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Journal Article
ACS Chemical Neuroscience, ISSN 1948-7193, 07/2015, Volume 6, Issue 9, pp. 1546 - 1559
Synthetic cannabinoid (SC) designer drugs based on indole and indazole scaffolds and featuring L-valinamide or L-tert-leucinamide side chains are encountered... 
FUBINACA | JWH-018 | Cannabinoid | PINACA | THC | SR 144528 | SELECTIVE ANTAGONIST | CHEMISTRY, MEDICINAL | BIOCHEMISTRY & MOLECULAR BIOLOGY | 1ST POTENT | RATS | ILLEGAL PRODUCTS | ALPHA-PVT | NEUROSCIENCES | RECEPTOR ANTAGONIST | INTERNATIONAL UNION | DERIVATIVES | SR141716A | Rats, Wistar | Receptor, Cannabinoid, CB2 - agonists | Body Temperature - drug effects | Indazoles - chemical synthesis | Humans | Indoles - chemical synthesis | Male | Receptor, Cannabinoid, CB2 - antagonists & inhibitors | Cannabinoid Receptor Agonists - chemistry | Cannabinoid Receptor Agonists - pharmacology | Receptor, Cannabinoid, CB2 - genetics | Dose-Response Relationship, Drug | Heart Rate - drug effects | Receptor, Cannabinoid, CB1 - agonists | Cannabinoid Receptor Antagonists - pharmacology | Indoles - pharmacology | Molecular Structure | Drug Evaluation, Preclinical | Membrane Potentials - drug effects | Designer Drugs - pharmacology | Indazoles - chemistry | Cannabinoid Receptor Agonists - chemical synthesis | Designer Drugs - chemical synthesis | Indazoles - pharmacology | Designer Drugs - chemistry | Receptor, Cannabinoid, CB1 - metabolism | Receptor, Cannabinoid, CB2 - metabolism | Animals | Receptor, Cannabinoid, CB1 - genetics | Cell Line, Tumor | Mice | Receptor, Cannabinoid, CB1 - antagonists & inhibitors | Indoles - chemistry | Cohort Studies
Journal Article
Anesthesia and Analgesia, ISSN 0003-2999, 02/2013, Volume 116, Issue 2, pp. 463 - 472
Journal Article
Biological Psychiatry, ISSN 0006-3223, 2015, Volume 77, Issue 5, pp. 475 - 487
Abstract Background Mixed cannabinoid receptor 1 and 2 (CB1 and CB2 ) agonists such as Δ9 -tetrahydrocannabinol (Δ9 -THC) can produce tolerance, physical... 
Psychiatry | Tolerance | CB2 | Precipitated withdrawal | Chemotherapy-induced neuropathic pain | Knockout mouse | Side effect | DORSAL-HORN NEURONS | MECHANICAL ALLODYNIA | PSYCHIATRY | VENTRAL ROOT TRANSECTION | CB2 RECEPTOR | INDUCED PERIPHERAL NEUROPATHY | SPINAL-CORD | NECROSIS-FACTOR-ALPHA | NERVE INJURY | NEUROSCIENCES | ACID AMIDE HYDROLASE | PRIMARY SENSORY NEURONS | Hyperalgesia - chemically induced | Tumor Necrosis Factor-alpha - metabolism | Spinal Cord - drug effects | Receptor, Cannabinoid, CB2 - agonists | Male | Cannabinoid Receptor Agonists - pharmacology | RNA, Messenger - metabolism | Receptor, Cannabinoid, CB2 - genetics | Paclitaxel - toxicity | Receptor, Cannabinoid, CB1 - agonists | Cannabinoid Receptor Antagonists - pharmacology | Female | Indoles - pharmacology | Chemokine CCL2 - metabolism | Chromones - pharmacology | Dronabinol - pharmacology | Disease Models, Animal | Mice, Inbred C57BL | Random Allocation | Hyperalgesia - physiopathology | Mice, Knockout | Antineoplastic Agents, Phytogenic - toxicity | Receptor, Cannabinoid, CB1 - metabolism | Receptor, Cannabinoid, CB2 - metabolism | Animals | Receptor, Cannabinoid, CB1 - genetics | Hyperalgesia - drug therapy | Spinal Cord - physiopathology | Receptor, Cannabinoid, CB1 - antagonists & inhibitors | Cannabinoids | Neurosciences | Paclitaxel
Journal Article
Journal of Pharmacology and Experimental Therapeutics, ISSN 0022-3565, 07/2013, Volume 346, Issue 1, pp. 54 - 66
The G protein-coupled receptor 55 (GPR55) is a lysophosphatidylinositol (LPI) receptor that is also responsive to certain cannabinoids. Although GPR55 has been... 
IN-VITRO | ACTIVATION | CANNABINOID RECEPTOR | ASSAY | L-ALPHA-LYSOPHOSPHATIDYLINOSITOL | LIGANDS | GPR55 | PHARMACOLOGY & PHARMACY | PROLIFERATION | IDENTIFICATION | CANCER | Azabicyclo Compounds - pharmacology | Endothelium, Vascular - cytology | Lysophospholipids - metabolism | Receptors, G-Protein-Coupled - metabolism | Humans | NFATC Transcription Factors - agonists | Endothelium, Vascular - drug effects | NF-kappa B - metabolism | Protein Transport - drug effects | Sulfones - antagonists & inhibitors | RNA, Messenger - metabolism | Receptor, Cannabinoid, CB2 - genetics | Sulfones - pharmacology | Recombinant Fusion Proteins - metabolism | HEK293 Cells | Platelet Aggregation - ethics | Cannabinoid Receptor Antagonists - pharmacology | Piperazines - antagonists & inhibitors | Platelet Aggregation - drug effects | Blood Platelets - drug effects | Wound Healing - drug effects | Gene Expression | NF-kappa B - antagonists & inhibitors | Lysophospholipids - antagonists & inhibitors | NFATC Transcription Factors - metabolism | Cells, Cultured | Piperazines - pharmacology | Receptor, Cannabinoid, CB1 - metabolism | Receptor, Cannabinoid, CB2 - metabolism | Receptor, Cannabinoid, CB1 - genetics | Blood Platelets - metabolism | Calcium Signaling - drug effects | Endothelium, Vascular - metabolism | Receptors, G-Protein-Coupled - antagonists & inhibitors | Benzoates - pharmacology | Ligands | Receptors, G-Protein-Coupled - genetics | NFATC Transcription Factors - antagonists & inhibitors | Receptors, G-Protein-Coupled - chemistry
Journal Article
Neuropharmacology, ISSN 0028-3908, 06/2013, Volume 69, pp. 115 - 126
Journal Article
Journal Article