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Nature neuroscience, ISSN 1546-1726, 2010, Volume 14, Issue 2, pp. 173 - 180
Journal Article
The Journal of cell biology, ISSN 0021-9525, 12/2008, Volume 183, Issue 5, pp. 849 - 863
.... In the case of ion channels in excitable cells, the dynamics of signaling to the nucleus are particularly important because the natural stimulus, surface membrane depolarization, is rapidly pulsatile... 
Depolarization | Electric potential | Phosphorylation | Neuroscience | Receptors | Calcium | Neurons | Sensors | Gene expression | Synapses | PRESYNAPTIC CALCIUM | TRANSMITTER RELEASE | ELECTRICAL-STIMULATION | RYANODINE RECEPTORS | DEPENDENT FACILITATION | CA2+ CHANNELS | DIHYDROPYRIDINE RECEPTORS | RAT SYMPATHETIC NEURONS | CALCIUM-CHANNEL | KINASE-II | CELL BIOLOGY | Transcription, Genetic - drug effects | Calcium - metabolism | Cell Nucleus - enzymology | Transfection | RNA Interference | Time Factors | Calcium-Calmodulin-Dependent Protein Kinase Type 2 - metabolism | Animals, Newborn | Cells, Cultured | Rats | Calcium Channel Blockers - pharmacology | Superior Cervical Ganglion - cytology | Calcium-Calmodulin-Dependent Protein Kinase Type 2 - antagonists & inhibitors | Rats, Sprague-Dawley | Calcium-Calmodulin-Dependent Protein Kinase Type 2 - genetics | Cell Membrane - enzymology | Animals | Calcium Channels, L-Type - genetics | Membrane Potentials | Signal Transduction - drug effects | Superior Cervical Ganglion - enzymology | Calcium Channels, L-Type - drug effects | Neurons - enzymology | Cyclic AMP Response Element-Binding Protein - metabolism | Superior Cervical Ganglion - drug effects | Protein Kinase Inhibitors - pharmacology | Calcium Channels, L-Type - metabolism | Cell Nucleus - drug effects | RNA, Small Interfering - metabolism | Excitation (Physiology) | Evaluation | Cyclic adenosine monophosphate response element-binding protein | Ion channels | Genetic transcription | Research | Properties
Journal Article
Circulation research, ISSN 0009-7330, 03/2012, Volume 110, Issue 5, pp. 669 - 674
Journal Article
Journal Article
Molecular neurobiology, ISSN 1559-1182, 2016, Volume 54, Issue 7, pp. 4996 - 5007
... depolarization to the influx of calcium—and other processes such as gene expression—in excitable cells. L-type calcium channels, one of the three major classes and probably... 
Calcium signaling | Neurology | Neurosciences | Voltage-gated calcium channels | Biomedicine | Estrogen non-genomic actions | DHP binding site | Neurobiology | Estrogen receptor-independent mechanisms | Estradiol | Cell Biology | CORTICAL SPREADING DEPRESSION | ALZHEIMERS-DISEASE | CA2+ CHANNELS | POTENTIAL INITIATION MECHANISM | NON-GENOMIC ACTIONS | SYNAPTIC PLASTICITY | HIPPOCAMPAL-NEURONS | NEUROSCIENCES | ESTROGEN-RECEPTOR-BETA | NEURONAL EXCITABILITY | ASTROCYTIC CELLS | Estradiol - metabolism | Animals | Signal Transduction - drug effects | Calcium - metabolism | Calcium Channels, L-Type - drug effects | Humans | Neurons - metabolism | Calcium Channels, L-Type - metabolism | Estrogens - metabolism | Neurons - drug effects | Estradiol - pharmacology | Nervous system diseases | Calcium channels | Phenols | Genetic transcription | Calcium-binding proteins | Protein binding | Brain | Regulators | Transcription | Calcium | Computational neuroscience | Estrogens | Estrogen receptors | Intracellular signalling | Kinases | Channels | Calcium influx | Depolarization | Calcium signalling | Proteins | Pathways | Cascades | Modulation | Membrane potential | Drug therapy | Calcium (intracellular) | Neurodegenerative diseases | Calcium channels (voltage-gated) | Gene expression | Neurological diseases | Sex hormones | Brain research | Calcium channels (L-type) | Intracellular | Regulatory proteins
Journal Article