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British Journal of Pharmacology, ISSN 0007-1188, 06/2012, Volume 166, Issue 4, pp. 1247 - 1260
Journal Article
Journal of Cell Science, ISSN 0021-9533, 2015, Volume 128, Issue 23, pp. 4442 - 4452
Journal Article
Neuron, ISSN 0896-6273, 01/2015, Volume 85, Issue 2, pp. 346 - 363
Journal Article
Circulation research, ISSN 0009-7330, 2014, Volume 115, Issue 7, pp. 650 - 661
Journal Article
Cancer Research, ISSN 0008-5472, 07/2017, Volume 77, Issue 13, pp. 3479 - 3490
Journal Article
Molecular Pharmacology, ISSN 0026-895X, 12/2012, Volume 82, Issue 6, pp. 1194 - 1204
Journal Article
eLife, ISSN 2050-084X, 04/2015, Volume 4, p. e06315
Many Mendelian traits are likely unrecognized owing to absence of traditional segregation patterns in families due to causation by de novo mutations,... 
human biology | incomplete penetrance | de novo mutation | voltage-gated calcium channel | exome sequencing | genes | chromosomes | adrenal gland | medicine | CaV3.2 | human | CHILDHOOD ABSENCE EPILEPSY | GENETIC-VARIATION | HYPERALDOSTERONISM TYPE-II | GLUCOCORTICOID-REMEDIABLE ALDOSTERONISM | BIOLOGY | WHOLE-GENOME ASSOCIATION | CA2+ CHANNEL | T-TYPE | GLOMERULOSA CELLS | FAMILIAL HYPERALDOSTERONISM | SOMATIC MUTATIONS | Recurrence | Aldosterone - biosynthesis | Calcium - metabolism | Humans | Hyperaldosteronism - pathology | Middle Aged | Hyperaldosteronism - complications | Child, Preschool | Molecular Sequence Data | Zona Glomerulosa - pathology | Infant | Male | Hyperaldosteronism - metabolism | Calcium Channels, T-Type - metabolism | Adult | Female | Calcium Channels, T-Type - genetics | Hypertension - genetics | Child | Calcium Signaling | Amino Acid Sequence | Gene Expression | Hyperaldosteronism - genetics | Aldosterone - secretion | Genotype | Zona Glomerulosa - metabolism | Hypertension - pathology | Hypertension - metabolism | Phenotype | Sequence Alignment | Membrane Potentials | Adolescent | Age of Onset | Hypertension - complications | Heterozygote | Mutation | Hypertension | Haplotypes | Pediatrics | Nephrology | Calcium (intracellular) | Statistical analysis | Genomics | Genes | Calcium channels (voltage-gated) | Genomes | Aldosterone | Morbidity | Calcium signalling | Consortia | Pathology | Children | Age
Journal Article
Pain, ISSN 0304-3959, 2017, Volume 158, Issue 3, pp. 417 - 429
Here, it is shown that paclitaxel-induced neuropathy is associated with the development of spontaneous activity (SA) and hyperexcitability in dorsal root... 
Rat DRG | Chemotherapy | Human DRG | Spontaneous activity | TLR4 | RAT | NOCICEPTORS | MODEL | NEUROSCIENCES | CLINICAL NEUROLOGY | SIGNAL-TRANSDUCTION | RESPONSES | TOLL-LIKE RECEPTOR-4 | PAIN | SENSITIZATION | ANESTHESIOLOGY | PRIMARY SENSORY NEURONS | UP-REGULATION | Pain Threshold - physiology | Peripheral Nervous System Diseases - chemically induced | Paclitaxel - pharmacology | Spinal Cord - drug effects | Spinal Cord - metabolism | Humans | Calcium Channel Blockers - therapeutic use | Male | Benzamides - therapeutic use | Calcium Channels, T-Type - metabolism | Peripheral Nervous System Diseases - prevention & control | Pain Threshold - drug effects | Azabicyclo Compounds - therapeutic use | Peripheral Nervous System Diseases - pathology | Sensory Receptor Cells - metabolism | Toll-Like Receptor 4 - antagonists & inhibitors | Calcitonin Gene-Related Peptide - metabolism | Peripheral Nervous System Diseases - complications | Antineoplastic Agents, Phytogenic - adverse effects | Disease Models, Animal | Paclitaxel - adverse effects | Rats | Rats, Sprague-Dawley | Toll-Like Receptor 4 - metabolism | Gene Expression Regulation - drug effects | Animals | Sensory Receptor Cells - drug effects | Sulfonamides - therapeutic use | Ganglia, Spinal - pathology | Hyperalgesia - etiology | Antineoplastic Agents, Phytogenic - pharmacology | spontaneous activity | chemotherapy | human DRG
Journal Article