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Neuropathology and applied neurobiology, ISSN 0305-1846, 2011, Volume 37, Issue 1, pp. 56 - 74
Journal Article
Journal Article
Journal Article
PloS one, ISSN 1932-6203, 2012, Volume 7, Issue 5, p. e36893
...) and neurofibrillary tangles do not completely account for cognitive impairment, therefore other factors such as cardiovascular and cerebrovascular pathologies, may contribute to AD... 
TYPE-4 ALLELE | DIAGNOSIS | MICROVASCULAR PATHOLOGY | RISK-FACTORS | VASCULAR DEMENTIA | MULTIDISCIPLINARY SCIENCES | CEREBRAL-BLOOD-FLOW | APOLIPOPROTEIN-E | SENILE PLAQUES | DEGENERATION | AGE | Tyrosine 3-Monooxygenase - metabolism | Capillaries - pathology | Humans | Cerebral Cortex - pathology | Microvessels - metabolism | Microvessels - pathology | Male | Cerebral Cortex - metabolism | Alzheimer Disease - pathology | Brain - blood supply | Microcirculation | Vasoconstriction - physiology | Vesicular Acetylcholine Transport Proteins - metabolism | Dementia - metabolism | Aged, 80 and over | Neurons, Afferent - pathology | Female | Vasodilation - physiology | Capillaries - metabolism | Apolipoprotein E4 - metabolism | Dementia - pathology | Plaque, Amyloid - pathology | Cholinergic Neurons - metabolism | Aging - pathology | Immunohistochemistry - methods | Alzheimer Disease - metabolism | Plaque, Amyloid - metabolism | Aged | Neurons, Afferent - metabolism | Cholinergic Neurons - pathology | Aging - metabolism | Cohort Studies | Immunohistochemistry | Brain | Advertising executives | Dilatation | Blood vessels | Apolipoproteins | Alzheimer's disease | Pathogenesis | Innervation | Cognitive ability | Cardiovascular disease | Biochemistry | Vasodilation | Consortia | Cerebrovascular system | Angiogenesis | Immunotherapy | Aging | Tyrosine | Medical research | Neurodegenerative diseases | Hydroxylase | Cortex | Carriers | Neurofibrillary tangles | Morphology | Biomarkers | Transporter | Dementia | Structural integrity | Neuropathology | Staining | Tyrosine 3-monooxygenase | Substantia grisea | Vesicular acetylcholine transporter | Macular degeneration | Cerebral amyloid angiopathy | Rodents | Dementia disorders | Evolution | Amyloid | Capillaries | Plaques | Age | Medical imaging | Review boards | Vasoconstriction | Ethnicity | Brain research | Acetylcholine | Norepinephrine | Microvasculature
Journal Article
Kidney international, ISSN 0085-2538, 2012, Volume 82, Issue 4, pp. 412 - 427
Endothelial progenitor cells are known to reverse acute kidney injury by paracrine mechanisms. We previously found that microvesicles released from these... 
exosome | ischemia–reperfusion | acute kidney injury | ischemia-reperfusion | HORIZONTAL TRANSFER | STEM-CELLS | CONTRIBUTE | REGENERATION | FAILURE | EXPERIMENTAL GLOMERULONEPHRITIS | REPAIR | EPITHELIAL-CELLS | MESSENGER-RNA | UROLOGY & NEPHROLOGY | PLATELET-ACTIVATING-FACTOR | Chemotaxis, Leukocyte | Cell Proliferation | Epithelial Cells - metabolism | Kidney - blood supply | Capillaries - pathology | Kidney - pathology | Rats, Wistar | Endothelial Cells - transplantation | Cell-Derived Microparticles - transplantation | Male | MicroRNAs - metabolism | Acute Kidney Injury - genetics | Stem Cells - metabolism | Cell Hypoxia | Stem Cell Transplantation | Kidney - metabolism | Transfection | RNA Interference | Time Factors | Cell-Derived Microparticles - pathology | Cell-Derived Microparticles - metabolism | Kidney Tubules - pathology | Kidney Tubules - metabolism | Capillaries - metabolism | Reperfusion Injury - genetics | Reperfusion Injury - metabolism | Disease Models, Animal | Ribonuclease III - genetics | Ribonuclease III - metabolism | Reperfusion Injury - pathology | Acute Kidney Injury - pathology | Endothelial Cells - metabolism | Cells, Cultured | Gene Expression Regulation | Rats | Epithelial Cells - pathology | Acute Kidney Injury - prevention & control | Regeneration | Oligonucleotides - metabolism | Animals | Reperfusion Injury - prevention & control | Fibrosis | Stem Cells - pathology | Acute Kidney Injury - metabolism | Endothelial Cells - pathology | Apoptosis | Cell proliferation | Intravenous administration | Paracrine signalling | mRNA | Leukocytes | Ribonuclease | Endothelial cells | Kidney | Angiogenesis | Ischemia | Stem cells | Hypoxia | miRNA | Capillaries | Injuries
Journal Article
Brain Research, ISSN 0006-8993, 2007, Volume 1157, Issue 1, pp. 126 - 137
Abstract The purpose of this study was to determine the ultrastructure of the blood–brain barrier (BBB) and blood–spinal cord barrier (BSCB) in G93A SOD1 mice... 
Neurology | Spinal cord | Mitochondria | Ultrastructure | Blood–spinal cord barrier | Brainstem | Motor neuron | Blood–brain barrier | G93A SOD1 mice | Endothelium | Basement membrane | Blood-brain barrier | Blood-spinal cord barrier | blood-brain barrier | MOTOR-NEURONS | endothelium | IMMUNOGLOBULIN SUPERFAMILY | blood-spinal cord barrier | basement membrane | WHITE-MATTER | mitochondria | IGG | AMYOTROPHIC-LATERAL-SCLEROSIS | ultrastructure | CEREBROSPINAL-FLUID | NEUROSCIENCES | motor neuron | MOUSE MODEL | DISEASE | OCCLUDIN | AQUAPORIN-4 | brainstem | spinal cord | Basement Membrane - ultrastructure | Superoxide Dismutase - genetics | Amyotrophic Lateral Sclerosis - physiopathology | Capillaries - pathology | Humans | Astrocytes - pathology | Blood-Brain Barrier - physiopathology | Basement Membrane - pathology | Mitochondria - ultrastructure | Spinal Cord - ultrastructure | Blood-Brain Barrier - ultrastructure | Motor Neurons - pathology | Motor Neurons - ultrastructure | Brain - blood supply | Endothelial Cells - ultrastructure | Spinal Cord - pathology | Brain - ultrastructure | Disease Models, Animal | Extracellular Space - physiology | Microscopy, Electron, Transmission | Capillaries - physiopathology | Capillaries - ultrastructure | Brain Edema - etiology | Brain Edema - pathology | Mice, Inbred C57BL | Mice, Transgenic | Mitochondria - pathology | Spinal Cord - blood supply | Mutation - genetics | Astrocytes - ultrastructure | Tight Junctions - ultrastructure | Blood-Brain Barrier - pathology | Amyotrophic Lateral Sclerosis - pathology | Animals | Brain Edema - physiopathology | Brain - pathology | Mice | Superoxide Dismutase-1 | Endothelial Cells - pathology | Tight Junctions - pathology
Journal Article
Journal Article
The Journal of cell biology, ISSN 1540-8140, 2009, Volume 187, Issue 6, pp. 761 - 772
Selective degeneration and death of one or more classes of neurons is the defining feature of human neurodegenerative disease. Although traditionally viewed as... 
Motor neurons | Nervous system diseases | Reviews | Neurodegenerative diseases | Neurons | Astrocytes | Motor neuron disease | Huntington disease | Parkinson disease | Amyotrophic lateral sclerosis | Prion diseases | MOTOR-NEURONS | NEURON-SPECIFIC EXPRESSION | CU/ZN-SUPEROXIDE-DISMUTASE | SLOW DISEASE PROGRESSION | MONOAMINE-OXIDASE-B | LINKED SOD1 MUTANTS | AMYOTROPHIC-LATERAL-SCLEROSIS | FRONTOTEMPORAL LOBAR DEGENERATION | TRANSGENIC MOUSE MODEL | GENOME-WIDE ASSOCIATION | CELL BIOLOGY | Neurons - pathology | Axonal Transport | Superoxide Dismutase - genetics | Capillaries - pathology | T-Lymphocytes - enzymology | Humans | Astrocytes - pathology | Stress, Physiological | Nerve Degeneration - genetics | Astrocytes - enzymology | Endoplasmic Reticulum - pathology | Cell Death | Superoxides - metabolism | Microglia - pathology | Amyotrophic Lateral Sclerosis - enzymology | T-Lymphocytes - pathology | Superoxide Dismutase - metabolism | Endoplasmic Reticulum - enzymology | Nerve Degeneration - enzymology | Amyotrophic Lateral Sclerosis - genetics | Microglia - enzymology | Capillaries - enzymology | Nerve Degeneration - pathology | Amyotrophic Lateral Sclerosis - pathology | Animals | Neurons - enzymology | Glutamic Acid - metabolism | Mutation | Superoxide Dismutase-1 | Care and treatment | Physiological aspects | Superoxide dismutase | Genetic aspects | Disease susceptibility | Research | Health aspects | Index Medicus
Journal Article