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British Journal of Pharmacology, ISSN 0007-1188, 02/2014, Volume 171, Issue 3, pp. 772 - 788
Background and Purpose Receptor activity‐modifying proteins (RAMPs) define the pharmacology of the calcitonin receptor‐like receptor (CLR). The interactions of... 
GPCR | adrenomedullin | RAMP | CGRP | receptor activity‐modifying protein | receptor activity-modifying protein | CRYSTAL-STRUCTURE | EXTRACELLULAR DOMAIN | N-TERMINUS | FAMILY | CORTICOTROPIN-RELEASING-FACTOR | STRUCTURAL BASIS | MOLECULAR RECOGNITION | PHARMACOLOGY & PHARMACY | CLASS-B GPCR | COUPLED-RECEPTOR | BINDING | Receptors, Adrenomedullin - chemistry | Humans | Adrenomedullin - chemistry | Cercopithecus aethiops | Receptor Activity-Modifying Protein 1 - chemistry | Receptor Activity-Modifying Protein 2 - chemistry | Receptor Activity-Modifying Protein 3 - genetics | Calcitonin Gene-Related Peptide - chemistry | Receptors, Calcitonin Gene-Related Peptide - metabolism | Recombinant Fusion Proteins - metabolism | Peptide Hormones - metabolism | Receptors, Calcitonin Gene-Related Peptide - chemistry | Peptide Hormones - chemistry | Protein Interaction Domains and Motifs | Calcitonin Gene-Related Peptide - metabolism | Cyclic AMP - metabolism | Peptide Fragments - genetics | Second Messenger Systems | Recombinant Proteins - metabolism | Peptide Fragments - metabolism | Receptors, Adrenomedullin - metabolism | Calcitonin Receptor-Like Protein - chemistry | Models, Molecular | Rats | Receptor Activity-Modifying Protein 1 - metabolism | Recombinant Proteins - chemistry | Mutant Proteins - metabolism | Receptor Activity-Modifying Protein 2 - metabolism | Adrenomedullin - metabolism | Receptor Activity-Modifying Protein 1 - genetics | Recombinant Fusion Proteins - chemistry | Calcitonin Receptor-Like Protein - metabolism | Peptide Fragments - chemistry | Animals | Receptor Activity-Modifying Protein 3 - chemistry | Calcitonin Receptor-Like Protein - genetics | Mutant Proteins - chemistry | Receptor Activity-Modifying Protein 3 - metabolism | Receptor Activity-Modifying Protein 2 - genetics | COS Cells | Proteins | Genetic research | Pharmacology | Genetic aspects | Algorithms | Analysis | Peptides | Mutation | Research Papers
Journal Article
Nature Medicine, ISSN 1078-8956, 10/2016, Volume 22, Issue 10, pp. 1160 - 1169
Orthopedic implants containing biodegradable magnesium have been used for fracture repair with considerable efficacy; however, the underlying mechanisms by... 
MEDICINE, RESEARCH & EXPERIMENTAL | GENE-RELATED PEPTIDE | VIVO | BIOCHEMISTRY & MOLECULAR BIOLOGY | INNERVATION | OSTEOPOROSIS | CELL BIOLOGY | IN-VITRO | TRAUMATIC BRAIN-INJURY | ALLOYS | DIFFERENTIATION | OSTEOARTHRITIS | EXPRESSION | Bone Nails | Humans | Fracture Fixation, Intramedullary | Ganglia, Spinal - cytology | Cyclic AMP Response Element-Binding Protein - drug effects | Gene Knockdown Techniques | Transcription Factors - drug effects | Stem Cells | Fracture Healing - drug effects | Osteoporosis, Postmenopausal | Cation Transport Proteins - metabolism | Female | Neurons - metabolism | Calcitonin Gene-Related Peptide - metabolism | Neurons - drug effects | Osteogenesis - genetics | Osteoporotic Fractures | Calcitonin Gene-Related Peptide - drug effects | Ovariectomy | Calcitonin Gene-Related Peptide - pharmacology | Osteogenesis - drug effects | Capsaicin - toxicity | Femoral Fractures | Rats | Femur - drug effects | Sensory System Agents - toxicity | Receptor Activity-Modifying Protein 1 - genetics | Magnesium - pharmacology | Gene Knock-In Techniques | Periosteum - cytology | Transcription Factors - metabolism | Animals | Cell Differentiation - drug effects | Calcitonin Receptor-Like Protein - genetics | Cyclic AMP Response Element-Binding Protein - metabolism | TRPM Cation Channels - metabolism | Fracture Healing - genetics | Denervation | Neuropeptides | Fracture fixation | Genetic aspects | Wound healing | Health aspects | Surgical implants | Femur | Transplants & implants | Biodegradability | Capsaicin | Genes | Menopause | Differentiation (biology) | Stem cell transplantation | Cortex (somatosensory) | Receptor activity modifying proteins | Proteins | Osteoporosis | Biomedical materials | Bone growth | Allografts | Dorsal root ganglia | Surgery | Calcitonin | Nerves | Sensory neurons | Biocompatibility | Ion channels | Magnesium | Bone (cortical) | Repair | Adenosine triphosphate | Biodegradation | Periosteum | Implantation | Bone healing | Ganglia | Calcitonin gene-related peptide | Bone implants | Fractures | Orthopedics | Stem cells | Healing | Bone | ATP | Transporter | Osteogenesis
Journal Article
British Journal of Pharmacology, ISSN 0007-1188, 01/2018, Volume 175, Issue 1, pp. 3 - 17
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 11/2012, Volume 109, Issue 46, pp. 18985 - 18990
Cortical spreading depression (CSD) is a key pathogenetic step in migraine with aura. Dysfunctions of voltage-dependent and receptor-operated channels have... 
Headache | Calcitonin gene related peptide receptors | Pain | Genetic variation | N methyl D aspartate receptors | Migraine | Imaging | Instructional materials | Rats | Depressive disorders | Epilepsy | Cortex | cortex | epilepsy | pain | RAT | MULTIDISCIPLINARY SCIENCES | MECHANISMS | PATHOPHYSIOLOGY | BLOOD-FLOW CHANGES | PROPHYLAXIS | DEPOLARIZATION | NEOCORTICAL SLICES | CGRP ANTAGONISTS | HEMIPLEGIC MIGRAINE TYPE-1 | headache | Receptors, N-Methyl-D-Aspartate - antagonists & inhibitors | Rats, Wistar | Cerebral Cortex - pathology | Receptors, N-Methyl-D-Aspartate - metabolism | Male | Anticonvulsants - pharmacology | Receptors, Calcitonin Gene-Related Peptide - metabolism | Cerebral Cortex - metabolism | Cerebral Cortex - physiopathology | Migraine Disorders - metabolism | Dose-Response Relationship, Drug | Fructose - pharmacology | Migraine Disorders - pathology | Migraine Disorders - physiopathology | Receptors, AMPA - metabolism | Carbamazepine - pharmacology | Calcitonin Gene-Related Peptide - pharmacology | Receptors, Calcitonin Gene-Related Peptide - antagonists & inhibitors | Calcitonin Gene-Related Peptide - pharmacokinetics | Migraine Disorders - drug therapy | Voltage-Gated Sodium Channels | Cortical Spreading Depression - drug effects | Fructose - analogs & derivatives | Animals | Receptors, AMPA - antagonists & inhibitors | Peptides | Calcitonin | Physiological aspects | Genetic aspects | Research | Gene expression | Health aspects | Biological Sciences
Journal Article
FRONTIERS IN PHARMACOLOGY, ISSN 1663-9812, 04/2019, Volume 10, p. 363
Migraine is a common neurologic disorder characterized by attacks consisting of unilateral, throbbing headache accompanied by photophobia, phonophobia, and... 
treatment | migraine | anti-CGRP | monoclonal antibodies anti-CGRP | RECEPTOR | ERENUMAB | TRIAL | PHARMACOKINETICS | DRUGS | PHARMACOLOGY & PHARMACY | CHANNELS | CGRP | pharmacology of migraine | Monoclonal antibodies | Peptides | Drug therapy | Migraine | Calcitonin | Genes
Journal Article
Circulation, ISSN 0009-7322, 07/2017, Volume 136, Issue 4, pp. 367 - 383
BACKGROUND:Research into the therapeutic potential of α-calcitonin gene–related peptide (α-CGRP) has been limited because of its peptide nature and short... 
heart failure | inflammation | hypertension | oxidative stress | receptors, calcitonin gene-related peptide | INDUCED RENAL DAMAGE | CARDIAC & CARDIOVASCULAR SYSTEMS | MYOCARDIAL-INFARCTION | ANGIOTENSIN-II | BLOOD-PRESSURE | II-INDUCED HYPERTENSION | MESSENGER-RNA | IN-VIVO | PERIPHERAL VASCULAR DISEASE | SMOOTH-MUSCLE-CELLS | NEUROGENIC VASODILATATION | Multiple Organ Failure - metabolism | Calcitonin Gene-Related Peptide - analogs & derivatives | Oxidative Stress - physiology | Cardiomegaly - pathology | Hypertension - drug therapy | Male | Cardiotonic Agents - therapeutic use | Multiple Organ Failure - pathology | Calcitonin Gene-Related Peptide - pharmacology | Mice, Inbred C57BL | Cardiomegaly - drug therapy | Heart Failure - metabolism | Cardiotonic Agents - pharmacology | Heart Failure - pathology | Hypertension - pathology | Heart Failure - drug therapy | Hypertension - metabolism | Animals | Multiple Organ Failure - prevention & control | Calcitonin Gene-Related Peptide - therapeutic use | Mice | Oxidative Stress - drug effects | Blood Flow Velocity - physiology | Blood Flow Velocity - drug effects | Cardiomegaly - metabolism | Heart failure | Hypertension | Complications and side effects | Control | Blood pressure | Cardiovascular diseases | Risk factors | 10095 | 10094 | 10101 | 10012 | 10111 | Original s
Journal Article
Journal of Neuroscience, ISSN 0270-6474, 03/2007, Volume 27, Issue 10, pp. 2693 - 2703
The neuropeptide calcitonin gene- related peptide ( CGRP) from the trigeminal ganglion has been established as a key player in the pathogenesis of migraine. In... 
Gene transfer | Transcription | Calcitonin | Migraine | Transgenic | GPCR | Neurogenic inflammation | cAMP | CGRP | Trigeminal | Cre-transgenic | NERVOUS-SYSTEM | AMYLIN RECEPTORS | migraine | calcitonin | gene transfer | transcription | ADRENOMEDULLIN RECEPTORS | ANTIMIGRAINE DRUG | NEUROSCIENCES | neurogenic inflammation | ANTAGONIST BIBN4096BS | MESSENGER-RNA | transgenic | SMOOTH-MUSCLE-CELLS | CEREBRAL-ARTERIES | trigeminal | BINDING-SITES | Inflammation - chemically induced | Intracellular Signaling Peptides and Proteins - pharmacology | Inflammation - pathology | Gene Expression - drug effects | Receptors, Calcitonin - metabolism | Humans | Intracellular Signaling Peptides and Proteins - drug effects | Nervous System - metabolism | Intracellular Signaling Peptides and Proteins - metabolism | Membrane Proteins - pharmacology | Calcitonin Receptor-Like Protein | Trigeminal Ganglion - drug effects | Receptors, Calcitonin Gene-Related Peptide - metabolism | Membrane Proteins - physiology | Membrane Proteins - metabolism | Neurons - metabolism | Intracellular Signaling Peptides and Proteins - genetics | Receptor Activity-Modifying Protein 1 | Subcutaneous Tissue - pathology | Cyclic AMP-Dependent Protein Kinases - metabolism | Gene Transfer Techniques | Trigeminal Ganglion - cytology | Receptors, Calcitonin - drug effects | Calcitonin Gene-Related Peptide - pharmacology | Membrane Proteins - genetics | Rats | Trigeminal Ganglion - metabolism | Mice, Transgenic | Rats, Sprague-Dawley | Animals | Promoter Regions, Genetic - physiology | Membrane Proteins - drug effects | Receptor Activity-Modifying Proteins | Receptors, Calcitonin Gene-Related Peptide - genetics | Mice | In Vitro Techniques | Intracellular Signaling Peptides and Proteins - physiology | Subcutaneous Tissue - drug effects
Journal Article
Journal Article