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Molecular Nutrition & Food Research, ISSN 1613-4125, 10/2014, Volume 58, Issue 10, pp. 1931 - 1940
Scope Dietary polyphenols are suggested to play a role in the prevention of Alzheimer's disease, of which accumulation of aggregated beta amyloid (Aβ) is a key... 
Amyloid β peptide | Caenorhabditis elegans | Alzheimer's disease | Autophagy | Proteasome | Unfolded protein response | ALZHEIMERS-DISEASE | MEDITERRANEAN DIET | FOOD SCIENCE & TECHNOLOGY | NEURODEGENERATION | MODEL | IMPAIRMENT | GENE-EXPRESSION | Amyloid beta peptide | PRECURSOR PROTEIN | STRESS | AGGREGATION | Humans | Caenorhabditis elegans Proteins - metabolism | Lysosomes - enzymology | Lysosomes - metabolism | RNA Interference | Alzheimer Disease - prevention & control | Amyloid beta-Peptides - genetics | Protein Aggregation, Pathological - prevention & control | Proteolysis | Amyloid beta-Peptides - metabolism | Quercetin - administration & dosage | Muscles - metabolism | Peptide Fragments - genetics | Disease Models, Animal | Nerve Tissue Proteins - antagonists & inhibitors | Paralysis - chemically induced | Peptide Fragments - metabolism | Protein Aggregation, Pathological - enzymology | Animals, Genetically Modified | Quercetin - therapeutic use | Unfolded Protein Response | Nerve Tissue Proteins - genetics | Nerve Tissue Proteins - metabolism | Amyloid beta-Peptides - antagonists & inhibitors | Antioxidants - therapeutic use | Animals | Caenorhabditis elegans Proteins - antagonists & inhibitors | Muscles - enzymology | Peptide Fragments - antagonists & inhibitors | Paralysis - prevention & control | Alzheimer Disease - metabolism | Antioxidants - administration & dosage | Proteasome Endopeptidase Complex - metabolism | Caenorhabditis elegans Proteins - genetics | Dietary Supplements | Protein Aggregation, Pathological - metabolism | Proteins | Analysis | Polyphenols | Genetic engineering | Mitochondrial DNA | Paralysis
Journal Article
PLoS Genetics, ISSN 1553-7390, 02/2008, Volume 4, Issue 2, p. e24
In many organisms, dietary restriction appears to extend lifespan, at least in part, by down-regulating the nutrient-sensor TOR (Target Of Rapamycin). TOR... 
CANINE KIDNEY-CELLS | MESSENGER-RNA | SIGNALING PATHWAY | INDUCED LONGEVITY | TOR | GENES | GENETICS & HEREDITY | NEMATODE CAENORHABDITIS-ELEGANS | SACCHAROMYCES-CEREVISIAE | MEMBRANE-TRANSPORT | 3-KINASE COMPLEX | Phosphotransferases (Alcohol Group Acceptor) - physiology | Genes, Helminth | rab GTP-Binding Proteins - genetics | Autophagy - physiology | Phosphatidylinositol 3-Kinases - antagonists & inhibitors | Trans-Activators - physiology | Caenorhabditis elegans - physiology | RNA Interference | Receptor, Insulin - genetics | Trans-Activators - genetics | Autophagy - genetics | Vesicular Transport Proteins | Caenorhabditis elegans - growth & development | Animals, Genetically Modified | Caenorhabditis elegans - genetics | Phosphotransferases (Alcohol Group Acceptor) - genetics | Longevity - genetics | Receptor, Insulin - physiology | Phosphatidylinositol 3-Kinases - genetics | rab GTP-Binding Proteins - physiology | Animals | Caenorhabditis elegans Proteins - antagonists & inhibitors | Diet | Receptors, Nicotinic - physiology | Models, Biological | Phosphatidylinositol 3-Kinases - physiology | rab GTP-Binding Proteins - antagonists & inhibitors | Caenorhabditis elegans Proteins - physiology | Mutation | Trans-Activators - antagonists & inhibitors | Caenorhabditis elegans Proteins - genetics | Longevity - physiology | Receptors, Nicotinic - genetics | Proteins | Medical research | Kinases | Metabolism
Journal Article
Cancer Cell, ISSN 1535-6108, 2011, Volume 19, Issue 1, pp. 17 - 30
and mutations occur frequently in gliomas and acute myeloid leukemia, leading to simultaneous loss and gain of activities in the production of α-ketoglutarate... 
BREAST-CANCER | TRANSCRIPTIONAL ACTIVITY | IDH2 MUTATIONS | ONCOLOGY | PROLYL HYDROXYLATION | INTEGRATED GENOMIC ANALYSIS | 2-OXOGLUTARATE OXYGENASES | ACUTE MYELOID-LEUKEMIA | HIF-ALPHA | HISTONE DEMETHYLATION | FAMILY | CELL BIOLOGY | Dioxygenases - metabolism | Histone Demethylases - antagonists & inhibitors | Gene Expression - genetics | Caenorhabditis elegans Proteins - chemistry | Humans | Ketoglutaric Acids - chemistry | Glioma - genetics | F-Box Proteins | Oxidoreductases, N-Demethylating - antagonists & inhibitors | Ketoglutaric Acids - pharmacology | Cytosine - metabolism | Hypoxia-Inducible Factor 1, alpha Subunit - metabolism | Glutarates - chemistry | Oxidoreductases, N-Demethylating - metabolism | DNA-Binding Proteins - antagonists & inhibitors | Glioma - enzymology | Endostatins - metabolism | Models, Molecular | Isocitrate Dehydrogenase - genetics | Histone Demethylases - metabolism | Dioxygenases - antagonists & inhibitors | Amino Acid Substitution - physiology | Procollagen-Proline Dioxygenase - genetics | Cell Line, Tumor | Isocitrate Dehydrogenase - metabolism | Glutarates - pharmacology | Histones - metabolism | Jumonji Domain-Containing Histone Demethylases - metabolism | Caenorhabditis elegans - enzymology | Cytosine - analogs & derivatives | Gene Expression - drug effects | Caenorhabditis elegans Proteins - metabolism | Isocitrate Dehydrogenase - antagonists & inhibitors | Glioma - metabolism | Procollagen-Proline Dioxygenase - metabolism | DNA-Binding Proteins - metabolism | Mixed Function Oxygenases | Biocatalysis - drug effects | Jumonji Domain-Containing Histone Demethylases - antagonists & inhibitors | Jumonji Domain-Containing Histone Demethylases - chemistry | Procollagen-Proline Dioxygenase - antagonists & inhibitors | Ketoglutaric Acids - metabolism | Oxalates - pharmacology | Binding, Competitive | Proto-Oncogene Proteins - metabolism | Proto-Oncogene Proteins - antagonists & inhibitors | Catalytic Domain | Proto-Oncogene Proteins - genetics | Hypoxia-Inducible Factor-Proline Dioxygenases | DNA-Binding Proteins - genetics | Homeodomain Proteins - genetics | Animals | 5-Methylcytosine - metabolism | Caenorhabditis elegans Proteins - antagonists & inhibitors | Glutarates - metabolism
Journal Article
PLoS Pathogens, ISSN 1553-7366, 2014, Volume 10, Issue 6, p. e1004200
Microsporidia comprise a phylum of over 1400 species of obligate intracellular pathogens that can infect almost all animals, but little is known about the host... 
TRIGGERED IMMUNITY | IMMUNE-RESPONSE | LIFE-SPAN | MICROBIOLOGY | CELL-PROLIFERATION | LARGE GENE LISTS | TRANSLATIONAL INHIBITION | CAENORHABDITIS-ELEGANS | VIROLOGY | INTRACELLULAR BACTERIA | TRANSCRIPTION FACTOR | PARASITOLOGY | INNATE IMMUNITY | Caenorhabditis elegans Proteins - immunology | SKP Cullin F-Box Protein Ligases - genetics | Ubiquitin - metabolism | Caenorhabditis elegans Proteins - metabolism | Microsporidia - immunology | Autophagy - immunology | RNA Interference | Base Sequence | Caenorhabditis elegans - parasitology | Autophagy - genetics | Caenorhabditis elegans - virology | SKP Cullin F-Box Protein Ligases - metabolism | SKP Cullin F-Box Protein Ligases - antagonists & inhibitors | Caenorhabditis elegans - immunology | Microsporidia - pathogenicity | Host-Pathogen Interactions | Animals | Sequence Analysis, RNA | Caenorhabditis elegans Proteins - antagonists & inhibitors | RNA, Small Interfering | Cullin Proteins - immunology | Transcription, Genetic - genetics | Caenorhabditis elegans Proteins - genetics | Ubiquitination - genetics | Caenorhabditis elegans Proteins - biosynthesis | Cullin Proteins - biosynthesis | Ubiquitin | Distribution | Genetic aspects | Research | Gene expression | Health aspects | Microsporidia | Medical research | Nematodes | Infections | Experiments | Autophagy | Viral infections | Worms
Journal Article
Journal Article
Journal Article
Journal Article
Developmental Cell, ISSN 1534-5807, 10/2017, Volume 43, Issue 2, pp. 212 - 226.e7
Both transcriptional regulation and signaling pathways play crucial roles in neuronal differentiation and plasticity. possesses 19 GABAergic motor neurons... 
motor neurons | transcription factor | differentiation | cAMP | gene regulatory network | developmental plasticity | IDENTITY | NERVOUS-SYSTEM | CELLS | CAENORHABDITIS-ELEGANS | NUCLEAR HORMONE-RECEPTOR | MAP KINASE PATHWAY | AXON REGENERATION REQUIRES | DLK-1 KINASE | DEVELOPMENTAL BIOLOGY | GENOME | HOMEODOMAIN PROTEIN | CELL BIOLOGY | Homeodomain Proteins - metabolism | Caenorhabditis elegans Proteins - metabolism | Receptors, Cell Surface - antagonists & inhibitors | GABAergic Neurons - cytology | Gene Expression Regulation, Developmental | Transcription, Genetic | GABAergic Neurons - metabolism | Nuclear Proteins - genetics | Cyclic AMP - metabolism | Caenorhabditis elegans - metabolism | Caenorhabditis elegans - growth & development | Caenorhabditis elegans - genetics | Receptors, Cell Surface - metabolism | Nuclear Proteins - metabolism | Receptors, Cytoplasmic and Nuclear - genetics | Animals, Genetically Modified - metabolism | Homeodomain Proteins - genetics | Animals | Animals, Genetically Modified - growth & development | Caenorhabditis elegans Proteins - antagonists & inhibitors | Animals, Genetically Modified - genetics | Homeodomain Proteins - antagonists & inhibitors | Nuclear Proteins - antagonists & inhibitors | Receptors, Cytoplasmic and Nuclear - antagonists & inhibitors | CRISPR-Cas Systems | Caenorhabditis elegans Proteins - genetics | Receptors, Cell Surface - genetics | Receptors, Cytoplasmic and Nuclear - metabolism | Chromatin | Neurons | GABA | Cyclic adenylic acid | Genetic aspects | Genetic transcription | DNA binding proteins | Anopheles | Genes | Chronic diseases
Journal Article
Nature, ISSN 0028-0836, 06/2010, Volume 465, Issue 7298, pp. 577 - 583
Journal Article
Nature, ISSN 0028-0836, 01/2009, Volume 457, Issue 7226, pp. 210 - 214
Many organisms can enter a dormant state or diapause to survive harsh environmental conditions for extended durations. When Caenorhabditis elegans larvae enter... 
ADIPOSE TRIGLYCERIDE LIPASE | LIFE-SPAN | LONGEVITY | METABOLISM | SIGNALING PATHWAY | MULTIDISCIPLINARY SCIENCES | TISSUE | GENE-EXPRESSION | C-ELEGANS | RESTRICTION | HYPODERMIS | Protein-Serine-Threonine Kinases - deficiency | AMP-Activated Protein Kinases - metabolism | Phosphorylation | Caenorhabditis elegans Proteins - metabolism | AMP-Activated Protein Kinases - deficiency | Lipase - antagonists & inhibitors | Time Factors | Subcutaneous Tissue - metabolism | Protein-Serine-Threonine Kinases - metabolism | Life Cycle Stages - physiology | Caenorhabditis elegans - metabolism | Caenorhabditis elegans - growth & development | Signal Transduction | Fasting - physiology | Larva - metabolism | Protein-Serine-Threonine Kinases - genetics | Adaptation, Physiological - physiology | Rats | Lipid Metabolism | Lipase - metabolism | Longevity - genetics | AMP-Activated Protein Kinases - chemistry | Triglycerides - metabolism | Animals | Caenorhabditis elegans Proteins - antagonists & inhibitors | Survival Analysis | Larva - physiology | Water-Electrolyte Balance - genetics | Caenorhabditis elegans Proteins - genetics | Longevity - physiology | AMP-Activated Protein Kinases - genetics | Caenorhabditis elegans | Analysis | Genetic aspects | Cellular signal transduction | Research | Gene expression | Phosphotransferases | Tissue | Genotype & phenotype | Signal transduction | Genetics | Excretory system | Kinases | Worms
Journal Article
Science, ISSN 0036-8075, 12/2008, Volume 322, Issue 5907, pp. 1543 - 1546
During cytokinesis, the guanosine triphosphatase (GTPase) RhoA orchestrates contractile ring assembly and constriction. RhoA signaling is controlled by the... 
Medical research | Phenotypes | Kinesis | Microtubules | Lead | Reports | Cytokinesis | Embryos | Anaphase | Animal cells | Family members | CONTRACTILE RING | MGCRACGAP | ORGANIZATION | COMPLEX | CAENORHABDITIS-ELEGANS | MULTIDISCIPLINARY SCIENCES | SPINDLE | GTPASE-ACTIVATING PROTEIN | GENE-PRODUCT | RHOA | REQUIRE | Embryo, Nonmammalian - cytology | Caenorhabditis elegans Proteins - chemistry | Embryo, Nonmammalian - metabolism | Genes, Helminth | Caenorhabditis elegans Proteins - metabolism | rac GTP-Binding Proteins - metabolism | rhoA GTP-Binding Protein - metabolism | GTPase-Activating Proteins - metabolism | rac GTP-Binding Proteins - antagonists & inhibitors | Spindle Apparatus - physiology | Spindle Apparatus - ultrastructure | Protein Structure, Tertiary | Caenorhabditis elegans - metabolism | Signal Transduction | Caenorhabditis elegans - genetics | GTPase-Activating Proteins - chemistry | Caenorhabditis elegans - embryology | Kinesin - metabolism | Animals | Caenorhabditis elegans - cytology | Caenorhabditis elegans Proteins - antagonists & inhibitors | GTPase-Activating Proteins - genetics | Mutation | Caenorhabditis elegans Proteins - genetics | Amino Acid Substitution | Physiological aspects | Cellular proteins | Caenorhabditis elegans | Research | Properties | Proteins | Nematodes | Signal transduction | Cellular biology | Molecular biology
Journal Article