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Cell Death and Differentiation, ISSN 1350-9047, 11/2010, Volume 17, Issue 11, pp. 1785 - 1794
Journal Article
Developmental Cell, ISSN 1534-5807, 10/2016, Volume 39, Issue 1, pp. 13 - 27
Journal Article
Journal of Biological Chemistry, ISSN 0021-9258, 04/2015, Volume 290, Issue 17, pp. 10840 - 10849
Journal Article
Journal of the American Society of Nephrology, ISSN 1046-6673, 09/2017, Volume 28, Issue 9, pp. 2631 - 2640
Journal Article
Cardiovascular Research, ISSN 0008-6363, 12/2011, Volume 92, Issue 3, pp. 504 - 513
Aims Activation of transient receptor potential vanilloid type-1 (TRPV1) channels may affect lipid storage and the cellular inflammatory response. Now, we... 
Transient receptor potential vanilloid type-1 | Capsaicin | Vascular smooth muscle cells | Atherosclerosis | CARDIAC & CARDIOVASCULAR SYSTEMS | DEPENDENT PROTEIN-KINASE | RECEPTOR | MECHANISMS | DEFICIENT MICE | CHOLESTEROL EFFLUX | CALCINEURIN | SMOOTH-MUSCLE-CELLS | CHANNELS | EXPRESSION | Immunohistochemistry | Dietary Fats - metabolism | Muscle, Smooth, Vascular - metabolism | Calcium - metabolism | Capsaicin - pharmacology | Atherosclerosis - genetics | Aorta - metabolism | RNA, Messenger - metabolism | Apolipoproteins E - metabolism | Aortic Diseases - metabolism | TRPV Cation Channels - deficiency | Dose-Response Relationship, Drug | TRPV Cation Channels - metabolism | Time Factors | Dietary Fats - administration & dosage | Polymerase Chain Reaction | ATP-Binding Cassette Transporters - metabolism | Aortic Diseases - prevention & control | ATP Binding Cassette Transporter 1 | Myocytes, Smooth Muscle - drug effects | Myocytes, Smooth Muscle - metabolism | Aortic Diseases - pathology | Disease Models, Animal | Muscle, Smooth, Vascular - drug effects | Cyclic AMP-Dependent Protein Kinases - metabolism | Diterpenes - pharmacology | Atherosclerosis - pathology | Tumor Suppressor Proteins - metabolism | Aorta - drug effects | Mice, Inbred C57BL | TRPV Cation Channels - agonists | Cells, Cultured | Receptors, LDL - metabolism | Atherosclerosis - metabolism | Blotting, Western | TRPV Cation Channels - genetics | Mice, Knockout | Aortic Diseases - genetics | Animals | Apolipoproteins E - genetics | Lipid Metabolism - drug effects | Mice | Atherosclerosis - prevention & control | Calcineurin - metabolism | Index Medicus
Journal Article
Cell Metabolism, ISSN 1550-4131, 01/2017, Volume 25, Issue 1, pp. 182 - 196
Journal Article
SCIENCE, ISSN 0036-8075, 09/2017, Volume 357, Issue 6357, pp. 1255 - 1255
Mitochondrial and lysosomal dysfunction have been implicated in substantia nigra dopaminergic neurodegeneration in Parkinson's disease (PD), but how these... 
GLUCOCEREBROSIDASE | PACEMAKING | DJ-1 | ALPHA-SYNUCLEIN | SUBSTANTIA-NIGRA | MULTIDISCIPLINARY SCIENCES | Mitochondria - enzymology | Mesencephalon - metabolism | Humans | Protein Deglycase DJ-1 - genetics | Substantia Nigra - metabolism | Melanins - metabolism | Glucosylceramidase - deficiency | Lysosomes - metabolism | Dopaminergic Neurons - metabolism | Tacrolimus - pharmacology | Parkinson Disease - metabolism | Dopamine - metabolism | Disease Models, Animal | Substantia Nigra - enzymology | Cell Line | Calcineurin Inhibitors - pharmacology | Oxidation-Reduction | Mesencephalon - enzymology | Mitochondria - metabolism | Antioxidants - pharmacology | Mitochondria - drug effects | Parkinson Disease - genetics | Mice, Knockout | Animals | Parkinson Disease - enzymology | Mice | Oxidative Stress - drug effects | alpha-Synuclein - metabolism | Oxidation-reduction reaction | Development and progression | Mitochondria | Dopamine | Parkinson's disease | Health aspects | Brain | Energy metabolism | Animal models | Target recognition | Mesencephalon | Pathogenesis | Substantia nigra | Parkinsons disease | Lysosomes | Synuclein | Accumulation | Pathways | Enzymatic activity | Neurodegeneration | Rodents | Oxidation | Degeneration | Species | Movement disorders | Dopamine receptors | Neurodegenerative diseases | Neurons | Medical treatment | Metabolism | Patients | Glucosylceramidase | Index Medicus
Journal Article
American Journal of Physiology - Regulatory, Integrative and Comparative Physiology, ISSN 0363-6119, 02/2011, Volume 300, Issue 2, pp. 408 - 417
Our aim was to analyze the role of phosphatidylinositol 3-kinase (PI3K)-AKT and MAPK signaling pathways in the regulation of muscle mass and slow-to-fast... 
Myosin heavy chain isoform | Low-frequency stimulation | Glycolytic metabolism | Mitogen-activated protein kinase pathway | SKELETAL-MUSCLE ATROPHY | PHYSIOLOGY | mitogen-activated protein kinase pathway | ACTIVATED PROTEIN-KINASE | RAT | glycolytic metabolism | HYPERTROPHY | RESPONSES | CONTRACTILE | GENE | myosin heavy chain isoform | CALCINEURIN | TNF-ALPHA | low-frequency stimulation | PLASTICITY | Phosphorylation - physiology | Electric Stimulation | Rats, Wistar | TOR Serine-Threonine Kinases - metabolism | Muscle Fibers, Fast-Twitch - metabolism | Protein Biosynthesis - physiology | JNK Mitogen-Activated Protein Kinases - metabolism | Male | Muscle, Skeletal - innervation | Muscle, Skeletal - metabolism | Muscle Fibers, Slow-Twitch - metabolism | Phosphatidylinositol 3-Kinases - metabolism | Extracellular Signal-Regulated MAP Kinases - metabolism | Hindlimb Suspension - physiology | Myosin Heavy Chains - metabolism | Tibial Nerve - physiology | Glycolysis - physiology | Protein Isoforms - metabolism | Forkhead Transcription Factors - metabolism | Up-Regulation - physiology | Muscular Atrophy - prevention & control | p38 Mitogen-Activated Protein Kinases - metabolism | Proto-Oncogene Proteins c-akt - metabolism | Muscular Atrophy - etiology | Muscular Atrophy - metabolism | Muscular Atrophy - pathology | Rats | Down-Regulation - physiology | Nerve Tissue Proteins - metabolism | Animals | Proteins - metabolism | Mitogen-Activated Protein Kinase 3 - metabolism | Muscle Fibers, Slow-Twitch - pathology | Signal Transduction - physiology | Muscle, Skeletal - pathology | Mitogen-Activated Protein Kinase 1 - metabolism | Proteins | Genotype & phenotype | Signal transduction | Rodents | Physiology | Kinases | Metabolism | Binding sites | Index Medicus
Journal Article
The Journal of Cell Biology, ISSN 0021-9525, 1/2005, Volume 168, Issue 2, pp. 193 - 199
Amyotrophic lateral sclerosis (ALS) is a progressive neurodegenerative disease characterized by a selective degeneration of motor neurons, atrophy, and... 
Protein isoforms | Motor neurons | Nervous system diseases | Spinal cord | Muscular diseases | Transgenic animals | Motor neuron disease | Muscles | Amyotrophic lateral sclerosis | Reports | Skeletal muscle | SKELETAL-MUSCLE | HYPERTROPHY | REGENERATION | DISEASE | CALCINEURIN | MICE | ASSOCIATION | GROWTH-FACTOR-I | GATA-2 | CELL BIOLOGY | Immunohistochemistry | Tumor Necrosis Factor-alpha - metabolism | Superoxide Dismutase - genetics | Central Nervous System - metabolism | Gene Expression - genetics | Neuromuscular Junction - metabolism | Homeodomain Proteins - metabolism | Humans | PAX7 Transcription Factor | Central Nervous System - pathology | Muscle, Skeletal - metabolism | Insulin-Like Growth Factor I - genetics | Glial Fibrillary Acidic Protein - metabolism | Myosin Heavy Chains - metabolism | Motor Neurons - pathology | Calcineurin - genetics | Muscle, Skeletal - chemistry | Central Nervous System - chemistry | Superoxide Dismutase - metabolism | Disease Models, Animal | Agrin - genetics | Receptors, Cholinergic - metabolism | Mice, Transgenic | Satellite Cells, Skeletal Muscle - metabolism | Survival Rate | Blotting, Western | Protein Isoforms - physiology | Amyotrophic Lateral Sclerosis - mortality | Motor Neurons - metabolism | Walking | Amyotrophic Lateral Sclerosis - pathology | Blotting, Northern | Animals | Insulin-Like Growth Factor I - physiology | Desmin - metabolism | Amyotrophic Lateral Sclerosis - metabolism | Satellite Cells, Skeletal Muscle - chemistry | Muscle Fibers, Skeletal - cytology | Mice | Muscle, Skeletal - pathology | Superoxide Dismutase-1 | Calcineurin - metabolism | Astrocytes - metabolism | Protein Isoforms - genetics | Receptors, Cholinergic - genetics | Neurons | Rodents | Motor ability | Models | Muscular system | Gene expression | Index Medicus | s
Journal Article
Journal Article
Nature Communications, ISSN 2041-1723, 12/2017, Volume 8, Issue 1, pp. 2270 - 2270
Drugs that mirror the cellular effects of starvation mimics are considered promising therapeutics for common metabolic disorders, such as obesity, liver... 
LOOP-HELIX PROTEIN | REGULATOR | ACTIVATED PROTEIN-KINASE | LIPID-METABOLISM | MULTIDISCIPLINARY SCIENCES | CALCIUM | CALCINEURIN | RECEPTOR | AUTOPHAGY | ENERGY-METABOLISM | LYSOSOMAL BIOGENESIS | Biguanides - pharmacology | Calcium - metabolism | Caloric Restriction | Longevity - drug effects | Humans | Metabolic Syndrome - metabolism | Autophagy - drug effects | Basic Helix-Loop-Helix Leucine Zipper Transcription Factors - agonists | Autophagosomes - drug effects | Lysosomes - metabolism | Liver - drug effects | Diet, High-Fat | Lactams - pharmacology | Lysosomes - drug effects | Starvation | Caenorhabditis elegans - metabolism | Fatty Liver - metabolism | Liver - metabolism | Enzyme Inhibitors - pharmacology | Mitochondria - metabolism | Autophagosomes - metabolism | Mitochondria - drug effects | Digoxin - pharmacology | Animals | Caenorhabditis elegans - drug effects | High-Throughput Screening Assays | Lipid Metabolism - drug effects | Mice | HeLa Cells | Drugs | Animal models | Digoxin | Calcium | Biodegradability | Liver | Disorders | Autophagy | Nanoparticles | Dietary restrictions | Mitochondria | Fatty liver | Biocompatibility | Lipid metabolism | Biodegradation | Metabolic diseases | Lead compounds | Metabolism | Steatosis | Mode of action | Life span | Nematodes | Aging (natural) | Metabolic disorders | Nanotechnology | Index Medicus
Journal Article
Cell Metabolism, ISSN 1550-4131, 12/2015, Volume 22, Issue 6, pp. 1020 - 1032
Chronic kidney disease (CKD) is a worldwide public health threat that increases risk of death due to cardiovascular complications, including left ventricular... 
SIGNAL-TRANSDUCTION | MORTALITY | FACTOR FAMILY | KLOTHO | POINT MUTATION | TYROSINE KINASE | ENDOCRINOLOGY & METABOLISM | CHRONIC KIDNEY-DISEASE | CANCER PROGRESSION | EXPRESSION | FGF RECEPTOR | CELL BIOLOGY | Receptor, Fibroblast Growth Factor, Type 4 - metabolism | Humans | Glucuronidase - metabolism | Fibroblast Growth Factors - genetics | Male | Renal Insufficiency, Chronic - metabolism | Fibroblast Growth Factors - metabolism | Hypertrophy, Left Ventricular - pathology | HEK293 Cells | Receptor, Fibroblast Growth Factor, Type 4 - deficiency | Female | Receptor, Fibroblast Growth Factor, Type 4 - genetics | Disease Models, Animal | Phospholipase C gamma - metabolism | Mutagenesis, Site-Directed | Myocytes, Cardiac - cytology | Signal Transduction | Hypertrophy, Left Ventricular - metabolism | Mice, Inbred C57BL | NFATC Transcription Factors - metabolism | Cells, Cultured | Rats | Rats, Sprague-Dawley | Gene Knock-In Techniques | Mice, Knockout | Renal Insufficiency, Chronic - pathology | Animals | Glucuronidase - genetics | Myocytes, Cardiac - metabolism | Mice | Calcineurin - metabolism | Hypertension | Medical colleges | Chronic kidney failure | Heart enlargement | Stem cells | Physiological aspects | Fibroblast growth factors | Phospholipases | Drug discovery | T cells | Health aspects | Heart | Resveratrol | Development and progression | Kidney diseases | Cells | Index Medicus
Journal Article