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Neuropharmacology, ISSN 0028-3908, 09/2012, Volume 63, Issue 4, pp. 653 - 666
Cannabinoids have been shown to exert neuroprotective effects in a plethora of neurodegenerative conditions. Over the past decade, some studies demonstrate... 
Neuroinflammation | CB2 | CB1 | WIN55212-2 | PPAR-γ | Beta-amyloid | NITRIC-OXIDE SYNTHASE | ALZHEIMERS-DISEASE | NEUROSCIENCES | PPAR-gamma | CELL-DEATH | CASPASE-3 ACTIVATION | CORTICAL-NEURONS | SIGNALING PATHWAY | DNA FRAGMENTATION | IN-VIVO | PHARMACOLOGY & PHARMACY | INFLAMMATORY RESPONSE | NF-KAPPA-B | Receptor, Cannabinoid, CB2 - agonists | Neuroglia - pathology | Injections, Intraventricular | Male | PPAR gamma - metabolism | Alzheimer Disease - pathology | Cannabinoid Receptor Agonists - therapeutic use | Neuroglia - immunology | Nootropic Agents - antagonists & inhibitors | Cannabinoid Receptor Antagonists - pharmacology | Neurons - metabolism | Alzheimer Disease - immunology | Morpholines - therapeutic use | Receptors, Cannabinoid - chemistry | Disease Models, Animal | Benzoxazines - administration & dosage | Nerve Tissue Proteins - agonists | Alzheimer Disease - drug therapy | Rats | Hippocampus - pathology | Random Allocation | PPAR gamma - antagonists & inhibitors | Signal Transduction - drug effects | Receptor, Cannabinoid, CB1 - antagonists & inhibitors | Naphthalenes - therapeutic use | Neurons - pathology | Rats, Wistar | Hippocampus - drug effects | Receptor, Cannabinoid, CB2 - antagonists & inhibitors | Cannabinoid Receptor Agonists - chemistry | Neuroglia - drug effects | Receptors, Cannabinoid - metabolism | Anti-Inflammatory Agents, Non-Steroidal - antagonists & inhibitors | Receptor, Cannabinoid, CB1 - agonists | Morpholines - antagonists & inhibitors | Benzoxazines - antagonists & inhibitors | Neurons - drug effects | Nerve Tissue Proteins - antagonists & inhibitors | Hippocampus - immunology | Morpholines - administration & dosage | Neurons - immunology | Nootropic Agents - therapeutic use | Cannabinoid Receptor Agonists - administration & dosage | Benzoxazines - therapeutic use | Nerve Tissue Proteins - metabolism | Hippocampus - metabolism | Receptor, Cannabinoid, CB1 - metabolism | Receptor, Cannabinoid, CB2 - metabolism | Animals | Anti-Inflammatory Agents, Non-Steroidal - therapeutic use | Nootropic Agents - administration & dosage | Naphthalenes - administration & dosage | Alzheimer Disease - metabolism | Anti-Inflammatory Agents, Non-Steroidal - administration & dosage | PPAR gamma - agonists | Neuroglia - metabolism | Naphthalenes - antagonists & inhibitors
Journal Article
British Journal of Pharmacology, ISSN 0007-1188, 06/2013, Volume 169, Issue 4, pp. 808 - 819
Background and purpose JZL184 is a selective inhibitor of monoacylglycerol lipase (MAGL), the enzyme that preferentially catabolizes the endocannabinoid... 
monoacylglycerol lipase | endocannabinoid | NF‐κB | TNF‐α | 2‐AG | brain | JZL184 | IL‐6 | IL‐1β | frontal cortex | IL‐10 | cytokines | plasma | IL-1β | IL-10 | IL-6 | NF-κB | TNF-α | 2-AG | CELLS | IL-1 | 2-ARACHIDONYL GLYCEROL | NF-B | RECEPTOR | NECROSIS-FACTOR-ALPHA | TNF | ENDOCANNABINOID 2-ARACHIDONOYLGLYCEROL | COX-2 EXPRESSION | IN-VIVO | PHARMACOLOGY & PHARMACY | NF-KAPPA-B | CANNABINOIDS | Benzodioxoles - antagonists & inhibitors | Spleen - immunology | Frontal Lobe - metabolism | Male | Spleen - drug effects | Prostaglandins - blood | Lipopolysaccharides | Monoacylglycerol Lipases - metabolism | Arachidonic Acids - metabolism | Glycerides - blood | Arachidonic Acids - blood | Enzyme Inhibitors - chemistry | Anti-Anxiety Agents - therapeutic use | Benzodioxoles - therapeutic use | Peritonitis - metabolism | Rats | Encephalitis - immunology | Random Allocation | Glycerides - metabolism | Enzyme Inhibitors - therapeutic use | Rats, Sprague-Dawley | Cannabinoid Receptor Antagonists - therapeutic use | Monoacylglycerol Lipases - blood | Peritonitis - drug therapy | Receptor, Cannabinoid, CB1 - antagonists & inhibitors | Endocannabinoids - metabolism | Nerve Tissue Proteins - blood | Monoacylglycerol Lipases - antagonists & inhibitors | Piperidines - antagonists & inhibitors | Receptor, Cannabinoid, CB2 - antagonists & inhibitors | Anti-Inflammatory Agents, Non-Steroidal - antagonists & inhibitors | Encephalitis - drug therapy | Encephalitis - metabolism | Cytokines - blood | Nerve Tissue Proteins - antagonists & inhibitors | Cytokines - metabolism | Peritonitis - immunology | Nerve Tissue Proteins - metabolism | Receptor, Cannabinoid, CB1 - metabolism | Receptor, Cannabinoid, CB2 - metabolism | Animals | Spleen - metabolism | Anti-Inflammatory Agents, Non-Steroidal - therapeutic use | Piperidines - therapeutic use | Frontal Lobe - drug effects | Cannabinoid Receptor Antagonists - chemistry | Frontal Lobe - immunology | Cytokines - antagonists & inhibitors | Endocannabinoids - blood | Prostaglandins - metabolism | Glycerin | Brain | Unsaturated fatty acids | Cytokines | Glycerol | Lipase | Mitogens | Plasma | Rodents | Cannabinoids 2012, Part Two | Themed Section
Journal Article
Neuropharmacology, ISSN 0028-3908, 10/2012, Volume 63, Issue 5, pp. 905 - 915
Mu-opioid and CB1-cannabinoid agonists produce analgesia; however, adverse effects limit use of drugs in both classes. Additive or synergistic effects... 
Analgesia | Morphine | Marijuana | G-protein | Adenylyl cyclase | CB1 receptors | AGONIST MORPHINE | ACTIVATION | PROTEIN-KINASE | CB1 CANNABINOID RECEPTOR | SYNERGISTIC INTERACTIONS | NEUROSCIENCES | INHIBITION | KAPPA | FULL AGONIST | ADENYLYL-CYCLASE | PHARMACOLOGY & PHARMACY | RAT-BRAIN | Pyrazoles - therapeutic use | Receptor, Cannabinoid, CB2 - agonists | Humans | Analgesics, Opioid - pharmacology | Receptors, Opioid, mu - agonists | Piperidines - pharmacology | Cannabinoid Receptor Agonists - therapeutic use | Recombinant Proteins - antagonists & inhibitors | Piperidines - metabolism | Receptors, Opioid, mu - metabolism | Morpholines - metabolism | Analgesics, Non-Narcotic - metabolism | Drug Inverse Agonism | Mice | Analgesics, Opioid - metabolism | Kinetics | Receptor, Cannabinoid, CB1 - antagonists & inhibitors | Analgesics, Opioid - antagonists & inhibitors | Cannabinoid Receptor Agonists - metabolism | Cricetulus | Analgesics, Non-Narcotic - pharmacology | Cannabinoid Receptor Agonists - pharmacology | Receptor, Cannabinoid, CB2 - genetics | Analgesics, Opioid - adverse effects | Narcotic Antagonists - metabolism | Receptor, Cannabinoid, CB1 - agonists | Analgesics, Non-Narcotic - adverse effects | Cannabinoid Receptor Agonists - adverse effects | Morpholines - adverse effects | CHO Cells | Pyrazoles - adverse effects | Pyrazoles - pharmacology | Binding, Competitive | Recombinant Proteins - metabolism | Cricetinae | Morpholines - pharmacology | Narcotic Antagonists - adverse effects | Recombinant Proteins - agonists | Analgesics, Non-Narcotic - therapeutic use | Pyrazoles - metabolism | Mice, Inbred Strains | Receptors, Opioid, mu - antagonists & inhibitors | Receptor, Cannabinoid, CB1 - metabolism | Receptor, Cannabinoid, CB2 - metabolism | Animals | Narcotic Antagonists - therapeutic use | Piperidines - therapeutic use | Narcotic Antagonists - pharmacology | Piperidines - adverse effects | Receptors, Opioid, mu - genetics | Index Medicus | Drugs | Pain perception | Opioid receptors | Cyclic AMP | Opioid receptors (type mu) | Antagonists | Cannabinoid CB1 receptors | Guanine nucleotide-binding protein | Antagonism | Cell activation | Side effects | Inverse agonists | CB1 Receptors | Adenylyl Cyclase
Journal Article
Methods in Enzymology, ISSN 0076-6879, 2017, Volume 593, pp. 199 - 215
PIMSR is among the first neutral antagonists for the CB1R and was demonstrated pharmacologically to bind to the CB1R, yet not alter calcium flux. It was... 
Glycemic control | Obesity | Neutral antagonist | Metabolic syndrome effects | Alcoholic steatosis | PIMSR | Modeling | CB1R | ENERGY-BALANCE | OBESITY | CANNABINOID RECEPTOR | INVERSE AGONISM | BINGE DRINKING | BIOCHEMISTRY & MOLECULAR BIOLOGY | PERSPECTIVES | BIOCHEMICAL RESEARCH METHODS | ENDOCANNABINOID SYSTEM
Journal Article
British Journal of Pharmacology, ISSN 0007-1188, 03/2013, Volume 168, Issue 6, pp. 1430 - 1444
Background and Purpose Palmitoylethanolamide (PEA) is an endogenous fatty acid amide displaying anti‐inflammatory and analgesic actions. To investigate the... 
intracellular calcium concentrations | PPAR transcription factor | pain | palmitoylethanolamide | transient receptor potential vanilloid type‐1 channels | capsaicin | bradykinin | transient receptor potential vanilloid type-1 channels | N-PALMITOYLETHANOLAMIDE | CA2+ INFLUX | RAT | ROOT GANGLION NEURONS | VR1 RECEPTORS | ANANDAMIDE | HUMAN VANILLOID RECEPTORS | CANNABINOID RECEPTOR | CALCIUM-CHANNELS | PHARMACOLOGY & PHARMACY | CELL-LINE | Ethanolamines - pharmacology | Cricetulus | Endocannabinoids - pharmacology | Membrane Transport Modulators - antagonists & inhibitors | Anti-Inflammatory Agents, Non-Steroidal - pharmacology | TRPV Cation Channels - metabolism | Anti-Inflammatory Agents, Non-Steroidal - antagonists & inhibitors | TRPV Cation Channels - antagonists & inhibitors | Membrane Transport Modulators - pharmacology | Sensory Receptor Cells - metabolism | Palmitic Acids - pharmacology | Action Potentials - drug effects | CHO Cells | Recombinant Proteins - metabolism | Tachyphylaxis | Cell Line | Nerve Tissue Proteins - antagonists & inhibitors | Cricetinae | PPAR alpha - antagonists & inhibitors | Recombinant Proteins - antagonists & inhibitors | Palmitic Acids - antagonists & inhibitors | Nerve Tissue Proteins - agonists | TRPV Cation Channels - agonists | Rats | Ethanolamines - antagonists & inhibitors | Recombinant Proteins - agonists | Nerve Tissue Proteins - genetics | TRPV Cation Channels - genetics | Nerve Tissue Proteins - metabolism | Animals | Sensory Receptor Cells - cytology | Sensory Receptor Cells - drug effects | Drug Antagonism | Calcium Signaling - drug effects | PPAR alpha - agonists | Mice | PPAR alpha - metabolism | Endocannabinoids - antagonists & inhibitors | Research Papers
Journal Article