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Journal of Neuroscience, ISSN 0270-6474, 04/2010, Volume 30, Issue 14, pp. 4981 - 4989
Journal Article
Circulation Research, ISSN 0009-7330, 02/2013, Volume 112, Issue 4, pp. 664 - 674
Journal Article
Circulation Research, ISSN 0009-7330, 01/2009, Volume 104, Issue 1, pp. 79 - 86
Journal Article
Journal Article
Journal of Clinical Investigation, ISSN 0021-9738, 05/2009, Volume 119, Issue 5, pp. 1230 - 1240
Journal Article
Journal Article
Hypertension, ISSN 0194-911X, 02/2015, Volume 65, Issue 2, pp. 335 - 344
Activation of Wnt signaling results in maladaptive cardiac remodeling and cardiomyopathy. Recently, calcium/calmodulin-dependent protein kinase II (CaMKII) was... 
Wnt signaling pathway | histone deacetylase 4 | dishevelled proteins | cardiomyopathies | calcium-calmodulin-dependent protein kinase type 2 | PRESSURE-OVERLOAD | ACTIVATION | MYOCARDIAL-INFARCTION | CATENIN DOWN-REGULATION | DELTA | HEART-FAILURE | CAMKII | PATHWAY | PERIPHERAL VASCULAR DISEASE | INDUCED CARDIAC-HYPERTROPHY | TRANSCRIPTION FACTOR | Hypertrophy, Left Ventricular - enzymology | Heart Failure - enzymology | Heart Failure - physiopathology | Wnt Signaling Pathway - physiology | MAP Kinase Signaling System | Heart Failure - prevention & control | Calcium-Calmodulin-Dependent Protein Kinase Type 2 - deficiency | Ventricular Remodeling | Hypertrophy, Left Ventricular - genetics | Ventricular Dysfunction, Left - genetics | Calcium-Calmodulin-Dependent Protein Kinase Type 2 - physiology | Ultrasonography | Ventricular Dysfunction, Left - enzymology | Phosphoproteins - physiology | beta Catenin - physiology | Dishevelled Proteins | Protein Kinase C - physiology | Myocardium - pathology | Sulfonamides - pharmacology | Calcium-Calmodulin-Dependent Protein Kinase Type 2 - antagonists & inhibitors | MEF2 Transcription Factors - physiology | Ventricular Dysfunction, Left - physiopathology | Mice, Knockout | Adaptor Proteins, Signal Transducing - physiology | Phenotype | Animals | Fibrosis | Mice | Wnt Proteins - physiology | Histone Deacetylases - physiology | Hypertrophy, Left Ventricular - physiopathology | Benzylamines - pharmacology | Apoptosis | Hypertrophy, Left Ventricular - diagnostic imaging | Index Medicus
Journal Article
Journal of Neurochemistry, ISSN 0022-3042, 12/2011, Volume 119, Issue 5, pp. 1108 - 1118
Journal Article
Circulation research, ISSN 0009-7330, 02/2015, Volume 116, Issue 5, pp. E28 - E39
Rationale: Sustained activation of Gq transgenic (Gq) signaling during pressure overload causes cardiac hypertrophy that ultimately progresses to dilated... 
Heart failure | Mitochondrial uncoupling protein 3 | Oxidative stress | G-protein | Calcium-calmodulin-dependent protein kinase type 2 | heart failure | OXYGEN SPECIES PRODUCTION | PRESSURE-OVERLOAD | CARDIAC & CARDIOVASCULAR SYSTEMS | UNCOUPLING PROTEINS | HEART-FAILURE | calcium-calmodulin-dependent protein kinase type 2 | KINASE-II | CARDIAC-HYPERTROPHY | MYOCARDIAL HYPERTROPHY | PERIPHERAL VASCULAR DISEASE | MICE | mitochondrial uncoupling protein 3 | PPAR-ALPHA AGONIST | DYSFUNCTION | HEMATOLOGY | oxidative stress | Reactive Oxygen Species | Oxidative Stress | GTP-Binding Protein alpha Subunits, Gq-G11 - deficiency | Heart Failure - physiopathology | Ion Channels - genetics | Male | Gene Expression Profiling | Ion Channels - physiology | Mitochondrial Proteins - genetics | Calcium-Calmodulin-Dependent Protein Kinase Type 2 - deficiency | Cardiomyopathy, Dilated - prevention & control | RNA, Messenger - biosynthesis | Transfection | RNA Interference | Calcium-Calmodulin-Dependent Protein Kinase Type 2 - physiology | Heart Failure - etiology | GTP-Binding Protein alpha Subunits, Gq-G11 - genetics | Ion Channels - biosynthesis | PPAR alpha - biosynthesis | RNA, Messenger - genetics | RNA, Small Interfering - pharmacology | Cells, Cultured | Cardiomegaly - physiopathology | Mitochondrial Proteins - physiology | Rats | Mice, Transgenic | PPAR alpha - genetics | GTP-Binding Protein alpha Subunits, Gq-G11 - physiology | Mitochondrial Proteins - biosynthesis | Cardiomegaly - enzymology | Sulfonamides - pharmacology | Pressure | Disease Progression | Mice, Knockout | Calcium-Calmodulin-Dependent Protein Kinase Type 2 - genetics | Point Mutation | Animals | Sequence Analysis, RNA | Cardiomyopathy, Dilated - etiology | Cardiomyopathy, Dilated - physiopathology | Mitochondria, Heart - physiology | Acetylcysteine - pharmacology | Myocytes, Cardiac - metabolism | Mice | Benzylamines - pharmacology | Uncoupling Protein 3 | Apoptosis | Index Medicus | UCP | calcium | Gq | calmodulin-dependent protein kinase II | peroxisome proliferator-activated receptor alpha and gamma
Journal Article
Psychopharmacology, ISSN 0033-3158, 1/2014, Volume 231, Issue 1, pp. 55 - 65
Contextual control over drug relapse depends on the successful reconsolidation and retention of context-response–cocaine associations in long-term memory... 
Context | Protein kinase A | Basolateral amygdala | Calcium/calmodulin-dependent protein kinase 2 | Self-administration | Neurosciences | Biomedicine | Memory reconsolidation | Cocaine-seeking | Pharmacology/Toxicology | Psychiatry | DRUG MEMORIES | NMDA RECEPTORS | PSYCHIATRY | CONDITIONED PLACE PREFERENCE | FEAR MEMORIES | NEUROSCIENCES | PYRAMIDAL NEURONS | DORSAL HIPPOCAMPUS | NUCLEUS-ACCUMBENS CORE | LATERAL AMYGDALA | SEEKING BEHAVIOR | PHARMACOLOGY & PHARMACY | Memory - drug effects | Data Interpretation, Statistical | Brain - anatomy & histology | Microinjections | Extinction, Psychological - drug effects | Rats | Male | Sulfonamides - pharmacology | Calcium-Calmodulin-Dependent Protein Kinase Type 2 - antagonists & inhibitors | Rats, Sprague-Dawley | Drug-Seeking Behavior - drug effects | Cocaine - pharmacology | Reinforcement Schedule | Cyclic AMP-Dependent Protein Kinases - physiology | Self Administration | Amygdala - enzymology | Animals | Calcium-Calmodulin-Dependent Protein Kinase Type 2 - physiology | Protein Kinase Inhibitors - pharmacology | Cyclic AMP-Dependent Protein Kinases - antagonists & inhibitors | Amygdala - physiology | Benzylamines - pharmacology | Food | Cocaine abuse | Care and treatment | Physiological aspects | Genetic aspects | Research | Protein kinases | Risk factors | Proteins | Kinases | Psychopharmacology | Addictive behaviors | Index Medicus | Amygdala | calcium | cocaine-seeking | calmodulin-dependent protein kinase 2 | self-administration | context | basolateral amygdala | protein kinase A | memory reconsolidation
Journal Article