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Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 11/2013, Volume 110, Issue 47, pp. 19071 - 19076
Cerebral arteriovenous malformations (AVMs) are common vascular malformations, which may result in hemorrhagic strokes and neurological deficits. Bone... 
Cardiovascular system | Ephrins | Kidneys | Transfection | Lungs | Small interfering RNA | Arteriovenous malformations | Immunoblotting | Ligands | Endothelial cells | DEFECTS | shunt | telangiectasia | angiogenesis | MULTIDISCIPLINARY SCIENCES | NOTCH | hereditary hemorrhagic | MICE LACKING | LETHALITY | LIGANDS | brain circulation | ENDOTHELIAL-CELLS | DIFFERENTIATION | animal model | RECEPTORS | EXPRESSION | Bone Morphogenetic Proteins - antagonists & inhibitors | RNA, Small Interfering - genetics | Extracellular Matrix Proteins - deficiency | Receptors, Notch - metabolism | X-Ray Microtomography | Intercellular Signaling Peptides and Proteins - metabolism | Serrate-Jagged Proteins | Gene Deletion | Jagged-2 Protein | Intracranial Arteriovenous Malformations - etiology | Membrane Proteins - metabolism | Receptors, Eph Family - metabolism | Cell Differentiation - physiology | Real-Time Polymerase Chain Reaction | Endothelial Cells - physiology | Jagged-1 Protein | Calcium-Binding Proteins - metabolism | Extracellular Matrix Proteins - genetics | Calcium-Binding Proteins - deficiency | Ephrin-B2 - metabolism | Intracranial Arteriovenous Malformations - prevention & control | Mice, Knockout | Animals | Analysis of Variance | Guanylate Kinases - metabolism | Mice | Adaptor Proteins, Signal Transducing - metabolism | Calcium-Binding Proteins - genetics | Physiological aspects | Bone morphogenetic proteins | Nervous system | Degeneration | Research | Health aspects | Neurophysiology | Proteins | Neurology | Molecules | Signal transduction | Gene expression | Veins & arteries | Index Medicus | Biological Sciences | hereditary hemorrhagic telangiectasia
Journal Article
Journal Article
Journal Article
Journal Article
Nature, ISSN 0028-0836, 2014, Volume 512, Issue 1, pp. 69 - 73
Rheumatoid arthritis is a chronic autoinflammatory disease that affects 1-2% of the world's population and is characterized by widespread joint inflammation.... 
RHEUMATOID-ARTHRITIS | COLLAGEN-INDUCED ARTHRITIS | ACTIVATION | 6Q23 | CASPASE-1 | MULTIDISCIPLINARY SCIENCES | SUSCEPTIBILITY | MICE | INHIBITOR | NF-KAPPA-B | ASSOCIATION | Inflammasomes - metabolism | NLR Family, Pyrin Domain-Containing 3 Protein | Caspase 1 - metabolism | Male | Intracellular Signaling Peptides and Proteins - metabolism | Intracellular Signaling Peptides and Proteins - deficiency | Arthritis, Rheumatoid - metabolism | Arthritis, Rheumatoid - prevention & control | Cysteine Endopeptidases - metabolism | Receptors, Interleukin-1 - deficiency | Cysteine Endopeptidases - deficiency | Female | Tumor Necrosis Factor alpha-Induced Protein 3 | DNA-Binding Proteins | Disease Models, Animal | Calcium-Binding Proteins - metabolism | Interleukin-1 - metabolism | Signal Transduction | Nuclear Proteins - metabolism | Apoptosis Regulatory Proteins - metabolism | Arthritis, Rheumatoid - pathology | Mice, Knockout | Receptors, Interleukin-1 - metabolism | Macrophages - metabolism | Phenotype | Animals | Carrier Proteins - metabolism | Caspase 1 - deficiency | Mice | Arthritis, Rheumatoid - immunology | Prevention | Rheumatoid arthritis | Physiological aspects | NLRP3 | Properties | Macrophages | Risk factors | Proteins | Pathology | Dehydrogenases | Histopathology | Pathogenesis | Rodents | Protein expression | Index Medicus | Nlrp3 | IL1R1 | caspase-1 | inflammasome | A20
Journal Article
Nature: international weekly journal of science, ISSN 0028-0836, 01/2014, Volume 506, Issue 7487, pp. 240 - 244
textabstractCells of the osteoblast lineage affect the homing and the number of long-term repopulating haematopoietic stem cells, haematopoietic stem cell... 
PROGENITOR CELLS | MULTIDISCIPLINARY SCIENCES | ACUTE LYMPHOBLASTIC-LEUKEMIA | BONE-MARROW NICHE | NOTCH ACTIVATION | STEM-CELL NICHE | MYELOID-LEUKEMIA | MICE | DIFFERENTIATION | EXPRESSION | MYELODYSPLASTIC SYNDROMES | Osteoblasts - secretion | Receptors, Notch - metabolism | Humans | Leukemia, Myeloid, Acute - metabolism | Hematopoietic Stem Cells - pathology | Anemia - pathology | Male | Cell Differentiation - genetics | Intercellular Signaling Peptides and Proteins - metabolism | Membrane Proteins - deficiency | Anemia - genetics | Cell Nucleus - metabolism | Tumor Microenvironment - genetics | Cell Transformation, Neoplastic - genetics | Serrate-Jagged Proteins | Base Sequence | Female | Membrane Proteins - metabolism | Intercellular Signaling Peptides and Proteins - deficiency | Jagged-1 Protein | Calcium-Binding Proteins - metabolism | Myelodysplastic Syndromes - metabolism | Signal Transduction | Leukemia, Myeloid, Acute - pathology | Membrane Proteins - genetics | Intercellular Signaling Peptides and Proteins - genetics | Calcium-Binding Proteins - deficiency | Hematopoietic Stem Cells - metabolism | Mutation - genetics | beta Catenin - metabolism | beta Catenin - genetics | Anemia - metabolism | Cell Lineage | Osteoblasts - pathology | Animals | Chromosome Aberrations | Myeloid Cells - metabolism | Ligands | Mice | Myelodysplastic Syndromes - genetics | Myeloid Cells - pathology | Cell Transformation, Neoplastic - pathology | Myelodysplastic Syndromes - pathology | Osteoblasts - metabolism | Calcium-Binding Proteins - genetics | Leukemia, Myeloid, Acute - genetics | Index Medicus
Journal Article
Cell Metabolism, ISSN 1550-4131, 06/2013, Volume 17, Issue 6, pp. 976 - 987
Journal Article
Nature, ISSN 0028-0836, 10/2012, Volume 490, Issue 7421, pp. 539 - 542
Journal Article
Cancer Research, ISSN 0008-5472, 09/2011, Volume 71, Issue 18, pp. 6061 - 6072
One important function of endothelial cells in glioblastoma multiforme (GBM) is to create a niche that helps promote self-renewal of cancer stem-like cells... 
INITIATING CELLS | DLL4 | ANGIOGENESIS | ONCOLOGY | VASCULAR NICHE | NEUROGENESIS | CENTRAL-NERVOUS-SYSTEM | SUBVENTRICULAR ZONE | PERIVASCULAR NICHE | INHIBITS TUMOR-GROWTH | BRAIN-TUMORS | RNA, Small Interfering - genetics | Nestin | Receptors, Notch - metabolism | Humans | Brain Neoplasms - pathology | Glycoproteins - metabolism | Cell Growth Processes - physiology | Intercellular Signaling Peptides and Proteins - biosynthesis | Receptors, Notch - genetics | Brain Neoplasms - metabolism | Antigens, CD - metabolism | Gene Knockdown Techniques | Membrane Proteins - deficiency | Peptides - metabolism | Neoplastic Stem Cells - metabolism | Receptors, Notch - biosynthesis | Serrate-Jagged Proteins | Neoplastic Stem Cells - pathology | Glioblastoma - metabolism | Intercellular Signaling Peptides and Proteins - deficiency | Receptors, Notch - deficiency | Calcium-Binding Proteins - biosynthesis | Stem Cell Niche | Endothelial Cells - metabolism | Membrane Proteins - genetics | Intercellular Signaling Peptides and Proteins - genetics | Calcium-Binding Proteins - deficiency | AC133 Antigen | Nerve Tissue Proteins - metabolism | Xenograft Model Antitumor Assays | Membrane Proteins - biosynthesis | Animals | Glioblastoma - pathology | Mice | Endothelial Cells - pathology | RNA, Small Interfering - administration & dosage | Intermediate Filament Proteins - metabolism | Calcium-Binding Proteins - genetics | Brain | glioblastoma multiforme | Tumor cells | Glioblastoma | Endothelial cells | Endothelium | Molecular modelling | Stem cells | Microenvironments | Microvasculature | Notch protein | neurospheres | Cancer | Tumors | Index Medicus | Notch ligand | Endothelial cell | niche | glioblastoma | cancer stem cell
Journal Article
Immunity, ISSN 1074-7613, 10/2015, Volume 43, Issue 4, pp. 751 - 763
The crosstalk between inflammation and tumorigenesis is now clearly established. However, how inflammation is elicited in the metastatic environment and the... 
colorectal cancer | cell death | innate immunity | metastasis | interleukin-18 | inflammasome | NLRP3 | Fas | natural killer (NK) cells | caspase-1 | immunosurveillance | Cell death | Natural killer (NK) cells | Colorectal cancer | Immunosurveillance | Innate immunity | Inflammasome | Metastasis | Interleukin-18 | Caspase-1 | COLON-CANCER | APOPTOSIS | ACTIVATION | NK CELLS | CASPASE-1 | INTERLEUKIN-1-BETA | HUMAN-MELANOMA | KUPFFER CELLS | RECEPTOR | CYTOKINE | IMMUNOLOGY | Immunologic Surveillance | Interleukin-18 - physiology | NLR Family, Pyrin Domain-Containing 3 Protein | Neoplasm Proteins - physiology | Radiation Tolerance | Tumor Microenvironment | DNA-Binding Proteins - deficiency | Apoptosis Regulatory Proteins - deficiency | Killer Cells, Natural - immunology | Liver Neoplasms - secondary | Inflammasomes - deficiency | Carrier Proteins - physiology | Interleukin-1beta - physiology | Adenocarcinoma - immunology | Mice, Inbred C57BL | Gastrointestinal Microbiome | Calcium-Binding Proteins - deficiency | Liver Neoplasms - immunology | Radiation Chimera | Immunity, Innate | Adenocarcinoma - secondary | Mice, Knockout | Fas Ligand Protein - physiology | Inflammasomes - physiology | Animals | Colorectal Neoplasms - immunology | Caspase 1 - deficiency | Cell Line, Tumor | Mice | Neoplasm Proteins - deficiency | Colorectal Neoplasms - pathology | Cytotoxicity, Immunologic | Lymphocytes, Tumor-Infiltrating - immunology | Killer cells | Microbiota (Symbiotic organisms) | Growth | Cytokines | Liver | Inflammation | Gene expression | Inflammatory bowel disease | Scholarships & fellowships | Rodents | Tumorigenesis | Immune system | Tumors | Apoptosis | Index Medicus
Journal Article
Nature, ISSN 0028-0836, 05/2016, Volume 534, Issue 7605, pp. 55 - 62
Somatic mutations have been extensively characterized in breast cancer, but the effects of these genetic alterations on the proteomic landscape remain poorly... 
PATHWAYS | HETEROGENEITY | PIK3CA MUTATIONS | PHOSPHORYLATION | MULTIDISCIPLINARY SCIENCES | GENES | BIOLOGY | RECEPTOR | EXPRESSION | SIGNATURE | REVEALS | Protein Kinases - metabolism | Focal Adhesion Kinase 1 - genetics | Receptor, Epidermal Growth Factor - genetics | Protein Kinases - genetics | Cyclin-Dependent Kinases - metabolism | Receptor, ErbB-2 - genetics | Receptors, G-Protein-Coupled - metabolism | Genomics | Humans | Gene Expression Regulation, Neoplastic | Receptor, ErbB-2 - metabolism | Phosphoproteins - metabolism | Receptor-Interacting Protein Serine-Threonine Kinase 2 - genetics | Tumor Suppressor Protein p53 - genetics | Breast Neoplasms - metabolism | Receptor-Interacting Protein Serine-Threonine Kinase 2 - metabolism | Breast Neoplasms - enzymology | Receptor, Epidermal Growth Factor - metabolism | Phosphoproteins - analysis | Mass Spectrometry | src-Family Kinases - metabolism | Female | Cyclin-Dependent Kinases - genetics | Focal Adhesion Kinase 1 - metabolism | Chromosomes, Human, Pair 5 - genetics | Breast Neoplasms - classification | Chromosome Deletion | p21-Activated Kinases - genetics | Signal Transduction | Molecular Sequence Annotation | Calcium-Binding Proteins - deficiency | Phosphoproteins - genetics | Mutation - genetics | S-Phase Kinase-Associated Proteins - metabolism | p21-Activated Kinases - metabolism | Phosphatidylinositol 3-Kinases - genetics | Breast Neoplasms - genetics | Class I Phosphatidylinositol 3-Kinases | Proteomics | S-Phase Kinase-Associated Proteins - genetics | Receptors, G-Protein-Coupled - genetics | src-Family Kinases - genetics | Calcium-Binding Proteins - genetics | Breast cancer | Genetic aspects | Research | Oncology, Experimental | Cancer | Physiological aspects | Methods | Mutation (Biology) | Proteins | Gene amplification | Peptides | Protein expression | Genomes | Mutation | Kinases | Deoxyribonucleic acid--DNA | Tumors | Index Medicus
Journal Article