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Journal Article
Journal of Infectious Diseases, ISSN 0022-1899, 2014, Volume 210, Issue 8, pp. 1296 - 1305
Journal Article
European Journal of Nutrition, ISSN 1436-6207, 3/2018, Volume 57, Issue 2, pp. 723 - 730
Maternal vitamin D deficiency during pregnancy is a widespread issue that may have long-lasting consequences on offspring adiposity. We sought to determine how... 
Pregnancy | Chemistry | DOHaD | Nutrition | Vitamin D | Gene expression | Fetal programming | Adipogenesis | CELLS | METABOLIC SYNDROME | C/EBP-BETA | FATTY LIVER-DISEASE | DIET-INDUCED OBESITY | WHITE ADIPOSE-TISSUE | NUTRITION & DIETETICS | INSULIN-RESISTANCE | CARDIOVASCULAR-DISEASE | ADIPOCYTE DIFFERENTIATION | BINDING | Prenatal Exposure Delayed Effects | Maternal Nutritional Physiological Phenomena | Adipose Tissue, White - immunology | Lactation | Adipose Tissue, White - metabolism | Male | PPAR gamma - metabolism | Vitamin D Deficiency - physiopathology | Receptors, Calcitriol - genetics | Adipokines - blood | Adiposity | Gene Expression Regulation, Developmental | Inflammation Mediators - metabolism | Female | Panniculitis - immunology | PPAR gamma - genetics | Adipose Tissue, White - pathology | Panniculitis - etiology | Mice, Inbred C57BL | Inflammation Mediators - blood | Fetal Development | Cell Size | Adipokines - metabolism | Pregnancy Complications - physiopathology | Random Allocation | Receptors, Calcitriol - metabolism | Pilot Projects | Animals | Panniculitis - pathology | Panniculitis - metabolism | Genes | Body weight | Calcifediol | Vitamin D deficiency | Anopheles | Pregnant women | Physiological aspects | Genetic research | Mice | Alfacalcidol | Progeny | Nutrient deficiency | Adipose tissue | Perinatal exposure | Vitamin deficiency | Peroxisome proliferator-activated receptors | Vitamin D receptors
Journal Article
Thorax, ISSN 0040-6376, 07/2015, Volume 70, Issue 7, pp. 617 - 624
Journal Article
PLoS ONE, ISSN 1932-6203, 2011, Volume 6, Issue 8, p. e23524
Aberrant activation of the Wnt/beta-catenin pathway is critical for the initiation and progression of most colon cancers. This activation provokes the... 
STEM-CELLS | APC | PHOSPHORYLATION | MULTIDISCIPLINARY SCIENCES | GROWTH | PHENOTYPE | E-CADHERIN | DIFFERENTIATION | BETA-CATENIN | WNT | EXPRESSION | Steroid Hydroxylases - metabolism | Colonic Neoplasms - genetics | Humans | Protein Transport - drug effects | Wnt Proteins - metabolism | Receptors, Calcitriol - genetics | Colonic Neoplasms - metabolism | Cell Nucleus - metabolism | RNA Interference | Cell Membrane - metabolism | Intestinal Neoplasms - genetics | Steroid Hydroxylases - genetics | Adenomatous Polyposis Coli Protein - genetics | Receptors, Calcitriol - deficiency | Signal Transduction | HCT116 Cells | Intestinal Neoplasms - metabolism | Vitamin D3 24-Hydroxylase | Tumor Burden | Reverse Transcriptase Polymerase Chain Reaction | beta Catenin - metabolism | Blotting, Western | Intestinal Neoplasms - pathology | Mice, Knockout | Gene Expression Regulation - drug effects | HT29 Cells | Animals | Calcitriol - pharmacology | Colonic Neoplasms - pathology | Cell Line, Tumor | Mice | Mutation | Phosphorylation | Transcription factors | Carcinoma | Wnt protein | Laboratories | Genes | Colorectal cancer | Calcitriol | Oncology | Genomes | Activation | Kinases | Small intestine | Nuclei | Accumulation | Proteins | β-catenin | Colon cancer | Vitamin D | Colon | Relocation | Phenotypes | Nutrient deficiency | Baldness | Breeding | Gene expression | Adenomatous polyposis coli | Signaling | Alleles | Stem cells | Ligands | Vitamin D receptors | Aberration | Tumors | Cancer
Journal Article
Journal Article
Journal of Steroid Biochemistry and Molecular Biology, ISSN 0960-0760, 01/2018, Volume 175, pp. 23 - 28
Vitamin D has emerged as a key regulator of innate immune responses to pathogen threat. The hormonal form of vitamin D signals through a nuclear receptor... 
Vitamin D deficiency | Intervention trials | Vitamin D receptor | 1α,25-dihydroxyvitamin D | Innate immunity | Crohn's disease | Autophagy | REDUCED RISK | VARIANTS | BIOCHEMISTRY & MOLECULAR BIOLOGY | SUSCEPTIBILITY LOCI | NOD2 | INFLAMMATORY-BOWEL-DISEASE | GENE | ENDOCRINOLOGY & METABOLISM | D-RECEPTOR | EPIDEMIOLOGY | 1 alpha,25-dihydroxyvitamin D-3 | GENOME-WIDE ASSOCIATION | Crohn Disease - genetics | Vitamin D Deficiency - complications | Humans | Vitamin D Response Element - immunology | Nod2 Signaling Adaptor Protein - genetics | Interleukin-1beta - genetics | Vitamin D Response Element - genetics | Receptors, Calcitriol - genetics | Crohn Disease - complications | Vitamin D - immunology | Transcription, Genetic | Vitamin D Deficiency - immunology | Vitamin D Deficiency - genetics | Immunity, Innate - drug effects | Signal Transduction | Gene Expression Regulation | Interleukin-1beta - immunology | Nod2 Signaling Adaptor Protein - immunology | Clinical Trials as Topic | Crohn Disease - diet therapy | Crohn Disease - immunology | Receptors, Calcitriol - immunology | Vitamin D - analogs & derivatives | Vitamin D - metabolism | Vitamin D Deficiency - diet therapy | Vitamin D - therapeutic use | Dietary Supplements | Medical research | Immune response | Vitamin D | Gastrointestinal diseases | Medicine, Experimental | Calcifediol | Disease susceptibility | Genetic transcription | Alfacalcidol
Journal Article