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Journal Article
Journal Article
Scientific Reports, ISSN 2045-2322, 12/2018, Volume 8, Issue 1, pp. 1004 - 13
We here describe a novel α-conopeptide, Eu1.6 from Conus eburneus, which exhibits strong anti-nociceptive activity by an unexpected mechanism of action. Unlike... 
Analgesics - pharmacology | Injections, Intravenous | Conotoxins - isolation & purification | Calcium - metabolism | Humans | Male | Neurons - cytology | Ganglia, Spinal - cytology | Calcium Channels, N-Type - metabolism | Conotoxins - chemical synthesis | Solid-Phase Synthesis Techniques | Peptides - chemical synthesis | Analgesics - chemical synthesis | HEK293 Cells | Oocytes - drug effects | Neurons - metabolism | Neurons - drug effects | Sciatic Neuropathy - drug therapy | Analgesics - isolation & purification | Amino Acid Sequence | Chronic Pain - drug therapy | Oocytes - metabolism | Mice, Inbred C57BL | Xenopus laevis | Rats | Injections, Intramuscular | Rats, Sprague-Dawley | Peptides - pharmacology | Conotoxins - pharmacology | Conus Snail - chemistry | Chronic Pain - metabolism | Patch-Clamp Techniques | Peptides - isolation & purification | Animals | Sciatic Neuropathy - physiopathology | Mice | Chronic Pain - physiopathology | Ganglia, Spinal - drug effects | Sciatic Neuropathy - metabolism | Ganglia, Spinal - metabolism | γ-Aminobutyric acid B receptors | Tetrodotoxin | Sodium channels | Calcium channels | Animal models | Respiratory function | Pain perception | Calcium channels (N-type) | Calcium channels (voltage-gated) | Neuropathy | Drug development | Calcium channels (T-type) | Depolarization | Dorsal root ganglia | Analgesics | Rodents | Acetylcholine receptors (nicotinic) | KCNQ1 protein | Potassium channels (voltage-gated) | Receptor mechanisms | Drug dependence | Heart diseases | Locomotor activity | Sciatic nerve
Journal Article
Journal Article
SCIENCE, ISSN 0036-8075, 10/2006, Volume 314, Issue 5799, pp. 615 - 620
Journal Article
Cell Metabolism, ISSN 1550-4131, 06/2010, Volume 11, Issue 6, pp. 543 - 553
Journal Article
Cardiovascular Research, ISSN 0008-6363, 10/2014, Volume 104, Issue 1, pp. 183 - 193
Aims Dysregulation of autonomic nervous system activity can trigger ventricular arrhythmias and sudden death in patients with heart failure. N-type Ca2+... 
Heart failure | Nervous system | Arrhythmia | Ion channel | N-type Ca2+ channel | Autonomic | SYSTEM | CARDIAC & CARDIOVASCULAR SYSTEMS | CILNIDIPINE | V 2.2 | DEATH | RELEASE | CALCIUM-CHANNEL | REVEALS | Anti-Arrhythmia Agents - pharmacology | Heart Failure - physiopathology | Death, Sudden, Cardiac - prevention & control | Death, Sudden, Cardiac - etiology | Calcium Channels, N-Type - metabolism | Autonomic Nervous System - physiopathology | Arrhythmias, Cardiac - physiopathology | Adrenergic beta-Antagonists - pharmacology | Cardiomyopathy, Dilated - drug therapy | Time Factors | Autonomic Nervous System - metabolism | Arrhythmias, Cardiac - genetics | Repressor Proteins - metabolism | Disease Models, Animal | Heart - innervation | Arrhythmias, Cardiac - metabolism | Cardiomyopathy, Dilated - genetics | Arrhythmias, Cardiac - prevention & control | Calcium Channels, N-Type - genetics | Calcium Channels, N-Type - drug effects | Autonomic Nervous System - drug effects | Ventricular Function, Left - drug effects | Repressor Proteins - genetics | Heart Failure - genetics | Mice, Transgenic | Heart Failure - metabolism | Nitrendipine - pharmacology | Calcium Channel Blockers - pharmacology | Cardiomyopathy, Dilated - metabolism | Heart Failure - drug therapy | Mice, Knockout | Animals | Cardiomyopathy, Dilated - physiopathology | Calcium Channels, L-Type - drug effects | Dihydropyridines - pharmacology | Calcium Channels, L-Type - metabolism
Journal Article