growth Hormone-Releasing Hormone - antagonists & inhibitors | Cell Movement | Somatotropin | Somatotropin releasing hormone | Lung cancer, Non-small cell | Analysis | Protein binding | Phosphorylation | Animal models | Transcription | Lung cancer | Cyclic AMP | Non-small cell lung carcinoma | Antagonists | Kinases | E-cadherin | Cell adhesion & migration | Signaling | Growth hormone-releasing hormone | Lungs | Pituitary | Animal tissues | Cyclin-dependent kinase inhibitor p27 | Growth hormones | Inhibition | Growth hormone | Viability | Cell migration | Tumors | Cancer | Apoptosis
Journal Article
Blood, ISSN 0006-4971, 2013, Volume 122, Issue 16, pp. 2911 - 2919
Journal Article
Cell and Tissue Research, ISSN 0302-766X, 5/2012, Volume 348, Issue 2, pp. 309 - 313
Journal Article
Journal of Clinical Investigation, ISSN 0021-9738, 12/2009, Volume 119, Issue 12, pp. 3626 - 3636
The polycomb group protein B lymphoma Mo-MLV insertion region 1 homolog (Bmi-1) is dysregulated in various cancers, and its upregulation strongly correlates... 
MEDICINE, RESEARCH & EXPERIMENTAL | BREAST-CANCER CELLS | DEVELOPMENTAL REGULATORS | CELLULAR MEMORY | E-CADHERIN EXPRESSION | HEMATOPOIETIC STEM-CELLS | PROSTATE-CANCER | MYC TRANSGENIC MICE | SELF-RENEWAL | NF-KAPPA-B | TRANSCRIPTION FACTOR | Nasopharyngeal Neoplasms - genetics | Neoplasm Transplantation | Nasopharyngeal Neoplasms - metabolism | Epithelial Cells - metabolism | Cadherins - metabolism | Humans | Glycogen Synthase Kinase 3 beta | Transplantation, Heterologous | Phosphatidylinositol 3-Kinases - metabolism | Mesoderm - cytology | Repressor Proteins - antagonists & inhibitors | Nasopharyngeal Neoplasms - pathology | Cadherins - genetics | Epithelial Cells - cytology | Nuclear Proteins - genetics | Proto-Oncogene Proteins c-akt - metabolism | Nasopharyngeal Neoplasms - etiology | Snail Family Transcription Factors | Nasopharynx - metabolism | Repressor Proteins - metabolism | Proto-Oncogene Proteins - metabolism | PTEN Phosphohydrolase - genetics | Proto-Oncogene Proteins - antagonists & inhibitors | Signal Transduction | Neoplasm Invasiveness | Down-Regulation | Gene Silencing | PTEN Phosphohydrolase - metabolism | Repressor Proteins - genetics | Nuclear Proteins - metabolism | Proto-Oncogene Proteins - genetics | Glycogen Synthase Kinase 3 - metabolism | Transcription Factors - metabolism | Animals | Polycomb Repressive Complex 1 | Mice, Nude | Nuclear Proteins - antagonists & inhibitors | Cell Line, Tumor | Mesoderm - metabolism | Mice | Nasopharynx - cytology | Chromatin | Care and treatment | Lymphomas | Research | Analysis | Cancer
Journal Article
The Prostate, ISSN 0270-4137, 09/2018, Volume 78, Issue 12, pp. 915 - 926
Background Therapeutic strategies should be designed to transform aggressive prostate cancer phenotypes to a chronic situation. To evaluate the effects of the... 
antagonists | GHRH‐R | prostate cancer | cell proliferation | angiogenesis | cell adhesion | GHRH-R | HUMAN BREAST | CELL-ADHESION | E-CADHERIN | MESENCHYMAL TRANSITION | BETA-CATENIN | SPLICE VARIANTS | P21 | INHIBITION | GHRH | ENDOCRINOLOGY & METABOLISM | UROLOGY & NEPHROLOGY | EXPRESSION | Cell Survival - drug effects | Prostatic Neoplasms - pathology | PC-3 Cells | Apoptosis - drug effects | beta Catenin - analysis | Humans | Receptors, Neuropeptide - antagonists & inhibitors | Male | Vascular Endothelial Growth Factor A - analysis | Resting Phase, Cell Cycle | Vascular Endothelial Growth Factor A - metabolism | Cell Adhesion - drug effects | Sermorelin - pharmacology | Cell Line, Tumor | Sermorelin - analogs & derivatives | Cell Proliferation - drug effects | Cell Cycle - drug effects | Receptors, Pituitary Hormone-Regulating Hormone - antagonists & inhibitors | Wound Healing - drug effects | Prostatic Neoplasms - drug therapy | Cell proliferation | Phenotypes | Bax protein | p53 Protein | Malignancy | E-cadherin | Gelatinase B | Cell adhesion & migration | Gelatinase A | Angiogenesis | Growth hormone-releasing hormone | Cell lines | Cell adhesion | Proliferating cell nuclear antigen | Growth hormones | Growth hormone | Vascular endothelial growth factor | Prostate cancer | Prostate | Cell migration | Apoptosis
Journal Article
Cancer Science, ISSN 1347-9032, 03/2017, Volume 108, Issue 3, pp. 478 - 487
Pancreatic ductal adenocarcinoma (PDAC) is one of the most aggressive human malignancies. The Yes‐associated protein‐1 (YAP) plays a critical role in cell... 
Angiogenesis | apoptosis | pancreatic cancer | vasculogenic mimicry | verteporfin | HUMAN CHOLANGIOCARCINOMA | HIPPO PATHWAY | DOWN-REGULATION | ANGIOPOIETIN-2 | CANCER | MOLECULAR PATHWAYS | HEPATOCELLULAR-CARCINOMA | IN-VITRO | ONCOLOGY | POOR-PROGNOSIS | CHEMORESISTANCE | Human Umbilical Vein Endothelial Cells | Cyclin D1 - metabolism | Apoptosis - drug effects | Humans | Male | Phosphoproteins - antagonists & inhibitors | Phosphoproteins - metabolism | DNA-Binding Proteins - metabolism | Pancreatic Neoplasms - drug therapy | Proto-Oncogene Proteins c-bcl-2 - metabolism | Vesicular Transport Proteins - antagonists & inhibitors | Adaptor Proteins, Signal Transducing - antagonists & inhibitors | Antineoplastic Agents - pharmacology | Cell Survival - drug effects | DNA-Binding Proteins - antagonists & inhibitors | Matrix Metalloproteinase 2 - metabolism | Pancreatic Neoplasms - pathology | Oncogene Proteins - metabolism | Nuclear Proteins - metabolism | Poly (ADP-Ribose) Polymerase-1 - metabolism | Transcription Factors - antagonists & inhibitors | Porphyrins - pharmacology | Carcinoma, Pancreatic Ductal - pathology | Transcription Factors - metabolism | Xenograft Model Antitumor Assays | Carcinoma, Pancreatic Ductal - drug therapy | Animals | Mice, Nude | Neovascularization, Pathologic - drug therapy | Nuclear Proteins - antagonists & inhibitors | Cell Line, Tumor | Cadherins - antagonists & inhibitors | Cell Proliferation - drug effects | Mice | Mice, Inbred BALB C | Cyclin E - metabolism | Adaptor Proteins, Signal Transducing - metabolism | G1 Phase Cell Cycle Checkpoints - drug effects | Antigens, CD | Adenocarcinoma | Proteins | Photochemotherapy | Analysis | Pancreatic cancer | Cancer | Cell proliferation | Metastasis | Cancer therapies | Mimicry | Cell adhesion & migration | Macular degeneration | Cell cycle | Xenografts | Conflicts of interest | G1 phase | Cell survival | Photodynamic therapy | Poly(ADP-ribose) polymerase | Gene expression | Cadherin | Gelatinase A | Yes-associated protein | Microscopy | Medical prognosis | Stem cells | Hypoxia | Apoptosis | Tumors | Original
Journal Article
Journal of Cell Science, ISSN 0021-9533, 2013, Volume 126, Issue 17, pp. 3990 - 3999
The crucial role the Crumbs and Par polarity complexes play in tight junction integrity has long been established, however very few studies have investigated... 
Scribble | Polarity | Tight junctions | Mammary epithelium | EMT | ERK | CELL-CELL JUNCTIONS | PROTEIN-KINASE-C | BARRIER FUNCTION | E-CADHERIN | MAMMALIAN EPITHELIAL-CELLS | BETA-CATENIN | MESENCHYMAL TRANSITION | TIGHT JUNCTION | CELL BIOLOGY | MALIGNANT PROGRESSION | TUMOR-SUPPRESSOR SCRIBBLE | MAP Kinase Signaling System - physiology | Phosphorylation | Epithelial-Mesenchymal Transition - physiology | Homeodomain Proteins - metabolism | Humans | Membrane Glycoproteins - biosynthesis | Mitogen-Activated Protein Kinase 3 - antagonists & inhibitors | Transcriptional Activation | Phosphatidylinositol 3-Kinases - antagonists & inhibitors | RNA Interference | Mitogen-Activated Protein Kinase 1 - genetics | HEK293 Cells | Transcription, Genetic | Flavonoids - pharmacology | Membrane Proteins - metabolism | Chromones - pharmacology | Tight Junctions - metabolism | MAP Kinase Kinase 1 - antagonists & inhibitors | Tumor Suppressor Proteins - metabolism | Homeodomain Proteins - biosynthesis | JNK Mitogen-Activated Protein Kinases - antagonists & inhibitors | Mitogen-Activated Protein Kinase 3 - genetics | Down-Regulation | Mitogen-Activated Protein Kinase 1 - antagonists & inhibitors | Morpholines - pharmacology | Transcription Factors - antagonists & inhibitors | Transcription Factors - biosynthesis | Anthracenes - pharmacology | Transcription Factors - metabolism | Membrane Proteins - biosynthesis | MAP Kinase Signaling System - drug effects | Mitogen-Activated Protein Kinase 3 - metabolism | Homeodomain Proteins - antagonists & inhibitors | Protein Kinase Inhibitors - pharmacology | RNA, Small Interfering | Tumor Suppressor Proteins - biosynthesis | Mitogen-Activated Protein Kinase 1 - metabolism | Zinc Finger E-box-Binding Homeobox 1 | Index Medicus
Journal Article
Cancer Research, ISSN 0008-5472, 01/2007, Volume 67, Issue 2, pp. 573 - 579
Journal Article
International Journal of Cancer, ISSN 0020-7136, 02/2013, Volume 132, Issue 4, pp. 755 - 765
New approaches are needed to the therapy of advanced prostate cancer. This study determined the effect of growth hormone‐releasing hormone (GHRH) antagonists,... 
prostate cancer | GHRH antagonists | metastasis | GHRH | angiogenesis | ACTIVATION | RECEPTOR | MICROVESSEL DENSITY | PROLIFERATION | E-CADHERIN | BETA-CATENIN | INVASION | ONCOLOGY | GENE-EXPRESSION | Cell Proliferation | Cadherins - metabolism | Humans | Male | Receptors, Neuropeptide - metabolism | Growth Hormone-Releasing Hormone - pharmacology | Matrix Metalloproteinase 9 - metabolism | Neoplasm Metastasis - drug therapy | Hypoxia-Inducible Factor 1, alpha Subunit - metabolism | Receptors, Pituitary Hormone-Regulating Hormone - metabolism | Sermorelin - pharmacology | Sermorelin - analogs & derivatives | Antineoplastic Agents - pharmacology | Prostatic Neoplasms - drug therapy | Prostatic Neoplasms - pathology | Matrix Metalloproteinase 2 - metabolism | Neoplasm Invasiveness | Random Allocation | Cell Adhesion | beta Catenin - metabolism | Xenograft Model Antitumor Assays | Animals | Mice, Nude | Neovascularization, Pathologic - drug therapy | Cell Line, Tumor | Vascular Endothelial Growth Factors - metabolism | Growth Hormone-Releasing Hormone - analogs & derivatives | Mice | Growth Hormone-Releasing Hormone - antagonists & inhibitors | Care and treatment | Pituitary hormones | Somatotropin releasing hormone | Growth | Oncology, Experimental | Development and progression | Metastasis | Research | Cells | Endothelium | Heterocyclic compounds | Analysis | Health aspects | Vascular endothelial growth factor | Prostate cancer | Enzyme-linked immunosorbent assay | Cancer | Growth hormones | Cancer therapies
Journal Article