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British Journal of Pharmacology, ISSN 0007-1188, 06/2012, Volume 166, Issue 4, pp. 1247 - 1260
Journal Article
Journal Article
Cancer Research, ISSN 0008-5472, 07/2017, Volume 77, Issue 13, pp. 3479 - 3490
Journal Article
Journal Article
Journal Article
Lancet, The, ISSN 0140-6736, 2013, Volume 381, Issue 9875, pp. 1371 - 1379
Summary Background Findings from family and twin studies suggest that genetic contributions to psychiatric disorders do not in all cases map to present... 
Internal Medicine | POPULATION | DEPRESSION | MEDICINE, GENERAL & INTERNAL | ATTENTION-DEFICIT/HYPERACTIVITY DISORDER | METAANALYSIS | GENE-EXPRESSION | SCHIZOPHRENIA | CACNA1C | AUTISM SPECTRUM DISORDERS | BIPOLAR DISORDER | ASSOCIATION | Child Development Disorders, Pervasive - epidemiology | Genome-Wide Association Study | Humans | Genetic Loci - genetics | Logistic Models | Bipolar Disorder - genetics | Depressive Disorder, Major - epidemiology | Schizophrenia - epidemiology | Calcium Channels, L-Type - genetics | Schizophrenia - genetics | Attention Deficit Disorder with Hyperactivity - genetics | Child Development Disorders, Pervasive - genetics | Depressive Disorder, Major - genetics | Age of Onset | Polymorphism, Single Nucleotide - genetics | Adult | Child | Bipolar Disorder - epidemiology | Attention Deficit Disorder with Hyperactivity - epidemiology | Physiological aspects | Genetic aspects | Research | Genetic variation | Mental illness | Risk factors | Medical research | Calcium channels | Genes | Depression, Mental | Genomics | Schizophrenia | Bipolar disorder | Child psychopathology | Quantitative genetics | Autism | Medicine, Experimental | Genetic research | Single nucleotide polymorphisms | Studies | Genetics | Children & youth | Attention deficit disorder | Enrichment | Brain | Calcium | Disease | Mental disorders | Hyperactivity | Mental health | Disorders | Genomes | Single-nucleotide polymorphism | Mental depression | Consortia | Datasets | Calcium signalling | Genetic effects | Signal transduction | Regression models | Autopsy | Classification | Psychopathology | Children | Chromosomes | Genotypes | Phenotypes | Channel gating | Attention deficit hyperactivity disorder | Calcium channels (voltage-gated) | Behavior disorders | Regression analysis | Risk analysis | Loci | Quantitative trait loci | Government grants | Calcium channels (L-type) | Diagnostic systems | Polygenic inheritance | Psychiatry | Polymorphism | Neurosciences | Calcium Channels | Pervasive | Attention Deficit Disorder with Hyperactivity | L-Type | Child Development Disorders | Depressive Disorder | Genetic Loci | Single Nucleotide | epidemiology | genetics | Bipolar Disorder | Major | Neurovetenskaper
Journal Article
eLife, ISSN 2050-084X, 04/2015, Volume 4, p. e06315
Many Mendelian traits are likely unrecognized owing to absence of traditional segregation patterns in families due to causation by de novo mutations,... 
human biology | incomplete penetrance | de novo mutation | voltage-gated calcium channel | exome sequencing | genes | chromosomes | adrenal gland | medicine | CaV3.2 | human | CHILDHOOD ABSENCE EPILEPSY | GENETIC-VARIATION | HYPERALDOSTERONISM TYPE-II | GLUCOCORTICOID-REMEDIABLE ALDOSTERONISM | BIOLOGY | WHOLE-GENOME ASSOCIATION | CA2+ CHANNEL | T-TYPE | GLOMERULOSA CELLS | FAMILIAL HYPERALDOSTERONISM | SOMATIC MUTATIONS | Recurrence | Aldosterone - biosynthesis | Calcium - metabolism | Humans | Hyperaldosteronism - pathology | Middle Aged | Hyperaldosteronism - complications | Child, Preschool | Molecular Sequence Data | Zona Glomerulosa - pathology | Infant | Male | Hyperaldosteronism - metabolism | Calcium Channels, T-Type - metabolism | Adult | Female | Calcium Channels, T-Type - genetics | Hypertension - genetics | Child | Calcium Signaling | Amino Acid Sequence | Gene Expression | Hyperaldosteronism - genetics | Aldosterone - secretion | Genotype | Zona Glomerulosa - metabolism | Hypertension - pathology | Hypertension - metabolism | Phenotype | Sequence Alignment | Membrane Potentials | Adolescent | Age of Onset | Hypertension - complications | Heterozygote | Mutation | Hypertension | Haplotypes | Pediatrics | Nephrology | Calcium (intracellular) | Statistical analysis | Genomics | Genes | Calcium channels (voltage-gated) | Genomes | Aldosterone | Morbidity | Calcium signalling | Consortia | Pathology | Children | Age
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