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PLoS ONE, ISSN 1932-6203, 11/2011, Volume 6, Issue 11, p. e27706
Many therapeutic interventions for spinal cord injury (SCI) using neurotrophic factors have focused on reducing the area damaged by secondary, post-injury... 
NEUROTROPHIC FACTOR | FORELIMB MOTONEURONS | HAND REPRESENTATION | BIOLOGY | DISYNAPTIC PYRAMIDAL EXCITATION | CORTICOSPINAL TRACT | PROPRIOSPINAL NEURONS | MOTOR CORTEX | UNILATERAL SECTION | CERVICAL LEVEL | MACAQUE MONKEY | Hepatocyte Growth Factor - therapeutic use | Recombinant Proteins - therapeutic use | Spinal Cord Injuries - drug therapy | Recovery of Function - drug effects | Pyramidal Tracts - drug effects | Humans | Motor Activity - drug effects | Fingers - physiopathology | Hepatocyte Growth Factor - pharmacology | Myelin Sheath - metabolism | Spinal Cord Injuries - pathology | Indoles - metabolism | Pyramidal Tracts - physiopathology | Time Factors | Spinal Cord - pathology | Calcitonin Gene-Related Peptide - metabolism | Motor Neurons - drug effects | Calcium-Calmodulin-Dependent Protein Kinase Type 2 - metabolism | Motion | Reproducibility of Results | Myelin Sheath - pathology | Callithrix | Spinal Cord - enzymology | Recombinant Proteins - pharmacology | Diffusion Tensor Imaging | Organ Size - drug effects | Animals | Wrist - physiopathology | Staining and Labeling | Spinal Cord Injuries - physiopathology | Primates | Spinal cord injuries | Genetic vectors | Health aspects | Spinal cord | Traumatic brain injury | Central nervous system | Recovery of function | Herpes simplex | Parenchyma | Viruses | Spinal cord injury | Motor task performance | Bone surgery | Fibers | Angiogenesis | Rodents | Calcitonin | Physiology | Degeneration | Damage | Growth factors | Recombinant | Pain perception | Human motion | Preservation | Hepatocyte growth factor | Therapeutic applications | Inflammation | Pyramidal tracts | Neurotrophic factors | Monkeys & apes | Medicine | Walking | Injury prevention | Brain research | Magnetic resonance imaging | Hepatocytes | Human performance | Orthopedics | Stem cells
Journal Article
Arteriosclerosis, Thrombosis, and Vascular Biology, ISSN 1079-5642, 01/2018, Volume 38, Issue 1, pp. 174 - 185
Objective The incidence of blindness is increasing because of the increase in abnormal ocular neovascularization. Anti-VEGF (vascular endothelial growth... 
heparin-binding EGF-like growth factor | retina | mice | endothelial cell | vascular diseases | macular degeneration | VEGF | FACTOR KNOCKOUT MICE | PROTEIN-COUPLED RECEPTORS | CHOROIDAL NEOVASCULARIZATION | EPITHELIAL-CELLS | PROLIFERATIVE DIABETIC-RETINOPATHY | TRANSACTIVATION | EPIDERMAL-GROWTH-FACTOR | ENDOTHELIAL-CELLS | PERIPHERAL VASCULAR DISEASE | HEMATOLOGY | Heparin-binding EGF-like Growth Factor - genetics | Cell Proliferation | ADAM12 Protein - metabolism | Humans | ADAM12 Protein - genetics | Neovascularization, Pathologic | Autocrine Communication - drug effects | Retinal Neovascularization - metabolism | Vascular Endothelial Growth Factor A - metabolism | Extracellular Signal-Regulated MAP Kinases - metabolism | Vascular Endothelial Growth Factor A - genetics | ADAM17 Protein - metabolism | Retinal Vessels - pathology | Choroidal Neovascularization - pathology | Retinal Neovascularization - prevention & control | Retinal Neovascularization - genetics | Disease Models, Animal | Retinal Vessels - metabolism | Signal Transduction | Callithrix | Endothelial Cells - metabolism | Cells, Cultured | Angiogenesis Inhibitors - pharmacology | Choroidal Neovascularization - prevention & control | Retinal Vessels - drug effects | Mice, Knockout | Heparin-binding EGF-like Growth Factor - metabolism | ADAM17 Protein - genetics | Bacterial Proteins - pharmacology | Animals | Choroidal Neovascularization - metabolism | Paracrine Communication - drug effects | Heparin-binding EGF-like Growth Factor - deficiency | Endothelial Cells - pathology | Choroidal Neovascularization - genetics | Retinal Neovascularization - pathology | Cell Movement
Journal Article
Journal Article
American Journal of Physical Anthropology, ISSN 0002-9483, 01/2013, Volume 150, Issue 1, pp. 21 - 28
Journal Article