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Molecules, ISSN 1420-3049, 02/2017, Volume 22, Issue 2, pp. 197 - 197
Cistanoside A (Cis A), an active phenylethanoid glycoside isolated from Cistanche deserticola Y. C. Ma, has received our attention because of its possible role... 
Cistanoside A | RANKL | Antiosteoporotic | Ovariectomized mice | TRAF6 | ANTIOSTEOPOROTIC ACTIVITY | DIAGNOSIS | BIOCHEMISTRY & MOLECULAR BIOLOGY | CHEMISTRY, MULTIDISCIPLINARY | antiosteoporotic | OSTEOPOROSIS | cistanoside A | ovariectomized mice | OSTEOBLASTS | PHENYLETHANOIDS | ECHINACOSIDE | EXTRACT | Bone and Bones - pathology | Mechanical Phenomena - drug effects | Cancellous Bone - metabolism | Ovariectomy | Osteogenesis - drug effects | Osteoporosis - drug therapy | Cancellous Bone - drug effects | Glycosides - pharmacology | Osteoporosis - etiology | X-Ray Microtomography | Bone and Bones - drug effects | Bone Density - drug effects | Animals | Cancellous Bone - diagnostic imaging | Osteoporosis - metabolism | Osteoporosis - pathology | Bone and Bones - diagnostic imaging | Bone and Bones - metabolism | Biomarkers | Bone Resorption - metabolism | Cancellous Bone - pathology | Catechols - pharmacology | Female | Mice | Intervention | Alkaline phosphatase | Body weight | AKT protein | Bone (trabecular) | Inactivation | Phosphatase | Osteoprotegerin | Bone resorption | Osteoporosis | Bone growth | Rodents | Deoxypyridinoline | TRANCE protein | Bone strength | NF-κB protein | Cathepsin K | TRAF6 protein | Bone turnover | Metabolism | Biological activity | 1-Phosphatidylinositol 3-kinase | Molecular modelling | Lymphocytes B | Tumor necrosis factor | Ligands | Bone mineral density | Acid phosphatase (tartrate-resistant) | Bone | Osteogenesis
Journal Article
Annals of the Rheumatic Diseases, ISSN 0003-4967, 03/2018, Volume 77, Issue Suppl 1, p. A55
IntroductionOsteoporosis predominantly affects elderly people and is characterised by bone loss, increased fracture risk and reduced regeneration ability.... 
Cell culture | Collagen (type I) | Adipose tissue | Arthritis | Interleukin 6 | Osteoporosis | Bone growth | Mineralization | Surgery | Bone marrow | Extracellular matrix | Bone loss | Age | Interleukin 8 | Autografts | Secretion | Bone remodelling | Inflammation | Bone turnover | Gene expression | Ribonucleic acid--RNA | Collagenase 3 | Hip | Collagen | Stem cells | Monocyte chemoattractant protein 1 | Bone (cancellous)
Journal Article
Journal of Bone and Mineral Metabolism, ISSN 0914-8779, 11/2018, Volume 36, Issue 6, pp. 628 - 639
Journal Article
Bone, ISSN 8756-3282, 2008, Volume 44, Issue 5, pp. 942 - 947
Journal Article
World's Poultry Science Journal, ISSN 0043-9339, 03/2012, Volume 68, Issue 1, pp. 71 - 82
Journal Article
Nature Medicine, ISSN 1078-8956, 09/2017, Volume 23, Issue 9, pp. 1072 - 1079
Aging is associated with increased cellular senescence, which is hypothesized to drive the eventual development of multiple comorbidities(1). Here we... 
MEDICINE, RESEARCH & EXPERIMENTAL | CELLS | SECRETORY PHENOTYPE | BIOCHEMISTRY & MOLECULAR BIOLOGY | MATURE | IDENTIFICATION | CELL BIOLOGY | OSTEOCLASTOGENESIS | ACTIVATOR | MASS | ESTROGEN | FRACTURES | DYSFUNCTION | Osteocytes - drug effects | Cancellous Bone - metabolism | beta-Galactosidase | Cellular Senescence - drug effects | Apoptosis - genetics | Cancellous Bone - drug effects | Gene Expression Profiling | Osteoclasts - cytology | Bone and Bones - drug effects | Caspase 8 - genetics | Flow Cytometry | Osteoporosis - metabolism | Bone and Bones - metabolism | Osteoporosis - genetics | Cell Differentiation | Osteoblasts - cytology | Real-Time Polymerase Chain Reaction | Pyrazoles - pharmacology | Cellular Senescence - genetics | Osteoblasts - drug effects | Mice, Transgenic | Absorptiometry, Photon | Cortical Bone - metabolism | Janus Kinases - antagonists & inhibitors | Culture Media, Conditioned | Animals | Cortical Bone - drug effects | Weight-Bearing | Mice | In Vitro Techniques | Osteoclasts - drug effects | Prevention | Osteoporosis | Aging | Physiological aspects | Genetic aspects | Research | Cells | Senescence | Bone (trabecular) | Suicide | Bone resorption | Bone growth | Biomedical materials | Mineralization | Rodents | Computer architecture | Biocompatibility | Inhibition | Conditioning | Bone loss | Bone (cortical) | Suicide genes | Age | Cardiovascular system | Cell survival | Aging (artificial) | Caspase | Pharmacology | Secretome | Inflammation | Insulin | Osteoclastogenesis | Caspase-8 | Bone mass | Cellular biology | Sensitivity enhancement | Bone | In vitro methods and tests | Osteogenesis | Disease prevention | Osteoclast | Drug therapy | Osteocyte
Journal Article
PLoS ONE, ISSN 1932-6203, 04/2012, Volume 7, Issue 4, p. e34647
Glucocorticoid (GC) induced osteoporosis (GIO) is caused by the long-term use of GC for treatment of autoimmune and inflammatory diseases. The GC related... 
TRABECULAR BONE | APPOSITION RATE | CELLS | OSTEOBLAST DIFFERENTIATION | INDUCED OSTEONECROSIS | MULTIDISCIPLINARY SCIENCES | BEAGLE | DANSHEN | EXPRESSION | MILTIORRHIZA | OSTEOPOROSIS | Neovascularization, Physiologic - drug effects | Gene Expression - drug effects | Adipogenesis - drug effects | Benzofurans - administration & dosage | Male | Adipocytes - drug effects | PPAR gamma - metabolism | Core Binding Factor Alpha 1 Subunit - metabolism | Bone and Bones - drug effects | Intercellular Signaling Peptides and Proteins - metabolism | Bone Morphogenetic Proteins - metabolism | Osteoporosis - metabolism | Bone and Bones - metabolism | Stromal Cells - drug effects | Bone Marrow Cells - drug effects | Cell Differentiation | Bone Morphogenetic Proteins - genetics | PPAR gamma - genetics | Prednisone - administration & dosage | Benzofurans - therapeutic use | Osteoporosis - chemically induced | Prednisone - adverse effects | Osteoporosis - prevention & control | Osteoblasts - drug effects | Osteogenesis - drug effects | Stromal Cells - metabolism | Bone and Bones - blood supply | Intercellular Signaling Peptides and Proteins - genetics | Rats | Rats, Sprague-Dawley | beta Catenin - metabolism | beta Catenin - genetics | Animals | Adipocytes - metabolism | Osteoblasts - metabolism | Bone Marrow Cells - metabolism | Core Binding Factor Alpha 1 Subunit - genetics | Osteoporosis | Corticosteroids | Bones | Prednisone | Cell differentiation | Density | Steroids | Oxidative stress | Transcription factors | Glucocorticoids | Laboratories | Differentiation (biology) | Stimulation | Adipocytes | Inflammatory diseases | β-catenin | Angiogenesis | Bone histomorphometry | Biomedical materials | Bone growth | Ischemia | Rodents | Bone marrow | Biocompatibility | Inhibition | Bone loss | Bone strength | Osteopenia | Adipogenesis | Cbfa-1 protein | Research & development--R&D | Medical treatment | Pharmacology | Gene expression | Medicine | Studies | Herbal medicine | Salvia miltiorrhiza | Osteoblastogenesis | Bone mass | Acids | Stem cells | Dilation | Bone mineral density | In vivo methods and tests | Dkk1 protein | Osteogenesis | Bone (cancellous) | Apoptosis | Herbal medicines | Guanylate cyclase | Osteoblasts | adipogenesis | stromal cells | Bone morphogenetic proteins | Differentiation | Research & development | R&D
Journal Article
Journal Article
Journal Article
Journal Article
Annals of the New York Academy of Sciences, ISSN 0077-8923, 01/2016, Volume 1364, Issue 1, pp. 11 - 24
Osteoimmunology is a field of research dedicated to the study of the interactions between the immune system and bone. Among the cells of the immune system that... 
T cells | Pacifici, R. 2015. T cells, osteoblasts, and osteocytes: interacting lineages key for the bone anabolic and catabolic activities of parathyroid hormone. In “MARROW,” ed. by M. Zaidi. Ann. N.Y. Acad. Sci. 1364: 11–24 | PTH | stromal cells | bone | osteoblasts | osteocytes | Pacifici, R. 2015. T cells, osteoblasts, and osteocytes: interacting lineages key for the bone anabolic and catabolic activities of parathyroid hormone. In “MARROW,” ed. by M. Zaidi. Ann. N.Y. Acad. Sci. 1364: 11-24 | Stromal cells | Osteocytes | Bone | T cells | Osteoblasts | C-REACTIVE PROTEIN | DIFFERENTIATED OSTEOBLASTS | CD40 LIGAND | BETA-CATENIN | TUMOR-NECROSIS-FACTOR | CANCELLOUS BONE | OSTEOCLAST FORMATION | IN-VIVO | ENDOCRINOLOGY & METABOLISM | TNF-ALPHA | POSTMENOPAUSAL WOMEN | Osteoblasts - secretion | Humans | T-Lymphocytes - metabolism | Parathyroid Hormone - metabolism | Bone Marrow Cells - immunology | Bone and Bones - metabolism | Osteocytes - immunology | Osteoblasts - cytology | Bone and Bones - cytology | Osteocytes - secretion | Parathyroid Hormone - secretion | T-Lymphocytes - secretion | Osteocytes - metabolism | Bone Marrow Cells - cytology | Osteocytes - cytology | Stromal Cells - metabolism | Bone Marrow Cells - pathology | Cytokines - secretion | Osteoblasts - immunology | Bone Morphogenetic Proteins - secretion | Cell Communication | Wnt Proteins - secretion | Stromal Cells - immunology | Genetic Markers | Parathyroid Hormone - immunology | Proto-Oncogene Proteins - secretion | Cell Lineage | Stromal Cells - secretion | Animals | T-Lymphocytes - cytology | Models, Biological | T-Lymphocytes - immunology | Bone and Bones - immunology | Osteoblasts - metabolism | Bone Remodeling | Bone Marrow Cells - metabolism | Stromal Cells - cytology | Parathyroid hormone | Immune systems | Biomedical materials | Lymphocytes | Biocompatibility | Bones | Mice | Hormones
Journal Article