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Circulation research, ISSN 1524-4571, 2009, Volume 104, Issue 6, pp. 733 - 741
Vascular calcification is a major risk factor for cardiovascular morbidity and mortality. To develop appropriate prevention and/or therapeutic strategies for... 
Vascular calcification | Smooth muscle cells | Genetic fate mapping | Lineage reprogramming | Runx2/Cbfa1 | WALL | CARDIAC & CARDIOVASCULAR SYSTEMS | vascular calcification | ATHEROSCLEROSIS | lineage reprogramming | genetic fate mapping | CARTILAGE | smooth muscle cells | IN-VITRO | DEFICIENT | CALCIFICATION | PERIPHERAL VASCULAR DISEASE | MICE | MUTATIONS | MATRIX-GLA-PROTEIN | HEMATOLOGY | EXPRESSION | Calcinosis - genetics | Vascular Diseases - pathology | Homeodomain Proteins - metabolism | Humans | Myocytes, Smooth Muscle - pathology | Extracellular Signal-Regulated MAP Kinases - metabolism | RNA, Messenger - metabolism | Stem Cells - metabolism | Wnt Proteins - metabolism | Core Binding Factor Alpha 1 Subunit - metabolism | Extracellular Signal-Regulated MAP Kinases - genetics | Arteries - metabolism | MAP Kinase Signaling System - genetics | Wnt Proteins - genetics | Mice, Mutant Strains | Bone Morphogenetic Protein 2 - metabolism | Calcinosis - metabolism | Myocytes, Smooth Muscle - metabolism | Chondrocytes - metabolism | SOX9 Transcription Factor - metabolism | Bone Morphogenetic Protein 2 - genetics | Chondrocytes - pathology | RNA, Messenger - genetics | Vascular Diseases - genetics | Arteries - pathology | Transcription Factors - genetics | Down-Regulation - genetics | Homeodomain Proteins - genetics | Mice, Knockout | Proteins - genetics | Transcription Factors - metabolism | Animals | Proteins - metabolism | Sp7 Transcription Factor | Stem Cells - pathology | Wnt3 Protein | Mice | Calcinosis - pathology | Vascular Diseases - metabolism | Wnt3A Protein | Cell Dedifferentiation - genetics | SOX9 Transcription Factor - genetics | Core Binding Factor Alpha 1 Subunit - genetics | Cbfa1 | Runx2
Journal Article
Arteriosclerosis, thrombosis, and vascular biology, ISSN 1524-4636, 2007, Volume 27, Issue 12, pp. 2589 - 2596
OBJECTIVE—Aortic calcification is prevalent in type II diabetes (T2DM), enhancing morbidity and tracking metabolic syndrome parameters. Ldlr mice fed high-fat... 
Aortic calcification | Diabetes | TNF-α | Metabolic syndrome | Wnt | TNF-alpha | BONE MORPHOGENETIC PROTEIN-2 | MATRIX GLA PROTEIN | VASCULAR CALCIFICATION | aortic calcification | DEFICIENT MICE | metabolic syndrome | INFLAMMATION | IN-VIVO | ARTERIAL CALCIFICATION | PERIPHERAL VASCULAR DISEASE | SMOOTH-MUSCLE-CELLS | DIFFERENTIATION | HEMATOLOGY | diabetes | EXPRESSION | Inflammation - pathology | Fibroblasts - enzymology | Homeodomain Proteins - metabolism | Tumor Necrosis Factor-alpha - blood | Tumor Necrosis Factor-alpha - genetics | Antibodies, Monoclonal - therapeutic use | Male | Aorta - metabolism | Haptoglobins - metabolism | RNA, Messenger - metabolism | Wnt Proteins - metabolism | Aortic Diseases - metabolism | Bone Morphogenetic Proteins - metabolism | Inflammation - metabolism | Calcinosis - etiology | Dietary Fats - administration & dosage | Anti-Inflammatory Agents - therapeutic use | Aortic Diseases - prevention & control | Disease Models, Animal | Fibroblasts - metabolism | Anti-Inflammatory Agents - pharmacology | Antibodies, Monoclonal - pharmacology | Receptors, LDL - metabolism | Mice, Transgenic | Mice, Knockout | Aorta - pathology | Muscle Proteins - genetics | Signal Transduction - drug effects | Wnt3 Protein | Calcinosis - prevention & control | Inflammation - prevention & control | Mice | Diabetes Mellitus, Type 2 - pathology | Calcinosis - pathology | Wnt3A Protein | Tumor Necrosis Factor-alpha - metabolism | Alkaline Phosphatase | Diabetes Mellitus, Type 2 - metabolism | DNA-Binding Proteins - metabolism | Intercellular Signaling Peptides and Proteins - metabolism | Wnt Proteins - genetics | Infliximab | Receptors, LDL - deficiency | Calcinosis - metabolism | Microfilament Proteins - genetics | Aorta - enzymology | Diabetes Mellitus, Type 2 - complications | Aortic Diseases - pathology | Promoter Regions, Genetic | Receptors, LDL - genetics | Bone Morphogenetic Protein 2 | Mice, Inbred C57BL | Cells, Cultured | DNA-Binding Proteins - genetics | Homeodomain Proteins - genetics | Animals | Diabetes Mellitus, Type 2 - chemically induced | Transforming Growth Factor beta - metabolism | Aortic Diseases - etiology | Tumor Necrosis Factor-alpha - antagonists & inhibitors
Journal Article
Arteriosclerosis, thrombosis, and vascular biology, ISSN 1524-4636, 2013, Volume 33, Issue 1, pp. 121 - 130
OBJECTIVE—Inflammatory activation of valve endothelium is an early phase of aortic valve disease pathogenesis, but subsequent mechanisms are poorly understood.... 
calcification | invasion | transforming growth factor-β | smooth muscle actin | tumor necrosis factor-α | PROGENITOR CELLS | ENDOCARDIAL CUSHION TISSUE | ATHEROSCLEROSIS | FACTOR-KAPPA-B | INTERSTITIAL-CELLS | transforming growth factor-beta | tumor necrosis factor-alpha | HEART | ARTERIAL | GROWTH | RECEPTOR ACTIVATOR | PERIPHERAL VASCULAR DISEASE | HEMATOLOGY | Tumor Necrosis Factor-alpha - metabolism | Inflammation - pathology | Heart Valve Diseases - pathology | Humans | Transforming Growth Factor beta1 - metabolism | NF-kappa B - metabolism | Epithelial-Mesenchymal Transition - genetics | Heart Valve Diseases - immunology | Aortic Valve - pathology | Inflammation - metabolism | Transfection | Time Factors | Swine | Inflammation Mediators - metabolism | Calcinosis - metabolism | Proto-Oncogene Proteins c-akt - metabolism | Interleukin-6 - metabolism | Protein-Serine-Threonine Kinases - metabolism | Aortic Valve - immunology | Recombinant Proteins - metabolism | Signal Transduction | Endothelial Cells - metabolism | Aortic Valve - embryology | Cells, Cultured | Gene Expression Regulation | Activin Receptors, Type I - metabolism | Inflammation - immunology | Quail | Aortic Valve - metabolism | Chick Embryo | Endothelial Cells - immunology | Animals | NF-kappa B - genetics | Receptors, Transforming Growth Factor beta - metabolism | Heart Valve Diseases - metabolism | Heart Valve Diseases - genetics | Inflammation - genetics | Calcinosis - immunology | Primary Cell Culture | Calcinosis - pathology | Endothelial Cells - pathology | Cell Movement
Journal Article
Arteriosclerosis, thrombosis, and vascular biology, ISSN 1524-4636, 2015, Volume 35, Issue 2, pp. 399 - 408
OBJECTIVE—Vascular and valvular calcifications are pathological processes regulated by resident cells, and depending on a complex interplay between calcification promoters and inhibitors, resembling skeletal metabolism... 
multivesicular bodies | aortic valve, calcification of | gene expression | vascular calcification | aortic valve | calcification of | AORTIC-STENOSIS | MATRIX VESICLES | VALVE | CARTILAGE | DISEASE | VITAMIN-K | PERIPHERAL VASCULAR DISEASE | HEMATOLOGY | EXPRESSION | FETUIN-A | INSIGHTS | Calcinosis - genetics | Aortic Valve Stenosis - genetics | Calcium - metabolism | Humans | Middle Aged | Vascular Calcification - genetics | Actins - metabolism | Osteocalcin - genetics | Male | Aorta - metabolism | Case-Control Studies | Aortic Valve - pathology | Coronary Vessels - metabolism | Aortic Valve Stenosis - prevention & control | Vascular Calcification - metabolism | Coronary Artery Disease - pathology | Aged, 80 and over | Adult | Female | Calcinosis - metabolism | Extracellular Matrix Proteins - metabolism | Calcium-Binding Proteins - metabolism | Coronary Vessels - pathology | Osteocalcin - metabolism | Tissue Culture Techniques | Coronary Artery Disease - metabolism | Extracellular Matrix Proteins - genetics | Coronary Artery Disease - prevention & control | Gene Expression Regulation | Vascular Calcification - prevention & control | Vascular Calcification - pathology | Aortic Valve - metabolism | Aorta - pathology | Proteins - genetics | Aortic Valve Stenosis - pathology | Proteins - metabolism | Coronary Artery Disease - genetics | alpha-2-HS-Glycoprotein - metabolism | Aortic Valve Stenosis - metabolism | Aged | Calcinosis - prevention & control | Calcinosis - pathology | Calcium-Binding Proteins - genetics
Journal Article
PLoS ONE, ISSN 1932-6203, 06/2013, Volume 8, Issue 6, p. e68197
Free fatty acids (FFAs), elevated in metabolic syndrome and diabetes, play a crucial role in the development of atherosclerotic cardiovascular disease, and... 
SUPPLEMENTATION | ARTERY ENDOTHELIAL-CELLS | PROTEIN-KINASE | MULTIDISCIPLINARY SCIENCES | DISEASE | CALCIUM | CALCIFICATION | RISK | FREE FATTY-ACID | DEPENDENT ACTIVATION | EXPRESSION | Calcinosis - genetics | Carotid Arteries - metabolism | Osteopontin - genetics | Palmitic Acid - metabolism | Coenzyme A Ligases - genetics | Muscle, Smooth, Vascular - metabolism | Calcium - metabolism | Gene Expression - genetics | Homeodomain Proteins - metabolism | Humans | Eicosapentaenoic Acid - genetics | Myocytes, Smooth Muscle - pathology | NF-kappa B - metabolism | Osteopontin - metabolism | Aorta - metabolism | Promoter Regions, Genetic - genetics | Coenzyme A Ligases - metabolism | Cell Differentiation - genetics | Caspases - metabolism | Bone Morphogenetic Protein 2 - metabolism | Calcinosis - metabolism | Plaque, Atherosclerotic - pathology | Cell Differentiation - physiology | Myocytes, Smooth Muscle - metabolism | Bone Morphogenetic Protein 2 - genetics | Macrophages - pathology | Plaque, Atherosclerotic - metabolism | Caspases - genetics | Cells, Cultured | Eicosapentaenoic Acid - metabolism | Homeodomain Proteins - genetics | Aorta - pathology | Osteoblasts - pathology | Macrophages - metabolism | Muscle, Smooth, Vascular - pathology | NF-kappa B - genetics | Carotid Arteries - pathology | Calcinosis - pathology | Osteoblasts - metabolism | Immunohistochemistry | Smooth muscle | Cardiovascular disease | Palmitic acid | Osteoblasts | Proteins | Mineralization | Biocompatibility | Aorta | Inhibition | NF-κB protein | Cytokines | Osteopontin | Metabolism | Gene expression | Fatty acids | Studies | Osteoblastogenesis | Inhibitors | Arteriosclerosis | Calcification | Cardiovascular diseases | Metabolic disorders | Calcification (ectopic) | Health sciences | Oxidative stress | Calcium | Laboratories | Chains | Lipids | Activation | Kinases | Macrophages | Carotid arteries | Arteries | Msx2 protein | Rodents | Atherosclerosis | Tumor necrosis factor-TNF | Carotid artery | Deposition | Plaques | University graduates | Bone morphogenetic protein 2 | Diabetes mellitus | Muscles | Caspase | siRNA | Medicine | Antibiotics | Kidney diseases | Diabetes | Differentiation | Eicosapentaenoic acid | Apoptosis | Pharmaceuticals
Journal Article
Atherosclerosis, ISSN 0021-9150, 2012, Volume 224, Issue 2, pp. 340 - 347
Abstract Objective The objective of this study is to develop a method for selective detection of the calcific (hydroxyapatite) component in human aortic smooth... 
Cardiovascular | Molecular imaging | Calcification | Osteocalcin | Hydroxyapatite | Atherosclerosis | ATHEROSCLEROTIC LESIONS | CARDIAC & CARDIOVASCULAR SYSTEMS | VASCULAR CALCIFICATION | DEFICIENT MICE | STEM-CELL DIFFERENTIATION | IN-VIVO | ARTERIAL CALCIFICATION | MINERALIZATION | PERIPHERAL VASCULAR DISEASE | BONE | ENERGY-METABOLISM | Carotid Arteries - metabolism | Heart Valve Diseases - pathology | Muscle, Smooth, Vascular - metabolism | Humans | Fluorescein-5-isothiocyanate - metabolism | Fluorescent Dyes - metabolism | Calcinosis - diagnostic imaging | Carbocyanines - metabolism | Aorta - metabolism | X-Ray Microtomography | Aortic Valve - pathology | Durapatite - metabolism | Vascular Calcification - metabolism | Molecular Probes - metabolism | Time Factors | Calcinosis - metabolism | Biomarkers - metabolism | Atherosclerosis - pathology | Aortic Valve - diagnostic imaging | Osteocalcin - metabolism | Cells, Cultured | Molecular Imaging - methods | Oligopeptides - metabolism | Plaque, Atherosclerotic | Vascular Calcification - pathology | Aortic Valve - metabolism | Atherosclerosis - metabolism | Aorta - pathology | Muscle, Smooth, Vascular - pathology | Heart Valve Diseases - metabolism | Protein Binding | Carotid Arteries - pathology | Kinetics | Magnetic Fields | Calcinosis - pathology | Microscopy, Fluorescence | Osteogenesis | Heart Valve Diseases - diagnostic imaging | Hydroxylapatite | atherosclerosis | hydroxyapatite | osteocalcin | calcification | molecular imaging
Journal Article
Arteriosclerosis, thrombosis, and vascular biology, ISSN 1524-4636, 2014, Volume 34, Issue 7, pp. 1399 - 1411
OBJECTIVE—S100A12 and fibroblast growth factor 23 are biomarkers of cardiovascular morbidity and mortality in patients with chronic kidney disease (CKD). We... 
renal insufficiency chronic | S100 proteins | glycosylation end products advanced | hypertrophy left ventricular | MORTALITY | PLASMA S100A12 LEVELS | renal insufficiency, chronic | VASCULAR CALCIFICATION | hypertrophy, left ventricular | ATHEROSCLEROSIS | RAGE | ARTERIAL CALCIFICATION | glycosylation end products, advanced | GROWTH-FACTOR 23 | HEMODIALYSIS-PATIENTS | PERIPHERAL VASCULAR DISEASE | HEMATOLOGY | EXPRESSION | SOLUBLE RECEPTOR | Calgranulin B - metabolism | Diastole | Humans | Calgranulin A - genetics | Renal Insufficiency, Chronic - complications | Sclerosis | S100A12 Protein | Fibroblast Growth Factors - metabolism | Inflammation - metabolism | S100 Proteins - genetics | Hypertrophy, Left Ventricular - genetics | Calcinosis - etiology | Time Factors | Myocardium - metabolism | Renal Insufficiency, Chronic - genetics | Receptor for Advanced Glycation End Products | Disease Models, Animal | Fibroblasts - metabolism | Signal Transduction | Receptors, Immunologic - deficiency | Genotype | Mice, Transgenic | S100 Proteins - metabolism | Mice, Knockout | Ventricular Dysfunction, Left - metabolism | Phenotype | Heart Valve Diseases - metabolism | Heart Valve Diseases - genetics | Mice | Hypertrophy, Left Ventricular - physiopathology | Receptors, Immunologic - genetics | Heart Valve Diseases - pathology | Ventricular Function, Left | Calgranulin A - metabolism | Leukocyte L1 Antigen Complex - metabolism | Aortic Valve - pathology | Renal Insufficiency, Chronic - metabolism | Inflammation - complications | Ventricular Remodeling | Inflammation Mediators - metabolism | Calgranulin B - genetics | Calcinosis - metabolism | Hypertrophy, Left Ventricular - etiology | Hypertrophy, Left Ventricular - metabolism | Mice, Inbred C57BL | Cells, Cultured | Ventricular Dysfunction, Left - etiology | Heart Valve Diseases - etiology | Aortic Valve - metabolism | Ventricular Dysfunction, Left - physiopathology | Animals | Inflammation - genetics | Leukocyte L1 Antigen Complex - genetics | Receptors, Immunologic - metabolism
Journal Article
Arteriosclerosis, thrombosis, and vascular biology, ISSN 1524-4636, 2012, Volume 32, Issue 11, pp. 2711 - 2720
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Journal Article
Circulation Research, ISSN 0009-7330, 06/2013, Volume 113, Issue 1, pp. 72 - 77
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Circulation research, ISSN 1524-4571, 2011, Volume 108, Issue 4, pp. 446 - 457
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