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1. Full Text M2 microglia and macrophages drive oligodendrocyte differentiation during CNS remyelination
by Miron, Veronique E and Boyd, Amanda and Zhao, Jing-Wei and Yuen, Tracy J and Ruckh, Julia M and Shadrach, Jennifer L and Van Wijngaarden, Peter and Wagers, Amy J and Williams, Anna and Franklin, Robin J.M and Ffrench-Constant, Charles
Nature Neuroscience, ISSN 1097-6256, 09/2013, Volume 16, Issue 9, pp. 1211 - 1218
The lack of therapies for progressive multiple sclerosis highlights the need to understand the regenerative process of remyelination that can follow CNS... 
PROGENITOR CELLS | ACTIVIN-A | REGENERATION | GENE-EXPRESSION | MONOCYTES | INJURY | GADOLINIUM CHLORIDE | IDENTIFICATION | NEUROSCIENCES | PROGRESSION | BETA | Clodronic Acid - pharmacology | Humans | Middle Aged | Central Nervous System - pathology | Culture Media, Conditioned - pharmacology | Male | Cell Differentiation - genetics | Microglia - physiology | Oligodendroglia - drug effects | Oligodendroglia - physiology | Aged, 80 and over | Adult | Female | Cadmium Chloride - pharmacology | Demyelinating Diseases - pathology | Cell Differentiation - physiology | Animals, Newborn | Macrophages - physiology | Mice, Inbred C57BL | Cells, Cultured | Rats | Mice, Transgenic | Regeneration - physiology | Rats, Sprague-Dawley | Regeneration - drug effects | Animals | Cell Differentiation - drug effects | Central Nervous System - drug effects | Aged | Mice | In Vitro Techniques | Myelin Proteins - metabolism | Physiological aspects | Multiple sclerosis | Myelination | Research | Regeneration (Biology) | Macrophages | Index Medicus
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2. Full Text Presynaptic α4β2 nicotinic acetylcholine receptors increase glutamate release and serotonin neuron excitability in the dorsal raphe nucleus
by Garduño, Julieta and Galindo-Charles, Luis and Jiménez-Rodríguez, Javier and Galarraga, Elvira and Tapia, Dagoberto and Mihailescu, Stefan and Hernandez-Lopez, Salvador
Journal of Neuroscience, ISSN 0270-6474, 10/2012, Volume 32, Issue 43, pp. 15148 - 15157
Several behavioral effects of nicotine are mediated by changes in serotonin (5-HT) release in brain areas that receive serotonergic afferents from the dorsal... 
Electric Stimulation | Rats, Wistar | Dihydro-beta-Erythroidine - pharmacology | Male | Nicotinic Antagonists - pharmacology | Raphe Nuclei - cytology | Excitatory Postsynaptic Potentials - drug effects | Nicotine - pharmacology | Excitatory Postsynaptic Potentials - physiology | Muscarinic Antagonists - pharmacology | Egtazic Acid - analogs & derivatives | 6-Cyano-7-nitroquinoxaline-2,3-dione - pharmacology | Indoles - pharmacology | Tetrodotoxin - pharmacology | Cadmium Chloride - pharmacology | Cholinergic Agonists - pharmacology | Ryanodine - pharmacology | Animals, Newborn | Serotonergic Neurons - physiology | Presynaptic Terminals - drug effects | Acetylcholine - pharmacology | Enzyme Inhibitors - pharmacology | Glutamic Acid - physiology | Rats | Atropine - pharmacology | Sodium Channel Blockers - pharmacology | Excitatory Amino Acid Antagonists - pharmacology | Presynaptic Terminals - physiology | Aconitine - analogs & derivatives | Bicuculline - pharmacology | Chelating Agents - pharmacology | Aconitine - pharmacology | GABA Antagonists - pharmacology | Physostigmine - pharmacology | Patch-Clamp Techniques | Animals | Egtazic Acid - pharmacology | Receptors, Nicotinic - physiology | Nicotine - analogs & derivatives | Serotonin - metabolism | In Vitro Techniques | Serotonergic Neurons - cytology | Index Medicus
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3. Full Text Oxidative phosphorylation, not glycolysis, powers presynaptic and postsynaptic mechanisms underlying brain information processing
by Hall, Catherine N and Klein-Flügge, Miriam C and Howarth, Clare and Attwell, David
Journal of Neuroscience, ISSN 0270-6474, 06/2012, Volume 32, Issue 26, pp. 8940 - 8951
Neural activity has been suggested to initially trigger ATP production by glycolysis, rather than oxidative phosphorylation, for three reasons: glycolytic... 
OXYGEN-CONSUMPTION | TISSUE OXYGENATION | GLUCOSE CONSUMPTION | GLUTAMATE RELEASE | RAT CEREBELLAR CORTEX | AEROBIC GLYCOLYSIS | SYNAPTIC ACTIVITY | NITRIC-OXIDE | NEURAL ACTIVITY | CEREBRAL-BLOOD-FLOW | NEUROSCIENCES | Oxygen - pharmacology | Enzyme Inhibitors | Electric Stimulation | Neurons - cytology | Excitatory Postsynaptic Potentials - drug effects | Oxygen - metabolism | Dose-Response Relationship, Drug | Oxidative Phosphorylation - drug effects | Glycolysis - physiology | Adenosine Triphosphate - metabolism | Neurons - physiology | Organic Chemicals - pharmacology | Statistics, Nonparametric | 6-Cyano-7-nitroquinoxaline-2,3-dione - pharmacology | Tetrodotoxin - pharmacology | Cadmium Chloride - pharmacology | Neurons - drug effects | Action Potentials - drug effects | NAD - metabolism | Animals, Newborn | Synapses - drug effects | Presynaptic Terminals - drug effects | Synapses - physiology | Lactic Acid - metabolism | Valine - analogs & derivatives | Rats | Sodium Channel Blockers - pharmacology | Excitatory Amino Acid Antagonists - pharmacology | Presynaptic Terminals - physiology | Hippocampus - cytology | Rats, Sprague-Dawley | Action Potentials - physiology | Animals | Models, Biological | Valine - pharmacology | Hippocampus - physiology | In Vitro Techniques | Index Medicus
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4. Full Text TMEM16C facilitates Na + -activated K + currents in rat sensory neurons and regulates pain processing
by Huang, Fen and Wang, Xidao and Ostertag, Eric M and Nuwal, Tulip and Huang, Bo and Jan, Yuh-Nung and Basbaum, Allan I and Jan, Lily Yeh
Nature Neuroscience, ISSN 1097-6256, 09/2013, Volume 16, Issue 9, pp. 1284 - 1290
TMEM16C belongs to the TMEM16 family, which includes the Ca2+-activated Cl- channels TMEM16A and TMEM16B and a small-conductance, Ca2+-activated, nonselective... 
ELECTROGENESIS | CALCIUM-CHANNELS | ROOT GANGLION NEURONS | PRIMARY AFFERENT NEURONS | SLACK | SODIUM-CHANNELS | CHLORIDE CHANNEL ACTIVITY | NEUROSCIENCES | MOLECULAR-MECHANISMS | POTASSIUM CHANNEL | FAMILY | Hyperalgesia - chemically induced | New Brunswick | Pain Threshold - physiology | Membrane Potentials - genetics | Sodium Chloride - pharmacology | Humans | Chloride Channels - deficiency | Ganglia, Spinal - cytology | Chloride Channels - genetics | Sodium - metabolism | Potassium Channels - metabolism | Mutation - physiology | Sensory Receptor Cells - physiology | Pain Threshold - drug effects | Spinal Cord - cytology | Membrane Potentials - drug effects | Rats, Transgenic | Gene Expression Regulation - genetics | RNA, Small Interfering - pharmacology | Cells, Cultured | Cadmium - pharmacology | Rats | Nerve Tissue Proteins - genetics | Potassium Channels - genetics | Chloride Channels - metabolism | Nerve Tissue Proteins - metabolism | Animals | Sensory Receptor Cells - drug effects | Hyperalgesia - genetics | Mice | Pain Measurement | Index Medicus
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5. Full Text Regulation of ion channel localization and phosphorylation by neuronal activity
by Zhen, Dongkai and Mohapatra, Durga P and Park, Eunice W and Trimmer, James S and Leung, Victor and Anderson, Anne E and Misonou, Hiroaki and Misonou, Kaori
Nature Neuroscience, ISSN 1097-6256, 07/2004, Volume 7, Issue 7, pp. 711 - 718
Voltage-dependent Kv2.1 K+ channels, which mediate delayed rectifier Kv currents (I-K), are expressed in large clusters on the somata and dendrites of... 
ALPHA-SUBUNITS | RAT HIPPOCAMPAL-NEURONS | MAMMALIAN BRAIN | ACTIVATION | GATED POTASSIUM CHANNELS | RECEPTOR | NEOCORTICAL PYRAMIDAL NEURONS | IDENTIFICATION | KV2.1 K+ CHANNEL | BINDING | NEUROSCIENCES | Potassium - metabolism | Okadaic Acid - pharmacology | Potassium Channels, Voltage-Gated | Pyramidal Cells - metabolism | Cyclosporine - pharmacology | Cell Count | Neuronal Plasticity - drug effects | Pyrethrins - pharmacology | Translocation, Genetic - drug effects | Patch-Clamp Techniques - methods | Seizures - chemically induced | Dose-Response Relationship, Drug | Potassium Channels - metabolism | Phosphoprotein Phosphatases - pharmacology | Seizures - physiopathology | Drug Interactions | Neuronal Plasticity - physiology | Time Factors | Dendrites - physiology | Potassium Chloride - pharmacology | Nitriles | Pyramidal Cells - drug effects | Phosphorylation - drug effects | Cadmium Chloride - pharmacology | Ionophores - pharmacology | Potassium Channel Blockers - pharmacology | Disease Models, Animal | Animals, Newborn | Membrane Potentials - drug effects | Dendrites - drug effects | Translocation, Genetic - physiology | Glutamic Acid - pharmacology | Kainic Acid - pharmacology | Cells, Cultured | Enzyme Inhibitors - pharmacology | Rats | Excitatory Amino Acid Antagonists - pharmacology | Nitrendipine - pharmacology | Calcium Channel Blockers - pharmacology | Delayed Rectifier Potassium Channels | Hippocampus - cytology | Membrane Potentials - physiology | Shab Potassium Channels | Calcimycin - pharmacology | Blotting, Western | Animals | Ion Channel Gating - drug effects | Physiological aspects | Phosphorylation | Ion channels | Research | Neurons | Index Medicus
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6. Full Text Enhanced biosynthesis of withanolides by elicitation and precursor feeding in cell suspension culture of Withania somnifera (L.) dunal in shake-flask culture and bioreactor
by Sivanandhan, Ganeshan and Selvaraj, Natesan and Ganapathi, Andy and Manickavasagam, Markandan
PLoS ONE, ISSN 1932-6203, 08/2014, Volume 9, Issue 8, pp. e104005 - e104005
The present study investigated the biosynthesis of major and minor withanolides of Withania somnifera in cell suspension culture using shake-flask culture and... 
SECONDARY METABOLITES | PATHWAYS | METHYL JASMONATE | MULTIDISCIPLINARY SCIENCES | PHENYLETHANOID GLYCOSIDES | GROWTH | ACCUMULATION | ROOT CULTURES | WITHAFERIN | STRATEGIES | Withanolides - metabolism | Withania - drug effects | Chlorides - pharmacology | Withanolides - chemistry | Glutamine - metabolism | Picloram - pharmacology | Cholesterol - metabolism | Mevalonic Acid - metabolism | Squalene - metabolism | Bioreactors | Sucrose - metabolism | Chitosan - pharmacology | Withanolides - classification | Aluminum Compounds - pharmacology | Withania - metabolism | Cell Culture Techniques | Cadmium Chloride - pharmacology | Culture Media - chemistry | Herbicides | Auxin | Chemical tests and reagents | Physiological aspects | Biosynthesis | Chemical properties | Glutamine | Cell culture | Biotechnology | Cadmium chlorides | Sucrose | Aluminum | Cadmium chloride | Signal transduction | Suspension culture | Aluminum chlorides | Mevalonic acid | Metabolites | Precursors | Physiology | Life sciences | Sugars | Cadmium | Aluminum chloride | Squalene | Metabolism | Cholesterol | Feeding | Herbal medicine | Picloram | Combined treatment | Genetic engineering | Chitosan | Apoptosis | Pharmaceuticals | Index Medicus
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7. Full Text Effect of cadmium chloride exposure during the induction of collagen induced arthritis
by Ansari, Md. Meraj and Neha and Khan, Haider A
Chemico-Biological Interactions, ISSN 0009-2797, 08/2015, Volume 238, pp. 55 - 65
The precise cause of autoimmune diseases such as rheumatoid arthritis remains uncertain. Collagen induced arthritis (CIA) in animals is the most commonly used... 
Cadmium exposure | Wistar rats | Pro-inflammatory cytokine | CIA | Rheumatoid arthritis | RHEUMATOID-ARTHRITIS | APOPTOSIS | GLYCOSAMINOGLYCANS | BIOCHEMISTRY & MOLECULAR BIOLOGY | RATS | PREVENTION | KAPPA-B | MODEL | NITRIC-OXIDE | PHARMACOLOGY & PHARMACY | MICE | TOXICOLOGY | STRESS | Knee Joint - metabolism | Tumor Necrosis Factor-alpha - metabolism | Rats, Wistar | Glutathione - metabolism | Knee Joint - drug effects | Nitrites - metabolism | Rats | Pancreatic Elastase - metabolism | Freund's Adjuvant - immunology | Arthritis, Experimental - pathology | Catalase - metabolism | Knee Joint - pathology | Animals | Transcription Factor RelA - metabolism | Arthritis, Experimental - chemically induced | Cyclooxygenase 2 - metabolism | Lipid Peroxidation - drug effects | Collagen Type II - toxicity | Female | Cadmium Chloride - pharmacology | Interleukin-6 - metabolism | Superoxide Dismutase - metabolism | Disease Models, Animal | Cadmium | Development and progression | Arthritis | Collagen | Index Medicus
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8. Full Text Trans-resveratrol Inhibits Tau Phosphorylation in the Brains of Control and Cadmium Chloride-Treated Rats by Activating PP2A and PI3K/Akt Induced-Inhibition of GSK3β
by Shati, Ali A and Alfaifi, Mohammad Y
Neurochemical Research, ISSN 0364-3190, 2/2019, Volume 44, Issue 2, pp. 357 - 373
This study investigated if resveratrol (RES) can protect against cadmium chloride (CdCl2)-induced memory loss and Tau protein hyperphosphorylation in rats and... 
Neurochemistry | Biochemistry, general | Neurology | Cadmium | Brain | Neurosciences | Biomedicine | Memory, PI3K/Akt | Resveratrol | Tau | Cell Biology | Resveratrol - pharmacology | tau Proteins - metabolism | Male | Phosphatidylinositol 3-Kinases - metabolism | Antioxidants - pharmacology | Rats, Sprague-Dawley | Brain - drug effects | Brain - metabolism | Animals | Signal Transduction - drug effects | Phosphatidylinositol 3-Kinases - drug effects | Phosphorylation - drug effects | Cadmium Chloride - pharmacology | Proto-Oncogene Proteins c-akt - metabolism | Proto-Oncogene Proteins c-akt - drug effects | Phosphorylation | Regulators | Reactive oxygen species | Cadmium chlorides | Rats | AKT protein | Pattern recognition | Kinases | Object recognition | Cadmium chloride | Malondialdehyde | 1-Phosphatidylinositol 3-kinase | Proteins | Antioxidants | Signal transduction | Signaling | Tau protein | Long term memory | Inhibition | Glutathione | Index Medicus
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9. Full Text Improved production of dibenzocyclooctadiene lignans in the elicited microshoot cultures of Schisandra chinensis (Chinese magnolia vine)
by Szopa, Agnieszka and Kokotkiewicz, Adam and Król, Agata and Luczkiewicz, Maria and Ekiert, Halina
Applied Microbiology and Biotechnology, ISSN 0175-7598, 1/2018, Volume 102, Issue 2, pp. 945 - 959
Dibenzocyclooctadiene lignans are a specific group of secondary metabolites that occur solely in Schisandra chinensis. The aim of the presented work was to... 
Life Sciences | Biotic elicitors | In vitro cultures | Biotechnology | Microbiology | Schisandra lignan production | Microbial Genetics and Genomics | Elicitation | Abiotic elicitors | Temporary-immersion bioreactor | BIOTECHNOLOGY & APPLIED MICROBIOLOGY | SUSPENSION-CULTURES | GROWTH | CELL-CULTURES | BIOREACTORS | Schisandra - metabolism | Dimethyl Sulfoxide - pharmacology | Lignans - biosynthesis | Chromatography, High Pressure Liquid | Cyclopentanes - pharmacology | Secondary Metabolism | Bioreactors | Plant Shoots - metabolism | Oxylipins - pharmacology | Chitosan - pharmacology | Cyclooctanes | Acetates - pharmacology | Cadmium Chloride - pharmacology | Culture Media - chemistry | Physiological aspects | Polyphenols | Schisandra | Biosynthesis | Methods | Cadmium | Secondary metabolites | Yeast | Metabolites | Lignans | Cultures | Chitosan | Supplementation | Flax | Cadmium chloride | Jasmonic acid | Methyl jasmonate | Index Medicus | Applied Microbial and Cell Physiology
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10. Full Text Endogenous H2S is required for hypoxic sensing by carotid body glomus cells
by Makarenko, Vladislav V and Nanduri, Jayasri and Raghuraman, Gayatri and Fox, Aaron P and Gadalla, Moataz M and Kumar, Ganesh K and Snyder, Solomon H and Prabhakar, Nanduri R
American Journal of Physiology - Cell Physiology, ISSN 0363-6143, 11/2012, Volume 303, Issue 9, pp. C916 - C923
Makarenko VV, Nanduri J, Raghuraman G, Fox AP, Gadalla MM, Kumar GK, Snyder SH, Prabhakar NR. Endogenous H2S is required for hypoxic sensing by carotid body... 
sensing | Arterial chemoreceptors | Voltage-gated Ca | Breathing | channels | Gasotransmitters | LOCALIZATION | PHYSIOLOGY | voltage-gated Ca2+ channels | RAT | breathing | CHEMORECEPTOR CELLS | CA2+ CHANNELS | HYDROGEN-SULFIDE | CELL BIOLOGY | gasotransmitters | CARBON-MONOXIDE | PATHWAY | O-2 sensing | GAMMA-CYSTATHIONASE | CAT | arterial chemoreceptors | CATECHOLAMINE RELEASE | Hydrogen Sulfide - metabolism | Glycine - analogs & derivatives | Nifedipine - pharmacology | Catecholamines - secretion | Enzyme Inhibitors - pharmacology | Rats | Cystathionine gamma-Lyase - metabolism | Male | Calcium - analysis | Calcium Channel Blockers - pharmacology | Cystathionine gamma-Lyase - antagonists & inhibitors | Rats, Sprague-Dawley | Carotid Body - metabolism | Hypoxia - metabolism | Animals | Glycine - pharmacology | Potassium Chloride - pharmacology | Alkynes - pharmacology | Carotid Body - drug effects | Mice | Cadmium Chloride - pharmacology | Sulfides - pharmacology | Index Medicus | O2 sensing
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11. Full Text Thin Dendrites of Cerebellar Interneurons Confer Sublinear Synaptic Integration and a Gradient of Short-Term Plasticity
by Abrahamsson, Therese and Cathala, Laurence and Matsui, Ko and Shigemoto, Ryuichi and DiGregorio, David A
Neuron, ISSN 0896-6273, 03/2012, Volume 73, Issue 6, pp. 1159 - 1172
Interneurons are critical for neuronal circuit function, but how their dendritic morphologies and membrane properties influence information flow within... 
PURKINJE-CELLS | PERMEABLE AMPA RECEPTORS | POSTSYNAPTIC POTENTIALS | COINCIDENCE DETECTION | STELLATE CELLS | MOLECULAR LAYER INTERNEURONS | GRANULE CELLS | HIPPOCAMPAL-NEURONS | NEUROSCIENCES | CA1 PYRAMIDAL NEURONS | LATERAL GENICULATE-NUCLEUS | Tetraethylammonium - pharmacology | Electric Stimulation | Age Factors | Dendrites - ultrastructure | Excitatory Postsynaptic Potentials - physiology | Neuronal Plasticity - physiology | Time Factors | Lasers | Dendrites - physiology | Statistics, Nonparametric | Indoles - pharmacology | Tetrodotoxin - pharmacology | Cadmium Chloride - pharmacology | Imaging, Three-Dimensional | Potassium Channel Blockers - pharmacology | Receptors, AMPA - metabolism | Animals, Newborn | Microscopy, Electron, Transmission | Benzodiazepines - pharmacology | Chlorides - pharmacology | Synapses - physiology | Interneurons - ultrastructure | Sodium Channel Blockers - pharmacology | Synapses - ultrastructure | Excitatory Amino Acid Antagonists - pharmacology | Biophysics | Receptors, AMPA - ultrastructure | Microscopy, Confocal | Cesium - pharmacology | Patch-Clamp Techniques | Animals | Cerebellum - cytology | Glutamates - pharmacology | Mice | Models, Neurological | In Vitro Techniques | Glutamate | Neurons | Information management | Studies | Microscopy | Cerebellum | Index Medicus | Life Sciences
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12. Full Text miR-181a and miR-630 regulate cisplatin-induced cancer cell death
by Galluzzi, Lorenzo and Morselli, Eugenia and Vitale, Ilio and Kepp, Oliver and Senovilla, Laura and Criollo, Alfredo and Servant, Nicolas and Paccard, Caroline and Hupé, Philippe and Robert, Thomas and Ripoche, Hugues and Lazar, Vladimir and Harel-Bellan, Annick and Dessen, Philippe and Barillot, Emmanuel and Kroemer, Guido
Cancer Research, ISSN 0008-5472, 03/2010, Volume 70, Issue 5, pp. 1793 - 1803
MicroRNAs (miRNA) are noncoding RNAs that regulate multiple cellular processes, including proliferation and apoptosis. We used microarray technology to... 
MITOCHONDRIAL-MEMBRANE PERMEABILIZATION | LUNG-CANCER | ONCOLOGY | DNA-DAMAGE | APAF-1 | BAX | MICRORNAS | TUMOR SUPPRESSORS | CHEMICAL INHIBITOR | DAMAGE-INDUCED APOPTOSIS | P53 | Cell Cycle - genetics | Lung Neoplasms - drug therapy | Phosphorylation | Apoptosis - drug effects | Humans | Lung Neoplasms - metabolism | Apoptosis - genetics | Lung Neoplasms - pathology | MicroRNAs - metabolism | Tumor Suppressor Protein p53 - genetics | Caspases - metabolism | Antineoplastic Agents - pharmacology | Cadmium Chloride - pharmacology | Carcinoma, Non-Small-Cell Lung - pathology | Lung Neoplasms - genetics | Carcinoma, Non-Small-Cell Lung - genetics | Oligonucleotide Array Sequence Analysis - methods | Carcinoma, Non-Small-Cell Lung - metabolism | Tumor Suppressor Protein p53 - metabolism | bcl-2-Associated X Protein - metabolism | Cisplatin - pharmacology | Sphingosine - pharmacology | Up-Regulation - drug effects | Sphingosine - analogs & derivatives | Cell Line, Tumor | MicroRNAs - genetics | Carcinoma, Non-Small-Cell Lung - drug therapy | DNA Damage | HeLa Cells | MicroRNAs - physiology | Cell Cycle - drug effects | Index Medicus
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13. Full Text A Sodium-Pump-Mediated Afterhyperpolarization in Pyramidal Neurons
by Gulledge, Allan T and Dasari, Sameera and