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Immunity, ISSN 1074-7613, 09/2012, Volume 37, Issue 3, pp. 563 - 573
Mice lacking junctional adhesion molecule A (JAM-A, encoded by ) exhibit enhanced intestinal epithelial permeability, bacterial translocation, and elevated... 
GROWTH-FACTOR-BETA | INFLAMMATORY-BOWEL-DISEASE | IMMUNOGLOBULIN-A | IN-VIVO | REGULATORY T-CELLS | JUNCTIONAL-ADHESION-MOLECULE | DEFICIENT MICE | E-CADHERIN | IMMUNOLOGY | ORAL ANTIGEN | EXPRESSION | Dextran Sulfate | Cell Adhesion Molecules - genetics | Intestinal Mucosa - metabolism | Colitis - genetics | Immunoglobulin A - genetics | Male | Homeodomain Proteins - immunology | Intestines - metabolism | Cell Adhesion Molecules - immunology | Intestines - immunology | Adaptive Immunity - genetics | CD4-Positive T-Lymphocytes - immunology | Epithelium - immunology | Flow Cytometry | Intestinal Mucosa - immunology | Colitis - chemically induced | Female | Adaptive Immunity - immunology | Colitis - immunology | Transforming Growth Factor beta - immunology | Gene Expression | Bacterial Translocation - genetics | Epithelium - metabolism | Mice, Inbred C57BL | CD4-Positive T-Lymphocytes - metabolism | Bacterial Translocation - immunology | Intestinal Mucosa - microbiology | Permeability | Reverse Transcriptase Polymerase Chain Reaction | Receptors, Cell Surface - immunology | Homeodomain Proteins - genetics | Mice, Knockout | Animals | Intestines - microbiology | Transforming Growth Factor beta - genetics | Immunoglobulin A - immunology | Mice | Transforming Growth Factor beta - metabolism | Receptors, Cell Surface - genetics | Disease susceptibility | Colitis | T cells | Transforming growth factors | Immunoglobulin A | Inflammatory bowel disease | Studies | Medical research | Nutrition research | Disease | Antibiotics | Lymphocytes | Rodents | Small intestine | Immune system | Cell adhesion & migration | Index Medicus | Medical and Health Sciences | Homeodomain Proteins/genetics/immunology | Medicin och hälsovetenskap | Cell Surface/genetics/immunology | Colitis/chemically induced/genetics/immunology | CD4-Positive T-Lymphocytes/immunology/metabolism | Bacterial Translocation/genetics/immunology | Receptors | Immunoglobulin A/genetics/immunology | Transforming Growth Factor beta/genetics/immunology/metabolism | Intestinal Mucosa/immunology/metabolism/microbiology | Adaptive Immunity/genetics/immunology | Basic Medicine | Intestines/immunology/metabolism/microbiology | Cell and Molecular Biology | Inbred C57BL | Knockout | Cell Adhesion Molecules/genetics/immunology | Epithelium/immunology/metabolism | Medicinska och farmaceutiska grundvetenskaper | Cell- och molekylärbiologi
Journal Article
Journal of Immunology, ISSN 0022-1767, 11/2015, Volume 195, Issue 9, pp. 4378 - 4386
Studies of influenza-specific immune responses in humans have largely assessed systemic responses involving serum Ab and peripheral blood T cell responses.... 
NONHUMAN-PRIMATES | LUNG | CHALLENGE | MEDIATED PROTECTION | VIRUS-INFECTION | DENDRITIC CELLS | IMMUNE-RESPONSES | VACCINE | E-CADHERIN | IMMUNOLOGY | EXPRESSION | Spleen - immunology | Humans | Influenza A Virus, H1N1 Subtype - physiology | Macaca mulatta - immunology | Interferon-gamma - metabolism | T-Lymphocytes - virology | Antigens, CD - metabolism | Host-Pathogen Interactions - immunology | Lectins, C-Type - metabolism | T-Lymphocytes - metabolism | Time Factors | Bone Marrow - metabolism | Integrin alpha Chains - immunology | B-Lymphocytes - virology | CD8-Positive T-Lymphocytes - metabolism | B-Lymphocytes - metabolism | Bone Marrow - virology | Antigens, Differentiation, T-Lymphocyte - metabolism | Lymph Nodes - metabolism | Lymph Nodes - immunology | B-Lymphocytes - immunology | T-Lymphocytes - immunology | CD8-Positive T-Lymphocytes - immunology | Orthomyxoviridae Infections - virology | Influenza A Virus, H1N1 Subtype - immunology | Spleen - virology | Antigens, CD - immunology | Bone Marrow - immunology | Age Factors | Lectins, C-Type - immunology | Macaca mulatta - virology | Lung - virology | Macaca mulatta - metabolism | Interleukin-2 - immunology | Lung - metabolism | Immunologic Memory - immunology | Interleukin-2 - metabolism | Antigens, Differentiation, T-Lymphocyte - immunology | Orthomyxoviridae Infections - metabolism | Cells, Cultured | Integrin alpha Chains - metabolism | Lymph Nodes - virology | Mediastinum - virology | Animals | Spleen - metabolism | CD8-Positive T-Lymphocytes - virology | Interferon-gamma - immunology | Lung - immunology | Orthomyxoviridae Infections - immunology | Index Medicus | Abridged Index Medicus | Infectious Disease and Host Response
Journal Article
Immunity, ISSN 1074-7613, 10/2013, Volume 39, Issue 4, pp. 687 - 696
Journal Article
Journal of Immunology, ISSN 0022-1767, 03/2015, Volume 194, Issue 6, pp. 2810 - 2818
Macrophages play a major role in joint inflammation. Estrogen is involved in rheumatoid arthritis and temporomandibular disorders. However, the underlying... 
RHEUMATOID-ARTHRITIS | PATHOGENESIS | PAIN | ENDOMETRIAL STROMAL CELLS | TISSUE | KAPPA-B | IMMUNOLOGY | OVARIECTOMIZED RATS | EXPRESSION | ALTERNATIVE ACTIVATION | DISEASE-ACTIVITY | Estradiol - analogs & derivatives | Receptors, Estrogen - metabolism | Estrogens - pharmacology | Arthritis - metabolism | Cadherins - metabolism | Gene Expression - drug effects | Arginase - metabolism | Arthritis - genetics | Temporomandibular Joint - pathology | Receptors, Estrogen - immunology | Estrogen Receptor Antagonists - pharmacology | Arginase - immunology | Inflammation - metabolism | Cadherins - immunology | Nitric Oxide Synthase Type II - immunology | Gene Expression - immunology | Tumor Necrosis Factor-alpha - immunology | Arthritis - immunology | Interleukin-10 - metabolism | Female | Cadherins - genetics | Estradiol - pharmacology | Macrophages - immunology | Nitric Oxide - immunology | Ovariectomy | Macrophage Activation - immunology | Receptors, Estrogen - antagonists & inhibitors | Arginase - genetics | Inflammation - immunology | Reverse Transcriptase Polymerase Chain Reaction | Rats, Sprague-Dawley | Blotting, Western | Estradiol - immunology | Temporomandibular Joint - drug effects | Microscopy, Confocal | Tumor Necrosis Factor-alpha - pharmacology | Macrophages - metabolism | Animals | Nitric Oxide Synthase Type II - genetics | Interleukin-10 - genetics | Temporomandibular Joint - immunology | Inflammation - genetics | Estrogens - immunology | Macrophages - drug effects | Macrophage Activation - drug effects | Nitric Oxide - metabolism | Interleukin-10 - immunology | Nitric Oxide Synthase Type II - metabolism | Index Medicus | Abridged Index Medicus
Journal Article
Immunity, ISSN 1074-7613, 2007, Volume 27, Issue 4, pp. 610 - 624
Journal Article
Immunity, ISSN 1074-7613, 04/2015, Volume 42, Issue 4, pp. 640 - 653
Acute lung injury (ALI) remains a serious health issue with little improvement in our understanding of the pathophysiology and therapeutic approaches. We... 
BACTERIAL LIPOPOLYSACCHARIDE | STERILE INFLAMMATION | SIGNALING PATHWAY | P2X RECEPTORS | ALPHA PRODUCTION | ENDOTHELIAL-CELLS | MOUSE MODEL | IN-VIVO | ACUTE LUNG INJURY | IMMUNOLOGY | INFLAMMATORY RESPONSE | Interleukin-1alpha - immunology | Macrophages, Alveolar - pathology | Calcium - metabolism | Humans | Capillary Permeability - immunology | Neutrophil Infiltration | Necrosis - pathology | Receptors, Purinergic P2X7 - genetics | Necrosis - immunology | Cadherins - immunology | Adenosine Triphosphate - metabolism | HEK293 Cells | Interleukin-1alpha - genetics | Cadherins - genetics | Receptors, Purinergic P2X7 - immunology | Macrophages, Alveolar - immunology | Neutrophils - pathology | Lung - pathology | Protein Precursors - genetics | Signal Transduction | Neutrophils - drug effects | Gene Expression Regulation | Neutrophils - immunology | Lipopolysaccharide Receptors - immunology | Mice, Transgenic | Protein Precursors - immunology | Acute Lung Injury - pathology | Necrosis - chemically induced | Endothelial Cells - immunology | Animals | Acute Lung Injury - chemically induced | Macrophages, Alveolar - drug effects | Lung - drug effects | Lipopolysaccharides - pharmacology | Lipopolysaccharide Receptors - genetics | Acute Lung Injury - immunology | Mice | Endothelial Cells - pathology | Intubation, Intratracheal | Lung - immunology | Endothelial Cells - drug effects | Index Medicus | endothelial cells | alveolar macrophage | Ca2+ influx | MyD88 | IL-1α | neutrophil | P2X7R | lung injury | necrosis | ARDS | ALI | sepsis | lung inflammation
Journal Article
Journal Article
Annals of the Rheumatic Diseases, ISSN 0003-4967, 06/2014, Volume 73, Issue 6, pp. 1223 - 1231
Journal Article
Journal of Allergy and Clinical Immunology, The, ISSN 0091-6749, 2015, Volume 136, Issue 2, pp. 423 - 432.e8
Background The immunomodulatory and anti-inflammatory functions of mesenchymal stem cells (MSCs) have been demonstrated in several autoimmune/inflammatory... 
Allergy and Immunology | Allergy | allergic conjunctivitis | histamine | mesenchymal stem cells | stems cells | B cells | mast cells | RECONSTRUCTION | CONTACT HYPERSENSITIVITY | RECEPTOR | IMMUNOLOGY | SUPPRESSION | MATURATION | TRANSPLANTATION | MURINE MODEL | RESPONSES | INFLAMMATION | VE-CADHERIN | Ambrosia - chemistry | RNA, Small Interfering - genetics | Tumor Necrosis Factor-alpha - genetics | Capillary Permeability - drug effects | Conjunctivitis, Allergic - drug therapy | Capillary Permeability - immunology | Culture Media, Conditioned - pharmacology | Pollen - immunology | Mesenchymal Stromal Cells - immunology | Histamine - metabolism | Conjunctiva - pathology | Immunoglobulin E - genetics | RNA, Small Interfering - immunology | Cyclooxygenase 2 - genetics | Bone Marrow Cells - immunology | Tumor Necrosis Factor-alpha - immunology | Bone Marrow Cells - drug effects | Cell Differentiation | B-Lymphocytes - pathology | Interleukin-4 - genetics | Cyclooxygenase 2 - immunology | Mesenchymal Stromal Cells - drug effects | Conjunctivitis, Allergic - chemically induced | Conjunctiva - immunology | Signal Transduction | Bone Marrow Cells - pathology | Gene Expression Regulation | Ambrosia - immunology | Conjunctivitis, Allergic - genetics | B-Lymphocytes - drug effects | Interleukin-4 - immunology | Tumor Necrosis Factor-alpha - pharmacology | Animals | B-Lymphocytes - immunology | Conjunctiva - drug effects | Mice | Mice, Inbred BALB C | Mesenchymal Stromal Cells - pathology | Primary Cell Culture | Conjunctivitis, Allergic - immunology | Index Medicus | Abridged Index Medicus
Journal Article
Journal Article
Scientific Reports, ISSN 2045-2322, 02/2016, Volume 6, Issue 1, pp. 21868 - 21868
Liver dysfunction is an early event in sepsis-related multi-organ failure. We here report the establishment and characterization of a microfluidically... 
RESPONSES | MACROPHAGES | MULTIDISCIPLINARY SCIENCES | LIPOPOLYSACCHARIDE | INFECTION | SEPSIS | INDUCTION | HEPATOTOXICITY | IDENTIFICATION | CULTURE | CELL-LINE | Antigens, CD - immunology | Cell Communication - immunology | Monocytes - cytology | Coculture Techniques | Humans | Capillaries - drug effects | Monocytes - immunology | Antigens, CD - genetics | Hepatocytes - cytology | Liver - immunology | Cadherins - immunology | Liver - drug effects | Multidrug Resistance-Associated Proteins - immunology | Multidrug Resistance-Associated Proteins - genetics | Cadherins - genetics | Cytokines - genetics | Hepatocytes - drug effects | Cytokines - immunology | Signal Transduction | Toll-Like Receptors - immunology | Gene Expression Regulation | Organoids - cytology | Apolipoproteins B - genetics | Inflammation | Lab-On-A-Chip Devices | Hepatocytes - immunology | Endothelial Cells - immunology | Monocytes - drug effects | Albumins - immunology | Capillaries - cytology | Endothelial Cells - cytology | Models, Biological | Toll-Like Receptors - genetics | Lipopolysaccharides - pharmacology | Organoids - drug effects | Capillaries - immunology | Liver - cytology | Organoids - immunology | Albumins - genetics | Apolipoproteins B - immunology | Endothelial Cells - drug effects | Liver diseases | Cytokines | Liver | Albumin | Cadherin | Apolipoproteins | Contamination | Apolipoprotein B | Urea | Monocytes | Organoids | Rodents | Toll-like receptors | Sepsis | Peritoneum | Index Medicus
Journal Article